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6. TOP-061-YI Whole blood thrombin generation improves the understanding of cirrhotic coagulopathy and predicts clinical outcomes in patients with cirrhosis: a prospective cohort study

9. Profiling plasma alterations of extracellular vesicles in patients with acutely decompensated cirrhosis and bacterial infection.

11. Whole blood thrombin generation shows a significant hypocoagulable state in patients with decompensated cirrhosis

15. Reply to “Peripheral versus central venous blood sampling does not influence the assessment of platelet activation in cirrhosis”

16. Assessing Clinically Meaningful Hypercoagulability after COVID-19 Vaccination: A Longitudinal Study

23. More Pronounced Hypercoagulable State and Hypofibrinolysis in Patients With Cirrhosis With Versus Without HCC

24. Coagulopathy is not predictive of bleeding in patients with acute decompensation of cirrhosis and acute‐on‐chronic liver failure

26. Determinants of increased thrombotic tendency in NASH cirrhosis: not there yet!

27. Partial F8 gene duplication (factor VIII Padua) associated with high factor VIII levels and familial thrombophilia

29. X-linked thrombophilia with a mutant factor IX (factor IX Padua)

30. Noninvasive Risk Stratification After HCV Eradication in Patients With Advanced Chronic Liver Disease

31. Influence of Hepatocellular Carcinoma on Platelet Aggregation in Cirrhosis

32. Thrombin generation in patients with COVID-19 with and without thromboprophylaxis

33. Direct Oral Anticoagulants in Patients With Inherited Thrombophilia and Venous Thromboembolism: A Prospective Cohort Study

34. Acute Kidney Injury in Decompensated Cirrhosis Is Associated With Both Hypo‐coagulable and Hyper‐coagulable Features

44. Endothelial portal venous damage is associated with alterations in local TM and EPCR expression in patients with cirrhosis and portal hypertension

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