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12. SARS-CoV-2 accessory proteins involvement in inflammatory and profibrotic processes through IL11 signaling

13. Individually expressed SARS-CoV-2 viroporin ORF3a alters inflammatory responses and cellular structure in lung epithelial cells

14. Metabolic and mitochondria alterations induced by SARS-CoV-2 accessory proteins ORF3a, ORF9b, ORF9c and ORF10

16. SARS-CoV-2 accessory proteins involvement in inflammatory and profibrotic processes through IL11 signaling

17. Impairment of antiviral immune response and disruption of cellular functions by SARS-CoV-2 ORF7a and ORF7b

18. SARS-CoV-2 accessory proteins involvement in inflammatory and profibrotic processes through IL11 signaling

19. Metabolic and mitochondria alterations induced by SARS-CoV-2 accessory proteins ORF3a, ORF9b, ORF9c and ORF10

20. SARS-CoV-2 accessory proteins involvement in inflammatory and profibrotic processes through IL11 signaling

21. IL11 involvement in inflammatory and pro-fibrotic alterations via STAT3-WNT5A signaling activation by SARS-CoV-2 accessory proteins

24. Identification of amino acid residues of OspA of Borrelia involved in binding to CD40 receptor

28. P128. Development and validation of a novel mucin-containing in vitro model of the piglet colon : effect of associated feed deprivation

29. Antiviral immune responses, cellular metabolism and adhesion are differentially modulated by SARS-CoV-2 ORF7a or ORF7b

31. Antiviral immune responses, cellular metabolism and adhesion are differentially modulated by SARS-CoV-2 ORF7a or ORF7b

34. SARS-CoV-2 Accessory Proteins in Viral Pathogenesis: Knowns and Unknowns

35. CPEB alteration and aberrant transcriptome-polyadenylation lead to a treatable SLC19A3 deficiency in Huntington’s disease

36. Saccharomyces Cerevisiae Var Boulardii CNCM I–1079 Reduces Expression of Genes Involved in Inflammatory Response in Porcine Cells Challenged by Enterotoxigenic E. Coli and Influences Bacterial Communities in an In Vitro Model of the Weaning Piglet Colon

37. I03 CPEB alteration and aberrant transcriptome-polyadenylation unveil a treatable vitamin B1 deficiency in huntington’s disease

39. Pathogen Challenge and Dietary Shift Alter Microbiota Composition and Activity in a Mucin-Associated in vitro Model of the Piglet Colon (MPigut-IVM) Simulating Weaning Transition

41. CPEB alteration and aberrant transcriptome-polyadenylation lead to a treatable SLC19A3 deficiency in Huntington’s disease

42. SARS-CoV-2 Accessory Proteins in Viral Pathogenesis: Knowns and Unknowns

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