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2. Biallelic variants in CSMD1 are implicated in a neurodevelopmental disorder with intellectual disability and variable cortical malformations

3. Pathogenic variants in KMT2C result in a neurodevelopmental disorder distinct from Kleefstra and Kabuki syndromes

4. Menke-Hennekam syndrome; delineation of domain-specific subtypes with distinct clinical and DNA methylation profiles

5. Genotype-phenotype associations in a large PTEN Hamartoma Tumor Syndrome (PHTS) patient cohort

6. LINE1-mediated epigenetic repression of androgen receptor transcription causes androgen insensitivity syndrome

7. Pathogenic variants in KMT2C result in a neurodevelopmental disorder distinct from Kleefstra and Kabuki syndromes

8. Biallelic variants in CSMD1 are implicated in a neurodevelopmental disorder with intellectual disability and variable cortical malformations

9. Deficiency of TET3 leads to a genome-wide DNA hypermethylation episignature in human whole blood

11. Genotype-phenotype associations in a large PTEN Hamartoma Tumor Syndrome (PHTS) patient cohort

12. Heterozygous Variants in the DNA-binding Domain of c-Myb May Affect Normal B/T Cell Development

13. Familial Male-limited Precocious Puberty (FMPP) and Testicular Germ Cell Tumors

14. The genetic basis of DOORS syndrome: an exome-sequencing study

15. Rare germline heterozygous missense variants in BRCA1-associated protein 1, BAP1, cause a syndromic neurodevelopmental disorder

16. Familial Male-Limited Precocious Puberty (FMPP) and Testicular Germ Cell Tumors

17. Heterozygous Variants in the DNA-binding Domain of c-Myb May Affect Normal B/T Cell Development

18. Author Correction: Deficiency of TET3 leads to a genome-wide DNA hypermethylation episignature in human whole blood (npj Genomic Medicine, (2021), 6, 1, (92), 10.1038/s41525-021-00256-y)

19. Rare germline heterozygous missense variants in BRCA1-associated protein 1, BAP1, cause a syndromic neurodevelopmental disorder

20. Author Correction: Deficiency of TET3 leads to a genome-wide DNA hypermethylation episignature in human whole blood (npj Genomic Medicine, (2021), 6, 1, (92), 10.1038/s41525-021-00256-y):Deficiency of TET3 leads to a genome-wide DNA hypermethylation episignature in human whole blood (npj Genomic Medicine, (2021), 6, 1, (92), 10.1038/s41525-021-00256-y)

21. Phenotypic variability in patients with ADA2 deficiency due to identical homozygous R169Q mutations

22. Author Correction: Deficiency of TET3 leads to a genome-wide DNA hypermethylation episignature in human whole blood

23. Deficiency of TET3 leads to a genome-wide DNA hypermethylation episignature in human whole blood

26. Disruption of ROBO2 is associated with urinary tract anomalies and confers risk of vesicoureteral reflux

27. A de novo variant in the human HIST1H4J gene causes a syndrome analogous to the HIST1H4C-associated neurodevelopmental disorder

30. Maternal risk associated with theVACTERLassociation: A case–control study

31. The clinical spectrum of missense mutations of the first aspartic acid of cbEGF-like domains in fibrillin-1 including a recessive family†

33. Polymorphic detection of a parthenogenetic maternal and double paternal contribution to a 46,XX/46,XY hermaphrodite

35. Phenotype delineation of ZNF462 related syndrome

36. A de novo variant in the human HIST1H4J gene causes a syndrome analogous to the HIST1H4C-associated neurodevelopmental disorder

37. Phenotype delineation of ZNF462 related syndrome

39. Phenotype delineation of ZNF462 related syndrome

42. Extreme phenotypic variability of a novel GLI2 mutation in a large family with panhypopituitarism and polydactyly: clinical implications

43. Mutations in N-acetylglucosamine (O-GlcNAc) transferase in patients with X-linked intellectual disability

44. Maternal risk associated with the VACTERL association: A case–control study.

45. Trichothiodystrophy causative TFIIEβ mutation affects transcription in highly differentiated tissue

46. Germline mutations affecting the histone H4 core cause a developmental syndrome by altering DNA damage response and cell cycle control

47. Germline mutations affecting the histone H4 core cause a developmental syndrome by altering DNA damage response and cell cycle control

48. Mutations in N-acetylglucosamine (O-GlcNAc) transferase in patients with X-linked intellectual disability

50. Trichothiodystrophy causative TFIIEβ mutation affects transcription in highly differentiated tissue

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