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1. Fundamental relations for the velocity dispersion of stars in the Milky Way

8. The K2-HERMES survey: Age and metallicity of the thick disc

9. The GALAH survey and Gaia DR2: Dissecting the stellar disc's phase space by age, action, chemistry, and location

10. The GALAH survey: An abundance, age, and kinematic inventory of the solar neighbourhood made with TGAS

12. The GALAH Survey: Second data release

14. COHORT study of the HSG. CAG repeat expansion in Huntington disease determines age at onset in a fully dominant fashion

15. Population stratification may bias analysis of PGC-1alpha as amodifiewr of age at Huntington disease motor onset

16. Population stratification may bias analysis of PGC-1? as a modifier of age at Huntington disease motor onset

17. TAA repeat variation in the GRIK2 gene does not influence age at onset in Huntington's disease

18. Mutations in HFE2 cause iron overload in chromosome 1q-linked juvenile hemochromatosis

19. A genome scan for modifiers of age at onset in Huntington disease: The HD MAPS study

20. The Salmonella Type III Effector SspH2 Specifically Exploits the NLR Co-chaperone Activity of SGT1 to Subvert Immunity

22. Mutant huntingtin binds the mitochondrial fission GTPase dynamin-related protein-1 and increases its enzymatic activity

23. A frequent mutation in the lipoprotein lipase gene (D9N) deteriorates the biochemical and clinical phenotype of familial hypercholesterolemia

24. Cholesterol biosynthesis pathway is disturbed in YAC128 mice and is modulated by huntingtin mutation

28. DETECTION OF THE PRO664-LEU MUTATION IN THE LOW-DENSITY-LIPOPROTEIN RECEPTOR AND ITS RELATION TO LIPOPROTEIN(A) LEVELS IN PATIENTS WITH FAMILIAL HYPERCHOLESTEROLEMIA OF DUTCH ANCESTRY FROM THE NETHERLANDS AND CANADA

30. Loss‐of‐function mutations in the Nav1.7 gene underlie congenital indifference to pain in multiple human populations

38. Predictive, pre-natal and diagnostic genetic testing for Huntington's disease: the experience in Canada from 1987 to 2000

44. Providing predictive testing for Huntington disease via telehealth: results of a pilot study in British Columbia, Canada.

46. What monozygotic twins discordant for phenotype illustrate about mechanisms influencing genetic forms of neurodegeneration.

48. Loss of both ABCA1 and ABCG1 results in increased disturbances in islet sterol homeostasis, inflammation, and impaired β-cell function.

49. Adoption and the communication of genetic risk: experiences in Huntington disease.

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