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182 results on '"H. Gottschalk"'

1. Dubrovnik

Author

H. Gottschalk

Subjects
Human ecology. Anthropogeography, GF1-900, Geography (General), G1-922, Cartography, GA101-1776
Abstract

No abstract available.

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2. Exploratory investigation of a patient‐informed low‐dose psilocybin pulse regimen in the suppression of cluster headache: Results from a randomized, double‐blind, placebo‐controlled trial

Author

Emmanuelle A. D. Schindler, R. Andrew Sewell, Christopher H. Gottschalk, Christina Luddy, L. Taylor Flynn, Yutong Zhu, Hayley Lindsey, Brian P. Pittman, Nicholas V. Cozzi, and Deepak C. D'Souza

Subjects
Neurology, Neurology (clinical)
Abstract

Using a patient-informed regimen, we conducted an exploratory randomized, double-blind, placebo-controlled study to systematically investigate the effects of psilocybin in cluster headache.Sustained reductions in cluster headache burden after limited quantities of psilocybin-containing mushrooms are anecdotally reported, although to date there are no controlled studies investigating these effects.Participants were randomized to receive psilocybin (0.143 mg/kg) or placebo (microcrystalline cellulose) in a pulse of three doses, each ~5 days apart. Participants maintained headache diaries starting 2 weeks before and continuing through 8 weeks after the first drug session. A total of 16 participants were randomized to receive experimental drug and 14 were included in the final analysis.In the 3 weeks after the start of the pulse regimen, the change in cluster attack frequency was 0.03 (95% confidence interval [CI] -2.6 to 2.6) attacks/week with placebo (baseline 8.9 [95% CI 3.8 to 14.0]) and -3.2 (95% CI -8.3 to 1.9) attacks/week with psilocybin (baseline 9.6 [95% CI 5.6 to 13.6]; p = 0.251). Group difference in change from baseline had a moderate effect size (d = 0.69). The effect size was small in episodic participants (d = 0.35) but large in chronic participants (d = 1.25), which remained over the entire 8-week period measured (d = 0.81). Changes in cluster attack frequency were not correlated with the intensity of acute psychotropic effects during psilocybin administration. Psilocybin was well-tolerated without any unexpected or serious adverse events.Findings from this initial, exploratory study provide valuable information for the development of larger, more definitive studies. Efficacy outcomes were negative, owing in part to the small number of participants. The separation of acute psychotropic effects and lasting therapeutic effects underscores the need for further investigation into the mechanism(s) of action of psilocybin in headache disorders.

Published
2022

4. Whole Exome Sequencing Is the Preferred Strategy to Identify the Genetic Defect in Patients With a Probable or Possible Mitochondrial Cause

Author

Tom E. J. Theunissen, Minh Nguyen, Rick Kamps, Alexandra T. Hendrickx, Suzanne C. E. H. Sallevelt, Ralph W. H. Gottschalk, Chantal M. Calis, Alphons P. M. Stassen, Bart de Koning, Elvira N. M. Mulder-Den Hartog, Kees Schoonderwoerd, Sabine A. Fuchs, Yvonne Hilhorst-Hofstee, Marianne de Visser, Jo Vanoevelen, Radek Szklarczyk, Mike Gerards, Irenaeus F. M. de Coo, Debby M. E. I. Hellebrekers, and Hubert J. M. Smeets

Subjects
mitochondrial disease, next-generation sequencing, mtDNA sequencing, whole-exome sequencing, diagnostic yield, Genetics, QH426-470
Abstract

Mitochondrial disorders, characterized by clinical symptoms and/or OXPHOS deficiencies, are caused by pathogenic variants in mitochondrial genes. However, pathogenic variants in some of these genes can lead to clinical manifestations which overlap with other neuromuscular diseases, which can be caused by pathogenic variants in non-mitochondrial genes as well. Mitochondrial pathogenic variants can be found in the mitochondrial DNA (mtDNA) or in any of the 1,500 nuclear genes with a mitochondrial function. We have performed a two-step next-generation sequencing approach in a cohort of 117 patients, mostly children, in whom a mitochondrial disease-cause could likely or possibly explain the phenotype. A total of 86 patients had a mitochondrial disorder, according to established clinical and biochemical criteria. The other 31 patients had neuromuscular symptoms, where in a minority a mitochondrial genetic cause is present, but a non-mitochondrial genetic cause is more likely. All patients were screened for pathogenic variants in the mtDNA and, if excluded, analyzed by whole exome sequencing (WES). Variants were filtered for being pathogenic and compatible with an autosomal or X-linked recessive mode of inheritance in families with multiple affected siblings and/or consanguineous parents. Non-consanguineous families with a single patient were additionally screened for autosomal and X-linked dominant mutations in a predefined gene-set. We identified causative pathogenic variants in the mtDNA in 20% of the patient-cohort, and in nuclear genes in 49%, implying an overall yield of 68%. We identified pathogenic variants in mitochondrial and non-mitochondrial genes in both groups with, obviously, a higher number of mitochondrial genes affected in mitochondrial disease patients. Furthermore, we show that 31% of the disease-causing genes in the mitochondrial patient group were not included in the MitoCarta database, and therefore would have been missed with MitoCarta based gene-panels. We conclude that WES is preferable to panel-based approaches for both groups of patients, as the mitochondrial gene-list is not complete and mitochondrial symptoms can be secondary. Also, clinically and genetically heterogeneous disorders would require sequential use of multiple different gene panels. We conclude that WES is a comprehensive and unbiased approach to establish a genetic diagnosis in these patients, able to resolve multi-genic disease-causes.

Published
2018
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5. Treatment of Chronic Migraine with Focus on Botulinum Neurotoxins

Author

Sara M. Schaefer, Christopher H. Gottschalk, and Bahman Jabbari

Subjects
botulinum toxin, migraine, headache, chronic migraine, chronic daily headache, neurotoxins, medication overuse headache, Medicine
Abstract

Migraine is the most common neurological disorder, and contributes to disability and large healthcare costs in the United States and the world. The treatment of migraine until recently has focused on medications, both abortive and prophylactic, but treatment of chronic migraine has been revolutionized with the introduction of botulinum toxin injection therapy. In this review, we explore the current understanding of migraine pathophysiology, and the evolution of the use of botulinum toxin therapy including proposed pathophysiological mechanisms through animal data. We also discuss the similarities and differences between three injection techniques.

Published
2015
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6. Probabilistic modeling of LCF failure times using a epidemiological crack percolation model

Author

M. Harder, P. Lion, L. Mäde, T. Beck, and H. Gottschalk

Abstract

The analysis of standardized low cycle fatigue (LCF) experiments shows that the failure times widely scatter. Furthermore, mechanical components often fail before the deterministic failure time is reached. A possibility to overcome these problems is to consider probabilistic failure times. Our approach for probabilistic life prediction is based on the microstructure of the metal. Since we focus on nickel-base alloys we consider a coarse grained microstructure, with random oriented FCC grains. This leads to random distributed Schmid factors and different anisotropic stress in each grain. To gain crack initiation times, we use Coffin-MansonBasquin and Ramberg-Osgood equation on stresses corrected with probabilistic Schmid factors. Using these single grain crack initiation times, we have developed an epidemiological crack growth model over multiple grains. In this mesoscopic crack percolation model, cracked grains induce a stress increase in neighboring grains. This stress increase is realized using a machine learning model trained on data generated from finite element simulations. The resulting crack clusters are evaluated with a failure criterion based on a multimodal stress intensity factor. From the generated failure times, we calculate surface dependent hazard rates using a Monte Carlo framework. We compare the obtained failure time distributions to data from LCF experiments and find good coincidence of predicted and measured scatter bands.

Published
2022

8. Endoscopic Repair of Obstructive Cor Triatriatum

Author

Satoru Fujii, Brian Evans, Ali Hage, Ivan Iglesias, Byron H. Gottschalk, and Michael W.A. Chu

Subjects
medicine.medical_specialty, business.industry, General Medicine, medicine.disease, Minimally invasive cardiac surgery, Cor triatriatum sinister, Surgery, Endoscopic Cor Triatriatum, Cor triatriatum, medicine, business, ComputingMethodologies_COMPUTERGRAPHICS
Abstract

Graphical abstract, Highlights • CTS is characterized by a membrane that divides the left atrium into two chambers. • This patient also presented with mitral regurgitation. • The authors present multimodality images and intraoperative video clips. • In this case, repair was performed with a minimally invasive approach. • Intraoperative echocardiography is an essential element of patient care.

Published
2019

10. Link between Brugada phenocopy and myocardial ischemia: Results from the International Registry on Brugada Phenocopy

Author

Sahil Agrawal, Aldo G. Carrizo, Byron H. Gottschalk, Antonio Bayés de Luna, Andrés Ricardo Pérez-Riera, Raimundo Barbosa-Barros, János Tomcsányi, Marek Jastrzębski, Adrian Baranchuk, Gregory Dendramis, and Grace Xu

Subjects
Adult, Male, medicine.medical_specialty, Myocardial ischemia, Myocardial Ischemia, Ischemia, 030204 cardiovascular system & hematology, Sudden cardiac death, Pathogenesis, Electrocardiography, 03 medical and health sciences, 0302 clinical medicine, Internal medicine, medicine, Humans, Registries, cardiovascular diseases, 030212 general & internal medicine, Aged, Brugada Syndrome, Brugada syndrome, Phenocopy, business.industry, General Medicine, Middle Aged, medicine.disease, Phenotype, Brugada ECG Pattern, Cardiology, Etiology, Female, Cardiology and Cardiovascular Medicine, business
Abstract

Background Brugada phenocopies clinical entities that have indistinguishable electrocardiographic (ECG) patterns from true congenital Brugada syndrome. However, they are induced by other clinical circumstances such as myocardial ischemia. The purpose of our study was to examine the clinical features and pathogenesis of ischemia-induced Brugada phenocopy (BrP). Methods Data from 17 cases of ischemia-induced BrP were collected from the International Registry (www.brugadaphenocopy.com). Data were extracted from these publications and authors were contacted to provide further insight into each case. Results Of the patients included in this study, 71% were male. Mean age was 59 ± 11 years (range: 38-76). Type-1 Brugada ECG pattern occurred in 15/17 (88%) of the cases, while a type-2 Brugada ECG pattern was observed in the other 2/17 (12%). In all cases, the Brugada ECG pattern resolved upon correction of the ischemia, indicating ischemia as the inducing circumstance. No arrhythmic events have been detected acutely or during the follow-up. Reported time to resolution ranged from 2 minutes to 5 hours. Provocative challenges using sodium channel blocking agents were performed in 7/17 cases (41%), and all failed to induce a Brugada ECG pattern (BrP Class A). The remaining 10/17 cases (59%) did not undergo provocative testing due to various clinical reasons. Conclusions Myocardial ischemia is a commonly reported etiology of BrP. Importantly, this study found no association between BrP induced by myocardial ischemia and sudden cardiac death or malignant ventricular arrhythmias.

Published
2019

12. The fine structure of dislocations in silicon

Author

H Gottschalk, H Alexander, and V Dietz

Subjects
Relaxation phenomena, Condensed Matter::Materials Science, Materials science, Condensed matter physics, Silicon, chemistry, Annealing (metallurgy), Relaxation (physics), Non-equilibrium thermodynamics, chemistry.chemical_element, Dislocation, Dissociation (chemistry), High stress
Abstract

For high stress deformed specimens containing dislocations in a nonequilibrium dissociation state the relaxation of the splitting width by annealing at very low temperatures (100°C to 140°C) is investigated by TEM measurements. As the formation of new double kinks is prevented by the low temperature, the motion of the partials is a result of the motion of existing kinks. Assuming the kink migration energy being equal for the 30° and for the 90° partial its value is found between 1 and 1.2 eV. Additional TEM observations of relaxation phenomena changing the dislocation arrangement at higher annealing temperatures are reported.

Published
2021

14. Generative Modeling of Turbulence

Author

C. Drygala, B. Winhart, F. di Mare, and H. Gottschalk

Subjects
Fluid Flow and Transfer Processes, Physics::Fluid Dynamics, FOS: Computer and information sciences, Mechanics of Materials, Mechanical Engineering, Computer Vision and Pattern Recognition (cs.CV), Computer Science - Computer Vision and Pattern Recognition, Computational Mechanics, Fluid Dynamics (physics.flu-dyn), FOS: Physical sciences, Physics - Fluid Dynamics, Condensed Matter Physics
Abstract

We present a mathematically well-founded approach for the synthetic modeling of turbulent flows using generative adversarial networks (GAN). Based on the analysis of chaotic, deterministic systems in terms of ergodicity, we outline a mathematical proof that GAN can actually learn to sample state snapshots from the invariant measure of the chaotic system. Based on this analysis, we study a hierarchy of chaotic systems starting with the Lorenz attractor and then carry on to the modeling of turbulent flows with GAN. As training data, we use fields of velocity fluctuations obtained from large-eddy simulations (LES). Two architectures are investigated in detail: we use a deep, convolutional GAN (DCGAN) to synthesize the turbulent flow around a cylinder. We furthermore simulate the flow around a low-pressure turbine stator using the pix2pixHD architecture for a conditional DCGAN being conditioned on the position of a rotating wake in front of the stator. The settings of adversarial training and the effects of using specific GAN architectures are explained. We thereby show that GAN are efficient in simulating turbulence in technically challenging flow problems on the basis of a moderate amount of training data. GAN training and inference times significantly fall short when compared with classical numerical methods, in particular, LES, while still providing turbulent flows in high resolution. We furthermore analyze the statistical properties of the synthesized and LES flow fields, which agree excellently. We also show the ability of the conditional GAN to generalize over changes of geometry by generating turbulent flow fields for positions of the wake that are not included in the training data.

Published
2021
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15. Identifying biomarkers of ventilator induced lung injury during one-lung ventilation surgery: a scoping review

Author

Allan J. G. Bruinooge, Ruochen Mao, Tania H. Gottschalk, Sadeesh K. Srinathan, Gordon Buduhan, Lawrence Tan, Andrew J. Halayko, and Biniam Kidane

Subjects
Pulmonary and Respiratory Medicine
Abstract

Ventilator-induced lung injury (VILI) can occur as a result of mechanical ventilation to two lungs. Thoracic surgery often requires one-lung ventilation (OLV). The potential for VILI is likely higher in OLV. The impact of OLV on development of post-operative pulmonary complications is not well understood. We aimed to perform a scoping review to determine reliable biomarkers of VILI after OLV.A scoping review was performed using Cochrane Collaboration methodology. We searched Medline, EMBASE and SCOPUS. Gray literature was searched. Studies of adult human or animal models without pre-existing lung damage exposed to OLV, with biomarker responses analyzed were included.After screening 5,613 eligible papers, 89 papers were chosen for full text review, with 29 meeting inclusion. Approximately half (52%, n=15) of studies were conducted in humans in an intra-operative setting. Bronchoalveolar lavage (BAL)serum analyses with enzyme-linked immunosorbent assay (ELISA)-based assays were most commonly used. The majority of analytes were investigated by a single study. Of the analytes that were investigated by two or more studies (n=31), only 16 were concordant in their findings. Across all sample types and studies 84% (n=66) of the 79 inflammatory markers and 75% (n=6) of the 8 anti-inflammatory markers tested were found to increase. Half (48%) of all studies showed an increase in TNF-α or IL-6.A scoping review of the state of the evidence demonstrated that candidate biomarkers with the most evidence and greatest reliability are general markers of inflammation, such as IL-6 and TNF-α assessed using ELISA assays. Studies were limited in the number of biomarkers measured concurrently, sample size, and studies using human participants. In conclusion these identified markers can potentially serve as outcome measures for studies on OLV.

Published
2020

16. New methodologies for measuring Brugada ECG patterns cannot differentiate the ECG pattern of Brugada syndrome from Brugada phenocopy

Author

Daniel D. Anselm, Atul Jaidka, Javier García-Niebla, Byron H. Gottschalk, A. Bayés de Luna, and Adrian Baranchuk

Subjects
medicine.medical_specialty, 030204 cardiovascular system & hematology, Sensitivity and Specificity, Sudden death, Diagnosis, Differential, Electrocardiography, 03 medical and health sciences, 0302 clinical medicine, Internal medicine, Positive predicative value, medicine, Humans, Diagnosis, Computer-Assisted, cardiovascular diseases, 030212 general & internal medicine, Brugada Syndrome, Brugada syndrome, Phenocopy, medicine.diagnostic_test, business.industry, Reproducibility of Results, medicine.disease, Brugada ECG Pattern, Cardiology, Cardiology and Cardiovascular Medicine, Beta angle, business, Algorithms
Abstract

Background Brugada phenocopies (BrP) are clinical entities characterized by ECG patterns that are identical to true Brugada syndrome (BrS), but are elicited by various clinical circumstances. A recent study demonstrated that the patterns of BrP and BrS are indistinguishable under the naked eye, thereby validating the concept that the patterns are identical. Objective The aim of our study was to determine whether recently developed ECG criteria would allow for discrimination between type-2 BrS ECG pattern and type-2 BrP ECG pattern. Methods Ten ECGs from confirmed BrS (aborted sudden death, transformation into type 1 upon sodium channel blocking test and/or ventricular arrhythmias, positive genetics) cases and 9 ECGs from confirmed BrP were included in the study. Surface 12-lead ECGs were scanned, saved in JPEG format for blind measurement of two values: (i) β-angle; and (ii) the base of the triangle. Cut-off values of ≥ 58° for the β-angle and ≥ 4 mm for the base of the triangle were used to determine the BrS ECG pattern. Results Mean values for the β-angle in leads V1 and V2 were 66.7 ± 25.5 and 55.4 ± 28.1 for BrS and 54.1 ± 26.5 and 43.1 ± 16.1 for BrP respectively (p = NS). Mean values for the base of the triangle in V1 and V2 were 7.5 ± 3.9 and 5.7 ± 3.9 for BrS and 5.6 ± 3.2 and 4.7 ± 2.7 for BrP respectively (p = NS). The β-angle had a sensitivity of 60%, specificity of 78% (LR + 2.7, LR − 0.5). The base of the triangle had a sensitivity of 80%, specificity of 40% (LR + 1.4, LR − 0.5). Conclusions New ECG criteria presented relatively low sensitivity and specificity, positive and negative predictive values to discriminate between BrS and BrP ECG patterns, providing further evidence that the two patterns are identical.

Published
2016

17. Letter by Gottschalk et al Regarding Article, 'Cardiac Arrest With ST-Segment-Elevation in V1 and V2: Differential Diagnosis'

Author

Adrian Baranchuk, Javier García-Niebla, and Byron H. Gottschalk

Subjects
medicine.medical_specialty, business.industry, Elevation, Arrhythmias, Cardiac, 030204 cardiovascular system & hematology, Heart Arrest, Diagnosis, Differential, 03 medical and health sciences, 0302 clinical medicine, Physiology (medical), Internal medicine, medicine, Cardiology, ST segment, Humans, 030212 general & internal medicine, Differential diagnosis, Cardiology and Cardiovascular Medicine, business
Published
2018

20. Ochronosis of Mitral Valve and Coronary Arteries

Author

Linrui Guo, Jonathan Blankenstein, and Byron H. Gottschalk

Subjects
Pulmonary and Respiratory Medicine, Aortic valve, medicine.medical_specialty, medicine.medical_treatment, 030204 cardiovascular system & hematology, Alkaptonuria, 03 medical and health sciences, 0302 clinical medicine, Thoracic Arteries, Internal medicine, Mitral valve, medicine, Humans, Coronary Artery Bypass, Vascular Calcification, Aged, Heart Valve Prosthesis Implantation, Ochronosis, business.industry, Mitral valve replacement, Mitral Valve Insufficiency, medicine.disease, Coronary Vessels, Tricuspid Valve Insufficiency, Coronary arteries, Stenosis, medicine.anatomical_structure, Dyspnea, Elective Surgical Procedures, cardiovascular system, Cardiology, Mitral Valve, Surgery, Female, Tricuspid Valve, Cardiology and Cardiovascular Medicine, business, 030217 neurology & neurosurgery, Artery
Abstract

Cardiac ochronosis is a rare complication of alkaptonuria, a disorder of tyrosine metabolism characterized by a triad of dark urine, pigmentation of tissues, and ochronotic arthropathies. When present, cardiac ochronosis generally affects the aortic valve, resulting in aortic stenosis. More rarely, it may affect the mitral valve and the coronary arteries. This report describes the case of a 67-year-old woman with a history of alkaptonuria with severe ochronosis of the coronary arteries and mitral valve who required coronary artery bypass and mitral valve replacement.

Published
2018

21. Differentiation Between Brugada Syndrome and Brugada Phenocopy ECG Patterns: Is It Possible?

Author

Byron H. Gottschalk and Adrian Baranchuk

Subjects
Phenocopy, medicine.medical_specialty, business.industry, 030204 cardiovascular system & hematology, medicine.disease, 03 medical and health sciences, 0302 clinical medicine, Brugada ECG Pattern, Internal medicine, medicine, Cardiology, 030212 general & internal medicine, business, Brugada syndrome
Abstract

The Brugada ECG pattern is an essential component of both Brugada Phenocopy (BrP) and Brugada Syndrome (BrS). Indeed, diagnostic suspicion for each condition begins with identification of the ECG pattern. As its introduction, a requirement of BrP is an ECG pattern that is identical and indistinguishable to that of BrS. The importance of this suggests that the two conditions cannot be differentiated solely based on the electrocardiogram on presentation. It requires that the clinician use a combination of clinical and electrocardiographic cues to arrive at a diagnosis. Although the concept of identical ECG patterns has been a fundamental conceptual assumption to BrP, it had not been quantitatively validated until recently in a series of two important studies. The first study set out to determine whether experts in the field of BrS can reliably diagnose BrS based on ECG pattern alone. The second study examined whether ECG criteria used to distinguish Brugada ECG patterns from similar patterns would allow one to distinguish BrP from BrS.

Published
2018

22. Brugada Phenocopy: Definition, Diagnosis, and Differentiation From True Brugada Syndrome

Author

Byron H. Gottschalk

Subjects
Phenocopy, congenital, hereditary, and neonatal diseases and abnormalities, medicine.medical_specialty, business.industry, fungi, Context (language use), medicine.disease, Internal medicine, Brugada ECG Pattern, Cardiology, Medicine, cardiovascular diseases, Abnormality, business, Brugada syndrome
Abstract

With the recognition that patients may present with a Brugada ECG pattern in the context conditions not attributable to Brugada syndrome (BrS), there was a need to further characterize these “phenocopies” to facilitate clinical decision-making and research. Brugada phenocopies (BrPs) are clinical entities that present with an ECG pattern identical to either the type-1 or type-2 Brugada patterns, yet differ etiologically from true congenital BrS. The pattern presents in association with an identifiable condition and, upon resolution of that condition, the ECG pattern normalizes. BrP may not be due to the same sodium channel abnormality as BrS, or may be only transient while the underlying condition persists. Indeed, the defining feature of BrP is the absence of true congenital BrS.

Published
2018

23. Precision measurement of the structure of the CMS inner tracking system using nuclear interactions

Author

Sirunyan, A.M. Tumasyan, A. Adam, W. Ambrogi, F. Asilar, E. Bergauer, T. Brandstetter, J. Brondolin, E. Dragicevic, M. Erö, J. Valle, A.E.D. Flechl, M. Frühwirth, R. Ghete, V.M. Grossmann, J. Hrubec, J. Jeitler, M. König, A. Krammer, N. Krätschmer, I. Liko, D. Madlener, T. Mikulec, I. Rad, N. Rohringer, H. Schieck, J. Schöfbeck, R. Spanring, M. Spitzbart, D. Steininger, H. Taurok, A. Waltenberger, W. Wittmann, J. Wulz, C.-E. Zarucki, M. Chekhovsky, V. Mossolov, V. Gonzalez, J.S. Beaumont, W. Wolf, E.A.D. Croce, D.D. Janssen, X. Lauwers, J. Pieters, M. Klundert, M.V.D. Haevermaet, H.V. Mechelen, P.V. Remortel, N.V. Zeid, S.A. Blekman, F. Bols, E.S. D'Hondt, J. Bruyn, I.D. Clercq, J.D. Deroover, K. Flouris, G. Lontkovskyi, D. Lowette, S. Marchesini, I. Moortgat, S. Moreels, L. Python, Q. Skovpen, K. Tavernier, S. Doninck, W.V. Mulders, P.V. Parijs, I.V. Allard, Y. Beghin, D. Bilin, B. Brun, H. Clerbaux, B. Lentdecker, G.D. Delannoy, H. Dorney, B. Fasanella, G. Favart, L. Goldouzian, R. Grebenyuk, A. Kalsi, A.K. Lenzi, T. Luetic, J. Moureaux, L. Postiau, N. Song, Z. Starling, E. Velde, C.V. Vanlaer, P. Vannerom, D. Wang, Q. Yang, Y. Cornelis, T. Dobur, D. Fagot, A. Gul, M. Khvastunov, I. Poyraz, D. Roskas, C. Trocino, D. Tytgat, M. Verbeke, W. Vermassen, B. Vit, M. Zaganidis, N. Bakhshiansohi, H. Bondu, O. Brochet, S. Bruno, G. Caputo, C. Caudron, A. David, P. Visscher, S.D. Delaere, C. Delcourt, M. Francois, B. Giammanco, A. Krintiras, G. Lemaitre, V. Magitteri, A. Mertens, A. Michotte, D. Musich, M. Piotrzkowski, K. Quertenmont, L. Saggio, A. Szilasi, N. Marono, M.V. Wertz, S. Zobec, J. Beliy, N. Caebergs, T. Daubie, E. Hammad, G.H. Alves, F.L. Alves, G.A. Brito, L. Silva, G.C. Hensel, C. Moraes, A. Pol, M.E. Teles, P.R. Chagas, E.B.B.D. Carvalho, W. Chinellato, J. Coelho, E. Costa, E.M.D. Silveira, G.G.D. Damiao, D.D.J. Martins, C.D.O. Souza, S.F.D. Malbouisson, H. Figueiredo, D.M. Almeida, M.M.D. Herrera, C.M. Mundim, L. Nogima, H. Silva, W.L.P.D. Rosas, L.J.S. Santoro, A. Sznajder, A. Thiel, M. Manganote, E.J.T. Araujo, F.T.D.S.D. Pereira, A.V. Ahuja, S. Bernardes, C.A. Calligaris, L. Tomei, T.R.F.P. Gregores, E.M. Mercadante, P.G. Novaes, S.F. Padula, S. Abad, D.R. Aleksandrov, A. Hadjiiska, R. Iaydjiev, P. Marinov, A. Misheva, M. Rodozov, M. Shopova, M. Sultanov, G. Dimitrov, A. Litov, L. Pavlov, B. Petkov, P. Fang, W. Gao, X. Yuan, L. Ahmad, M. Bian, J.G. Chen, G.M. Chen, H.S. Chen, M. Chen, Y. Jiang, C.H. Leggat, D. Liao, H. Liu, Z. Romeo, F. Shaheen, S.M. Spiezia, A. Tao, J. Wang, C. Wang, Z. Yazgan, E. Zhang, H. Zhao, J. Ban, Y. Chen, G. Li, J. Li, Q. Liu, S. Mao, Y. Qian, S.J. Wang, D. Xu, Z. Wang, Y. Avila, C. Cabrera, A. Montoya, C.A.C. Sierra, L.F.C. Florez, C. Hernández, C.F.G. Delgado, M.A.S. Courbon, B. Godinovic, N. Lelas, D. Puljak, I. Sculac, T. Antunovic, Z. Kovac, M. Brigljevic, V. Ceci, S. Ferencek, D. Kadija, K. Mesic, B. Starodumov, A. Susa, T. Ather, M.W. Attikis, A. Mavromanolakis, G. Mousa, J. Nicolaou, C. Ptochos, F. Razis, P.A. Rykaczewski, H. Finger, M. Finger, M. Ayala, E. Jarrin, E.C. Elgammal, S. Kamel, A.E. Salama, E. Ahmed, I. Bhowmik, S. Oliveira, A.C.A.D. Dewanjee, R.K. Ehataht, K. Kadastik, M. Perrini, L. Raidal, M. Veelken, C. Eerola, P. Kirschenmann, H. Pekkanen, J. Voutilainen, M. Havukainen, J. Heikkilä, J.K. Järvinen, T. Karimäki, V. Kinnunen, R. Lampén, T. Lassila-Perini, K. Laurila, S. Lehti, S. Lindén, T. Luukka, P. Mäenpä, T. Siikonen, H. Tuominen, E. Tuominiemi, J. Tuuva, T. Besancon, M. Couderc, F. Dejardin, M. Denegri, D. Faure, J.L. Ferri, F. Ganjour, S. Givernaud, A. Gras, P. De Monchenault, G.H. Jarry, P. Leloup, C. Locci, E. Malcles, J. Negro, G. Rander, J. Rosowsky, A. Sahin, M.Ö. Titov, M. Abdulsalam, A. Amendola, C. Antropov, I. Beaudette, F. Busson, P. Charlot, C. De Cassagnac, R.G. Kucher, I. Lisniak, S. Lobanov, A. Blanco, J.M. Nguyen, M. Ochando, C. Ortona, G. Pigard, P. Salerno, R. Sauvan, J.B. Sirois, Y. Leiton, A.G.S. Yilmaz, Y. Zabi, A. Zghiche, A. 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Bat, A. Boran, F. Cerci, S. Damarseckin, S. Demiroglu, Z.S. Dozen, C. Dumanoglu, I. Girgis, S. Gokbulut, G. Guler, Y. Gurpinar, E. Hos, I. Kangal, E.E. Kara, O. Topaksu, A.K. Kiminsu, U. Oglakci, M. Onengut, G. Ozdemir, K. Ozturk, S. Cerci, D.S. Tali, B. Tok, U.G. Turkcapar, S. Zorbakir, I.S. Zorbilmez, C. Isildak, B. Karapinar, G. Yalvac, M. Zeyrek, M. Atakisi, I.O. Gülmez, E. Kaya, M. Kaya, O. Tekten, S. Yetkin, E.A. Agaras, M.N. Atay, S. Cakir, A. Cankocak, K. Komurcu, Y. Sen, S. Grynyov, B. Levchuk, L. Alexander, T. Ball, F. Beck, L. Brooke, J.J. Burns, D. Clement, E. Cussans, D. Davignon, O. Flacher, H. Goldstein, J. Heath, G.P. Heath, H.F. Kreczko, L. Newbold, D.M. Paramesvaran, S. Penning, B. Sakuma, T. Nasr-Storey, S.S.E. Smith, D. Smith, V.J. Taylor, J. Bell, K.W. Belyaev, A. Brew, C. Brown, R.M. Cieri, D. Cockerill, D.J.A. Coughlan, J.A. Harder, K. Harper, S. Linacre, J. Manolopoulos, K. Olaiya, E. Petyt, D. Shepherd-Themistocleous, C.H. Thea, A. Tomalin, I.R. Williams, T. 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Hakala, J. Heintz, A. Heintz, U. Hinton, N. Hogan, J.M. Kwok, K.H.M. Laird, E. Landsberg, G. Lee, J. Mao, Z. Narain, M. Pazzini, J. Piperov, S. Sagir, S. Scotti, E. Spencer, E. Syarif, R. Yu, D. Band, R. Brainerd, C. Breedon, R. Burns, D. Sanchez, M.C.D.L.B. Chertok, M. Conway, J. Conway, R. Cox, P.T. Erbacher, R. Flores, C. Funk, G. Ko, W. Kukral, O. Lander, R. McLean, C. Mulhearn, M. Pellett, D. Pilot, J. Shalhout, S. Shi, M. Stolp, D. Taylor, D. Thomson, J. Tos, K. Tripathi, M. Wang, Z. Zhang, F. Bachtis, M. Bravo, C. Cousins, R. Dasgupta, A. Florent, A. Hauser, J. Ignatenko, M. McColl, N. Regnard, S. Saltzberg, D. Schnaible, C. Valuev, V. Bouvier, E. Burt, K. Clare, R. Gary, J.W. Shirazi, S.M.A.G. Hanson, G. Karapostoli, G. Kennedy, E. Lacroix, F. Long, O.R. Negrete, M.O. Paneva, M.I. Si, W. Wang, L. Wei, H. Wimpenny, S. Yates, B.R. Branson, J.G. Cittolin, S. Derdzinski, M. Gerosa, R. Gilbert, D. Hashemi, B. Holzner, A. Klein, D. Kole, G. Krutelyov, V. Letts, J. Masciovecchio, M. Olivito, D. Padhi, S. Pieri, M. Sani, M. Sharma, V. Simon, S. Tadel, M. Vartak, A. Wasserbaech, S. Wood, J. Würthwein, F. Yagil, A. Porta, G.Z.D. Amin, N. Bhandari, R. Bradmiller-Feld, J. Campagnari, C. Citron, M. Colegrove, O. Dishaw, A. Dutta, V. Sevilla, M.F. Gouskos, L. Heller, R. Incandela, J. Kyre, S. Ovcharova, A. Qu, H. Richman, J. Stuart, D. Suarez, I. Wang, S. White, D. Yoo, J. Anderson, D. Bornheim, A. Bunn, J. Lawhorn, J.M. Newman, H.B. Nguyen, T.Q. Spiropulu, M. Vlimant, J.R. Wilkinson, R. Xie, S. Zhang, Z. Zhu, R.Y. Andrews, M.B. Ferguson, T. Mudholkar, T. Paulini, M. Sun, M. Vorobiev, I. Weinberg, M. Cumalat, J.P. Ford, W.T. Jensen, F. Johnson, A. Krohn, M. Leontsinis, S. Macdonald, E. Mulholland, T. Stenson, K. Ulmer, K.A. Wagner, S.R. Alexander, J. Chaves, J. Cheng, Y. Chu, J. Datta, A. McDermott, K. Mirman, N. Patterson, J.R. Quach, D. Rinkevicius, A. Ryd, A. Skinnari, L. Soffi, L. Tan, S.M. Tao, Z. Thom, J. Tucker, J. Wittich, P. Zientek, M. Abdullin, S. Albrow, M. Alyari, M. Apollinari, G. Apresyan, A. Apyan, A. Banerjee, S. Bauerdick, L.A.T. Beretvas, A. Berryhill, J. Bhat, P.C. Bolla, G. Burkett, K. Butler, J.N. Canepa, A. Cerati, G.B. Cheung, H.W.K. Chlebana, F. Chramowicz, J. Cooper, W. Cremonesi, M. Derylo, G. Duarte, J. Elvira, V.D. Freeman, J. Gecse, Z. Gingu, C. Gonzalez, H. Gottschalk, E. Gray, L. Green, D. Grünendahl, S. Gutsche, O. Hanlon, J. Harris, R.M. Hasegawa, S. Hirschauer, J. Hu, Z. Jayatilaka, B. Jindariani, S. Johnson, M. Joshi, U. Klima, B. Kortelainen, M.J. Kreis, B. Lammel, S. Lei, C.M. Lincoln, D. Lipton, R. Liu, M. Liu, T. Sá, R.L.D. Los, S. Lykken, J. Maeshima, K. Magini, N. Marraffino, J.M. Mason, D. Matulik, M. McBride, P. Merkel, P. Mrenna, S. Nahn, S. O'Dell, V. Olsen, J. Pedro, K. Pena, C. Prokofyev, O. Prosser, A. Rakness, G. Ristori, L. Rivera, R. Savoy-Navarro, A. Schneider, B. Sexton-Kennedy, E. Soha, A. Spalding, W.J. Spiegel, L. Stoynev, S. Strait, J. Strobbe, N. Taylor, L. Tkaczyk, S. Tran, N.V. Uplegger, L. Vaandering, E.W. Vernieri, C. Verzocchi, M. Vidal, R. Voirin, E. Wang, M. Weber, H.A. Whitbeck, A. Acosta, D. Avery, P. Bortignon, P. Bourilkov, D. Brinkerhoff, A. Cadamuro, L. Carnes, A. Carver, M. Curry, D. Field, R.D. Gleyzer, S.V. Joshi, B.M. Konigsberg, J. Korytov, A. Ma, P. Matchev, K. Mei, H. Mitselmakher, G. Shi, K. Sperka, D. Thomas, L. Wang, J. Wang, S. Joshi, Y.R. Linn, S. Ackert, A. Adams, T. Askew, A. Hagopian, S. Hagopian, V. Johnson, K.F. Kolberg, T. Martinez, G. Perry, T. Prosper, H. Saha, A. Santra, A. Sharma, V. Yohay, R. Baarmand, M.M. Bhopatkar, V. Colafranceschi, S. Hohlmann, M. Noonan, D. Roy, T. Yumiceva, F. Adams, M.R. Apanasevich, L. Berry, D. Betts, R.R. Cavanaugh, R. Chen, X. Dittmer, S. Evdokimov, A. Evdokimov, O. Gerber, C.E. Hangal, D.A. Hofman, D.J. Jung, K. Kamin, J. Macauda, S. Mills, C. Gonzalez, I.D.S. Tonjes, M.B. Varelas, N. Wang, H. Wu, Z. Zhang, J. Alhusseini, M. Bilki, B. Clarida, W. Dilsiz, K. Durgut, S. Gandrajula, R.P. Haytmyradov, M. Khristenko, V. Merlo, J.-P. Mestvirishvili, A. Moeller, A. Nachtman, J. Ogul, H. Onel, Y. Ozok, F. Penzo, A. Rude, C. Snyder, C. Tiras, E. Wetzel, J. Yi, K. Anderson, I. Blumenfeld, B. Cocoros, A. Eminizer, N. Fehling, D. Feng, L. Gritsan, A.V. Hung, W.T. Maksimovic, P. Martin, C. Roskes, J. Sarica, U. Swartz, M. Xiao, M. You, C. Al-Bataineh, A. Baringer, P. Bean, A. Boren, S. Bowen, J. Castle, J. Flowers, Z. Gibson, E. Khalil, S. Kropivnitskaya, A. Majumder, D. McBrayer, W. Murray, M. Rogan, C. Sanders, S. Schmitz, E. Takaki, J.D.T. Wang, Q. Wilson, G. Ivanov, A. Kaadze, K. Maravin, Y. Mendis, D.R. Mitchell, T. Modak, A. Mohammadi, A. Saini, L.K. Skhirtladze, N. Taylor, R. Rebassoo, F. Wright, D. Baden, A. Baron, O. Belloni, A. Eno, S.C. Feng, Y. Ferraioli, C. Hadley, N.J. Jabeen, S. Jeng, G.Y. Kellogg, R.G. Kunkle, J. Mignerey, A.C. Ricci-Tam, F. Shin, Y.H. Skuja, A. Tonwar, S.C. Wong, K. Abercrombie, D. Allen, B. Azzolini, V. Barbieri, R. Baty, A. Bauer, G. Bi, R. Brandt, S. Busza, W. Cali, I.A. D'Alfonso, M. Demiragli, Z. Ceballos, G.G. Goncharov, M. Harris, P. Hsu, D. Hu, M. Iiyama, Y. Innocenti, G.M. Klute, M. Kovalskyi, D. Lee, Y.-J. Levin, A. Luckey, P.D. Maier, B. Marini, A.C. McGinn, C. Mironov, C. Narayanan, S. Niu, X. Paus, C. Roland, C. Roland, G. Stephans, G.S.F. Sumorok, K. Tatar, K. Velicanu, D. Wang, J. Wang, T.W. Wyslouch, B. Zhaozhong, S. Benvenuti, A.C. Chatterjee, R.M. Evans, A. Hansen, P. Kalafut, S. Kubota, Y. Lesko, Z. Mans, J. Nourbakhsh, S. Ruckstuhl, N. Rusack, R. Turkewitz, J. Wadud, M.A. Acosta, J.G. Cremaldi, L.M. Oliveros, S. Perera, L. Summers, D. Avdeeva, E. Bloom, K. Claes, D.R. Fangmeier, C. Golf, F. Suarez, R.G. Kamalieddin, R. Kravchenko, I. Monroy, J. Siado, J.E. Snow, G.R. Stieger, B. Godshalk, A. Harrington, C. Iashvili, I. Kharchilava, A. Nguyen, D. Parker, A. Rappoccio, S. Roozbahani, B. Alverson, G. Barberis, E. Freer, C. Hortiangtham, A. Morse, D.M. Orimoto, T. Lima, R.T.D. Wamorkar, T. Wang, B. Wisecarver, A. Wood, D. Bhattacharya, S. Charaf, O. Hahn, K.A. Mucia, N. Odell, N. Schmitt, M.H. Sevova, S. Sung, K. Trovato, M. Velasco, M. Bucci, R. Dev, N. Hildreth, M. Anampa, K.H. Jessop, C. Karmgard, D.J. Kellams, N. Lannon, K. Li, W. Loukas, N. Marinelli, N. Meng, F. Mueller, C. Musienko, Y. Planer, M. Reinsvold, A. Ruchti, R. Siddireddy, P. Smith, G. Taroni, S. Wayne, M. Wightman, A. Wolf, M. Woodard, A. Alimena, J. Antonelli, L. Bylsma, B. Durkin, L.S. Flowers, S. Francis, B. Hart, A. Hill, C. Ji, W. Ling, T.Y. Luo, W. Winer, B.L. Wulsin, H.W. Cooperstein, S. Elmer, P. Hardenbrook, J. Hebda, P. Higginbotham, S. Kalogeropoulos, A. Lange, D. Luo, J. Marlow, D. Mei, K. Ojalvo, I. Olsen, J. Palmer, C. Piroué, P. Salfeld-Nebgen, J. Stickland, D. Tully, C. Malik, S. Norberg, S. Vargas, J.E.R. Barker, A. Barnes, V.E. Das, S. Gutay, L. Jones, M. Jung, A.W. Khatiwada, A. Miller, D.H. Neumeister, N. Peng, C.C. Qiu, H. Schulte, J.F. Sun, J. Thieman, J. Wang, F. Xiao, R. Xie, W. Cheng, T. Dolen, J. Parashar, N. Chen, Z. Ecklund, K.M. Freed, S. Geurts, F.J.M. Guilbaud, M. Kilpatrick, M. Li, W. Michlin, B. Nussbaum, T. Padley, B.P. Roberts, J. Rorie, J. Shi, W. Tu, Z. Zabel, J. Zhang, A. Betchart, B. Bodek, A. De Barbaro, P. Demina, R. Duh, Y.T. Dulemba, J.L. Fallon, C. Ferbel, T. Galanti, M. Garcia-Bellido, A. Han, J. Hindrichs, O. Khukhunaishvili, A. Lo, K.H. Petrillo, G. Tan, P. Taus, R. Verzetti, M. Agapitos, A. Bartz, E. Chou, J.P. Gershtein, Y. Espinosa, T.A.G. Halkiadakis, E. Heindl, M. Hughes, E. Kaplan, S. Elayavalli, R.K. Kyriacou, S. Lath, A. Montalvo, R. Nash, K. Osherson, M. Saka, H. Salur, S. Schnetzer, S. Sheffield, D. Somalwar, S. Stone, R. Thomas, S. Thomassen, P. Walker, M. Delannoy, A.G. Heideman, J. Riley, G. Rose, K. Spanier, S. Thapa, K. Bouhali, O. Hernandez, A.C. Celik, A. Dalchenko, M. Mattia, M.D. Delgado, A. Dildick, S. Eusebi, R. Gilmore, J. Huang, T. Kamon, T. Mueller, R. Osipenkov, I. Pakhotin, Y. Patel, R. Perloff, A. Perniè, L. Rathjens, D. Safonov, A. Tatarinov, A. Akchurin, N. Damgov, J. Guio, F.D. Dudero, P.R. Faulkner, J. Kunori, S. Lamichhane, K. Lee, S.W. Mengke, T. Muthumuni, S. Peltola, T. Undleeb, S. Volobouev, I. Wang, Z. D'Angelo, P. Greene, S. Gurrola, A. Janjam, R. Johns, W. Maguire, C. Melo, A. Ni, H. Padeken, K. Alvarez, J.D.R. Sheldon, P. Tuo, S. Velkovska, J. Xu, Q. Arenton, M.W. Barria, P. Cox, B. Hirosky, R. Joyce, M. Ledovskoy, A. Li, H. Neu, C. Sinthuprasith, T. Wang, Y. Wolfe, E. Xia, F. Harr, R. Karchin, P.E. Poudyal, N. Sturdy, J. Thapa, P. Zaleski, S. Brodski, M. Buchanan, J. Caillol, C. Carlsmith, D. Dasu, S. Dodd, L. Duric, S. Gomber, B. Grothe, M. Herndon, M. Hervé, A. Hussain, U. Klabbers, P. Lanaro, A. Levine, A. Long, K. Loveless, R. Maurisset, A. Ruggles, T. Savin, A. Smith, N. Smith, W.H. Woods, N.

Subjects
Physics::Instrumentation and Detectors, Physics::Accelerator Physics, High Energy Physics::Experiment, Nuclear Experiment
Abstract

The structure of the CMS inner tracking system has been studied using nuclear interactions of hadrons striking its material. Data from proton-proton collisions at a center-of-mass energy of 13 TeV recorded in 2015 at the LHC are used to reconstruct millions of secondary vertices from these nuclear interactions. Precise positions of the beam pipe and the inner tracking system elements, such as the pixel detector support tube, and barrel pixel detector inner shield and support rails, are determined using these vertices. These measurements are important for detector simulations, detector upgrades, and to identify any changes in the positions of inactive elements. © 2018 CERN for the benefit of the CMS collaboration.

Published
2018

24. International Registry and Educational Portal on Brugada Phenocopies

Author

Daniel D. Anselm, Byron H. Gottschalk, and Grace Xu

Subjects
Natural history, Phenocopy, medicine.medical_specialty, Myocardial ischemia, business.industry, education, medicine, Etiology, General hospital, Intensive care medicine, business, Pericardial disease
Abstract

Since recognizing the Brugada Phenocopy (BrP) concept, associations with various clinical conditions including myocardial ischemia, pericardial disease, and metabolic derangements continue to emerge. Baranchuk et al. at Queen's University and Kingston General Hospital in Canada developed an initiative to investigate the etiology, pathophysiology, and long-term natural history of BrP. They created an international registry and online educational portal at http://www.brugadaphenocopy.com/ which allowed them to collate these cases, identify patterns, and assess associations with morbidity and mortality on a longitudinal basis.

Published
2018

25. List of Contributors

Author

Bryce Alexander, Pavel Antiperovitch, Daniel D. Anselm, Raimundo Barbosa-Barros, Héctor Barajas-Martinez, Adrian Baranchuk, Antoni Bayés de Luna, David G. Benditt, Eugene H. Chung, Gregory Dendramis, Luis Alberto Escobar-Robledo, Javier García-Niebla, Byron H. Gottschalk, Dan Hu, Ankit Maheshwari, Albert Massó van Roessel, Andrés R. Pérez-Riera, Pieter G. Postema, Guillem Serra, Shiva Sreenivasan, Grace Xu, and Zhong-Qun Zhan

Published
2018

26. Atypical Chest Pain and a Blood Blister: More Than Meets the Eye

Author

Michael W.A. Chu, Philip M. Jones, Satoru Fujii, and Byron H. Gottschalk

Subjects
Male, medicine.medical_specialty, Chest Pain, Saphenous vein graft, 030204 cardiovascular system & hematology, Coronary Angiography, 03 medical and health sciences, 0302 clinical medicine, Aneurysm, Percutaneous Coronary Intervention, Medicine, Humans, 030212 general & internal medicine, Aged, Multimodal imaging, business.industry, Coronary Aneurysm, Atypical chest pain, medicine.disease, Surgery, medicine.anatomical_structure, Blood blister, Echocardiography, Cardiology and Cardiovascular Medicine, business, Artery
Abstract

Brief summary We present a series of multimodal imaging and videos of an interesting case of a 76-year-old patient who presented 15 years after coronary artery bypass with a ruptured saphenous vein graft aneurysm that had eroded though the chest wall and skin.

Published
2017

27. The Use of Fontaine Leads in the Diagnosis of Arrhythmogenic Right Ventricular Dysplasia

Author

Michael Gysel, Andrés Ricardo Pérez-Riera, Guy Fontaine, Byron H. Gottschalk, Li Zhang, Adrian Baranchuk, Ricardo Paulo De Sousa Rocha, and Raimundo Barbosa-Barros

Subjects
medicine.medical_specialty, business.industry, Diagnostic accuracy, General Medicine, Emergency department, medicine.disease, Ventricular tachycardia, Arrhythmogenic right ventricular dysplasia, Sustained ventricular tachycardia, Physiology (medical), Internal medicine, cardiovascular system, Cardiology, medicine, Palpitations, Ventricular outflow tract, In patient, cardiovascular diseases, medicine.symptom, Cardiology and Cardiovascular Medicine, business
Abstract

We report a case of a 68-year-old man admitted to the emergency department with syncope preceded by rapid palpitations. His admission ECG demonstrated a sustained ventricular tachycardia (VT) originating from the right ventricular outflow tract (RVOT). This report highlights the importance of distinguishing ventricular tachycardia caused by arrhythmogenic right ventricular dysplasia (ARVD) from the more benign idiopathic RVOT-VT. Furthermore, we demonstrate the utility of the Fontaine leads placement in increasing the sensitivity for uncovering epsilon waves, a highly specific electrocardiographic feature that increases diagnostic accuracy in patients with ARVD.

Published
2014

28. Brugada phenocopy due to hypokalemia

Author

Pavel Antiperovitch, Byron H. Gottschalk, and Adrian Baranchuk

Subjects
Phenocopy, business.industry, lcsh:R, MEDLINE, lcsh:Medicine, 030204 cardiovascular system & hematology, Bioinformatics, Letter to Editor, Hypokalemia, 03 medical and health sciences, 0302 clinical medicine, medicine, 030212 general & internal medicine, medicine.symptom, business
Published
2018

29. Expert cardiologists cannot distinguish between Brugada phenocopy and Brugada syndrome electrocardiogram patterns

Author

Hanno L. Tan, Arthur A.M. Wilde, Pablo A. Chiale, Marcelo V. Elizari, Byron H. Gottschalk, Antoni Bayés de Luna, Daniel D. Anselm, Andrew D. Krahn, Josep Brugada, Pieter G. Postema, Pedro Brugada, Adrian Baranchuk, Andrés Ricardo Pérez-Riera, Amsterdam Cardiovascular Sciences, Cardiology, Clinical sciences, and Cardio-vascular diseases

Subjects
medicine.medical_specialty, Pathology, Visual interpretation, 030204 cardiovascular system & hematology, Sensitivity and Specificity, Diagnosis, Differential, Electrocardiography, 03 medical and health sciences, Cardiologists, 0302 clinical medicine, Clinical history, Physiology (medical), Internal medicine, medicine, Humans, 030212 general & internal medicine, cardiovascular diseases, Brugada Syndrome, Brugada syndrome, Brugada ECG pattern, Medicine(all), Phenocopy, medicine.diagnostic_test, business.industry, Brugada phenocopy, fungi, medicine.disease, Phenotype, Brugada ECG Pattern, Cardiology, Cardiology and Cardiovascular Medicine, business
Abstract

Aims Brugada phenocopies (BrPs) are electrocardiogram (ECG) patterns that are identical to true Brugada syndrome (BrS) but are induced by various clinical conditions. The concept that both ECG patterns are visually identical has not been formally demonstrated. The aim of our study was to determine if experts on BrS were able to accurately distinguish between the BrS and BrP ECG patterns. Methods and results Six ECGs from confirmed cases of BrS and six ECGs from previously published cases of BrP were included in the study. Surface 12-lead ECGs were scanned, saved in JPEG format, and sent to 10 international experts on BrS for evaluation (no clinical history provided). Evaluators were asked to label each case as a Brugada ECG pattern or non-Brugada ECG pattern by visual interpretation alone. The overall accuracy was 53 ± 33% for all cases. Within the BrS cases, the mean accuracy was 63 ± 34% and within the BrP cases, the mean accuracy was 43 ± 33%. Intra-observer repeatability was moderate ( κ = 0.56) and inter-observer agreement was fair ( κ = 0.36) while evaluator accuracy vs. the true diagnosis was only marginally better than chance ( κ = 0.05). Similarly, diagnostic operating characteristics were poor (sensitivity 62%, specificity 43%, +LR 1.1, −LR 0.9). Conclusion Our results provide strong evidence that BrP and BrS ECG patterns are visually identical and indistinguishable. These findings support the use of systematic diagnostic criteria for differentiating BrP vs. BrS as an erroneous diagnosis may have a negative impact on patient morbidity and mortality.

Published
2016

30. Relationship between Brugada Phenocopy and Myocardial Ischemia: Results from the International Registry on Brugada Phenocopy

Author

Raimundo Barbosa-Barros, Adrian Baranchuk, Sahil Agrawal, Antonio Bayés de Luna, Byron H. Gottschalk, Gregory Dendramis, Aldo G. Carrizo, Grace Xu, and Andrés Ricardo Pérez-Riera

Subjects
Phenocopy, medicine.medical_specialty, Myocardial ischemia, business.industry, Internal medicine, Cardiology, Medicine, Cardiology and Cardiovascular Medicine, business
Published
2018

31. Relation of the Brugada Phenocopy to Hyperkalemia (From the International Registry on Brugada Phenocopy)

Author

Shiva Sreenivasan, Ankit Maheshwari, Grace Xu, Daniel D. Anselm, Hector Barajas-Martinez, Sam G. Aznaurov, Gregory Dendramis, Raed Abu Shama, David G. Benditt, Adrian Baranchuk, Byron H. Gottschalk, and José Manuel Rubio Campal

Subjects
Adult, Male, medicine.medical_specialty, Hyperkalemia, 030204 cardiovascular system & hematology, Sudden cardiac death, Electrocardiography, 03 medical and health sciences, Hyperphosphatemia, 0302 clinical medicine, Sodium channel blocker, Internal medicine, medicine, Humans, Hypocalcaemia, Registries, 030212 general & internal medicine, cardiovascular diseases, Aged, Brugada Syndrome, Brugada syndrome, Aged, 80 and over, Phenocopy, business.industry, nutritional and metabolic diseases, Middle Aged, medicine.disease, Phenotype, Cardiology, Female, medicine.symptom, Cardiology and Cardiovascular Medicine, business, Hyponatremia
Abstract

Brugada phenocopies (BrPs) are clinical entities that differ in etiology from true congenital Brugada syndrome but have identical electrocardiographic (ECG) patterns. Hyperkalemia is known to be one of the causes of BrP. The aim of this study was to determine the clinical characteristics and evolution of hyperkalemia-induced BrP. Data from 27 cases of hyperkalemia-induced BrP were collected from the International Registry at www.brugadaphenocopy.com . Data were extracted from publications. Of the 27 patients included in the analysis, 18 (67%) were male; mean age was 53 ± 15 years (range 31 to 89). Mean serum potassium concentration was 7.45 ± 0.89 mmol/L. Type-1 Brugada ECG pattern was observed in 21 cases (78%), whereas 6 cases (22%) showed a type-2 Brugada ECG pattern. The Brugada ECG pattern resolved once the hyperkalemia was corrected, with no arrhythmic events. Estimated time to resolution was 7 ± 3 hours. In 4 cases (16%), a concurrent metabolic abnormality was detected: 3 (11%) presented with acidosis, 2 (7%) with hyponatremia, 1 (4%) with hypocalcaemia, 1 (4%) with hyperphosphatemia, and 1 (4%) with hyperglycemia. In 7 cases (26%), provocative testing using sodium channel blockers was performed, and all failed to reproduce a BrS ECG pattern (BrP class A). Additionally, no sudden cardiac death or malignant ventricular arrhythmias were detected. Hyperkalemia was found a common cause of BrP in our International Registry. The Brugada ECG pattern appears to occur at high serum potassium concentrations (>6.5 mmol/L). The ECG normalizes within hours of correcting the electrolyte imbalance. Importantly, hyperkalemia-induced BrP has not been associated with sudden cardiac death or ventricular arrhythmia.

Published
2018

32. MINIMALLY INVASIVE PERIAREOLAR APPROACH TO REPAIR OF COR TRIATRIATUM

Author

Byron H. Gottschalk, A. Grant, Satoru Fujii, Ali Hage, Michael W.A. Chu, and Ivan Iglesias

Subjects
medicine.medical_specialty, business.industry, Cor triatriatum, Medicine, Cardiology and Cardiovascular Medicine, business, medicine.disease, Periareolar, Surgery
Published
2018

33. Not All Brugada Electrocardiogram Patterns are Brugada Syndrome or Brugada Phenocopy

Author

Adrian Baranchuk, Umut Kocabaş, Byron H. Gottschalk, and Grace Xu

Subjects
medicine.medical_specialty, congenital, hereditary, and neonatal diseases and abnormalities, Adolescent, Midazolam, lcsh:Medicine, Suicide, Attempted, 030204 cardiovascular system & hematology, Electrocardiography, 03 medical and health sciences, 0302 clinical medicine, Propafenone, Seizures, Internal medicine, medicine, Humans, cardiovascular diseases, Letter to the Editor, Brugada Syndrome, Brugada syndrome, Chicago, Phenocopy, business.industry, lcsh:R, General Medicine, medicine.disease, Treatment Outcome, 030228 respiratory system, Adolescent Behavior, Electrocardiography, Ambulatory, Cardiology, Anticonvulsants, Female, business, Sodium Channel Blockers
Abstract

Brugada syndrome is an inherited arrhythmogenic disease that may cause sudden cardiac death due to ventricular fibrillation in young adults. Brugada syndrome caused by propafenone intoxication has been noted rarely in the literature. We report a rare case, Brugada phenocopy due to propafenone intoxication and its treatment.A 15-year-old girl having a seizure was brought to the emergency room. She took 1.5 g propafenone (Rythmol, Abbott, Chicago, IL, USA) for suicidal intention. She had metabolic acidosis. Long QRS interval and ST elevation in leads V1 through V3 were seen on electrocardiography. After bicarbonate infusion for 4 hours, haemodynamic and neurologic findings were recovered, and all electrocardiography abnormalities disappeared. The Brugada-like electrocardiography pattern was not recognized in her surface and 24-hour Holter electrocardiography at follow-up. Ajmaline challenge test was negative 2 weeks later.Absence of symptoms and documented ventricular tachycardia, negative challenge test, and a negative family history demonstrated that the Brugada phenocopy was a transient finding in this case and related to propafenone intoxication.

Published
2017

35. Methods for Improving the Diagnosis of a Brugada ECG Pattern

Author

Byron H, Gottschalk, Javier, Garcia-Niebla, Daniel D, Anselm, Benedict, Glover, and Adrian, Baranchuk

Subjects
Adult, Diagnosis, Differential, Male, congenital, hereditary, and neonatal diseases and abnormalities, Electrocardiography, fungi, Humans, cardiovascular diseases, Case Reports, Brugada Syndrome
Abstract

Brugada syndrome (BrS) is an inherited channelopathy that predisposes individuals to malignant arrhythmias and can lead to sudden cardiac death. The condition is characterized by two electrocardiography (ECG) patterns: the type‐1 or “coved” ECG and the type‐2 or “saddleback” ECG. Although the type‐1 Brugada ECG pattern is diagnostic for the condition, the type‐2 Brugada ECG pattern requires differential diagnosis from conditions that produce a similar morphology. In this article, we present a case that is suspicious but not diagnostic for BrS and discuss the application of ECG methodologies for increasing or decreasing suspicion for a diagnosis of BrS.

Published
2015

36. Coronary anomalies resulting in ischemia induced Brugada Phenocopy

Author

Daniel D. Anselm, Adrian Baranchuk, and Byron H. Gottschalk

Subjects
Phenocopy, Male, medicine.medical_specialty, business.industry, Ischemia, medicine.disease, Coronary Vessels, Arterio-Arterial Fistula, Internal medicine, medicine, Cardiology, Humans, Cardiology and Cardiovascular Medicine, business, Brugada syndrome, Brugada Syndrome
Published
2015

37. Hypophosphatemia as a novel etiology of Brugada Phenocopy

Author

Usama Boles, Adrian Baranchuk, Byron H. Gottschalk, Jennifer Meloche, and Stephen A. LaHaye

Subjects
Phenocopy, medicine.medical_specialty, medicine.diagnostic_test, business.industry, 030204 cardiovascular system & hematology, medicine.disease, 03 medical and health sciences, 0302 clinical medicine, Endocrinology, 030228 respiratory system, Electrolyte imbalance, Internal medicine, Brugada ECG Pattern, medicine, Cardiology, Etiology, Cardiology and Cardiovascular Medicine, business, Electrocardiography, Hypophosphatemia, Brugada syndrome
Published
2016

38. Suspected Brugada phenocopy in the context of hypothermia

Author

Adrian Baranchuk, Daniel D. Anselm, and Byron H. Gottschalk

Subjects
Phenocopy, medicine.medical_specialty, business.industry, Context (language use), General Medicine, Hypothermia, medicine.disease, Electrocardiography, Internal medicine, Cardiology, medicine, Humans, Female, medicine.symptom, Cardiology and Cardiovascular Medicine, business, Brugada syndrome
Abstract

(2014). Suspected Brugada phenocopy in the context of hypothermia. Acta Cardiologica: Vol. 69, No. 4, pp. 454-455.

Published
2014

39. Brugada phenocopies: consideration of morphologic criteria and early findings from an international registry

Author

Adrian Baranchuk, Byron H. Gottschalk, and Daniel D. Anselm

Subjects
Phenocopy, medicine.medical_specialty, Myocardial ischemia, medicine.diagnostic_test, business.industry, Precordial examination, medicine.disease, Sudden cardiac death, Diagnosis, Differential, Electrocardiography, Internal medicine, medicine, Cardiology, Humans, cardiovascular diseases, Registries, Cardiology and Cardiovascular Medicine, business, Brugada syndrome, Brugada Syndrome
Abstract

Brugada syndrome (BrS) is an inherited sudden cardiac death syndrome characterized by type 1 or type 2 electrocardiographic (ECG) patterns in precordial leads V1-V3 that predispose individuals to malignant ventricular arrhythmias and sudden cardiac death. Brugada phenocopies (BrPs) are clinical entities that have ECG patterns that are identical to true congenital BrS but are elicited by various other clinical or technical factors such as myocardial ischemia, metabolic abnormalities, mechanical mediastinal compression, or poor electrocardiographic (ECG) filters. This article discusses the conceptual emergence of BrP as a new ECG phenomenon, reviews the current BrP diagnostic criteria, and provides a new morphologic classification that more precisely categorizes future BrP cases. Preliminary results from the International Registry of Brugada Phenocopies (www.brugadaphenocopy.com) are also discussed.

Published
2014

40. Phosphine Poisoning is Emerging as an Important Cause of Brugada Phenocopy

Author

Daniel D. Anselm, Adrian Baranchuk, and Byron H. Gottschalk

Subjects
Phenocopy, medicine.medical_specialty, Injury control, Accident prevention, business.industry, PHOSPHINE POISONING, Poison control, 030208 emergency & critical care medicine, Context (language use), General Medicine, 030204 cardiovascular system & hematology, medicine.disease, 03 medical and health sciences, 0302 clinical medicine, Brugada ECG Pattern, medicine, Medical emergency, Cardiology and Cardiovascular Medicine, Intensive care medicine, business, Brugada syndrome
Abstract

We read the report by Prabhu et al. with great interest and noted their description of a Brugada ECG pattern in the context of phosphide poisoning. Their case is important as it contributes to our understanding of the varying etiologies of Brugada Phenocopy (BrP). This article is protected by copyright. All rights reserved. Language: en

Published
2015

41. Brugada phenocopies are the leading differential diagnosis of Brugada syndrome

Author

Daniel D. Anselm, Byron H. Gottschalk, and Adrian Baranchuk

Subjects
Phenocopy, Pediatrics, medicine.medical_specialty, business.industry, medicine, General Medicine, Differential diagnosis, medicine.disease, business, Brugada syndrome
Abstract

OVERVIEW Please submit letters for the editor9s consideration within three weeks of receipt of Clinical Medicine. Letters should ideally be limited to 350 words, and sent by email to: clinicalmedicine@rcplondon.ac.uk

Published
2015

42. Brugada phenocopy in the context of intracranial hemorrhage

Author

Adrian Baranchuk, Claudio Hadid, Mirza Rivero, Daniel D. Anselm, Byron H. Gottschalk, Juan J. Fuselli, and Carlos Labadet

Subjects
Phenocopy, medicine.medical_specialty, business.industry, Internal medicine, Intracranial Hemorrhages, Brugada ECG Pattern, medicine, Cardiology, Context (language use), Cardiology and Cardiovascular Medicine, medicine.disease, business, Brugada syndrome
Published
2014

43. Brugada phenocopy induced by ischemia or Brugada syndrome unmasked by ischemia?

Author

Adrian Baranchuk, Byron H. Gottschalk, and Daniel D. Anselm

Subjects
Male, medicine.medical_specialty, medicine.diagnostic_test, business.industry, medicine.medical_treatment, Ischemia, Atrial fibrillation, Catheter ablation, medicine.disease, Pulmonary embolism, Electrocardiography, Internal medicine, Coronary vasospasm, Atrial Fibrillation, Troponin I, Catheter Ablation, Cardiology, Humans, Medicine, Cardiology and Cardiovascular Medicine, business, Brugada Syndrome, Brugada syndrome
Abstract

We read the casereport by Jiang et al. with great interest and notedthat their patient presented with a Type-1 Brugada ECG pattern after aradiofrequency catheter ablation procedure for atrial fibrillation [1].The caseis important asit contributesto thegrowingbodyof literatureon Brugada phenocopy (BrP) [2,3].BrP are clinical entities that present with ECGs identical to those oftrueBrugadasyndrome(BrS)butareelicitedbyvariousclinicalcircum-stances [2,3]. They may be induced by a number of clinical conditionsthat are characterized into six etiological categories: (i) metabolicconditions; (ii) mechanical compression; (iii) myocardial ischemia p (iv) myocardial & pericardial disease; (v) ECGmodulations; and (vi) miscellaneous [4]. The hallmark to diagnosislies in a series of electrocardiographic and clinical features (Table 1)that aim to differentiate BrP from BrS [5,6]. More information onthe condition and a stepwise diagnostic approach can be found at theinternational registry and online educational portal for BrP (www.brugadaphenocopy.com).In the case presented by Jiang et al. [1], the patient developed aBrugada ECG pattern and a marked increase in troponin I (TnI) follow-ing an ablation procedure for atrial fibrillation. We agree with theauthors that the mechanism behind this phenomenon was unlikelydue to coronary vasospasm as the ST-elevations were sustained over anumber of days. Michael et al. described a similar case of ST-segmentelevations that occurred during an ablation procedure for atrialfibrillation [7]. An urgent coronary angiogram revealed a sub-totalocclusion within the inferior branch of the 1st diagonal. We suspectthat ischemia was alsothelikely mechanismbehindST-segment eleva-tions and increased TnI in the present case [1]. Blood clots may beextremely small, and lysis of these clots can occur in a matter of mi-nutes.Itisconceivabletherefore,thatthiswasanembolicphenomenonthat was not captured by coronary angiography.As previously discussed by our group [3], ischemia may eitherinduce a BrP [5] or modulate myocardial sodium channels resulting intheunmaskingoftrueBrS[8].Aprovocativetestusingasodiumchannelblocker was therefore mandatory in this case. The negative result,coupled with the patient's lack of personal or family history suggestiveof BrS, allows us to confirm the diagnosis of BrP in this case. It will beincluded in our database as a Type 1A BrP under the category of ische-mia and pulmonary embolism. We recommend the authors use theterminologyBrugadaphenocopyinthefutureforconsistencyintheliter-ature and to facilitate future research on this clinical phenomenon.Conflict of interestNoneReferences

Published
2014

44. ptandxFdependence of the polarization ofΣ+hyperons produced by 800 GeV/cprotons

Author

A. Morelos, I. F. Albuquerque, N. F. Bondar, R. Carrigan, D. Chen, P. S. Cooper, Dai Lisheng, A. S. Denisov, A. V. Dobrovolsky, T. Dubbs, A. M. F. Endler, C. O. Escobar, M. Foucher, V. L. Golovtsov, H. Gottschalk, P. Gouffon, V. T. Grachev, A. V. Khanzadeev, M. A. Kubantsev, N. P. Kuropatkin, J. Lach, null Lang Pengfei, V. N. Lebedenko, null Li Chengze, null Li Yunshan, M. Luksys, J. R. P. Mahon, E. McCliment, C. Newsom, M. C. Pommot Maia, V. M. Samsonov, V. A. Schegelsky, null Shi Huanzhang, V. J. Smith, Tang Fukun, N. K. Terentyev, S. Timm, I. I. Tkatch, L. N. Uvarov, A. A. Vorobyov, null Yan Jie, Zhao Wenheng, null Zheng Shuchen, and null Zhong Yuanyuan

Subjects
Physics, Nuclear physics, Hyperon, Sigma, Nuclear Experiment, Polarization (waves), Lambda
Abstract

We utilize the angle and momentum resolution of our apparatus to study the polarization of 375 GeV/c Sigma(+) hyperons produced by 800 GeV/c protons incident on a Cu target. By examining in detail two of our high statistics data samples, we find evidence for structure in the p(t) dependence of Sigma(+) polarization and are able to extract the x(F) dependence of the Sigma(+) polarization and compare it with x(F) behavior in the Lambda(0) and Xi(-) systems.

Published
1995

45. Measurement of the branching ratio and asymmetry parameter for theΣ+→pγ radiative decay

Author

Lang Pengfei, N. P. Kuropatkin, V. T. Grachev, Vincent J Smith, Shi Huanzhang, M. C. Pommot Maia, I. I. Tkatch, N. F. Bondar, A. Morelos, A. V. Dobrovolsky, M. A. Kubantsev, M. Luksys, Yan Jie, V.A. Schegelsky, T. Dubbs, V. L. Golovtsov, Zhao Wenheng, Zheng Shuchen, Alexei Khanzadeev, I. F. Albuquerque, Li Chengze, An.A. Vorobyov, A. S. Denisov, P. Gouffon, Carlos Escobar, E. McCliment, J. R. P. Mahon, M. Foucher, L. N. Uvarov, Dai Lisheng, R.A. Carrigan, P. S. Cooper, Tang Fukun, A. M. F. Endler, C. R. Newsom, H. Gottschalk, Li Yunshan, D Chen, N. K. Terentyev, S. Timm, J. Lach, Zhong Yuanyuan, V. M. Samsonov, and J. Langland

Subjects
Nuclear physics, Physics, Particle physics, Branching fraction, media_common.quotation_subject, Hyperon, Radiative decay, Sigma, High Energy Physics::Experiment, Asymmetry, media_common
Abstract

We have measured the branching ratio for the hyperon radiative decay ${\mathrm{\ensuremath{\Sigma}}}^{+}$\ensuremath{\rightarrow}p\ensuremath{\gamma} using the Fermilab polarized charged hyperon beam. This measurement and our previously published result on the asymmetry parameter in the same decay are part of Fermilab experiment E761. We find B(${\mathrm{\ensuremath{\Sigma}}}^{+}$\ensuremath{\rightarrow}p\ensuremath{\gamma})/B(${\mathrm{\ensuremath{\Sigma}}}^{+}$\ensuremath{\rightarrow}p${\mathrm{\ensuremath{\pi}}}^{0}$) to be [2.32\ifmmode\pm\else\textpm\fi{}0.11(stat)\ifmmode\pm\else\textpm\fi{}0.10(syst)]\ifmmode\times\else\texttimes\fi{}${10}^{\mathrm{\ensuremath{-}}3}$ with a sample of 31 901 events. The higher statistics and careful attention to systematic uncertainties make these significant improvements over previous measurements. We describe how our measurements were performed and briefly review the theoretical implications of these results.

Published
1995

46. Determination of transition curves of high-Tc superconductors by phase contrast microscopy

Author

H. Alexander, D. Wohlleben, H. Gottschalk, C.-L. Kriebel, R. Borowski, and O. Hoffels

Subjects
Physics, Superconductivity, Phase transition, Condensed matter physics, General Physics and Astronomy, Microscopic scale, law.invention, Magnetic field, law, Meissner effect, Transmission electron microscopy, Condensed Matter::Superconductivity, Electron microscope, Intensity (heat transfer)
Abstract

A new method which allows the detection of the superconducting phase transition of high-Tc superconductors (HTSC) on a microscopic scale is reported. Micro-size holes in thin foils of superconducting material are examined in a transmission electron microscope at varying temperatures. The superconducting transition induces small changes in the image intensity within the holes, which can be detected by using electronic image analysis. Superconducting transition curves are then obtained for various types of high-Tc superconductors and for given values of the applied magnetic field.

Published
1995

47. Measurement of the branching ratio for theΞ−→Σ−γ radiative decay

Author

Yan Jie, Dai Lisheng, Shi Huanzhang, Lang Pengfei, D. Chen, M. C. Pommot Maia, A. V. Dobrovolsky, M. Luksys, J. R. P. Mahon, N. K. Terentyev, E. McCliment, Tang Fukun, A. M. F. Endler, H. Gottschalk, A. V. Khanzadeev, N. F. Bondar, V.A. Schegelsky, A. A. Vorobyov, Zhong Yuanyuan, P. S. Cooper, M. A. Kubantsev, M. Foucher, A. Morelos, J. Lach, P. Gouffon, A. S. Denisov, C. R. Newsom, I. F. Albuquerque, S. Timm, L. N. Uvarov, Zhao Wenheng, Li Yunshan, Zheng Shuchen, N. P. Kuropatkin, V. T. Grachev, Li Chengze, I. I. Tkatch, T. Dubbs, Carlos Escobar, R. Carrigan, Vincent J Smith, V. L. Golovtsov, and V. M. Samsonov

Subjects
Physics, Baryon, Particle physics, Particle decay, Branching fraction, Hadron, Hyperon, General Physics and Astronomy, Lambda, Xi baryon, Sigma baryon
Abstract

We have measured the branching ratio for the hyperon radiative decay [Xi][sup [minus]][r arrow][Sigma][sup [minus]][gamma] from a sample of 211[plus minus]33 events obtained in the polarized 375 GeV/[ital c] charged hyperon beam at Fermilab. We find [ital B]([Xi][sup [minus]][r arrow][Sigma][sup [minus]][gamma]/[Xi][sup [minus]][r arrow][Lambda][sup 0][pi][sup [minus]])=(1.22[plus minus]0.23[plus minus]0.06)[times]10[sup [minus]4] where the quoted errors are statistical and systematic, respectively. We have also obtained an indication that the sign of the asymmetry parameter of this decay is positive.

Published
1994

48. Measurement of the magnetic moments of theΣ+and Σ¯−hyperons

Author

N. P. Kuropatkin, V. T. Grachev, P. Gouffon, I. F. Albuquerque, M. Foucher, Lang Pengfei, Carlos Escobar, Tkatch, V. M. Samsonov, L. N. Uvarov, A. V. Dobrovolsky, Dai Lisheng, Li Chengze, Li Yunshan, E. McCliment, A. S. Denisov, M. Luksys, V.A. Schegelsky, N. K. Terentyev, P. S. Cooper, R.A. Carrigan, C. R. Newsom, S. Timm, A. Morelos, Zhong Yuanyuan, M. A. Kubantsev, N. F. Bondar, Shi Huanzhang, An.A. Vorobyov, T. Dubbs, J. Lach, Pommot Maia Mc, Tang Fukun, A. M. F. Endler, H. Gottschalk, Alexei Khanzadeev, D Chen, Zhao Wenheng, Zheng Shuchen, V. L. Golovtsov, Vincent J Smith, Yan Jie, and Mahon

Subjects
Baryon, Physics, Antiparticle, Proton, Magnetic moment, CPT symmetry, General Physics and Astronomy, Sigma, High Energy Physics::Experiment, Fermion, Statistical physics, Atomic physics, Sigma baryon
Abstract

We have measured the magnetic moments of the SIGMA+ and SIGMABAR- hyperons produced by 800-GeV protons incident on a Cu target. We determine the SIGMA+ magnetic moment to be (2.4613 +/- 0.0034 +/- 0.0040)mu(N) where the uncertainties are statistical and systematic, respectively. In this first measurement we determine the magnetic moment of the SIGMABAR- to be -(2.428 +/- 0.036 +/- 0.007)mu(N). The magnetic moments of the SIGMA+ and SIGMABAR- are consistent with each other in magnitude but opposite in sign as required by CPT invariance.

Published
1993

49. Polarization ofΣ+and Σ¯−hyperons produced by 800-GeV/cprotons

Author

P. Gouffon, M. Foucher, M. Luksys, V.A. Schegelsky, A. V. Dobrovolsky, Li Yunshan, Zhao Wenheng, Zheng Shuchen, I. I. Tkatch, Lang Pengfei, Yan Jie, Vincent J Smith, J. Lach, N. P. Kuropatkin, V. T. Grachev, J. R. P. Mahon, Tang Fukun, A. M. F. Endler, Dai Lisheng, H. Gottschalk, Shi Huanzhang, A. A. Vorobyov, N. K. Terentyev, A. V. Khanzadeev, M. A. Kubantsev, A. S. Denisov, V. M. Samsonov, E. McCliment, A. Morelos, S. Timm, R.A. Carrigan, Zhong Yuanyuan, D Chen, T. Dubbs, V. L. Golovtsov, I. F. Albuquerque, Li Chengze, L. N. Uvarov, M. C. Pommot Maia, N. F. Bondar, Carlos Escobar, P. S. Cooper, and C. R. Newsom

Subjects
Physics, Hyperon, General Physics and Astronomy, Sigma, High Energy Physics::Experiment, Elementary particle, Atomic physics, Nuclear Experiment, Polarization (waves), Sigma baryon
Abstract

We have measured the polarization of 375-GeV/c SIGMA+ and SIGMABAR- hyperons produced by 800-GeV/c protons incident on a Cu target. We find that the SIGMA+ polarization rises with increasing p(t) to a maximum of 16% at p(t) = 1.0 GeV/c and then decreases to 10% at p(t) = 1.8 GeV/c. We compare this SIGMA+ polarization with data at lower energies. The SIGMABAR- polarization has been measured for the first time. It has the same sign as the SIGMA+ but smaller magnitude in a similar kinematical region.

Published
1993

50. Constricted dislocations and their use for TEM measurements of the velocities of edge and 60° dislocations in silicon. A new approach to the problem of kink migration

Author

S. Sauerland, Petra Specht, N. Hiller, H. Alexander, and H. Gottschalk

Subjects
Deformation (mechanics), Silicon, Condensed matter physics, Chemistry, chemistry.chemical_element, Collective motion, Activation energy, Edge (geometry), Dislocation, Condensed Matter Physics, Electronic, Optical and Magnetic Materials
Abstract

A new method of measuring dislocation velocities in the bulk by TEM is presented. Jogs on dislocations are strong obstacles against dislocation motion during a low temperature deformation. They can be used as markers for the initial course of the dislocation lines and allow to measure the paths travelled by the free dislocation segments between the jogs. First results for the activation energy of the motion of screws and 60° dislocations (2 eV) and for the edges (1.8 eV) are reported. An edge dislocation is assumed to be a series of geometrical kinks. Its motion is interpreted as the collective motion of kinks and the activation energy is tentatively attributed to the kink migration energy. Eine neue Methode, Versetzungsgeschwindigkeiten im Inneren eines Kristalls mit TEM zu messen, wird vorgestellt. Jogs auf Versetzungen sind Hindernisse fur die Versetzungsbewegung bei einer Niedertemperatur-Verformung. Sie markieren die Ausgangslage der Versetzungen und ermoglichen, den Laufweg der freien Segmente zwischen den Jogs zu messen. Erste Ergebnisse fur die Aktivierungs-energien von Schrauben-, 60° - (2 eV), und Stufenversetzungen (1.8 eV) werden mitgeteilt. Es wird angenommen, eine Stufenversetzung sei eine Aneinanderreihung geometrischer Kinken und ihre Bewegung sei korrelierter Kinkenbewegung zuzuschreiben. Deshalb wird ihre Aktivierungsenergie versuchsweise der Kinkenwanderungsenergie zugeordnet.

Published
1993

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