558 results on '"Gessler Manfred"'
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2. Hallmark discoveries in the biology of Wilms tumour
3. Genomic characterization of DICER1-associated neoplasms uncovers molecular classes
4. Treatment-independent miRNA signature in blood of wilms tumor patients
5. Retinoic acid pathway activity in wilms tumors and characterization of biological responses in vitro
6. The impact of the route to diagnosis in nephroblastoma
7. How to improve initial diagnostic accuracy of kidney tumours in childhood?—A non‐invasive approach
8. Sarcoma classification by DNA methylation profiling
9. Wilms tumour
10. MYCN and MAX alterations in Wilms tumor and identification of novel N-MYC interaction partners as biomarker candidates
11. High-risk blastemal Wilms tumor can be modeled by 3D spheroid cultures in vitro
12. Hallmark discoveries in the biology of Wilms tumour
13. TRIM28 inactivation in epithelial nephroblastoma is frequent and often associated with predisposing TRIM28 germline variants
14. Array-based DNA-methylation profiling in sarcomas with small blue round cell histology provides valuable diagnostic information
15. TRIM28 inactivation in epithelial nephroblastoma is frequent and often associated with predisposing TRIM28 germline variants.
16. Primary intracranial spindle cell sarcoma with rhabdomyosarcoma-like features share a highly distinct methylation profile and DICER1 mutations
17. Supplementary Data from Loss of Heterozygosity at 2q37 in Sporadic Wilms' Tumor: Putative Role for miR-562
18. Supplementary Data from Subtype-Specific FBXW7 Mutation and MYCN Copy Number Gain in Wilms' Tumor
19. Supplementary Figure Legends from Survival in Patients with High-Risk Prostate Cancer Is Predicted by miR-221, Which Regulates Proliferation, Apoptosis, and Invasion of Prostate Cancer Cells by Inhibiting IRF2 and SOCS3
20. Data from The Notch Pathway Inhibits TGFβ Signaling in Breast Cancer through HEYL-Mediated Crosstalk
21. Supplementary Methods from The Notch Pathway Inhibits TGFβ Signaling in Breast Cancer through HEYL-Mediated Crosstalk
22. Supplementary Table 1 from Survival in Patients with High-Risk Prostate Cancer Is Predicted by miR-221, Which Regulates Proliferation, Apoptosis, and Invasion of Prostate Cancer Cells by Inhibiting IRF2 and SOCS3
23. Supplementary Table 2 from Survival in Patients with High-Risk Prostate Cancer Is Predicted by miR-221, Which Regulates Proliferation, Apoptosis, and Invasion of Prostate Cancer Cells by Inhibiting IRF2 and SOCS3
24. Supplementary Figure 7 from Survival in Patients with High-Risk Prostate Cancer Is Predicted by miR-221, Which Regulates Proliferation, Apoptosis, and Invasion of Prostate Cancer Cells by Inhibiting IRF2 and SOCS3
25. Supplementary Figure 3 from Survival in Patients with High-Risk Prostate Cancer Is Predicted by miR-221, Which Regulates Proliferation, Apoptosis, and Invasion of Prostate Cancer Cells by Inhibiting IRF2 and SOCS3
26. Supplementary Figure Legends from The Notch Pathway Inhibits TGFβ Signaling in Breast Cancer through HEYL-Mediated Crosstalk
27. Supplementary Figure 2 from Survival in Patients with High-Risk Prostate Cancer Is Predicted by miR-221, Which Regulates Proliferation, Apoptosis, and Invasion of Prostate Cancer Cells by Inhibiting IRF2 and SOCS3
28. Supplementary Figures S1-S10 from The Notch Pathway Inhibits TGFβ Signaling in Breast Cancer through HEYL-Mediated Crosstalk
29. Supplementary Figure 6 from Survival in Patients with High-Risk Prostate Cancer Is Predicted by miR-221, Which Regulates Proliferation, Apoptosis, and Invasion of Prostate Cancer Cells by Inhibiting IRF2 and SOCS3
30. Supplementary Figure 5 from Survival in Patients with High-Risk Prostate Cancer Is Predicted by miR-221, Which Regulates Proliferation, Apoptosis, and Invasion of Prostate Cancer Cells by Inhibiting IRF2 and SOCS3
31. Supplementary Figure 4 from Survival in Patients with High-Risk Prostate Cancer Is Predicted by miR-221, Which Regulates Proliferation, Apoptosis, and Invasion of Prostate Cancer Cells by Inhibiting IRF2 and SOCS3
32. Supplementary Figure 1 from Survival in Patients with High-Risk Prostate Cancer Is Predicted by miR-221, Which Regulates Proliferation, Apoptosis, and Invasion of Prostate Cancer Cells by Inhibiting IRF2 and SOCS3
33. Tumor biology, biomarkers, and liquid biopsy in pediatric renal tumors
34. Mechanisms of epigenetic and cell-type specific regulation of Hey target genes in ES cells and cardiomyocytes
35. POSITION PAPER: Rationale for the treatment of Wilms tumour in the UMBRELLA SIOP-RTSG 2016 protocol
36. Nierentumoren
37. The transcriptional repressor Hes1 attenuates inflammation by regulating transcription elongation
38. Publisher Correction: The UMBRELLA SIOP–RTSG 2016 Wilms tumour pathology and molecular biology protocol
39. The variable clinical spectrum of nephroblastomatosis – results from the German Society of Pediatric Oncology and Hematology (GPOH) childhood kidney tumor group.
40. The genomic landscape of pediatric renal cell carcinomas
41. The DGCR8 E518K mutation found in Wilms tumors leads to a partial miRNA processing defect that alters gene expression patterns and biological processes
42. Characteristics of Nephroblastoma/Nephroblastomatosis in Children with a Clinically Reported Underlying Malformation or Cancer Predisposition Syndrome
43. Mosaic variegated aneuploidy in mouse BubR1 deficient embryos and pregnancy loss in human
44. Mutually exclusive BCOR internal tandem duplications and YWHAE-NUTM2 fusions in clear cell sarcoma of kidney: not the full story
45. Characteristics of Nephroblastoma / Nephroblastomatosis in Children With a Clinically Reported Underlying Malformation or Cancer Predisposition Syndrome
46. The landscape of genomic alterations across childhood cancers
47. Author Correction: The landscape of genomic alterations across childhood cancers
48. Cloning of Breakpoints of a Chromosome Translocation Identifies the an2 Locus
49. Hey bHLH Transcription Factors
50. Characteristics of nephroblastoma/nephroblastomatosis in children with a clinically reported underlying malformation or cancer predisposition syndrome
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