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1. Alzheimer’s disease induced neurons bearing PSEN1 mutations exhibit reduced excitability

2. Familial Alzheimer’s Disease Neurons Bearing Mutations in PSEN1 Display Increased Calcium Responses to AMPA as an Early Calcium Dysregulation Phenotype

3. Neuronal hyperexcitability in Alzheimer’s disease: what are the drivers behind this aberrant phenotype?

4. If Human Brain Organoids Are the Answer to Understanding Dementia, What Are the Questions?

5. Selective ferroptosis vulnerability due to familial Alzheimer's disease presenilin mutations

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