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1. Anti-GluA3 autoantibodies define a new sub-population of frontotemporal lobar degeneration patients with distinct neuropathological features

2. Missense variants in RPH3A cause defects in excitatory synaptic function and are associated with a clinically variable neurodevelopmental disorder

9. Detection of TDP-43 seeding activity in the olfactory mucosa from patients with frontotemporal dementia

13. Detection of TDP‐43 seeding activity in the olfactory mucosa from patients with frontotemporal dementia

16. Intensive exercise ameliorates motor and cognitive symptoms in experimental Parkinson’s disease restoring striatal synaptic plasticity

20. Intensive exercise ameliorates motor and cognitive symptoms in experimental Parkinson's disease restoring striatal synaptic plasticity

22. The levels of the NMDA receptor co-agonist D-serine are reduced in the substantia nigra of MPTP-lesioned macaques and in the cerebrospinal fluid of Parkinson’s disease patients

25. Anti-GluA3 autoantibodies recapitulate frontotemporal dementia with synaptic loss and tau accumulation: from animal model to rescue strategy

27. Molecular pathogenesis of Alzheimer’s disease in human peripheral cells: platelets show it all!

28. The development of ADAM10 endocytosis inhibitors for the treatment of Alzheimer’s disease

30. NMDA receptors

33. Detrimental effects of soluble a-synuclein oligomers at excitatory glutamatergic synapses.

37. Analysis of mRNA and Protein Levels of CAP2, DLG1 and ADAM10 Genes in Post-Mortem Brain of Schizophrenia, Parkinson’s and Alzheimer’s Disease Patients

41. Control of protein synthesis and memory by GluN3A-NMDA receptors through inhibition of GIT1/mTORC1 assembly

42. Author response: Control of protein synthesis and memory by GluN3A-NMDA receptors through inhibition of GIT1/mTORC1 assembly

44. Control of protein synthesis and memory by GluN3A-NMDA receptors through inhibition of GIT1/mTORC1 assembly

45. Pathophysiology of L-dopa-induced motor and non-motor complications in Parkinsonʼs disease

48. Experimental Disease-Modifying Agents for Frontotemporal Lobar Degeneration

49. Endocytosis of synaptic ADAM10 in neuronal plasticity and Alzheimer's disease

50. Transcranial Magnetic Stimulation Exerts “Rejuvenation” Effects on Corticostriatal Synapses after Partial Dopamine Depletion

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