258 results on '"Haan, Claude"'
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2. Many ways to resistance: How melanoma cells evade targeted therapies
3. Kinase inhibitor library screening identifies synergistic drug combinations effective in sensitive and resistant melanoma cells
4. A new ALK isoform transported by extracellular vesicles confers drug resistance to melanoma cells
5. Regulation of JAKs: Insights Gleaned from the Functional Protein Domains
6. CCT196969 effectively inhibits growth and survival of melanoma brain metastasis cells
7. Inhibition of Wee1 induces antitumoral effects in preclinical models of hepatocellular carcinoma independently of p53 mutational status
8. Oncostatin M induces RIG‐I and MDA5 expression and enhances the double‐stranded RNA response in fibroblasts
9. Effects of inflammatory cytokines and hypoxia on hepatocytes and HCC cell lines: ID: 197
10. IL-27 elicits responses similar to Interferon-gamma in non-hematopoietic cells: ID: 202
11. Role of microRNAs in signal transduction pathways of the inflammatory cytokine Interleukin-6: Relevance for liver diseases: ID: 194
12. Constitutive membrane binding of the multi-site docking protein Gab1 in JAK2-V617F expressing cells: ID: 79
13. Systematic transcriptional profiling of responses to STAT1- and STAT3- activating cytokines in different cancer types
14. Co-immunoprecipitation Protocol to Investigate Cytokine Receptor-Associated Proteins, e.g., Janus Kinases or Other Associated Signaling Proteins
15. Detection of Activated STAT Species Using Electrophoretic Mobility Shift Assay (EMSA) and Potential Pitfalls Arising from the Use of Detergents
16. Systematic Transcriptional Profiling of Responses to STAT1- and STAT3-Activating Cytokines in Different Cancer Types
17. Cooperative effects of Janus and Aurora kinase inhibition by CEP701 in cells expressing Jak2V617F
18. Perspectives for the use of structural information and chemical genetics to develop inhibitors of Janus kinases
19. Oncostatin M up-regulates the ER chaperone Grp78/BiP in liver cells
20. Hypoxia-inducible factor 1α is upregulated by oncostatin M via Jak/STAT3 and the MEK/Erk1/2 pathway
21. Hypoxia-inducible factor 1α is up-regulated by oncostatin M and participates in oncostatin M signaling#
22. Additional file 8: of Kinase inhibitor library screening identifies synergistic drug combinations effective in sensitive and resistant melanoma cells
23. Additional file 7: of Kinase inhibitor library screening identifies synergistic drug combinations effective in sensitive and resistant melanoma cells
24. Additional file 3: of Kinase inhibitor library screening identifies synergistic drug combinations effective in sensitive and resistant melanoma cells
25. Additional file 4: of Kinase inhibitor library screening identifies synergistic drug combinations effective in sensitive and resistant melanoma cells
26. Additional file 2: of Kinase inhibitor library screening identifies synergistic drug combinations effective in sensitive and resistant melanoma cells
27. Additional file 6: of Kinase inhibitor library screening identifies synergistic drug combinations effective in sensitive and resistant melanoma cells
28. Jaks and cytokine receptors—An intimate relationship
29. JAK2 mutants (e.g., JAK2V617F) and their importance as drug targets in myeloproliferative neoplasms
30. JAK2-V617F-induced MAPK activity is regulated by PI3K and acts synergistically with PI3K on the proliferation of JAK2-V617F-positive cells
31. Kinase inhibitor library screening identifies synergistic drug combinations effective in sensitive and resistant melanoma cells
32. Modulation of the IL-6 signaling pathway in liver cells by miRNAs targeting gp130, JAK1 and/or STAT3
33. Phosphorylation of the pyruvate dehydrogenase complex precedes HIF-1-mediated effects and pyruvate dehydrogenase kinase 1 upregulation during the first hours of hypoxic treatment in hepatocellular carcinoma cells
34. Additional file 1: of A new ALK isoform transported by extracellular vesicles confers drug resistance to melanoma cells
35. Additional file 2: of A new ALK isoform transported by extracellular vesicles confers drug resistance to melanoma cells
36. A new ALK isoform transported by extracellular vesicles confers drug resistance to melanoma cells
37. Additional file 1: Figure S1. of ROS production induced by BRAF inhibitor treatment rewires metabolic processes affecting cell growth of melanoma cells
38. Additional file 2: Figure S2. of ROS production induced by BRAF inhibitor treatment rewires metabolic processes affecting cell growth of melanoma cells
39. SIAH2 antagonizes TYK2-STAT3 signaling in lung carcinoma cells
40. Termination of IL-6-induced STAT activation is independent of receptor internalization but requires de novo protein synthesis
41. The PD-L1- and IL6-mediated dampening of the IL27/STAT1 anticancer responses are prevented by α-PD-L1 or α-IL6 antibodies
42. Crosstalk between different family members: IL27 recapitulates IFNγ responses in HCC cells, but is inhibited by IL6-type cytokines
43. The multi-site docking protein Gab1 is constitutively phosphorylated independent from its recruitment to the plasma membrane in Jak2-V617Fpositive cells and mediates proliferation of human erythroleukaemia cells
44. Additional file 6 of The role of HIF-1 in oncostatin M-dependent metabolic reprogramming of hepatic cells
45. Additional file 4 of The role of HIF-1 in oncostatin M-dependent metabolic reprogramming of hepatic cells
46. Additional file 5 of The role of HIF-1 in oncostatin M-dependent metabolic reprogramming of hepatic cells
47. Additional file 1 of The role of HIF-1 in oncostatin M-dependent metabolic reprogramming of hepatic cells
48. Additional file 3 of The role of HIF-1 in oncostatin M-dependent metabolic reprogramming of hepatic cells
49. Additional file 2 of The role of HIF-1 in oncostatin M-dependent metabolic reprogramming of hepatic cells
50. The multi-site docking protein Gab1 is constitutively phosphorylated independent from its recruitment to the plasma membrane in Jak2-V617F-positive cells and mediates proliferation of human erythroleukaemia cells
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