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1. Pannexin-1 opening in neuronal edema causes cell death but also leads to protection via increased microglia contacts.

2. KCC2 drives chloride microdomain formation in dendritic blebbing.

3. Mitochondrial Ca 2+ uptake by the MCU facilitates pyramidal neuron excitability and metabolism during action potential firing.

4. Ca 2+ transients in astrocyte fine processes occur via Ca 2+ influx in the adult mouse hippocampus.

5. Ca2+ removal by the plasma membrane Ca2+-ATPase influences the contribution of mitochondria to activity-dependent Ca2+ dynamics in Aplysia neuroendocrine cells.

6. PKC enhances the capacity for secretion by rapidly recruiting covert voltage-gated Ca2+ channels to the membrane.

7. Ca2+-induced uncoupling of Aplysia bag cell neurons.

8. Voltage-gated Ca2+ influx and mitochondrial Ca2+ initiate secretion from Aplysia neuroendocrine cells.

9. Separate Ca2+ sources are buffered by distinct Ca2+ handling systems in aplysia neuroendocrine cells.

10. Mitochondrial Ca2+ activates a cation current in Aplysia bag cell neurons.

11. Persistent Ca2+ current contributes to a prolonged depolarization in Aplysia bag cell neurons.

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