Search

(1) Background : the aim of this study was to explore parental competences and stress levels in the mothers of children with autism in relation to the mothers of neurotypical children. (2) Methods : the study used the Parental Competence Test and the PSS-10 scale to assess the intensity of stress related to one's own life situation over the past month. Forty mothers of children with ASD ( n = 20) and neurotypical children ( n = 20) participated in the study. (3) Results : the mothers of children with ASD showed higher levels of stress ( p = 0.0002). The mothers of neurotypical children achieved higher scores in parental competences (r = -0.49). The competence of mothers of children with ASD was correlated with rigour (r = 0.50), permissiveness (r = -0.60), overprotectiveness (r = 0.71), and helplessness (r = -0.77). (4) Conclusions : mothers of children with autism demonstrate lower parental competences than mothers of neurotypical children. Mothers of children with autism are less rigorous but more permissive, overprotective, and helpless. They tend to become heavily involved with their child. An overprotective attitude and greater tolerance for antisocial behaviours among parents of children with ASDs protect them from excessive stress.

The aim of the study was to evaluate whether the most common polymorphisms in the IL-6 and HSP70 genes affect the circulating heat shock protein 70 (HSP70), as well as inflammatory and prooxidant-antioxidant parameters in healthy men undergoing chronic endurance training. The subjects were randomly assigned to a 12-week swimming training (ST group) or control group (CON). Fasting blood samples were collected pre- and post-study period to assessment: superoxide dismutase (SOD) and glutathione peroxidase (GPx) activities, serum levels of lipid hydroperoxides (LHs), tumor necrosis factor α (TNFα), and HSP70. Subjects were genotyped for IL-6 -174G/C, HSPA1A + 190 G/C and HSPA1B + 1538 A/G single nucleotide polymorphisms (SNPs) by real-time PCR. After a 12-week study period, a decrease in TNFα, HSP70, and GPx was observed in the ST group, but not the CON group. IL-6 SNP affected serum TNFα levels (main effect of genotype). Higher TNFα levels (pre- and post-study period) was observed in CC CON than in other IL-6 genotypes of CON and ST groups. However, a post-training decrease in TNFα was observed in both GG and CC IL-6 genotypes of ST group. In turn, only GG IL-6 genotype of the ST group was related to a post-training decrease in HSP70 (main time and genotype interaction). Moreover, pre- and post-training LHs were lower in GG than GC/CC HSPA1A genotypes of the ST group (main genotype effect). In conclusion, polymorphisms within the IL-6 and HSPA1A genes seem to affect baseline levels of some inflammatory parameters and prooxidant-antioxidant status and/or their changes after chronic swimming training. However, the results should be confirmed in a study with a larger sample size, one that includes individuals with sedentary lifestyles.

A great number of studies focusing on the effects of dry-land resistance training interventions on swimming performance remain inconclusive. It is suggested that transferability of dry-land strength gains to swimming performance appear when dry-land resistance training programs are swim-specific. The main aim of this study was to compare the effects of specific dry-land resistance training on an ergometer with traditional dry-land exercises, and to determine how much of the resistance training effects were transferred to specific swimming conditions. The study included a group of 26 youth competitive male swimmers (age 15.7 ± 0.5 years, height 174.6 ± 6.6 cm, weight 68.4 ± 8.2 kg, training experience 5.8±0.7 years) of regional level. They were randomly allocated to one of two groups: experimental (E) and control (T). Both groups were involved in a 12-week dry-land resistance training concentrated on increasing muscular strength and power output of the upper limbs. Group E used a specialized ergometer (JBA - Zbigniew Staniak), while group T performed traditional resistance exercises. The program consisted of 10 sets of 30 s of exercise with 30 s rest intervals between each set. A two-way repeated measures ANOVA with Tukey HSD post hoc comparisons was used to determine if any significant differences existed between training groups across pretest and posttest conditions. The significance level was set at p ≤ 0.05. Dry-land resistance training modalities were the only differences in training between both groups. Our findings show that rates of transfer are much higher in group E than in group T, which resulted in a significant increase in swimming velocity (by 4.32%, p<0.001; ES=1.23, and 2.78%, p<0.003, ES=0.31, respectively)., (© 2020 Jerzy Sadowski, Andrzej Mastalerz, Wilhelm Gromisz, published by Sciendo.)

Background: Systematic physical activity can permanently prevent disadvantageous developments in the human body. This is very important especially for women, for whom the maintenance of a lean body in good shape is sometimes a primary consideration. However, in most cases, this activity is taken randomly and does not produce the desired effects such as reducing body fat. The purpose of the study was to evaluate changes in female body composition induced by 12 weeks of swimming training compared to sedentary controls., Methods: Training sessions occurred three times per week (60 min/session). Height, body mass, and waist/hip circumference and waist/hips ratio (WHR) were measured. Body cell mass (BCM), total body water (TBW), extracellular (ECW) and intracellular water (ICW), fat mass (FM), lean mass (FFM), and muscle mass (MM) were measured using bioelectrical impedance (pre/post)., Results: Training elicited decreases in hip circumference and increase in WHR. No changes were recorded in BCM, TBW, ECW, ICW, FM, FFM, and MM. Controls experienced decreases in values of BCM, ICW, and MM and increases in ECW., Conclusion: The applied swimming training did not significantly affect the body composition parameters. Inactivity also triggered a tendency toward unhealthy movement of water from the intracellular to extracellular space.

The aim of the study was to analyze the effect of SOD2 Val16Ala polymorphism on blood biochemical response to chronic swimming training. Healthy men (students of physical education) participated in a swimming training program (ST group) or served as a control group (CON group). The swimming training program lasted 12 weeks (1.5h per day; 4 days per week). Fasting blood samples were obtained prior to (pre) and after (post) a 12-week study period, to measure the biomarkers of oxidative stress, muscle damage and lipid profile. No significant changes in the study parameters were documented in CON group after a 12-week study period, either overall or among carriers of specific SOD2 Val16Ala genotypes. In ST group, post-training decrease in serum lipid hydroperoxides (p < 0.05) and creatine kinase activity (p < 0.05) was associated with Ala/Ala genotype of SOD2 Val16Ala polymorphism. In turn, the increase in serum activity of superoxide dismutase (p < 0.05) was associated with Val carriers, and Val/Val genotype additionally predisposed to the post-training increase in total glutathione level in whole blood (p < 0.05). Moreover, in ST group, a 12-week swimming training program induced an increase in serum concentration of total cholesterol (p < 0.05), which resulted from an increase in both high density (p < 0.05) and low density lipoprotein cholesterol (p < 0.05). The change in high density lipoprotein cholesterol level was irrespective of the genotype. Also, a tendency to post-training increase in both total and low density lipoprotein cholesterol was observed in all three genotypes, although these changes were significant solely in Ala/Val genotype carriers (p < 0.05). In conclusion, 12-week swimming training induces changes in oxidative stress and muscle damage parameters, as well as in lipid profile. These changes seem to be associated with the presence of SOD2 Val16Ala polymorphism. Presence of Ala allele, especially as homozygote, is associated with some beneficial post-training changes, such as a decrease in lipid peroxidation and less pronounced muscle damage. In turn, the influence of SOD2 Val16Ala polymorphism on the changes in lipid profile in response to chronic swimming training should be verified in further study., (Copyright © 2017 Elsevier Inc. All rights reserved.)

The aim of the study was to evaluate the effects of the dry-land power training on swimming force, swimming performance and strength in youth swimmers. Twenty six male swimmers, free from injuries and training regularly at least 6 times a week, were enrolled in the study and randomly assigned to one of two groups: experimental (n=14, mean age 14.0 ± 0.5 yrs, mean height 1.67±0.08 m and mean body mass 55.71 ±9.55 kg) and control (n=12, mean age 14.1 ± 0.5 yrs, mean height 1.61±0.11 m and mean body mass 49.07 ±8.25 kg). The experimental group took part in a combined swimming and dry-land power training. The control group took part in swimming training only. The training programmes in water included a dominant aerobic work in front crawl. In this research the experimental group tended to present slightly greater improvements in sprint performance. However, the stroke frequency insignificantly decreased (-4.30%, p>0.05) in the experimental group and increased (6.28%, p>0.05) in the control group. The distance per stroke insignificantly increased in the experimental group (5.98%, p>0.05) and insignificantly decreased in the control group (-5.36%, p>0.05). A significant improvement of tethered swimming force for the experimental group (9.64%, p<0.02) was found, whereas the increase was not statistically significant in the control group (2.86%, p>0.05). The main data cannot clearly state that power training allowed an enhancement in swimming performance, although a tendency to improve swimming performance in tethered swimming was noticed.