146 results on '"He, Zhini"'
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2. Metabolism-dependent mutagenicity of two structurally similar tobacco-specific nitrosamines (N-nitrosonornicotine and N-nitrosoanabasine) in human cells, partially different CYPs being activating enzymes
3. Geniposide reduced oxidative stress-induced apoptosis in HK-2 cell through PI3K/AKT3/FOXO1 by m6A modification
4. Geniposide alleviated hydrogen peroxide-induced apoptosis of human hepatocytes via altering DNA methylation
5. Global burden and drivers of hyperglycemia: Estimates and predictions from 1990 to 2050
6. CpG site-specific methylation as epi-biomarkers for the prediction of health risk in PAHs-exposed populations
7. Correlations of Aflatoxin Exposure from Cooking Oil and Dietary Foods During Pregnancy with Birth Weight and Gestational Age at Birth in Guangxi, China
8. Correlations of Aflatoxin Exposure from Cooking Oil and Dietary Foods During Pregnancy with Birth Weight and Gestational Age at Birth in Guangxi, China
9. Strain differences between CD-1 and C57BL/6 mice in expression of metabolic enzymes and DNA methylation modifications of the primary hepatocytes
10. Exploring the Antibacterial Potential and Underlying Mechanisms of Prunella vulgaris L. on Methicillin-Resistant Staphylococcus aureus.
11. Assessment of the Adverse Health Effects of Aflatoxin Exposure from Unpackaged Peanut Oil in Guangdong, China
12. Association between Residential Greenness and Human Microbiota: Evidence from Multiple Countries
13. Aberrant methylation of RUNX3 is present in Aflatoxin B1-induced transformation of the L02R cell line
14. Influence of benzene exposure, fat content, and their interactions on erythroid-related hematologic parameters in petrochemical workers: a cross-sectional study
15. Supplementary Figure 2 from CpG Site–Specific Hypermethylation of p16INK4α in Peripheral Blood Lymphocytes of PAH-Exposed Workers
16. Supplementary Figure Legends 1-2 from CpG Site–Specific Hypermethylation of p16INK4α in Peripheral Blood Lymphocytes of PAH-Exposed Workers
17. Supplementary Figure 1 from CpG Site–Specific Hypermethylation of p16INK4α in Peripheral Blood Lymphocytes of PAH-Exposed Workers
18. Supplementary Tables 1-3 from CpG Site–Specific Hypermethylation of p16INK4α in Peripheral Blood Lymphocytes of PAH-Exposed Workers
19. Specific long non-coding RNAs response to occupational PAHs exposure in coke oven workers
20. Hyperoside attenuates Cd-induced kidney injury via inhibiting NLRP3 inflammasome activation and ROS/MAPK/NF-κB signaling pathway in vivo and in vitro
21. Associations of long-term cadmium exposure with peripheral white blood cell subtype counts and indices in residents of cadmium-polluted areas
22. Associations between DNA methylation in DNA damage response-related genes and cytokinesis-block micronucleus cytome index in diesel engine exhaust-exposed workers
23. Benzene Exposure Leads to Lipodystrophy and Alters Endocrine Activity In Vivo and In Vitro
24. Cadmium chloride-induced apoptosis of HK-2 cells via interfering with mitochondrial respiratory chain
25. Specific histone modification responds to arsenic-induced oxidative stress
26. Cadmium induces renal inflammation by activating the NLRP3 inflammasome through ROS/MAPK/NF-κB pathway in vitro and in vivo
27. Global and MGMT promoter hypomethylation independently associated with genomic instability of lymphocytes in subjects exposed to high-dose polycyclic aromatic hydrocarbon
28. Frontiers in applications of porous materials in CO2gas separation membranes: Mechanisms, membrane properties, and future perspectives of porous aromatic frameworks (PAFs)
29. Aberrant Expression of miR-638 Contributes to Benzo(a)pyrene-Induced Human Cell Transformation
30. Association between H3K36me3 modification and methylation of LINE-1 and MGMT in peripheral blood lymphocytes of PAH-exposed workers
31. Influence of benzene exposure, fat content, and their interactions on erythroid-related hematologic parameters in petrochemical workers: a cross-sectional study
32. Influence of benzene exposure, fat content, and their interactions on erythroid-related hematologic parameters in petrochemical workers: a cross-sectional study
33. FLT1 hypermethylation is involved in polycyclic aromatic hydrocarbons-induced cell transformation
34. Benzene-induced mouse hematotoxicity is regulated by a protein phosphatase 2A complex that stimulates transcription of cytochrome P4502E1
35. The effects of Nrf2 knockout on regulation of benzene-induced mouse hematotoxicity
36. Enhanced H3K4me3 modifications are involved in the transactivation of DNA damage responsive genes in workers exposed to low-level benzene
37. Hypermethylation of PGCP gene is associated with human bronchial epithelial cells immortalization
38. TRIM36 hypermethylation is involved in polycyclic aromatic hydrocarbons-induced cell transformation
39. Aberrant methylation of RUNX3 is present in Aflatoxin B 1 -induced transformation of the L02R cell line
40. Persistent phosphorylation at specific H3 serine residues involved in chemical carcinogen-induced cell transformation
41. MGMThypomethylation is associated with DNA damage in workers exposed to low-dose benzene
42. Association between H3K36me3 modification and methylation of LINE-1and MGMTin peripheral blood lymphocytes of PAH-exposed workers
43. Aberrant methylation of nucleotide excision repair genes is associated with chronic arsenic poisoning
44. Specific histone modifications were associated with the PAH-induced DNA damage response in coke oven workers
45. Associations between DNA methylation in DNA damage response-related genes and cytokinesis-block micronucleus cytome index in diesel engine exhaust-exposed workers
46. CpG site-specific RASSF1a hypermethylation is associated with occupational PAH exposure and genomic instability
47. Specific histone modifications regulate the expression of AhR in 16HBE cells exposed to benzo(a)pyrene
48. Application of human cell transformation assay on assessment of carcinogenic potential of river organic pollutants
49. MGMT hypomethylation is associated with DNA damage in workers exposed to low-dose benzene.
50. Aberrant methylation of nucleotide excision repair genes is associated with chronic arsenic poisoning.
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