153 results on '"Head, Stewart I."'
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2. Calcium and strontium contractile activation properties of single skinned skeletal muscle fibres from elderly women 66–90 years of age
3. Absence of the Z-disc protein α-actinin-3 impairs the mechanical stability of Actn3KO mouse fast-twitch muscle fibres without altering their contractile properties or twitch kinetics
4. Six weeks of N-acetylcysteine antioxidant in drinking water decreases pathological fiber branching in MDX mouse dystrophic fast-twitch skeletal muscle
5. Quantitative Ratiometric Ca2+ Imaging to Assess Cell Viability
6. Antioxidant therapy in a mouse model of Duchenne muscular dystrophy: some promising results but with a weighty caveat
7. Calcium and strontium contractile activation properties of single skinned skeletal muscle fibres from elderly women 66-90 years of age
8. Sarcoplasmic reticulum calcium handling in unbranched, immediately post‐necrotic fast‐twitch mdx fibres is similar to wild‐type littermates
9. Lifespan Analysis of Dystrophic mdx Fast-Twitch Muscle Morphology and Its Impact on Contractile Function
10. Loss of α-actinin-3 confers protection from eccentric contraction damage in fast-twitch EDL muscles from aged mdx dystrophic mice by reducing pathological fibre branching
11. Absence of the Z-disc Protein α-actinin-3 Impairs the Mechanical Stability of Actn3KO Mouse Fast- twitch Muscle Fibres without Altering their Contractile Properties or Twitch Kinetics
12. Calcium and strontium contractile activation properties of single skinned skeletal muscle fibres from elderly women 66-90 years of age
13. Dystrophin-negative slow-twitch soleus muscles are not susceptible to eccentric contraction induced injury over the lifespan of the mdx mouse
14. Minocycline Treatment Reduces Mass and Force Output From Fast-Twitch Mouse Muscles and Inhibits Myosin Production in C2C12 Myotubes
15. Abnormalities in Structure and Function of Limb Skeletal Muscle Fibres of Dystrophic mdx Mice
16. Loss of α-actinin-3 confers protection from eccentric contraction damage in fast-twitch EDL muscles from aged mdx dystrophic mice by reducing pathological fibre branching.
17. Ca2+- and Sr2+-activation properties of muscle fibres from a muscle receptor organ and the associated extrafusal muscle of the crab and crayfish
18. Old men still have the skeletal muscle contractile function to get up and go even after they have had their leg in a cast
19. Acute inhibitory effects of clenbuterol on force, Ca2+ transients and action potentials in rat soleus may not involve the β2-adrenoceptor pathway
20. Deficiency of α-actinin-3 is associated with increased susceptibility to contraction-induced damage and skeletal muscle remodeling
21. PROPERTIES OF EXTENSOR DIGITORUM LONGUS MUSCLE AND SKINNED FIBERS FROM ADULT AND AGED MALE AND FEMALE ACTN3 KNOCKOUT MICE
22. Isolated extensor digitorum longus muscles from old mdx dystrophic mice show little force recovery 120 minutes after eccentric damage
23. Branched fibres in old dystrophic mdx muscle are associated with mechanical weakening of the sarcolemma, abnormal Ca2+ transients and a breakdown of Ca2+ homeostasis during fatigue
24. ENHANCED HOMOSYNAPTIC LTD IN CEREBELLAR PURKINJE CELLS OF THE DYSTROPHIC MDX MOUSE
25. An Actn3 knockout mouse provides mechanistic insights into the association between α-actinin-3 deficiency and human athletic performance
26. EDL and soleus muscles of the C57BL6J/dy2j laminin-α2-deficient dystrophic mouse are not vulnerable to eccentric contractions
27. Single muscle fibre biomechanics and biomechatronics – The challenges, the pitfalls and the future
28. Branched fibers from old fast-twitch dystrophic muscles are the sites of terminal damage in muscular dystrophy
29. The Effect of ACTN3 Gene Doping on Skeletal Muscle Performance
30. Analysis of theACTN3heterozygous genotype suggests that α-actinin-3 controls sarcomeric composition and muscle function in a dose-dependent fashion
31. Altered Ca2+ Kinetics Associated with α-Actinin-3 Deficiency May Explain Positive Selection for ACTN3 Null Allele in Human Evolution
32. Membrane Injury and Repair in the Muscular Dystrophies
33. RhoA/ROCK Signaling and Pleiotropic α1A-Adrenergic Receptor Regulation of Cardiac Contractility
34. Aged skeletal muscle retains the ability to fully regenerate functional architecture
35. Analysis of the ACTN3 heterozygous genotype suggests that a-actinin-3 controls sarcomeric composition and muscle function in a dose-dependent fashion.
36. Membrane Injury and Repair in the Muscular Dystrophies.
37. Incubating Isolated Mouse EDL Muscles with Creatine Improves Force Production and Twitch Kinetics in Fatigue Due to Reduction in Ionic Strength
38. Properties of extensor digitorum longus muscle and skinned fibers from adult and aged male and female Actn3 knockout mice
39. Age- and Gender-Related Changes in Contractile Properties of Non-Atrophied EDL Muscle
40. Branched fibres in old dystrophicmdxmuscle are associated with mechanical weakening of the sarcolemma, abnormal Ca2+transients and a breakdown of Ca2+homeostasis during fatigue
41. Enhanced homosynaptic LTD in cerebellar Purkinje cells of the dystrophic MDX mouse
42. Altered Ca2+ Kinetics Associated with α-Actinin-3 Deficiency May Explain Positive Selection for ACTN3 Null Allele in Human Evolution.
43. Contractile properties of single-skinned skeletal muscle fibres of the extensor digitorum longus muscle of the Australian short-nosed echidna
44. Long-term depression is reduced in cerebellar Purkinje cells of dystrophin-deficient mdx mice
45. EDL and soleus muscles of the C57BL6J/dy2jlaminin-α2-deficient dystrophic mouse are not vulnerable to eccentric contractions
46. Growth and maturation of cardiac myocytes in fetal sheep in the second half of gestation
47. Altered inhibitory input to Purkinje cells of dystrophin-deficient mice
48. Morphology and organization of muscle fibres in the thoracic coxal muscle receptor organ and the associated promotor muscle, in a crayfish, Cherax destructor, and mud crab, Scylla serrata
49. The role of nitric oxide in diaphragmatic dysfunction in endotoxemic rats
50. Morphology and organization of muscle fibres in the thoracic coxal muscle receptor organ and the associated promotor muscle, in a crayfish, Cherax destructor, and mud crab, Scylla serrata
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