142 results on '"Hamaï, Ahmed"'
Search Results
2. mTOR inhibition suppresses salinomycin-induced ferroptosis in breast cancer stem cells by ironing out mitochondrial dysfunctions
3. Cell Plasticity in a Mouse Model of Benign Prostate Hyperplasia Drives Amplification of Androgen-Independent Epithelial Cell Populations Sensitive to Antioxidant Therapy
4. Non-coding RNAs as new autophagy regulators in cancer progression
5. Mitochondrial dynamics and metabolic regulation control T cell fate in the thymus
6. Ferroptosis Inducers Upregulate PD-L1 in Recurrent Triple-Negative Breast Cancer
7. Ferroptosis Inducers Up-Regulate PD-L1 in Recurrent Triple Negative Breast Cancer
8. Cell Plasticity in a Mouse Model of Benign Prostate Hyperplasia Drives Amplification of Androgen-Independent Epithelial Cell Populations Sensitive to Antioxidant Therapy
9. Ferroptosis Inducers Upregulate PD-L1 in Recurrent Triple-Negative Breast Cancer.
10. Association of FTH1-Expressing Circulating Tumor Cells With Efficacy of Neoadjuvant Chemotherapy for Patients With Breast Cancer: A Prospective Cohort Study.
11. Adverse Crosstalk between Extracellular Matrix Remodeling and Ferroptosis in Basal Breast Cancer
12. Association of FTH1-Expressing Circulating Tumor Cells With Efficacy of Neoadjuvant Chemotherapy for Patients With Breast Cancer: A Prospective Cohort Study
13. mTOR Inhibition Suppresses Salinomycin-Induced Ferroptosis in Breast Cancer Stem Cells by Ironing Out Mitochondrial Dysfunctions
14. Autophagy and Inflammation
15. Crosstalk between autophagy and metabolic regulation of cancer stem cells
16. Autophagy and Tumor Cell Metabolism
17. Supplementary Methods from Human TH17 Immune Cells Specific for the Tumor Antigen MAGE-A3 Convert to IFN-γ–Secreting Cells as They Differentiate into Effector T Cells In Vivo
18. Supplementary Figure 1 from Human TH17 Immune Cells Specific for the Tumor Antigen MAGE-A3 Convert to IFN-γ–Secreting Cells as They Differentiate into Effector T Cells In Vivo
19. Supplementary Table 1 from Human TH17 Immune Cells Specific for the Tumor Antigen MAGE-A3 Convert to IFN-γ–Secreting Cells as They Differentiate into Effector T Cells In Vivo
20. Supplementary Figure Legend from Human TH17 Immune Cells Specific for the Tumor Antigen MAGE-A3 Convert to IFN-γ–Secreting Cells as They Differentiate into Effector T Cells In Vivo
21. Supplementary Table 2 from Human TH17 Immune Cells Specific for the Tumor Antigen MAGE-A3 Convert to IFN-γ–Secreting Cells as They Differentiate into Effector T Cells In Vivo
22. Supplementary Figure 1 from ICAM-1 Has a Critical Role in the Regulation of Metastatic Melanoma Tumor Susceptibility to CTL Lysis by Interfering with PI3K/AKT Pathway
23. Salinomycin kills cancer stem cells by sequestering iron in lysosomes
24. The HP1γ epigenetic silencer dampens IFN-γ response at the gut epithelial barrier
25. Autophagy, Cell Death, and Cancer
26. Editorial: The role of iron in cancer progression
27. Autophagy-Associated Immunogenic Modulation and Its Applications in Cancer Therapy
28. Predictive value of circulating tumor cells FTH1 gene on the efficacy of neoadjuvant chemotherapy in non-metastatic breast cancer.
29. GNS561, a clinical-stage PPT1 inhibitor, is efficient against hepatocellular carcinoma via modulation of lysosomal functions
30. Ferroptosis: Cancer Stem Cells Rely on Iron until “to Die for” It
31. GNS561, a clinical-stage PPT1 inhibitor, is efficient against hepatocellular carcinoma via modulation of lysosomal functions
32. hSMG-1 is a granzyme B-associated stress-responsive protein kinase
33. Chemical targeting of NEET proteins reveals their function in mitochondrial morphodynamics
34. Autophagy and Inflammation
35. GNS561, a clinical-stage PPT1 inhibitor, is efficient against hepatocellular carcinoma via modulation of lysosomal functions
36. GNS561, a clinical-stage PPT1 inhibitor, is efficient against hepatocellular carcinoma via modulation of lysosomal functions.
37. GNS561, a clinical-stage PPT1 inhibitor, is efficient against hepatocellular carcinoma viamodulation of lysosomal functions
38. A promising new approach to cancer therapy: Targeting iron metabolism in cancer stem cells
39. An iron hand over cancer stem cells
40. Homéostasie du fer et autophagie
41. Autophagy: A Druggable Process
42. Autophagy and cancer stem cells or tumor-initiating cells in human breast cancer
43. Reactive Oxygen Species, AMP-activated Protein Kinase, and the Transcription Cofactor p300 Regulate α-Tubulin Acetyltransferase-1 (αTAT-1/MEC-17)-dependent Microtubule Hyperacetylation during Cell Stress
44. Expression of MAGE-A3/6 in Primary Breast Cancer is Associated With Hormone Receptor Negative Status, High Histologic Grade, and Poor Survival
45. Cancer stem cells and autophagy: Facts and Perspectives
46. Autophagy modulates cell migration and β1 integrin membrane recycling
47. Autophagy regulation and its role in cancer
48. Inhibition of the autophagic flux by salinomycin in breast cancer stem-like/progenitor cells interferes with their maintenance
49. New Targets for Acetylation in Autophagy
50. Human TH17 Immune Cells Specific for the Tumor Antigen MAGE-A3 Convert to IFN-γ–Secreting Cells as They Differentiate into Effector T Cells In Vivo
Catalog
Books, media, physical & digital resources
Discovery Service for Jio Institute Digital Library
For full access to our library's resources, please sign in.