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2. Dominance is common in mammals and is associated with trans-acting gene expression and alternative splicing.

5. Contributors

6. Collagen IV deficiency causes hypertrophic remodeling and endothelium-dependent hyperpolarization in small vessel disease with intracerebral hemorrhage

9. Favourable HDL composition in endurance athletes is not associated with changes in HDL in vitro antioxidant and endothelial anti-inflammatory function.

13. Ldlr−/− and ApoE−/− mice better mimic the human metabolite signature of increased carotid intima media thickness compared to other animal models of cardiovascular disease

17. Vascular Collagen Type-IV in Hypertension and Cerebral Small Vessel Disease.

18. T-Cell–Derived miRNA-214 Mediates Perivascular Fibrosis in Hypertension

19. Dominance is common in mammals and is associated with trans-acting gene expression and alternative splicing

21. Pathophysiological relationship between lipoproteins and cation transport properties of platelets

26. PS-B01-7: VASCULAR SMOOTH MUSCLE CELL NOX5 REGULATES FIBROBLAST PHENOTYPIC SWITCH AND CARDIOVASCULAR FIBROSIS

28. Vascular Collagen Type-IV in Hypertension and Cerebral Small Vessel Disease

34. NOX5 EXPRESSION IN A VASCULAR SMOOTH MUSCLE CELL-SPECIFIC MANNER INDUCES FIBROBLAST PHENOTYPIC SWITCH AND PRO-FIBROTIC EFFECTS

39. Osteoprotegerin regulates vascular function through syndecan-1 and NADPH oxidase-derived reactive oxygen species

44. Selective Inhibition of the C-Domain of ACE (Angiotensin-Converting Enzyme) Combined With Inhibition of NEP (Neprilysin): A Potential New Therapy for Hypertension

45. Genes From a Translational Analysis Support a Multifactorial Nature of White Matter Hyperintensities

46. Th1‐type immune responses to Porphyromonas gingivalis antigens exacerbate angiotensin II‐dependent hypertension and vascular dysfunction

50. Sphingolipids in HDL - Potential markers for adaptation to pregnancy?

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