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19. Canine ceroid-lipofuscinoses: A review and classification

Author

Jolly, R. D., primary, Palmer, D. N., additional, Studdert, V. P., additional, Sutton, R. H., additional, Kelly, W. R., additional, Koppang, N., additional, Dahme, G., additional, Hartley, W. J., additional, Patterson, J. S., additional, and Riis, R. C., additional

Published
1994
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26. Comparative Toxicity of Selenium from Seleno-DL-methionine, Sodium Selenate, and Astragalus bisulcatus in Pigs.

Author

PANTER, K. E., HARTLEY, W. J., JAMES, L. F., MAYLAND, H. F., STEGELMEIER, B. L., and KECHELE, P. O.

Subjects
SELENIUM poisoning, SWINE diseases, SELENIUM, SODIUM selenate, LABORATORY swine, ANIMAL models of toxicology, METHIONINE
Abstract

Selenium is an essential micronutrient, although ingestion in excess in pigs can cause disease conditions including neurological dysfunction and chronic skin and hoof lesions. Controlled feeding trials in growing swine, using the same Se content in feed sources, resulted in higher concentrations (p < 0.05) of Se in blood and organs of pigs fed seleno-DL-methionine compared with those receiving Astragalus bisulcatus or sodium selenate. Clinical signs of Se toxicity including neurological signs of paralysis were more severe and occurred sooner in the A. bisulcatus group than in the sodium selenate or seleno-DL-methionine groups. All five pigs fed A. bisulcatus developed neurological signs of paralysis, and in four the signs occurred within 5 days of the start of treatment. Four of five pigs fed sodium selenate also developed paralysis, but this occurred 4 to 21 days after treatment began. The fifth pig in the group developed signs of chronic selenosis. Two of five pigs fed seleno-DL-methionine developed paralysis on 9 and 24 days, respectively, and the remaining three developed chronic selenosis. Selenium fed to pigs in three forms [plant (A. bisulcatus), sodium selenate, or seleno-DL-methionine] resulted in neurological dysfunction and lesions of symmetrical poliomyelomalacia. These were most severe in the A. bisulcatus group, which also had polioencephalo-malacia. Although seleno-DL-methionine caused the greater increase in tissue and blood Se concentrations, this did not correlate with severity of pathological changes, since animals fed A. bisulcatus developed more severe and disseminated lesions. [ABSTRACT FROM AUTHOR]

Published
1996
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40. Generalised ceroid-lipofuscinosis and brown bowel syndrome in Cocker spaniel dogs

Author

Jolly, R. D., Hartley, W. J., Jones, B. R., Johnstone, A. C., Palmer, A. C., and Blakemore, W. F.

Abstract

The ceroid-lipofuscinoses comprise a group of inherited storage diseases of human beings and animals that are characterised by progressive neurodegenerative disease and the accumulation within cells of a fluorescent lipopigment. A distinct syndrome occurs in Cocker spaniel dogs in which there is a generalised accumulation of a lipofuscin-like pigment, with such a heavy accumulation in smooth muscle that the intestine and other organs have a brown discolouration. Such discolouration is not observed in other forms of ceroid-lipofuscinosis. Dogs are clinically affected in adulthood and show progressive hind limb paresis, into-ordination, and deficient postural reactions and proprioception. Spinal reflexes may be exaggerated. Behavioural or temperament changes, seizures or blindness may occur.

Published
1994
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41. Akabane Disease of Cattle in Australia

Author

Hartley, W. J.

Subjects
Veterinary medicine, Disease, Biology, Cattle Diseases
Abstract

Paper presented at the XIIIth World Congress on Cattle Diseases, Durban, S. Africa, Sept. 17-21, 1984., The Bovine Practitioner, No. 20 (1985 November)

Published
1985
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