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In this article, we explore publication patterns in the Journal of Corporate Finance and discuss scientometric results from 1994 to 2017. We document evidence on the number and size of published papers, collaborative research in terms of coauthorship, editorial board characteristics, and citations as approximations of scientific impact. We identify the most prolific individuals and institutions and discuss how published research has exhibited an increasing number of authors per paper (2.38 on average), how the percentage of USA-based affiliations is gradually decreasing, and how 10.32% of published papers have been written by a member of the editorial board. In an attempt to map the social structure of collaboration in the Journal of Corporate Finance, we studied the social network of coauthors and found that the most prolific authors and institutions are also among the most central ones. We also discovered that the network of coauthorship spreads across 470 connected groups of nodes, the largest of which spans 24.63% of the network.

Modeling and experimental results suggest a role for Planar Cell Polarity-dependent multi-cellular rosette structures in ensuring correct epithelial cell migration in the mouse visceral endoderm., The visceral endoderm (VE) is a simple epithelium that forms the outer layer of the egg-cylinder stage mouse embryo. The anterior visceral endoderm (AVE), a specialised subset of VE cells, is responsible for specifying anterior pattern. AVE cells show a stereotypic migratory behaviour within the VE, which is responsible for correctly orientating the anterior-posterior axis. The epithelial integrity of the VE is maintained during the course of AVE migration, which takes place by intercalation of AVE and other VE cells. Though a continuous epithelial sheet, the VE is characterised by two regions of dramatically different behaviour, one showing robust cell movement and intercalation (in which the AVE migrates) and one that is static, with relatively little cell movement and mixing. Little is known about the cellular rearrangements that accommodate and influence the sustained directional movement of subsets of cells (such as the AVE) within epithelia like the VE. This study uses an interdisciplinary approach to further our understanding of cell movement in epithelia. Using both wild-type embryos as well as mutants in which AVE migration is abnormal or arrested, we show that AVE migration is specifically linked to changes in cell packing in the VE and an increase in multi-cellular rosette arrangements (five or more cells meeting at a point). To probe the role of rosettes during AVE migration, we develop a mathematical model of cell movement in the VE. To do this, we use a vertex-based model, implemented on an ellipsoidal surface to represent a realistic geometry for the mouse egg-cylinder. The potential for rosette formation is included, along with various junctional rearrangements. Simulations suggest that while rosettes are not essential for AVE migration, they are crucial for the orderliness of this migration observed in embryos. Our simulations are similar to results from transgenic embryos in which Planar Cell Polarity (PCP) signalling is disrupted. Such embryos have significantly reduced rosette numbers, altered epithelial packing, and show abnormalities in AVE migration. Our results show that the formation of multi-cellular rosettes in the mouse VE is dependent on normal PCP signalling. Taken together, our model and experimental observations suggest that rosettes in the VE epithelium do not form passively in response to AVE migration. Instead, they are a PCP-dependent arrangement of cells that acts to buffer the disequilibrium in cell packing generated in the VE by AVE migration, enabling AVE cells to migrate in an orderly manner., Author Summary The mouse visceral endoderm (VE) is a simple epithelium in the egg cylinder stage mouse embryo. Many functions associated with epithelia require them to undergo extensive remodelling through changes in the shape and relative positions of constituent cells, a process about which we understand relatively little. The anterior visceral endoderm (AVE) is a specialized group of cells in the simple epithelium of the VE, and their stereotypic migratory behaviour is essential for establishing the orientation of the anterior-posterior axis in the early mouse embryo. We show that AVE migration is linked to changes in cell packing in the VE and an increase in “rosettes,” which are striking collections of five or more cells meeting at a central point. To probe the role of rosettes during AVE migration, we have developed a mathematical model of cell movement in the VE. Simulations suggest that rosettes are not essential for AVE migration, but are crucial for the orderliness of this migration. We also explored the role of Planar Cell Polarity (PCP) signalling, which is known to coordinate cell polarization and rearrangement in many different tissues. We find that mutants in which PCP signalling is disrupted have fewer rosettes, altered epithelial packing, and abnormal AVE migration. We suggest that rosettes in the mouse VE are a PCP-dependent arrangement of cells that act to buffer the disturbances in cell packing generated by AVE migration, thereby enabling AVE cells to migrate in an orderly manner.