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2. Sofosbuvir improves HCV-induced insulin resistance by blocking IRS1 degradation

3. Sofosbuvir improves HCV-induced insulin resistance by blocking IRS1 degradation

6. Erratum to: 'Significant fibrosis predicts new-onset diabetes mellitus and arterial hypertension in patients with NASH (J Hepatol 2020; 73: 17-25)

21. Genetic and functional analysis of FGF21 in NAFLD/NASH

22. Angiopoietin-like protein 8 is a novel vitamin D receptor-targeted lipogenic gene associated with non-alcoholic fatty liver

24. Comprehensive NAFLD diagnosis based on a noninvasive metabolomic approach in a liquid biopsy

25. Metabolic and biochemical phenotypes can distinguish different patterns of histological severity in biopsy-proven NAFLD

26. A pragmatic algorithm to rule out Non-alcoholic steatohepatitis in patients at risk of non-alcoholic fatty liver disease in spite of a normal ultrasound

27. SAT-507 - Metabolically unhealthy status impacts on the risk of significant liver injury in biopsy-proven NAFLD patients beyond obesity

28. SAT-508 - Clinical Outcomes in biopsy-proven NAFLD patients from the HEPAmet Spanish Registry

30. THU-472 - Genetic and functional analysis of FGF21 in NAFLD/NASH

31. PS-179 - Hepamet Score: a new non-invasive method for NAFLD-related fibrosis screening in clinical practice

32. Foxa1 reduces lipid accumulation in human hepatocytes and is down-regulated in nonalcoholic fatty liver

35. The FTO rs1421085 T > C Polymorphism is Associated with the Severity of Non-Alcoholic Fatty Liver

36. Impact of GWAS-Identified Common Variants on Histopathological Features of Non-Alcoholic Fatty Liver Disease Patients

38. Liver growth factor antifibrotic activity in vivo is associated with a decrease in activation of hepatic stellate cells

42. THU-365 - A pragmatic algorithm to rule out Non-alcoholic steatohepatitis in patients at risk of non-alcoholic fatty liver disease in spite of a normal ultrasound

43. THU-353 - Metabolic and biochemical phenotypes can distinguish different patterns of histological severity in biopsy-proven NAFLD

44. Impaired autophagic flux is associated with increased endoplasmic reticulum stress during the development of NAFLD

45. The pathogen receptor liver and lymph node sinusoidal endotelial cell C-type lectin is expressed in human Kupffer cells and regulated by PU.1

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