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1. The lipopolysaccharide-TLR4 axis regulates hepatic glutaminase 1 expression promoting liver ammonia build-up as steatotic liver disease progresses to steatohepatitis

2. SUMOylation controls Hu antigen R posttranscriptional activity in liver cancer

3. Anti-miR-873-5p improves alcohol-related liver disease by enhancing hepatic deacetylation via SIRT1

4. Anti-miR-518d-5p overcomes liver tumor cell death resistance through mitochondrial activity.

5. Hepatic levels of S-adenosylmethionine regulate the adaptive response to fasting

7. Targeting Hepatic Glutaminase 1 Ameliorates Non-alcoholic Steatohepatitis by Restoring Very-Low-Density Lipoprotein Triglyceride Assembly

8. FRI-466-YI Implications and therapeutic potential of neddylation for pediatric liver cancer: hepatoblastoma

9. The spike of SARS-CoV-2 promotes metabolic rewiring in hepatocytes

10. Neddylation tunes peripheral blood mononuclear cells immune response in COVID-19 patients

11. Methionine adenosyltransferase 1a antisense oligonucleotides activate the liver-brown adipose tissue axis preventing obesity and associated hepatosteatosis

12. Restoring cellular magnesium balance through Cyclin M4 protects against acetaminophen-induced liver damage

13. Novel Emerging Mechanisms in Acetaminophen (APAP) Hepatotoxicity.

14. Neddylation inhibition ameliorates steatosis in NAFLD by boosting hepatic fatty acid oxidation via the DEPTOR-mTOR axis

15. Boosting mitochondria activity by silencing MCJ overcomes cholestasis-induced liver injury

16. SUMOylation controls Hu antigen R posttranscriptional activity in liver cancer

17. Potential Role of the mTORC1-PGC1α-PPARα Axis under Type-II Diabetes and Hypertension in the Human Heart

18. Enhanced mitochondrial activity reshapes a gut microbiota profile that delays NASH progression

19. Data from E2F1 and E2F2-Mediated Repression of CPT2 Establishes a Lipid-Rich Tumor-Promoting Environment

20. Supplementary Information from E2F1 and E2F2-Mediated Repression of CPT2 Establishes a Lipid-Rich Tumor-Promoting Environment

21. Supplementary Table 1 from E2F1 and E2F2-Mediated Repression of CPT2 Establishes a Lipid-Rich Tumor-Promoting Environment

22. Supplementary Data from E2F1 and E2F2-Mediated Repression of CPT2 Establishes a Lipid-Rich Tumor-Promoting Environment

23. Supplementary Table 2 from E2F1 and E2F2-Mediated Repression of CPT2 Establishes a Lipid-Rich Tumor-Promoting Environment

24. Supplementary Figures from E2F1 and E2F2-Mediated Repression of CPT2 Establishes a Lipid-Rich Tumor-Promoting Environment

25. Supplementary Table 3 from E2F1 and E2F2-Mediated Repression of CPT2 Establishes a Lipid-Rich Tumor-Promoting Environment

26. The outcome of boosting mitochondrial activity in alcohol-associated liver disease is organ-dependent

27. The outcome of boosting mitochondrial activity in alcohol-associated liver disease is organ-dependent.

28. Potential role of the mTOR-C1-PGC1α-PPARα axis under type-II diabetes and hypertension in the human heart

29. Mitochondrial bioenergetics boost macrophage activation, promoting liver regeneration in metabolically compromised animals

30. Methionine adenosyltransferase 1a antisense oligonucleotides activate the liver-brown adipose tissue axis preventing obesity and associated hepatosteatosis

31. Role of mitochondria in liver diseases

32. Restoring cellular magnesium balance through Cyclin M4 protects against acetaminophen-induced liver damage

33. Restoring cellular magnesium balance through Cyclin M4 protects against acetaminophen-induced liver damage

34. Mitochondrial bioenergetics boost macrophage activation, promoting liver regeneration in metabolically compromised animals

35. Methionine Cycle Rewiring by Targeting miR-873-5p Modulates Ammonia Metabolism to Protect the Liver from Acetaminophen

36. Role of mitochondria in liver diseases

37. Mitochondrial bioenergetics boost macrophage activation, promoting liver regeneration in metabolically compromised animals

38. Mitochondrial physiology: Gnaiger Erich et al ― MitoEAGLE Task Group

39. Magnesium accumulation upon cyclin M4 silencing activates microsomal triglyceride transfer protein improving NASH

40. Anti-miR-518d-5p Overcomes Liver Tumor Cell Death Resistance Through Mitochondrial Activity

41. Neddylation inhibition ameliorates steatosis in NAFLD by boosting hepatic fatty acid oxidation via DEPTOR-mTOR axis

42. Magnesium Accumulation Upon Cyclin M4 Silencing Activates Microsomal Triglyceride Transfer Protein Improving NASH

43. Neddylation inhibition ameliorates steatosis in NAFLD by boosting hepatic fatty acid oxidation via the DEPTOR-mTOR axis

44. E2F1 and E2F2-mediated repression of CPT2 establishes a lipid-rich tumor-promoting environment

45. Magnesium accumulation upon cyclin M4 silencing activates microsomal triglyceride transfer protein improving NASH

46. E2F1 and E2F2-Mediated Repression of CPT2 Establishes a Lipid-Rich Tumor-Promoting Environment

47. Nutraceutical Properties of Polyphenols against Liver Diseases

48. Multi-Omics Integration Highlights the Role of Ubiquitination in CCl4-Induced Liver Fibrosis

49. Mitochondrial bioenergetics boost macrophage activation, promoting liver regeneration in metabolically compromised animals.

50. Multi-Omics Integration Highlights the Role of Ubiquitination in CCl4-Induced Liver Fibrosis

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