12 results on '"Herrera, Waldo"'
Search Results
2. Tipos de prótesis sobre implantes en pacientes edéntulos planificados en la clínica dental docente de la Universidad Peruana Cayetano Heredia
- Author
-
Balarezo Razzeto, José Antonio, Díaz Sarabia, Edinson Antonio, Brignardello Cedrón, Fernando Ítalo, Paz Mayurí, Carlos Alberto, Santos Escalante, Claver Clemente, Ronquillo Herrera, Waldo Jesús, Leon Manco, Roberto, and Quintana del Solar, Martín
- Abstract
Objective:To determine the frequency of types of prostheses on planned implants in edentulous patients in the postgraduate services of Oral Rehabilitation and Integral Oral Implantology of the Teaching Dental Clinic of the Universidad Peruana Cayetano Heredia during the years 2016 and 2017.Materials and methods:The research was descriptive, retrospective, observational and transversal, the variables: the planned treatments, classifying them by gender, age, seat, service, type of prosthesis on implants, classification of edentulism and edentulous zone. The database of all the digitalized medical histories was used with the presentation of the planned case and accepted by the tutor and commitment certificate accepted by the patient.Results:We found 296 different types of prostheses on implants, 68.58% in the female sex, the average age was 54.23 years, the highest percentage between 60 and 69 years, the San Isidro headquarters were planned 61.15%, in the service of RO 60.81%, the highest percentage of prostheses on implants was made in the upper jaw, the type of prosthesis on implant with the highest percentage was the unitary prosthesis 58.78%, according to the classification of edentulism the highest percentage was found in the partial edentulous, the The highest percentage in total edentulism was the hybrid prosthesis with 59.46% and in partial edentulism it was the unitary prosthesis with 66.80%.Conclusions:Unitary prostheses were the most frequent, according to gender was female, according to San Isidro and according to RO service, according to the upper edentulous area, in total edentulous the hybrid prosthesis and in partial edentulous the unitary prosthesis. Objetivo: Determinar la frecuencia de tipos de prótesis sobre implantes planificados en pacientes edéntulos en los servicios del Posgrado de Rehabilitación Oral e Implantología Oral Integral de la Clínica Dental Docente de la Universidad Peruana Cayetano Heredia durante los años 2016 y 2017. Material y métodos: La investigación fue descriptiva, retrospectiva, observacional y transversal, las variables: los tratamientos planificados, clasificándolos por género, edad, sede, servicio, tipo de prótesis sobre implantes, clasificación de edentulismo y zona edéntula. Se empleó la base de datos de todas las historias clínicas digitalizadas con presentación de caso planificada y aceptada por el docente tutor y acta de compromiso aceptada por el paciente. Resultados: Se encontraron 296 diversos tipos de prótesis sobre implantes, 68.58% en el sexo femenino, la edad promedio fue de 54,23 años, el mayor porcentaje entre los 60 y 69 años, la sede de San Isidro fueron planificados 61,15%, en el servicio de RO 60,81%, el mayor porcentaje de prótesis sobre implantes se realizó en el maxilar superior, el tipo de prótesis sobre implante con mayor porcentaje fue la prótesis unitaria 58,78%, según clasificación de edentulismo se encontró el mayor porcentaje en el edéntulo parcial, el mayor porcentaje en el edentulismo total fue la prótesis híbrida con 59,46% y en el edentulismo parcial fue la prótesis unitaria con un 66,80%. Conclusiones: La mayor frecuencia fue para las prótesis unitarias, según género fue el femenino, según sede fue San Isidro y según servicio RO, según zona edéntula el maxilar superior, en edéntulos totales la prótesis híbrida y en edéntulos parciales la prótesis unitaria.
- Published
- 2019
3. Outpatient Interventions That May Enhance the Care of a Patient with Co-existing Moyamoya and Down Syndromes
- Author
-
Lwanga, Anita, primary, Herrera, Waldo, additional, Cruz Madrid, Katya, additional, and Irungu, Antony, additional
- Published
- 2018
- Full Text
- View/download PDF
4. Leber congenital amaurosis caused by Lebercilin (LCA5) mutation: Retained photoreceptors adjacent to retinal disorganization
- Author
-
Jacobson, Samuel G., Aleman, Tomas S., Cideciyan, Artur V., Sumaroka, Alexander, Schwartz, Sharon B., Windsor, Elizabeth A.M., Swider, Malgorzata, Herrera, Waldo, and Stone, Edwin M.
- Subjects
Adult ,Spectroscopy, Near-Infrared ,genetic structures ,Adolescent ,Infant ,Pupil ,Retinal Pigment Epithelium ,Middle Aged ,Blindness ,eye diseases ,Retina ,Child, Preschool ,Mutation ,Humans ,sense organs ,Child ,Eye Proteins ,Microtubule-Associated Proteins ,Tomography, Optical Coherence ,Research Article ,Photoreceptor Cells, Vertebrate - Abstract
Purpose To determine the retinal disease expression in the rare form of Leber congenital amaurosis (LCA) caused by Lebercilin (LCA5) mutation. Methods Two young unrelated LCA patients, ages six years (P1) and 25 years (P2) at last visit, both with the same homozygous mutation in the LCA5 gene, were evaluated clinically and with noninvasive studies. En face imaging was performed with near-infrared (NIR) reflectance and autofluorescence (AF); cross-sectional retinal images were obtained with optical coherence tomography (OCT). Dark-adapted thresholds were measured in the older patient; and the transient pupillary light reflex was recorded and quantified in both patients. Results Both LCA5 patients had light perception vision only, hyperopia, and nystagmus. P1 showed a prominent central island of retinal pigment epithelium (RPE) surrounded by alternating elliptical-appearing areas of decreased and increased pigmentation. Retinal laminar architecture at and near the fovea was abnormal in both patients. Foveal outer nuclear layer (ONL) was present in P1 and P2 but to different degrees. With increasing eccentricity, there was retinal laminar disorganization. Regions of pericentral and midperipheral retina in P1, but not P2, could retain measurable ONL and less laminopathy. P2 had a small central island of perception with >5 log units of sensitivity loss. Pupillary responsiveness was present in both LCA5 patients; the thresholds were abnormally elevated by ≥5.5 log units. Conclusions LCA5 patients had evidence of retained photoreceptors mainly in the central retina. Retinal remodeling was present in pericentral regions in both patients. The NIR reflectance and NIR-AF imaging in the younger patient suggested preserved RPE in retinal regions with retained photoreceptors. Detailed phenotype studies in other LCA5 patients with longitudinal follow-up will help determine the feasibility of future intervention in this rare disease.
- Published
- 2009
5. Page Kidney: A Case of Acute or Chronic Renal Failure & Refractory HTN Presenting after Renal Biopsy
- Author
-
Meyer, Stephen, primary, Nukala, Aswin, additional, Maniar, Nikita, additional, and Herrera, Waldo, additional
- Published
- 2015
- Full Text
- View/download PDF
6. Satisfacción laboral de docentes universitarios del Departamento Académico de Clínica Estomatológica
- Author
-
Olivares Preciado, Joan, primary, Matta Morales, Carlos, additional, Choy Lisung, José, additional, Ronquillo Herrera, Waldo, additional, and Maldonado Mendoza, María de las Mercedes, additional
- Published
- 2014
- Full Text
- View/download PDF
7. Normal Central Retinal Function and Structure Preserved in Retinitis Pigmentosa
- Author
-
Jacobson, Samuel G., primary, Roman, Alejandro J., additional, Aleman, Tomas S., additional, Sumaroka, Alexander, additional, Herrera, Waldo, additional, Windsor, Elizabeth A. M., additional, Atkinson, Lori A., additional, Schwartz, Sharon B., additional, Steinberg, Janet D., additional, and Cideciyan, Artur V., additional
- Published
- 2010
- Full Text
- View/download PDF
8. CERKLMutations Cause an Autosomal Recessive Cone-Rod Dystrophy with Inner Retinopathy
- Author
-
Aleman, Tomas S., primary, Soumittra, Nagasamy, additional, Cideciyan, Artur V., additional, Sumaroka, Alexander M., additional, Ramprasad, Vedam Lakshmi, additional, Herrera, Waldo, additional, Windsor, Elizabeth A. M., additional, Schwartz, Sharon B., additional, Russell, Robert C., additional, Roman, Alejandro J., additional, Inglehearn, Chris F., additional, Kumaramanickavel, Govindasamy, additional, Stone, Edwin M., additional, Fishman, Gerald A., additional, and Jacobson, Samuel G., additional
- Published
- 2009
- Full Text
- View/download PDF
9. Retinal Laminar Architecture in Human Retinitis Pigmentosa Caused byRhodopsinGene Mutations
- Author
-
Aleman, Tomas S., primary, Cideciyan, Artur V., additional, Sumaroka, Alexander, additional, Windsor, Elizabeth A. M., additional, Herrera, Waldo, additional, White, D. Alan, additional, Kaushal, Shalesh, additional, Naidu, Anjani, additional, Roman, Alejandro J., additional, Schwartz, Sharon B., additional, Stone, Edwin M., additional, and Jacobson, Samuel G., additional
- Published
- 2008
- Full Text
- View/download PDF
10. CERKL mutations cause an autosomal recessive cone-rod dystrophy with inner retinopathy.
- Author
-
Aleman TS, Soumittra N, Cideciyan AV, Sumaroka AM, Ramprasad VL, Herrera W, Windsor EA, Schwartz SB, Russell RC, Roman AJ, Inglehearn CF, Kumaramanickavel G, Stone EM, Fishman GA, and Jacobson SG
- Subjects
- Adolescent, Adult, Child, Child, Preschool, Electroretinography, Female, Fluorescence, Humans, Male, Middle Aged, Phenotype, Retinal Degeneration diagnosis, Retinal Degeneration physiopathology, Tomography, Optical Coherence, Visual Acuity physiology, Visual Field Tests, Visual Fields physiology, Genes, Recessive, Mutation, Phosphotransferases (Alcohol Group Acceptor) genetics, Photoreceptor Cells, Vertebrate pathology, Retinal Degeneration genetics
- Abstract
Purpose: To define the phenotype of the retinal degeneration associated with mutations in the CERKL gene., Methods: Six patients (ages, 26-54 years) from three unrelated families with CERKL mutations were studied clinically and by electroretinography, kinetic, and chromatic static perimetry, autofluorescence (AF) imaging, and optical coherence tomography (OCT)., Results: Three siblings were homozygotes for p.R257X mutation; two siblings were compound heterozygotes for p.R257X and a novel p.C362X mutation; and one patient had only p.R257X mutation identified to date. There was a spectrum of severity: from mild visual acuity loss to light perception; from full kinetic fields with relative central scotomas to remnant peripheral islands; from reduced ERGs (some with negative waveforms) to nondetectable signals. Maculopathy showed residual foveal islands or extensive central rod and cone scotomas. With AF imaging, there was evidence of hyperautofluorescence at earlier and hypoautofluorescence at later disease stages. Peripheral function was generally less affected than central function. With OCT there were small foveal islands of outer nuclear layer (ONL) in those with preserved acuity. Eccentric to an annular region with no discernible ONL, there could be ONL in the midperiphery. At early disease stages, ganglion cell layer thickness was less affected than ONL. Later disease stages were accompanied by inner nuclear layer and nerve fiber layer abnormalities., Conclusions: CERKL mutations are associated with widespread retinal degeneration with prominent maculopathy. The clinical presentation is that of an autosomal recessive cone-rod dystrophy. Photoreceptor loss appears at all stages of disease and inner laminopathy complicates the phenotype at later stages.
- Published
- 2009
- Full Text
- View/download PDF
11. Retinal disease in Usher syndrome III caused by mutations in the clarin-1 gene.
- Author
-
Herrera W, Aleman TS, Cideciyan AV, Roman AJ, Banin E, Ben-Yosef T, Gardner LM, Sumaroka A, Windsor EA, Schwartz SB, Stone EM, Liu XZ, Kimberling WJ, and Jacobson SG
- Subjects
- Adolescent, Adult, Aged, Female, Fluorescence, Humans, Male, Middle Aged, Photoreceptor Cells, Vertebrate physiology, Retinitis Pigmentosa diagnosis, Retinitis Pigmentosa physiopathology, Tomography, Optical Coherence, Usher Syndromes diagnosis, Usher Syndromes physiopathology, Visual Acuity physiology, Visual Field Tests, Visual Fields physiology, Membrane Proteins genetics, Mutation, Retinitis Pigmentosa genetics, Usher Syndromes genetics
- Abstract
Purpose: To determine the retinal phenotype of Usher syndrome type III (USH3A) caused by clarin-1 (CLRN1) gene mutations in a non-Finnish population., Methods: Patients with USH3A (n = 13; age range, 24-69) representing 11 different families were studied and the results compared with those from patients with USH2A (n = 24; age range, 17-66). The patients were evaluated by ocular examination, kinetic and static perimetry, near-infrared autofluorescence, and optical coherence tomography (OCT)., Results: Ten of 11 families had Ashkenazi Jewish origins and the N48K CLRN1 mutation. Rod function was lost in the peripheral field in the first two decades of life, but central rod function could be retained for another decade. Peripheral cone function was detectable into the third decade of life. Central cone function had a slower decline that extended for decades. Photoreceptor layer loss and features of retinal remodeling were present in retinal regions with severe visual dysfunction, even at the youngest ages tested. Central retinal structure could be normal in younger patients but structural integrity was lost in older patients. RPE disease generally paralleled photoreceptor degeneration. Comparisons between USH3A and USH2A suggested a common rod and cone phenotype but a more accelerated time course of rod loss in USH3A., Conclusions: USH3A and USH2A share patterns of rod and cone dysfunction and retinal structural abnormalities. Peripheral function measurements showed USH3A to be more rapidly progressive than USH2A.
- Published
- 2008
- Full Text
- View/download PDF
12. Retinal laminar architecture in human retinitis pigmentosa caused by Rhodopsin gene mutations.
- Author
-
Aleman TS, Cideciyan AV, Sumaroka A, Windsor EA, Herrera W, White DA, Kaushal S, Naidu A, Roman AJ, Schwartz SB, Stone EM, and Jacobson SG
- Subjects
- Adolescent, Adult, Aged, Animals, Child, Child, Preschool, Dark Adaptation, Electroretinography, Female, Fluorescence, Genes, Dominant, Humans, Male, Mice, Mice, Transgenic, Middle Aged, Retinitis Pigmentosa physiopathology, Sensory Thresholds physiology, Vision Disorders physiopathology, Visual Field Tests, Visual Fields physiology, Mutation, Retina pathology, Retinitis Pigmentosa diagnosis, Retinitis Pigmentosa genetics, Rhodopsin genetics, Tomography, Optical Coherence
- Abstract
Purpose: To determine the underlying retinal micropathology in subclasses of autosomal dominant retinitis pigmentosa (ADRP) caused by rhodopsin (RHO) mutations., Methods: Patients with RHO-ADRP (n = 17, ages 6-73 years), representing class A (R135W and P347L) and class B (P23H, T58R, and G106R) functional phenotypes, were studied with optical coherence tomography (OCT), and colocalized visual thresholds were determined by dark- and light-adapted chromatic perimetry. Autofluorescence imaging was performed with near-infrared light. Retinal histology in hT17M-rhodopsin mice was compared with the human results., Results: Class A patients had only cone-mediated vision. The outer nuclear layer (ONL) thinned with eccentricity and was not detectable within 3 to 4 mm of the fovea. Scotomatous extracentral retina showed loss of ONL, thickening of the inner retina, and demelanization of RPE. Class B patients had superior-inferior asymmetry in function and structure. The superior retina could have normal rod and cone vision, normal lamination (including ONL) and autofluorescence of the RPE melanin; laminopathy was found in the scotomas. With Fourier-domain-OCT, there was apparent inner nuclear layer (INL) thickening in regions with ONL thinning. Retinal regions without ONL had a thick hyporeflective layer that was continuous with the INL from neighboring regions with normal lamination. Transgenic mice had many of the laminar abnormalities found in patients., Conclusions: Retinal laminar abnormalities were present in both classes of RHO-ADRP and were related to the severity of colocalized vision loss. The results in human class B and the transgenic mice support the following disease sequence: ONL diminution with INL thickening; amalgamation of residual ONL with the thickened INL; and progressive retinal remodeling with eventual thinning.
- Published
- 2008
- Full Text
- View/download PDF
Catalog
Discovery Service for Jio Institute Digital Library
For full access to our library's resources, please sign in.