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1. Author Correction: Common and rare variant association analyses in amyotrophic lateral sclerosis identify 15 risk loci with distinct genetic architectures and neuron-specific biology (Nature Genetics, (2021), 53, 12, (1636-1648), 10.1038/s41588-021-00973-1)

2. Common and rare variant association analyses in amyotrophic lateral sclerosis identify 15 risk loci with distinct genetic architectures and neuron-specific biology

3. Association of Variants in the SPTLC1 Gene with Juvenile Amyotrophic Lateral Sclerosis

4. Genome-wide study of DNA methylation shows alterations in metabolic, inflammatory, and cholesterol pathways in ALS

5. Author Correction: Common and rare variant association analyses in amyotrophic lateral sclerosis identify 15 risk loci with distinct genetic architectures and neuron-specific biology

6. Tissue Engineering, Embryonic, Organ and Other Tissue Specific Stem Cells: SAFETY AND EFFICACY OF FIRST-IN-HUMAN INTRATHECAL TRANSPLANTATION OF HUMAN ASTROCYTES (ASTRORX) DERIVED FROM EMBRYONIC STEM CELLS IN ALS PATIENTS: FROM BENCH TO BEDSIDE

7. Common and rare variant association analyses in amyotrophic lateral sclerosis identify 15 risk loci with distinct genetic architectures and neuron-specific biology (vol 53, pg 1636, 2021)

8. Author Correction: Common and rare variant association analyses in amyotrophic lateral sclerosis identify 15 risk loci with distinct genetic architectures and neuron-specific biology (Nature Genetics, (2021), 53, 12, (1636-1648), 10.1038/s41588-021-00973-1)

9. Cell environment shapes TDP-43 function with implications in neuronal and muscle disease

11. Common and rare variant association analyses in amyotrophic lateral sclerosis identify 15 risk loci with distinct genetic architectures and neuron-specific biology

12. Common and rare variant association analyses in amyotrophic lateral sclerosis identify 15 risk loci with distinct genetic architectures and neuron-specific biology

13. Association of Variants in the SPTLC1 Gene With Juvenile Amyotrophic Lateral Sclerosis

15. Reconsidering the causality of TIA1 mutations in ALS

19. Genome-wide Analyses Identify KIF5A as a Novel ALS Gene

20. Genome-wide Analyses Identify KIF5A as a Novel ALS Gene

32. 801 - Tissue Engineering, Embryonic, Organ and Other Tissue Specific Stem Cells: SAFETY AND EFFICACY OF FIRST-IN-HUMAN INTRATHECAL TRANSPLANTATION OF HUMAN ASTROCYTES (ASTRORX) DERIVED FROM EMBRYONIC STEM CELLS IN ALS PATIENTS: FROM BENCH TO BEDSIDE.

35. Common and rare variant association analyses in amyotrophic lateral sclerosis identify 15 risk loci with distinct genetic architectures and neuron-specific biology

36. Genome-wide Analyses Identify KIF5A as a Novel ALS Gene

37. Autosomal recessive VWA1 -related disorder: comprehensive analysis of phenotypic variability and genetic mutations.

38. Peribulbar Corticosteroids for Ocular Myasthenia Gravis.

39. Mutations in the tail and rod domains of the neurofilament heavy-chain gene increase the risk of ALS.

40. DNMT3B splicing dysregulation mediated by SMCHD1 loss contributes to DUX4 overexpression and FSHD pathogenesis.

42. Genetic variability in sporadic amyotrophic lateral sclerosis.

43. SPTSSA variants alter sphingolipid synthesis and cause a complex hereditary spastic paraplegia.

44. Large-scale analyses of CAV1 and CAV2 suggest their expression is higher in post-mortem ALS brain tissue and affects survival.

45. Safety and efficacy of first-in-man intrathecal injection of human astrocytes (AstroRx®) in ALS patients: phase I/IIa clinical trial results.

46. Telomere length analysis in amyotrophic lateral sclerosis using large-scale whole genome sequence data.

47. Pearls & Oy-sters: Reversible Postpartum Pseudocoma State Associated With Magnesium Therapy: A Report of 2 Cases.

48. Predicting functional impairment trajectories in amyotrophic lateral sclerosis: a probabilistic, multifactorial model of disease progression.

49. Common genetic basis of ALS patients and soccer players may contribute to disease risk.

50. Author Correction: Common and rare variant association analyses in amyotrophic lateral sclerosis identify 15 risk loci with distinct genetic architectures and neuron-specific biology.

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