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257 results on '"Guatimosim, Cristina"'

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1. A suitable model to investigate acute neurological consequences of coronavirus infection

3. Weight-drop model as a valuable tool to study potential neurobiological processes underlying behavioral and cognitive changes secondary to mild traumatic brain injury

4. Neuromuscular defects after infection with a beta coronavirus in mice

7. Fast and slow-twitching muscles are differentially affected by reduced cholinergic transmission in mice deficient for VAChT: A mouse model for congenital myasthenia

8. Neuromuscular synapse degeneration without muscle function loss in the diaphragm of a murine model for Huntington's Disease

11. Renin-Angiotensin System in Huntington′s Disease: Evidence from Animal Models and Human Patients

12. Neuroprotective effect of CTK 01512‐2 recombinant toxin at the spinal cord in a model of Huntington's disease

17. A Biosafety Level 2 Mouse Model for Studying Betacoronavirus-Induced Acute Lung Damage and Systemic Manifestations

19. A suitable murine model for studying respiratory coronavirus infection and therapeutic countermeasures in BSL-2 laboratories

25. Increased Cholinergic Tone Causes Pre-synaptic Neuromuscular Degeneration and is Associated with Impaired Diaphragm Function

39. Evaluation of the neuromuscular junction in a middle‐aged mouse model of congenital myasthenic syndrome

42. Abnormalities in the Motor Unit of a Fast-Twitch Lower Limb Skeletal Muscle in Huntington’s Disease

43. Increased oxidative stress and CaMKIIactivity contribute to electro‐mechanical defects in cardiomyocytes from a murine model of Huntington's disease

46. Corrigendum to “Neuromuscular synapse degeneration without muscle function loss in the diaphragm of a murine model for Huntington's Disease” [Neurochem. Int. 116 (2018) 30–42]

47. Muscle atrophy is associated with cervical spinal motoneuron loss in BACHD mouse model for Huntington's disease

48. Increased oxidative stress and CaMKII activity contribute to electro‐mechanical defects in cardiomyocytes from a murine model of Huntington's disease.

49. VAChT overexpression increases acetylcholine at the synaptic cleft and accelerates aging of neuromuscular junctions

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