49 results on '"Gu, Ruimin"'
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2. Norepinephrine-Induced Stimulation of Kir4.1/Kir5.1 Is Required for the Activation of NaCl Transporter in Distal Convoluted Tubule
3. Bradykinin Stimulates Renal Na+ and K+ Excretion by Inhibiting the K+ Channel (Kir4.1) in the Distal Convoluted Tubule
4. Efficient Adsorption of Tl(I) from Aqueous Solutions Using Al and Fe-Based Water Treatment Residuals
5. In Situ Assay of Interfacial Interaction between ZnO Nanoparticles and Live Cell Disturbed by Surfactants
6. Budget Impact Analysis of Trastuzumab Biosimilar for the Treatment of Breast Cancer and Gastric Cancer in China
7. Thrombin and Its Receptor Enhance ST-Segment Elevation in Acute Myocardial Infarction by Activating the KATP Channel
8. Adenosine stimulates the basolateral 50 pS K channels in the thick ascending limb of the rat kidney
9. Dual effect of insulin-like growth factor on the apical 70-pS K channel in the thick ascending limb of rat kidney
10. PGF2α stimulates the 10-pS Cl− channel and thiazide-sensitive Na+-Cl− cotransporter in the distal convoluted tubule
11. Kir4.1/Kir5.1 Activity in the DCT is Essential for Angiotensin II Induced Stimulation of Thiazide‐Sensitive NCC Expression/Activity.
12. AT2R-mediated regulation of Na-Cl cotransporter (NCC) and renal K excretion depends on the K channel, Kir4.1
13. Inhibition of AT2R and BK2R attenuates the effect of high K intake (HK) on thiazide‐sensitive Na‐Cl cotransporter
14. Norepinephrine-Induced Stimulation of Kir4.1/Kir5.1 Is Required for the Activation of NaCl Transporter in Distal Convoluted Tubule
15. Bradykinin Stimulates Renal Na + and K + Excretion by Inhibiting the K + Channel (Kir4.1) in the Distal Convoluted Tubule
16. Kir4.1 is involved in Bradykinin‐induced inhibition of NCC and natriuresis
17. AT2R (Angiotensin II Type 2 Receptor)-Mediated Regulation of NCC (Na-Cl Cotransporter) and Renal K Excretion Depends on the K Channel, Kir4.1
18. PGF2αstimulates the 10-pS Cl−channel and thiazide-sensitive Na+-Cl−cotransporter in the distal convoluted tubule
19. Effect of dietary K intake on apical small-conductance K channel in CCD: role of protein tyrosine kinase
20. Role of 20-HETE in mediating the effect of dietary K intake on the apical K channels in the mTAL
21. Effects of adenosine stimulation on the mRNA expression of CLCNKB in the basolateral medullary thick ascending limb of the rat kidney
22. Disruption of KCNJ10 (Kir4.1) stimulates the expression of ENaC in the collecting duct
23. Vasopressin-induced stimulation of the Na+-activated K+ channels is responsible for maintaining the basolateral K+ conductance of the thick ascending limb (TAL) in EAST/SeSAME syndrome
24. ADH-induced Stimulation of Na-activated K Channels is Responsible for Maintaining Basolateral K Conductance of the Thick Ascending Limb (TAL) in EAST/Sesame Syndrome
25. Bradykinin Stimulates Renal Na+ and K+ Excretion by Inhibiting the K+ Channel (Kir4.1) in the Distal Convoluted Tubule.
26. AT2R (Angiotensin II Type 2 Receptor)-Mediated Regulation of NCC (Na-Cl Cotransporter) and Renal K Excretion Depends on the K Channel, Kir4.1.
27. Angiotensin II stimulates basolateral 50-pS K channels in the thick ascending limb
28. Angiotensin II Stimulates Basolateral 10-pS Cl Channels in the Thick Ascending Limb
29. Angiotensin II stimulates basolateral 10 pS Cl channels, a main type of Cl channels in the thick ascending limb (TAL)
30. Insulin-like growth factor-1 (IGF-1) inhibits the basolateral Cl channels in the thick ascending limb of the rat kidney
31. Stimulation of adenosine A1 receptor inhibits the basolateral 50 pS K channels in the thick ascending limb of rat kidney
32. Stimulation of Ca2+-sensing receptor inhibits the basolateral 50-pS K channels in the thick ascending limb of rat kidney
33. Stimulation of A2a adenosine receptor abolishes the inhibitory effect of arachidonic acid on the basolateral 50-pS K channel in the thick ascending limb
34. PGE2 inhibits basolateral 50pS potassium channels in the thick ascending limb of the rat kidney
35. PGE 2 inhibits the basolateral 50‐pS K channels in the thick ascending limb (TAL) of rat kidney by PKC and MAPK
36. Regulation of ROMK1 Channels by Protein-tyrosine Kinase and -tyrosine Phosphatase
37. The Role of 20-Hete in Mediating the Effect of Diatary K Intake on the Apical K Channels in the Mtal.
38. Protein-tyrosine Phosphatase Reduces the Number of Apical Small Conductance K+ Channels in the Rat Cortical Collecting Duct
39. PGE2 inhibits basolateral 50 pS potassium channels in the thick ascending limb of the rat kidney.
40. In SituAssay of Interfacial Interaction between ZnO Nanoparticles and Live Cell Disturbed by Surfactants
41. Stimulation of A2aadenosine receptor abolishes the inhibitory effect of arachidonic acid on the basolateral 50-pS K channel in the thick ascending limb
42. Protein-tyrosine Phosphatase Reduces the Number of Apical Small Conductance K+Channels in the Rat Cortical Collecting Duct*
43. Stimulation of Ca2+-sensing receptor inhibits the basolateral 50-pS K channels in the thick ascending limb of rat kidney
44. PGE2 inhibits basolateral 50 pS potassium channels in the thick ascending limb of the rat kidney.
45. Role of Kir4.1/Kir5.1 in mediating Angiotensin-II (Ang-II)-induced stimulation of thiazide-sensitive Na-Cl cotransporter.
46. PGF 2α stimulates the 10-pS Cl - channel and thiazide-sensitive Na + -Cl - cotransporter in the distal convoluted tubule.
47. Disruption of KCNJ10 (Kir4.1) stimulates the expression of ENaC in the collecting duct.
48. Angiotensin II stimulates basolateral 50-pS K channels in the thick ascending limb.
49. Stimulation of A(₂a) adenosine receptor abolishes the inhibitory effect of arachidonic acid on the basolateral 50-pS K channel in the thick ascending limb.
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