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1. A Concept for Mining Transitive Sequential Patterns from Pancreatic Cancer Patient Journeys

3. Framework for Federated Artificial Intelligence for the Optimization of Pancreatic Cancer Treatment

13. Making use of synergistic targeting to treat DNA damage repair deficient pancreatic cancer

14. Fibroblast drug scavenging increases intratumoural gemcitabine accumulation in murine pancreas cancer

20. ATM-Defizienz führt zu genomischer Instabilität im duktalen Pankreaskarzinom und sensibilisiert für neue Therapieoptionen

21. Fibroblast drug scavenging increases intratumoural gemcitabine accumulation in murine pancreas cancer

40. KRAS G 12C -inhibitor-based combination therapies for pancreatic cancer: insights from drug screening.

41. A Concept for Mining Transitive Sequential Patterns from Pancreatic Cancer Patient Journeys.

42. A Novel AMPK Inhibitor Sensitizes Pancreatic Cancer Cells to Ferroptosis Induction.

43. Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancreatic ductal adenocarcinoma.

44. Expression of gemcitabine metabolizing enzymes and stromal components reveal complexities of preclinical pancreatic cancer models for therapeutic testing.

45. A single-cell strategy for the identification of intronic variants related to mis-splicing in pancreatic cancer.

46. An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies.

47. Epigenetic control of pancreatic cancer metastasis.

48. Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive Pancreatic Ductal Adenocarcinoma.

49. Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer.

50. NFATc1 Is a Central Mediator of EGFR-Induced ARID1A Chromatin Dissociation During Acinar Cell Reprogramming.

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