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Childhood behavior problems are associated with reduced labor market participation and lower earnings in adulthood, but little is known about the pathways and mechanisms that explain these associations. Drawing on a 33‐year prospective birth cohort of White males from low‐income backgrounds (n = 1040), we conducted a path analysis linking participants' teacher‐rated behavior problems at age 6 years—that is, inattention, hyperactivity, aggression‐opposition, and low prosociality—to employment earnings at age 35–39 years obtained from tax records. We examined three psychosocial mediators at age 11–12 years (academic, behavioral, social) and two mediators at age 25 years (non‐high school graduation, criminal convictions). Our findings support the notion that multiple psychosocial pathways—especially low education attainment—link kindergarten behavior problems to lower employment earnings decades later. [ABSTRACT FROM AUTHOR]

Using a sample of 767 children (403 girls, 364 boys), this study aimed to (a) identify groups with distinct trajectories of peer victimization over a 6-year period from primary school through the transition to secondary school, and (b) examine the associated personal (i.e., aggression or internalizing problems) and familial (family status, socioeconomic status, the parent-child relationship) predictors. Peer victimization was assessed via self-reports from Grades 4 through 9 (ages 10 through 15 years), aggression and internalizing problems were assessed in Grade 4 via peer nominations, and the parent-child relationship was assessed in Grade 7 (i.e., right after the transition to secondary school) via parent-reports. Growth Mixture modeling revealed 1 group (62%) who experienced little victimization in primary school and even less in secondary school, another group (31%) who was victimized in primary but not or much less in secondary school, and a third group (7%) who was chronically victimized in both school contexts. Boys were more likely than girls to follow any elevated victimization trajectory. Chronic victimization across primary and secondary school was predicted by nonintact family status and a combination of both internalizing problems and aggression compared with nonvictimized youth. In contrast, transitory victimization during primary but not in secondary school was predicted by aggression, but not internalizing problems. Support as well as conflict in the parent-child relationship also showed significant, albeit distinct associations with the different peer victimization trajectories.

Exposure to deviant friends has been found to be a powerful source of influence on children's and adolescents' aggressive behavior. However, the contribution of deviant friends may have been overestimated because of a possible non-accounted gene-environment correlation (rGE). In this study, we used a cross-lagged design to test whether friends' physical aggression at age 10 predicts an increase in participants' physical aggression from age 10 to age 13 years. Participants were 201 pairs of monozygotic twins who are part of the Quebec Longitudinal Twin Study. We performed two sets of analyses. In the first set of analyses, using twins as singletons, we found that teacher-rated friends' physical aggression predicted an increase in each twin's self-reported physical aggression from age 10 to age 13, above and beyond auto-regressive and concurrent links. Second, we used within-pair differences in regard to friends' physical aggression to predict an increase in within-pair differences in physical aggression, thus accounting for family-wide influences, including a likely rGE at age 10. No significant association was found, however. These results suggest that part of the influence attributed to friends in past studies may have been due to common underlying genetic effects on both physical aggression and association with physically aggressive friends.

Background: Bullying victimisation has been associated with increased risk of suicide ideation and attempt throughout the lifespan, but no study has yet examined whether it translates to a greater risk of death by suicide. We aimed to determine the association of bullying victimisation with suicide mortality. Methods: Participants were drawn from the 1958 British birth cohort, a prospective follow-up of all births in 1 week in Britain in 1958. We conducted logistic regressions on 14 946 participants whose mothers reported bullying victimisation at 7 and 11 years with linked information on suicide deaths through the National Health Service Central Register. Results: Fifty-five participants (48 males) had died by suicide between the age 18 and 52 years. Bullying victimisation was associated with suicide mortality; a one standard deviation increases in bullying victimisation linked to an increased odds for suicide mortality [odds ratio (OR) 1.29; 1.02–1.64] during adulthood. The OR attenuated by 11% after adjustment for individual (e.g. behavioural and emotional problems) and familial characteristics (e.g. adverse childhood experiences, 1.18; 0.92–1.51). Analysis of bullying victimisation frequency categories yields similar results: compared with individuals who had not been bullied, those who had been frequently bullied had an increased odds for suicide mortality (OR 1.89; 0.99–3.62). Conclusion: Our study suggests that individuals who have been frequently bullied have a small increased risk of dying by suicide, when no other risk factors is considered. Suicide prevention might start in childhood, with bullying included in a range of inter-correlated vulnerabilities encompassing behavioural and emotional difficulties and adverse experiences within the family. [ABSTRACT FROM AUTHOR]

This study assessed the genetic and environmental contributions to peer difficulties in the early school years. Twins' peer difficulties were assessed longitudinally in kindergarten (796 twins, "M"[subscript age] = 6.1 years), Grade 1 (948 twins, "M"[subscript age] = 7.1 years), and Grade 4 (868 twins, "M"[subscript age] = 10 years) through multiple informants. The multivariate results revealed that genetic factors accounted for a strong part of both yearly and stable peer difficulties. At the univariate level, the genetic contributions emerged progressively, as did a growing consensus among informants with respect to those who experienced peer difficulties. These results underline the need to intervene early and persistently, and to target the child and the peer context to prevent peer difficulties and their consequences. (Contains 3 tables and 3 figures.)

The goal of this study was to examine how different types of social experiences in kindergarten relate to Grade 1 academic achievement, while controlling for possible genetic and shared environmental influences through the use of the monozygotic (MZ) twin difference method. Social experiences in kindergarten included relationship quality with the larger peer group (i.e., rejection and victimization), relationship quality with one's best friend, and relationship quality with the teacher. Control variables included parental hostility-coercion, child cognitive skills and externalizing problems, and equivalent social experiences in Grade 1. Participants consisted of 223 MZ twin pairs ages 6 years at Time 1 (T1) and 7 years at Time 2 (T2). Results showed that within-pair differences in peer rejection and in poor teacher-child relationship quality at T1 uniquely predicted differences in MZ twins' academic achievement at T2. Mechanisms that could account for the possible causal role of these social experiences in regard to children's school achievement are discussed. (Contains 2 figures and 6 tables.)

In this study, we examine whether preventive enrichment of pre-math skills has an influence on number knowledge in preschool children from low income families. Our data analyses use two methods to examine the influence of two independent programs implemented during junior kindergarten and kindergarten. The first implies the traditional approach comparing the self-selected treated group to a self-selected comparison group. The second approach uses a self-selected dosage approach to the treatment condition, considering that implementation of the program varied across teachers. The results regarding the enrichment program in kindergarten were inconclusive. Nevertheless, the pre-math program implemented in junior kindergarten showed a positive influence on children's core requisite skills for later learning of arithmetic. Although children were receptive during both preschool years, a junior kindergarten program that elaborates upon conceptual issues beyond the number line appears both feasible and beneficial over the short-term.

Using a genetic design of 234 six-year-old twins, this study examined (a) the contribution of genes and environment to social versus physical aggression, and (b) whether the correlation between social and physical aggression can be explained by similar genetic or environmental factors or by a directional link between the phenotypes. For social aggression, substantial (shared and unique) environmental effects but only weak genetic effects were found. For physical aggression, significant effects of genes and unique environment were found. Bivariate modeling suggests that social and physical aggression share most of their underlying genes but only very few overlapping environmental factors. The correlation between the two phenotypes can also be explained by a directional effect from physical to social aggression.

Background: Peer victimisation has been associated with depressive symptoms during adolescence, however not all peer victimised adolescents will exhibit such symptoms. This study tested whether having a genetic predisposition to developing depression increased the risk of experiencing depressive symptoms in peer victimised youth. To date, no study has explored such gene–environment interaction using a polygenic risk score for depression (PRS‐depression) in the context of peer victimisation and depressive symptoms in adolescence. Methods: The sample included 748 participants born in 1997/98 from the Quebec Longitudinal Study of Child Development with genotype data and prospectively collected information on peer victimisation (12–13 years) obtained from both self‐ and teacher‐reports, as well as self‐reported depressive symptoms (15–17 years). The PRS‐depression was based on the genome‐wide association meta‐analysis of broad depression by Howard et al. (2019). Results: Self‐ and teacher‐reported peer victimisation in early adolescence were both associated with depressive symptoms in adolescence (β = 0.34, p <.001; β = 0.14, p =.001 respectively), and this association remained significant when accounting for PRS‐depression (β = 0.33, p <.001; β = 0.13, p =.002 respectively). PRS‐depression was independently associated with depressive symptoms, but there was no significant PRS‐depression by peer victimisation interaction (self‐reported and teacher‐reported). PRS‐depression was correlated with self‐reported, but not teacher‐reported, peer victimisation. Conclusions: Our findings suggested that a partial measure of an individual's genetic predisposition to depression, as measured by PRS‐depression, and being exposed to peer victimisation (self‐ and teacher‐reported) were independently associated with depressive symptoms in adolescence. Furthermore, PRS‐depression did not exacerbate the risk of depressive symptoms among adolescents who had been peer victimised. Lastly, we found evidence of a gene–environment correlation between PRS‐depression and self‐reported peer victimisation. Future studies are needed to replicate this finding and to further understand the role of genetic predispositions in experiencing depressive symptoms following peer victimisation. [ABSTRACT FROM AUTHOR]