47 results on '"Gamsizkan M"'
Search Results
2. Effects of human amniotic fluid on costal cartilage regeneration: OP23-4
- Author
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Kavakli, K., Gurkok, S., Caylak, H., Genc, O., Gamsizkan, M., Yucel, O., and Karasahin, E.
- Published
- 2011
3. Measurement of the cross section for production of bb¯X decaying to muons in pp collisions at √s = 7 TeV
- Author
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Chatrchyan, S., Khachatryan, V., Sirunyan, A. M., Tumasyan, A., Adam, W., Bergauer, T., Dragicevic, M., Erö, J., Fabjan, C., Friedl, M., Frühwirth, R., Ghete, V. M., Hammer, J., Hoch, M., Hörmann, N., Hrubec, J., Jeitler, M., Kiesenhofer, W., Krammer, M., Liko, D., Mikulec, I., Pernicka, M., Rahbaran, B., Rohringer, C., Rohringer, H., Schöfbeck, R., Strauss, J., Taurok, A., Teischinger, F., Wagner, P., Waltenberger, W., Walzel, G., Widl, E., Wulz, C. E., Mossolov, V., Shumeiko, N., Suarez, Gonzalez, Bansal, J., Benucci, S., Cornelis, L., Wolf, De, E. A., Janssen, Luyckx, X., Maes, S., Mucibello, T., Ochesanu, L., Roland, S., Rougny, B., Selvaggi, R., Van, Haevermaet, Van, Mechelen, Van, Remortel, Van, Spilbeeck, Blekman, A., Blyweert, F., D'Hondt, S., Gonzalez, Suarez, Kalogeropoulos, R., Maes, A., Olbrechts, M., Van, Doninck, Van, Mulders, Van, Onsem, G. P., Villella, Charaf, I., Clerbaux, O., Lentdecker, De, Dero, G., Gay, V., A. P. R., Hammad, G. H., Hreus, Léonard, T., Marage, A., P. E., Thomas, Vander, Velde, Vanlaer, C., Wickens, P., Adler, J., Beernaert, V., Cimmino, K., Costantini, A., Garcia, S., Grunewald, G., Klein, M., Lellouch, B., Marinov, J., Mccartin, A., Ocampo, Rios, A. A., Ryckbosch, Strobbe, D., Thyssen, N., Tytgat, F., Vanelderen, M., Verwilligen, L., Walsh, P., Yazgan, S., Zaganidis, E., Basegmez, N., Bruno, S., Ceard, G., De Favereau De Jeneret, Delaere, J., Pree, Du, Favart, T., Forthomme, D., Giammanco, L., Grégoire, A., Hollar, G., Lemaitre, J., Liao, V., Militaru, J., Nuttens, O., Pagano, C., Pin, D., Piotrzkowski, A., Schul, K., Beliy, N., Caebergs, N., Daubie, T., Alves, E., G. A., Correa Martins, J. r., De Jesus Damiao, Martins, D., Pol, T., E. M., Souza, M. H. G., Aldá, J. r., W. L., Carvalho, Custódio, W., Costa, Da, E. M., De Oliveira Martins, Fonseca De Souza, Matos, Figueiredo, Mundim, D., Nogima, L., Oguri, H., Prado Da Silva, W. L., Santoro, Silva Do Amaral, S. M., Soares, Jorge, Sznajder, L., Anjos, A., T. S., Bernardes, C. A., Dias, F. A., Fernandez Perez Tomei, T. R., Gregores, E. M., Lagana, Marinho, C., Mercadante, F., P. G., Novaes, S. F., Padula, S. S., Genchev, Iaydjiev, V., Piperov, P., Rodozov, S., Stoykova, M., Sultanov, S., Tcholakov, G., Trayanov, V., Vutova, R., Dimitrov, M., Hadjiiska, A., Karadzhinova, R., Kozhuharov, A., Litov, V., Pavlov, L., Petkov, B., Bian, P., J. G., Chen, G. M., Chen, H. S., Jiang, C. H., Liang, Liang, D., Meng, S., Tao, X., Wang, J., Wang, X., Xiao, Z., Xu, H., Zang, M., Zhang, J., Asawatangtrakuldee, Z., Ban, C., Guo, Y., Guo, S., Li, Y., Liu, W., Mao, S., Qian, Y., J. S., Teng, Wang, H., Zhu, S., Zou, B., Cabrera, W., Gomez, Moreno, Osorio, Oliveros, A. F., Sanabria, J. C., Godinovic, Lelas, N., Plestina, D., Polic, R., Puljak, D., Antunovic, I., Dzelalija, Z., Kovac, M., Brigljevic, M., Duric, V., Kadija, S., Luetic, K., Morovic, J., Attikis, S., Galanti, A., Mousa, M., Nicolaou, J., Ptochos, C., Razis, F., P. A., Finger, Finger, J. r., Assran, M., Ellithi, Kamel, Khalil, A., Mahmoud, S., M. A., Radi, Hektor, A., Kadastik, A., Müntel, M., Raidal, M., Rebane, M., Tiko, L., Azzolini, A., Eerola, V., Fedi, P., Voutilainen, G., Czellar, M., Härkönen, S., Heikkinen, J., Karimäki, A., Kinnunen, V., Kortelainen, R., M. J., Lampén, Lassila, Perini, Lehti, K., Lindén, S., Luukka, T., Mäenpää, P., Peltola, T., Tuominen, T., Tuominiemi, E., Tuovinen, J., Ungaro, E., Wendland, D., Banzuzi, L., Korpela, K., Tuuva, A., Sillou, T., Besancon, D., Choudhury, M., Dejardin, S., Denegri, M., Fabbro, D., Faure, B., L. J., Ferri, Ganjour, F., Givernaud, S., Gras, A., Hamel De Monchenault, Jarry, G., Locci, P., Malcles, E., Millischer, J., Rander, L., Rosowsky, J., Shreyber, A., Titov, I., Baffioni, M., Beaudette, S., Benhabib, F., Bianchini, L., Bluj, L., Broutin, M., Busson, C., Charlot, P., Daci, C., Dahms, N., Dobrzynski, T., Elgammal, L., Granier De Cassagnac, Haguenauer, R., Miné, M., Mironov, P., Ochando, C., Paganini, C., Sabes, P., Salerno, D., Sirois, R., Thiebaux, Y., Veelken, C., Zabi, C., Agram, A., J. L., Andrea, Bloch, J., Bodin, D., Brom, D., J. M., Cardaci, Chabert, M., E. C., Collard, Conte, C., Drouhin, E., Ferro, F., Fontaine, C., J. C., Gelé, Goerlach, D., Juillot, U., Karim, P., Bihan, Le, A. C., Van, Hove, Fassi, P., Mercier, F., Baty, D., Beauceron, C., Beaupere, S., Bedjidian, N., Bondu, M., Boudoul, O., Boumediene, G., Brun, D., Chasserat, H., Chierici, J., Contardo, R., Depasse, D., Mamouni, El, Falkiewicz, H., Fay, A., Gascon, J., Gouzevitch, S., Ille, M., Kurca, B., Grand, Le, Lethuillier, T., Mirabito, M., Perries, L., Sordini, S., Tosi, V., Tschudi, S., Verdier, Y., Viret, P., Lomidze, S., Anagnostou, D., Beranek, G., Edelhoff, S., Feld, M., Heracleous, L., Hindrichs, N., Jussen, O., Klein, R., Merz, K., Ostapchuk, J., Perieanu, A., Raupach, A., Sammet, F., Schael, J., Sprenger, S., Weber, D., Wittmer, H., Zhukov, B., Ata, V., Caudron, M., Dietz, Laursonn, Erdmann, E., Güth, M., Hebbeker, A., Heidemann, T., Hoepfner, C., Klimkovich, K., Klingebiel, T., Kreuzer, D., Lanske, P., Lingemann, D., Magass, J., Merschmeyer, C., Meyer, M., Olschewski, A., Papacz, M., Pieta, P., Reithler, H., Schmitz, H., S. 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P., Schmanau, Schott, M., Simonis, G., H. J., Stober, F. M., Troendle, Wagner, Kuhr, Weiler, J., Zeise, T., Ziebarth, M., E. B., Daskalakis, Geralis, G., Kesisoglou, T., Kyriakis, S., Loukas, A., Manolakos, D., Markou, I., Markou, A., Mavrommatis, C., Ntomari, C., Gouskos, E., Mertzimekis, L., T. J., Panagiotou, Saoulidou, A., Stiliaris, N., Evangelou, E., Foudas, I., Kokkas, C., Manthos, P., Papadopoulos, N., Patras, I., Triantis, V., F. A., Aranyi, Bencze, A., Boldizsar, G., Hajdu, L., Hidas, C., Horvath, P., Kapusi, D., Krajczar, A., Sikler, K., Veszpremi, F., Vesztergombi, V., Beni, G., Molnar, N., Palinkas, J., Szillasi, J., Karancsi, Z., Raics, J., Trocsanyi, P., L. Z., Ujvari, Beri, B., S. B., Bhatnagar, Dhingra, V., Gupta, N., Jindal, R., Kaur, M., Kohli, M., J. M., Mehta, M. Z., Nishu, Saini, N., L. K., Sharma, Singh, A., A. P., Singh, Singh, J., S. P., Ahuja, Choudhary, S., B. C., Kumar, Kumar, A., Malhotra, A., Naimuddin, S., Ranjan, M., Sharma, K., Shivpuri, V., R. 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A., Martelli, Massironi, A., Menasce, A., Moroni, D., Paganoni, L., Pedrini, M., Ragazzi, D., Redaelli, S., Sala, N., Tabarelli De Fatis, Buontempo, T., Carrillo, Montoya, C. A., Cavallo, Cosa, De, Dogangun, A., Fabozzi, O., Iorio, F., A. O. M., Lista, Merola, L., Paolucci, M., Azzi, P., Bacchetta, P., Bellan, N., Bisello, P., Branca, D., Carlin, A., Checchia, R., Dorigo, P., Dosselli, T., Gasparini, U., Gasparini, F., Gozzelino, U., Kanishchev, A., Lacaprara, K., Lazzizzera, S., Margoni, I., Mazzucato, M., Meneguzzo, M., A. T., Montecassiano, Nespolo, F., Perrozzi, M., Pozzobon, L., Ronchese, N., Simonetto, P., Torassa, F., Tosi, E., Vanini, M., Zotto, S., Zumerle, P., Baesso, G., Berzano, P., Gabusi, U., Ratti, M., S. P., Riccardi, Torre, C., Vitulo, P., Viviani, P., Biasini, C., Bilei, M., G. 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V., Vinogradov, Azhgirey, A., Bayshev, I., Bitioukov, I., Grishin, S., Kachanov, V., Konstantinov, V., Korablev, D., Krychkine, A., Petrov, V., Ryutin, V., Sobol, R., Tourtchanovitch, A., Troshin, L., Tyurin, S., Uzunian, N., Volkov, A., Adzic, A., Djordjevic, P., Ekmedzic, M., Krpic, M., Milosevic, D., Aguilar, Benitez, Alcaraz, Maestre, Arce, J., Battilana, P., Calvo, C., Cerrada, E., Chamizo, Llatas, Colino, M., De La Cruz, Delgado, Peris, Diez, Pardos, Domínguez, Vázquez, Fernandez, Bedoya, Fernández, Ramos, J. P., Ferrando, Flix, A., Fouz, J., M. C., Garcia, Abia, Gonzalez, Lopez, Goy, Lopez, Hernandez, S., J. M., Josa, M. I., Merino, Puerta, Pelayo, Redondo, J., Romero, I., Santaolalla, L., Soares, J., M. S., Willmott, Albajar, C., Codispoti, C., Trocóniz, De, J. F., Cuevas, Fernandez, Menendez, Folgueras, J., Gonzalez, Caballero, Lloret, Iglesias, Piedra, Gomez, Vizan, Garcia, J. M., Brochero, Cifuentes, J. A., Cabrillo, I. J., Calderon, Chuang, A., S. 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F., Innocente, Janot, V., Kaadze, P., Karavakis, K., Kousouris, E., Lecoq, K., Lenzi, P., Lourenço, P., Mäki, C., Malberti, T., Malgeri, M., Mannelli, L., Masetti, M., Mavromanolakis, L., Meijers, G., Mersi, F., Meschi, S., Moser, E., Mozer, R., M. U., Mulders, Nesvold, M., Nguyen, E., Orimoto, M., Orsini, T., Palencia, Cortezon, Perez, E., Petrilli, E., Pfeiffer, A., Pierini, A., Pimiä, M., Piparo, M., Polese, D., Quertenmont, G., Racz, L., Reece, A., Rodrigues, Antunes, Rolandi, J., Rommerskirchen, G., Rovelli, T., Rovere, C., Sakulin, M., Santanastasio, H., Schäfer, F., Schwick, C., Segoni, C., Sharma, I., Siegrist, A., Silva, P., Simon, P., Sphicas, M., Spiga, P., Spiropulu, D., Stoye, M., Tsirou, M., Veres, A., G. I., Vichoudis, Wöhri, P., H. K., Worm, S. D., Zeuner, W. D., Bertl, Deiters, W., Erdmann, K., Gabathuler, W., Horisberger, K., Ingram, R., Kaestli, Q., H. C., König, Kotlinski, S., Langenegger, D., Meier, U., Renker, F., Rohe, D., Sibille, T., Bäni, J., Bortignon, L., Buchmann, P., M. A., Casal, Chanon, B., Chen, N., Deisher, Z., Dissertori, A., Dittmar, G., Dünser, M., Eugster, M., Freudenreich, J., Grab, K., Lecomte, C., Lustermann, P., Martinez Ruiz Del Arbol, Mohr, P., Moortgat, N., Nägeli, F., Nef, C., Nessi, Tedaldi, Pape, F., Pauss, L., Peruzzi, F., Ronga, M., F. J., Rossini, Sala, M., Sanchez, L., A. K., Sawley, M. C., Starodumov, Stieger, A., Takahashi, B., Tauscher, M., Thea, L., Theofilatos, A., Treille, K., Urscheler, D., Wallny, C., Weber, R., H. A., Wehrli, Weng, L., Aguilo, J., Amsler, E., Chiochia, C., Visscher, De, Favaro, S., Ivova, Rikova, Millan, Mejias, Otiougova, B., Robmann, P., Snoek, P., Verzetti, H., Chang, M., Y. H., Chen, K. H., Kuo, C. M., Li, S. W., Lin, Z. K., Lu, Y. J., Mekterovic, Volpe, D., Yu, R., S. S., Bartalini, Chang, P., Y. H., Chang, Y. W., Chao, Chen, Y., K. F., Dietz, Grundler, C., Hou, U., W. 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- Subjects
Hadron-Hadron scattering ,Nuclear and High Energy Physics ,CMS experiment ,LHC ,High Energy Physics::Experiment ,Nuclear Experiment - Published
- 2012
4. Shape, transverse size, and charged hadron multiplicity of jets in pp collisions at 7 TeV
- Author
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Chatrchyan, S., Khachatryan, V., Sirunyan, A. M., Tumasyan, A., Adam, W., Bergauer, T., Dragicevic, M., Erö, J., Fabjan, C., Friedl, M., Frühwirth, R., Ghete, V. M., Hammer, J., Hoch, M., Hörmann, N., Hrubec, J., Jeitler, M., Kiesenhofer, W., Krammer, M., Liko, D., Mikulec, I., Pernicka, M., Rahbaran, B., Rohringer, C., Rohringer, H., Schöfbeck, R., Strauss, J., Taurok, A., Teischinger, F., Wagner, P., Waltenberger, W., Walzel, G., Widl, E., Wulz, C. E., Mossolov, V., Shumeiko, N., Suarez, Gonzalez, Bansal, J., Benucci, S., Wolf, De, E. A., Janssen, Luyckx, X., Maes, S., Mucibello, T., Ochesanu, L., Roland, S., Rougny, B., Selvaggi, R., Van, Haevermaet, Van, Mechelen, Van, Remortel, Van, Spilbeeck, Blekman, A., Blyweert, F., D'Hondt, S., Gonzalez, Suarez, Kalogeropoulos, R., Maes, A., Olbrechts, M., Van, Doninck, Van, Mulders, Van, Onsem, G. P., Villella, Charaf, I., Clerbaux, O., Lentdecker, De, Dero, G., Gay, V., A. P. R., Hammad, G. H., Hreus, Léonard, T., Marage, A., P. 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I, Stupak, J, Sturm, P, Soh, Da, Su, D, Subramania, Hs, Succurro, A, Sugaya, Y, Sugimoto, T, Suhr, C, Suita, K, Suk, M, Sulin, Vv, Sultansoy, S, Sumida, T, Sun, X, Sundermann, Je, Suruliz, K, Sushkov, S, Susinno, G, Sutton, Mr, Suzuki, Y, Svatos, M, Sviridov, Ym, Swedish, S, Sykora, I, Sykora, T, Szeless, B, Sanchez, J, Ta, D, Tackmann, K, Taffard, A, Tafirout, R, Taiblum, N, Takahashi, Y, Takai, H, Takashima, R, Takeda, H, Takeshita, T, Talby, M, Talyshev, A, Tamsett, Mc, Tanaka, J, Tanaka, R, Tanaka, S, Tanaka, Y, Tani, K, Tannoury, N, Tappern, Gp, Tapprogge, S, Tardif, D, Tarem, S, Tarrade, F, Tartarelli, Gf, Tas, P, Tasevsky, M, Tassi, E, Tatarkhanov, M, Tayalati, Y, Taylor, C, Taylor, Fe, Taylor, Gn, Taylor, W, Teinturier, M, Castanheira, Mtd, Teixeira Dias, P, Temming, Kk, Ten Kate, H, Teng, Pk, Terada, S, Terashi, K, Terron, J, Terwort, M, Testa, M, Teuscher, Rj, Thadome, J, Therhaag, J, Laboratoire d'Annecy de Physique des Particules (LAPP), Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université Savoie Mont Blanc (USMB [Université de Savoie] [Université de Chambéry])-Centre National de la Recherche Scientifique (CNRS), Institut de Recherches sur les lois Fondamentales de l'Univers (IRFU), Commissariat à l'énergie atomique et aux énergies alternatives (CEA)-Université Paris-Saclay, Laboratoire Leprince-Ringuet (LLR), Centre National de la Recherche Scientifique (CNRS)-École polytechnique (X)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3), Département Recherches Subatomiques (DRS-IPHC), Institut Pluridisciplinaire Hubert Curien (IPHC), Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université de Strasbourg (UNISTRA)-Centre National de la Recherche Scientifique (CNRS)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université de Strasbourg (UNISTRA)-Centre National de la Recherche Scientifique (CNRS), Centre de Calcul de l'IN2P3 (CC-IN2P3), Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS), CMS, Institut de Physique des 2 Infinis de Lyon (IP2I Lyon), Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Centre National de la Recherche Scientifique (CNRS)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Centre National de la Recherche Scientifique (CNRS), Institut de Physique Nucléaire de Lyon (IPNL), Centre National de la Recherche Scientifique (CNRS)-Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3), Laboratoire d'Annecy de Physique des Particules (LAPP/Laboratoire d'Annecy-le-Vieux de Physique des Particules), Centre National de la Recherche Scientifique (CNRS)-Université Savoie Mont Blanc (USMB [Université de Savoie] [Université de Chambéry])-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3), Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-École polytechnique (X)-Centre National de la Recherche Scientifique (CNRS), Centre National de la Recherche Scientifique (CNRS)-Université de Strasbourg (UNISTRA)-Centre National de la Recherche Scientifique (CNRS)-Université de Strasbourg (UNISTRA), Centre National de la Recherche Scientifique (CNRS)-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3), Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS)-Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS), UAM. Departamento de Física Teórica, V. KHACHATRYAN, A.M. SIRUNYAN, A. TUMASYAN, W. ADAM, T. BERGAUER, M. DRAGICEVIC, J. ER¨O, C. FABJAN, M. FRIEDL, R. FRUHWIRTH, V.M. GHETE, J. HAMMER, S. H¨ANSEL, C. HARTL, M. HOCH, N. H N(ORMANN, J. HRUBEC, M. JEITLER, G. KASIECZKA, W. KIESENHOFER, M. KRAMMER, D. LIKO, I. MIKULEC, M. PERNICKA, H. ROHRINGER, R. SCH N(OFBECK, J. STRAUSS, A. TAUROK, F. TEISCHINGER, W.WALTENBERGER, G.WALZEL, E.WIDL, C.-E.WULZ, V. MOSSOLOV, N. SHUMEIKO, J. SUAREZ GONZALEZ, L. BENUCCI, L. CEARD, E.A. DE WOLF, X. JANSSEN, T. MAES, L. MUCIBELLO, S. OCHESANU, B. ROLAND, R. ROUGNY, M. SELVAGGI, H. VAN HAEVERMAET, P. VAN MECHELEN, N. VAN REMORTEL, V. ADLER, S. BEAUCERON, S. BLYWEERT, J. D $B!G (BHONDT, O. DEVROEDE, A. KALOGEROPOULOS, J. MAES, M. MAES, S. TAVERNIER, W. VAN DONINCK, P. VAN MULDERS, I. VILLELLA, E.C. CHABERT, O. CHARAF, B. CLERBAUX, G. DE LENTDECKER, V. DERO, A.P.R. GAY, G.H. HAMMAD, T. HREUS, P.E. MARAGE, C. VANDER VELDE, P. VANLAER, J.WICKENS, S. COSTANTINI, M. GRUNEWALD, B. KLEIN, A. MARINOV, D. RYCKBOSCH, F. THYSSEN, M. TYTGAT, L. VANELDEREN, P. VERWILLIGEN, S.WALSH, N. ZAGANIDIS, S. BASEGMEZ, G. BRUNO, J. CAUDRON, J. DE FAVEREAU DE JENERET, C. DELAERE, P. DEMIN, D. FAVART, A. GIAMMANCO, G. GR N4EGOIRE, J. HOLLAR, V. LEMAITRE, O. MILITARU, S. OVYN, D. PAGANO, A. PIN, K. PIOTRZKOWSKI, L. QUERTENMONT, N. SCHUL, N. BELIY, T. CAEBERGS, E. DAUBIE, G.A. ALVES, D. DE JESUS DAMIAO, M.E. POL, M.H.G. SOUZA, W. CARVALHO, E.M. DA COSTA, C. DE OLIVEIRA MARTINS, S. FONSECA DE SOUZA, L. MUNDIM, H. NOGIMA, V. OGURI, J.M. OTALORA GOICOCHEA, W.L. PRADO DA SILVA, A. SANTORO, S.M. SILVA DO, AMARAL, A. SZNAJDER, F. TORRES DA SILVA DE ARAUJO, F.A. DIAS, M.A.F. DIAS, T.R. FERNANDEZ PEREZ TOMEI, E. M. GREGORES2, F. MARINHO, S.F. NOVAES, SANDRA S. PADULA, N. DARMENOV, L. DIMITROV, V. GENCHEV, P. IAYDJIEV, S. PIPEROV, M. RODOZOV, S. STOYKOVA, G. SULTANOV, V. TCHOLAKOV, R. TRAYANOV, I. VANKOV, M. DYULENDAROVA, R. HADJIISKA, V. KOZHUHAROV, L. LITOV, E. MARINOVA, M. MATEEV, B. PAVLOV, P. PETKOV, J.G. BIAN, G.M. CHEN, H.S. CHEN, C.H. JIANG, D. LIANG, S. LIANG, J. WANG, X. WANG, Z.WANG, M. YANG, J. ZANG, Z. ZHANG, Y. BAN, S. GUO, Z. HU, W. LI, Y. MAO, S.J. QIAN, H. TENG, B. ZHU, A. CABRERA, B. GOMEZ MORENO, A.A. OCAMPO RIOS, A.F. OSORIO OLIVEROS, J.C. SANABRIA, N. GODINOVIC, D. LELAS, K. LELAS, R. PLESTINA3, D. POLIC, I. PULJAK, Z. ANTUNOVIC, M. DZELALIJA, V. BRIGLJEVIC, S. DURIC, K. KADIJA, S. MOROVIC, A. ATTIKIS, R. FEREOS, M. GALANTI, J. MOUSA, C. NICOLAOU, F. PTOCHOS, P.A. RAZIS, H. RYKACZEWSKI, Y. ASSRAN, M.A. MAHMOUD, A. HEKTOR, M. KADASTIK, K. KANNIKE, M. M N(UNTEL, M. RAIDAL, L. REBANE, V. AZZOLINI, P. EEROLA, S. CZELLAR, J. H N(ARK N(ONEN, A. HEIKKINEN, V. KARIM N(AKI, R. KINNUNEN, J. KLEM, M.J. KORTELAINEN, T. LAMP N4EN, K. LASSILA-PERINI, S. LEHTI, T. LIND N4EN, P. LUUKKA, T. M N(AENP N(A N(A, E. TUOMINEN, J. TUOMINIEMI, E. TUOVINEN, D. UNGARO, L.WENDLAND, K. BANZUZI, A. KORPELA, T. TUUVA, D. SILLOU, M. BESANCON, M. DEJARDIN, D. DENEGRI, J. DESCAMPS, B. FABBRO, J.L. FAURE, F. FERRI, S. GANJOUR, F.X. GENTIT, A. GIVERNAUD, P. GRAS, G. HAMEL DE MONCHENAULT, P. JARRY, E. LOCCI, J. MALCLES, M. MARIONNEAU, L. MILLISCHER, J. RANDER, A. ROSOWSKY, D. ROUSSEAU, M. TITOV, P. VERRECCHIA, S. BAFFIONI, L. BIANCHINI, M. BLUJ, C. BROUTIN, P. BUSSON, C. CHARLOT, L. DOBRZYNSKI, R. GRANIER DE, CASSAGNAC, M. HAGUENAUER, P. MIN N4E, C. MIRONOV, C. OCHANDO, P. PAGANINI, D. SABES, R. SALERNO, Y. SIROIS, C. THIEBAUX, A. ZABI, J.-L. AGRAM, A. BESSON, D. BLOCH, D. BODIN, J.-M. BROM, M. CARDACI, E. CONTE, F. DROUHIN, C. FERRO, J.-C. FONTAINE, D. GEL N4E, U. GOERLACH, S. GREDER, P. JUILLOT, M. KARIM, A.-C. LE BIHAN, Y. MIKAMI, P. VAN HOVE, F. FASSI, D. MERCIER, C. BATY, N. BEAUPERE, M. BEDJIDIAN, O. BONDU, G. BOUDOUL, D. BOUMEDIENE, H. BRUN, N. CHANON, R. CHIERICI, D. CONTARDO, P. DEPASSE, H. EL MAMOUNI, A. FALKIEWICZ, J. FAY, S. GASCON, B. ILLE, T. KURCA, T. LE GRAND, M. LETHUILLIER, L. MIRABITO, S. PERRIES, V. SORDINI, S. TOSI, Y. TSCHUDI, P. VERDIER, H. XIAO, V. ROINISHVILI, G. ANAGNOSTOU, M. EDELHOFF, L. FELD, N. HERACLEOUS, O. HINDRICHS, R. JUSSEN, K. KLEIN, J. MERZ, N. MOHR, A. OSTAPCHUK, A. PERIEANU, F. RAUPACH, J. SAMMET, S. SCHAEL, D. SPRENGER, H. WEBER, M.WEBER, B.WITTMER, M. ATA, W. BENDER, M. ERDMANN, J. FRANGENHEIM, T. HEBBEKER, A. HINZMANN, K. HOEPFNER, C. HOF, T. KLIMKOVICH, D. KLINGEBIEL, P. KREUZER1, D. LANSKEY, C. MAGASS, M. MERSCHMEYER, A. MEYER, P. PAPACZ, H. PIETA, H. REITHLER, S.A. SCHMITZ, L. SONNENSCHEIN, J. STEGGEMANN, D. TEYSSIER, M. BONTENACKELS, M. DAVIDS, M. DUDA, G. FL N(UGGE, H. GEENEN, M. GIFFELS, W. HAJ AHMAD, D. HEYDHAUSEN, T. KRESS, Y. KUESSEL, A. LINN, A. NOWACK, L. PERCHALLA, O. POOTH, J. RENNEFELD, P. SAUERLAND, A. STAHL, M. THOMAS, D. TORNIER, M.H. ZOELLER, M. ALDAYA MARTIN, W. BEHRENHOFF, U. BEHRENS, M. BERGHOLZ, K. BORRAS, A. CAMPBELL, E. CASTRO, D. DAMMANN, G. ECKERLIN, A. FLOSSDORF, G. FLUCKE, A. GEISER, I. GLUSHKOV, J. HAUK, H. JUNG, M. KASEMANN, I. KATKOV, P. KATSAS, C. KLEINWORT, H. KLUGE, A. KNUTSSON, D. KR N( UCKER, E. KUZNETSOVA, W. LANGE, W. LOHMANN, R. MANKEL, M. MARIENFELD, I.-A. MELZER-PELLMANN, A.B. MEYER, J. MNICH, A. MUSSGILLER, J. OLZEM, A. PARENTI, A. RASPEREZA, A. RAVAL, R. SCHMIDT, T. SCHOERNER-SADENIUS, N. SEN, M. STEIN, J. TOMASZEWSKA, D. VOLYANSKYY, R.WALSH, C.WISSING, C. AUTERMANN, S. BOBROVSKYI, J. DRAEGER, D. ECKSTEIN, H. ENDERLE, U. GEBBERT, K. KASCHUBE, G. KAUSSEN, R. KLANNER, B. MURA, S. NAUMANN-EMME, F. NOWAK, N. PIETSCH, C. SANDER, H. SCHETTLER, P. SCHLEPER, M. SCHR N(ODER, T. SCHUM, J. SCHWANDT, A.K. SRIVASTAVA, H. STADIE, G. STEINBR N(UCK, J. THOMSEN, R.WOLF, J. BAUER, V. BUEGE, A. CAKIR, T. CHWALEK, D. DAEUWEL, W. DE BOER, A. DIERLAMM, G. DIRKES, M. FEINDT, J. GRUSCHKE, C. HACKSTEIN, F. HARTMANN, M. HEINRICH, H. HELD, K.H. HOFFMANN, S. HONC, T. KUHR, D. MARTSCHEI, S. MUELLER, TH. M N(ULLER, M.B. NEULAND, M. NIEGEL, O. OBERST, A. OEHLER, J. OTT, T. PEIFFER, D. PIPARO, G. QUAST, K. RABBERTZ, F. RATNIKOV, M. RENZ, A. SABELLEK, C. SAOUT, A. SCHEURER, P. SCHIEFERDECKER, F.-P. SCHILLING, G. SCHOTT, H.J. SIMONIS, F.M. STOBER, D. TROENDLE, J. WAGNER-KUHR, M. ZEISE, V. ZHUKOV, E.B. ZIEBARTH, G. DASKALAKIS, T. GERALIS, S. KESISOGLOU, A. KYRIAKIS, D. LOUKAS, I. MANOLAKOS, A. MARKOU, C. MARKOU, C. MAVROMMATIS, E. PETRAKOU, L. GOUSKOS, T. MERTZIMEKIS, A. PANAGIOTOU, I. EVANGELOU, P. KOKKAS, N. MANTHOS, I. PAPADOPOULOS, V. PATRAS, F.A. TRIANTIS, A. ARANYI, G. BENCZE, L. BOLDIZSAR, G. DEBRECZENI, C. HAJDU1, D. HORVATH, A. KAPUSI, K. KRAJCZAR1, A. LASZLO, F. SIKLER, G. VESZTERGOMBI1, N. BENI, J. MOLNAR, J. PALINKAS, Z. SZILLASI, V. VESZPREMI, P. RAICS, Z.L. TROCSANYI, B. UJVARI, S. BANSAL, S.B. BERI, V. BHATNAGAR, M. JINDAL, M. KAUR, J.M. KOHLI, M.Z. MEHTA, N. NISHU, L.K. SAINI, A. SHARMA, R. SHARMA, A.P. SINGH, J.B. SINGH, S.P. SINGH, S. AHUJA, S. BHATTACHARYA, S. CHAUHAN, B.C. CHOUDHARY, P. GUPTA, S. JAIN, A. KUMAR, R.K. SHIVPURI, R.K. CHOUDHURY, D. DUTTA, S. KAILAS, S.K. KATARIA, A.K. MOHANTY1, L.M. PANT, P. SHUKLA, P. SUGGISETTI, T. AZIZ, M. GUCHAIT12, A. GURTU, M. MAITY13, D. MAJUMDER, G. MAJUMDER, K. MAZUMDAR, G.B. MOHANTY, A. SAHA, K. SUDHAKAR, N.WICKRAMAGE, S. BANERJEE, S. DUGAD, N.K. MONDAL, H. ARFAEI, H. BAKHSHIANSOHI, S.M. ETESAMI, A. FAHIM, M. HASHEMI, A. JAFARI, M. KHAKZAD, A. MOHAMMADI, M. MOHAMMADI NAJAFABADI, S. PAKTINAT MEHDIABADI, B. SAFARZADEH, M. ZEINALI, M. ABBRESCIA, L. BARBONE, C. CALABRIA, A. COLALEO, D. CREANZA, N. DE FILIPPIS, M. DE, PALMA, A. DIMITROV, F. FEDELE, L. FIORE, G. IASELLI, L. LUSITO, G. MAGGI, M. MAGGI, N. MANNA, B. MARANGELLI, S. MY, S. NUZZO, G.A. PIERRO, A. POMPILI, G. PUGLIESE, F. ROMANO, G. ROSELLI, G. SELVAGGI, L. SILVESTRIS, R. TRENTADUE, S. TUPPUTI, G. ZITO, G. ABBIENDI, A.C. BENVENUTI, D. BONACORSI, S. BRAIBANT, P. CAPILUPPI, A. CASTRO, F.R. CAVALLO, M. CUFFIANI, G.M. DALLAVALLE, F. FABBRI, A. FANFANI, D. FASANELLA, P. GIACOMELLI, M. GIUNTA, C. GRANDI, S. MARCELLINI, G. MASETTI, M. MENEGHELLI, A. MONTANARI, F.L. NAVARRIA, F. ODORICI, A. PERROTTA, A.M. ROSSI, T. ROVELLI, G. SIROLI, R. TRAVAGLINI, S. ALBERGO, G. CAPPELLO, M. CHIORBOLI, S. COSTA, A. TRICOMI, C. TUVE, G. BARBAGLI, G. BROCCOLO, V. CIULLI, C. CIVININI, R. D'ALESSANDRO, E. FOCARDI, S. FROSALI, E. GALLO, P. LENZI, M. MESCHINI, S. PAOLETTI, G. SGUAZZONI, A. TROPIANO, L. BENUSSI, S. BIANCO, S. COLAFRANCESCHI, D. PICCOLO, P. FABBRICATORE, R. MUSENICH, A. BENAGLIA, G.B. CERATI, F. DE GUIO, L. DI MATTEO, A. GHEZZI, P. GOVONI, M. MALBERTI, S. MALVEZZI, A. MARTELLI, A. MASSIRONI, D. MENASCE, V. MICCIO, L. MORONI, M. PAGANONI, D. PEDRINI, S. RAGAZZI, N. REDAELLI, S. SALA, T. TABARELLI DE, FATIS, V. TANCINI, S. BUONTEMPO, C.A. CARRILLO MONTOYA, A. CIMMINO, A. DE COSA, M. DE GRUTTOLA, F. FABOZZI, A.O.M. IORIO, L. LISTA, P. NOLI, P. PAOLUCCI, P. AZZI, N. BACCHETTA, P. BELLAN, D. BISELLO, A. BRANCA, R. CARLIN, P. CHECCHIA, E. CONTI, M. DE MATTIA, T. DORIGO, U. DOSSELLI, F. FANZAGO, F. GASPARINI, U. GASPARINI, P. GIUBILATO, A. GRESELE, S. LACAPRARA, I. LAZZIZZERA, M. MARGONI, M. MAZZUCATO, A.T. MENEGUZZO, L. PERROZZI, N. POZZOBON, P. RONCHESE, F. SIMONETTO, E. TORASSA, M. TOSI, S. VANINI, P. ZOTTO, G. ZUMERLE, P. BAESSO, U. BERZANO, C. RICCARDI, P. TORRE, P. VITULO, C. VIVIANI, M. BIASINI, G.M. BILEI, B. CAPONERI, L. FAN`O, P. LARICCIA, A. LUCARONI, G. MANTOVANI, M. MENICHELLI, A. NAPPI, A. SANTOCCHIA, L. SERVOLI, S. TARONI, M. VALDATA, R. VOLPE, P. AZZURRI, G. BAGLIESI, J. BERNARDINI, T. BOCCALI, R. CASTALDI, R.T. D'AGNOLO, R. DELL'ORSO, F. FIORI, L. FO`A, A. GIASSI, A. KRAAN, F. LIGABUE, T. LOMTADZE, L. MARTINI, A. MESSINEO, F. PALLA, F. PALMONARI, S. SARKAR, G. SEGNERI, A.T. SERBAN, P. SPAGNOLO, R. TENCHINI, G. TONELLI, A. VENTURI, P.G. VERDINI, L. BARONE, F. CAVALLARI, D. DEL RE, E. DI MARCO, M. DIEMOZ, D. FRANCI, M. GRASSI, E. LONGO, G. ORGANTINI, A. PALMA, F. PANDOLFI, R. PARAMATTI, S. RAHATLOU, N. AMAPANE, R. ARCIDIACONO, S. ARGIRO, M. ARNEODO, C. BIINO, C. BOTTA, N. CARTIGLIA, R. CASTELLO, M. COSTA, N. DEMARIA, A. GRAZIANO, C. MARIOTTI, M. MARONE, S. MASELLI, E. MIGLIORE, G. MILA, V. MONACO, M. MUSICH, M.M. OBERTINO, N. PASTRONE, M. PELLICCIONI, A. ROMERO, M. RUSPA, R. SACCHI, V. SOLA, A. SOLANO, A. STAIANO, D. TROCINO, A. VILELA PEREIRA, F. AMBROGLINI, S. BELFORTE, F. COSSUTTI, G. DELLA RICCA, B. GOBBO, D. MONTANINO, A. PENZO, S.G. HEO, S. CHANG, J. CHUNG, D.H. KIM, G.N. KIM, J.E. KIM, D.J. KONG, H. PARK, D. SON, D.C. SON, Z.KIM, J.Y. KIM, S. SONG, S. CHOI, B. HONG, M. JO, H. KIM, J.H. KIM, T.J. KIM, K.S. LEE, D.H. MOON, S.K. PARK, H.B. RHEE, E. SEO, S. SHIN, K.S. SIM, M. CHOI, S. KANG, C. PARK, I.C. PARK, S. PARK, G. RYU, Y. CHOI, Y.K. CHOI, J. GOH, J. LEE, S. LEE, H. SEO, I. YU, M.J. BILINSKAS, I. GRIGELIONIS, M. JANULIS, D. MARTISIUTE, P. PETROV, T. SABONIS, H. CASTILLA VALDEZ, E. DE LA CRUZ BURELO, R. LOPEZ-FERNANDEZ, A. S N4ANCHEZ HERN N4ANDEZ, L.M. VILLASENOR-CENDEJAS, S. CARRILLO MORENO, F. VAZQUEZ VALENCIA, H.A. SALAZAR IBARGUEN, E. CASIMIRO LINARES, A. MORELOS PINEDA, M.A. REYES-SANTOS, P. ALLFREY, D. KROFCHECK, J. TAM, P.H. BUTLER, R. DOESBURG, H. SILVERWOOD, M. AHMAD, I. AHMED, M.I. ASGHAR, H.R. HOORANI, W.A. KHAN, T. KHURSHID, S. QAZI, M. CWIOK, W. DOMINIK, K. DOROBA, A. KALINOWSKI, M. KONECKI, J. KROLIKOWSKI, T. FRUEBOES, R. GOKIELI, M. G N4ORSKI, M. KAZANA, K. NAWROCKI, M. SZLEPER, G. WROCHNA, P. ZALEWSKI, N. ALMEIDA, A. DAVID, P. FACCIOLI, P.G. FERREIRA PARRACHO, M. GALLINARO, P. MARTINS, G. MINI, P. MUSELLA, A. NAYAK, L. RAPOSO, P.Q. RIBEIRO, J. SEIXAS, P. SILVA, D. SOARES, J. VARELA, H.K.W N(OHRI, I. BELOTELOV, P. BUNIN, M. FINGER, M. FINGER JR., I. GOLUTVIN, A. KAMENEV, V. KARJAVIN, G. KOZLOV, A. LANEV, P. MOISENZ, V. PALICHIK, V. PERELYGIN, S. SHMATOV, V. SMIRNOV, A. VOLODKO, A. ZARUBIN, N. BONDAR, V. GOLOVTSOV, Y. IVANOV, V. KIM, P. LEVCHENKO, V. MURZIN, V. ORESHKIN, I. SMIRNOV, V. SULIMOV, L. UVAROV, S. VAVILOV, A. VOROBYEV, YU. ANDREEV, S. GNINENKO, N. GOLUBEV, M. KIRSANOV, N. KRASNIKOV, V. MATVEEV, A. PASHENKOV, A. 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CAVANAUGH, C. DRAGOIU, E.J. GARCIA-SOLIS, C.E. GERBER, D.J. HOFMAN, S. KHALATYAN, F. LACROIX, C. O $B!G (BBRIEN, E. SHABALINA, C. SILVESTRE, A. SMORON, D. STROM, N. VARELAS, U. AKGUN, E.A. ALBAYRAK, B. BILKI, K. CANKOCAK, W. CLARIDA, F. DURU, C.K. LAE, E. MCCLIMENT, J.-P. MERLO, H. MERMERKAYA, A. MESTVIRISHVILI, A. MOELLER, J. NACHTMAN, C.R. NEWSOM, E. NORBECK, J. OLSON, Y. ONEL, F. OZOK, S. SEN, J.WETZEL, T. YETKIN, K. YI, B.A. BARNETT, B. BLUMENFELD, A. BONATO, C. ESKEW, D. FEHLING, G. GIURGIU, A.V. GRITSAN, Z.J. GUO, G. HU, P. MAKSIMOVIC, S. RAPPOCCIO, M. SWARTZ, N.V. TRAN, A. WHITBECK, P. BARINGER, A. BEAN, G. BENELLI, O. GRACHOV, M. MURRAY, D. NOONAN, V. RADICCI, S. SANDERS, J.S.WOOD, V. ZHUKOVA, D. BANDURIN, T. BOLTON, I. CHAKABERIA, A. IVANOV, M. MAKOUSKI, Y. MARAVIN, S. SHRESTHA, I. SVINTRADZE, Z.WAN, J. GRONBERG, D. LANGE, D. WRIGHT, A. BADEN, M. BOUTEMEUR, S.C. ENO, D. FERENCEK, J.A. GOMEZ, N.J. HADLEY, R.G. KELLOGG, M. KIRN, Y. LU, A.C. MIGNEREY, K. ROSSATO, P. RUMERIO, F. SANTANASTASIO, A. SKUJA, J. TEMPLE, M.B. TONJES, S.C. TONWAR, E. TWEDT, B. ALVER, G. BAUER, J. BENDAVID, W. BUSZA, E. BUTZ, I.A. CALI, M. CHAN, V. DUTTA, P. EVERAERTS, G. GOMEZ CEBALLOS, M. GONCHAROV, K.A. HAHN, P. HARRIS, Y. KIM, M. KLUTE, Y.-J. LEE, C. LOIZIDES, P.D. LUCKEY, T. MA, S. NAHN, C. PAUS, C. ROLAND, G. ROLAND, M. RUDOLPH, G.S.F. STEPHANS, K. SUMOROK, K. SUNG, E.A. WENGER, B. WYSLOUCH, S. XIE, Y. YILMAZ, A.S. YOON, M. ZANETTI, P. COLE, S.I. COOPER, P. CUSHMAN, B. DAHMES, A. DE BENEDETTI, P.R. DUDERO, G. FRANZONI, J. HAUPT, K. KLAPOETKE, Y. KUBOTA, J. MANS, V. REKOVIC, R. RUSACK, M. SASSEVILLE, A. SINGOVSKY, L.M. CREMALDI, R. GODANG, R. KROEGER, L. PERERA, R. RAHMAT, D.A. SANDERS, D. SUMMERS, K. BLOOM, S. BOSE, J. BUTT, D.R. CLAES, A. DOMINGUEZ, M. EADS, J. KELLER, T. KELLY, I. KRAVCHENKO, J. LAZO-FLORES, C. LUNDSTEDT, H. MALBOUISSON, S. MALIK, G.R. SNOW, U. BAUR, A. GODSHALK, I. IASHVILI, A. KHARCHILAVA, K. SMITH, J. ZENNAMO, G. ALVERSON, E. BARBERIS, D. BAUMGARTEL, O. BOERIU, M. CHASCO, K. KAADZE, S. REUCROFT, J. SWAIN, D. WOOD, J. ZHANG, A. ANASTASSOV, A. KUBIK, N. ODELL, R.A. OFIERZYNSKI, B. POLLACK, A. POZDNYAKOV, S. STOYNEV, M. VELASCO, S.WON, L. ANTONELLI, D. BERRY, M. HILDRETH, C. JESSOP, D.J. KARMGARD, J. KOLB, T. KOLBERG, K. LANNON, W. LUO, S. LYNCH, N. MARINELLI, D.M. MORSE, T. PEARSON, R. RUCHTI, J. SLAUNWHITE, N. VALLS, J. WARCHOL, M.WAYNE, J. ZIEGLER, B. BYLSMA, L.S. DURKIN, J. GU, P. KILLEWALD, T.Y. LING, M. RODENBURG, G.WILLIAMS, N. ADAM, E. BERRY, P. ELMER, D. GERBAUDO, V. HALYO, P. HEBDA, A. HUNT, J. JONES, E. LAIRD, D. LOPES PEGNA, D. MARLOW, T. MEDVEDEVA, M. MOONEY, J. OLSEN, P. PIROU´E, H. SAKA, D. STICKLAND, C. TULLY, J.S.WERNER, A. ZURANSKI, J.G. ACOSTA, X.T. HUANG, A. LOPEZ, H. MENDEZ, S. OLIVEROS, J.E. RAMIREZ VARGAS, A. ZATSERKLYANIY, E. ALAGOZ, V.E. BARNES, G. BOLLA, L. BORRELLO, D. BORTOLETTO, A. EVERETT, A.F. GARFINKEL, Z. GECSE, L. GUTAY, M. JONES, O. KOYBASI, A.T. LAASANEN, N. LEONARDO, C. LIU, V. MAROUSSOV, M. MEIER, P. MERKEL, D.H. MILLER, N. NEUMEISTER, K. POTAMIANOS, I. SHIPSEY, D. SILVERS, A. SVYATKOVSKIY, H.D. YOO, J. ZABLOCKI, Y. ZHENG, P. JINDAL, N. PARASHAR, C. BOULAHOUACHE, V. CUPLOV, K.M. ECKLUND, F.J.M. GEURTS, J.H. LIU, J. MORALES, B.P. PADLEY, R. REDJIMI, J. ROBERTS, J. ZABEL, B. BETCHART, A. BODEK, Y.S. CHUNG, P. DE BARBARO, R. DEMINA, Y. ESHAQ, H. FLACHER, A. GARCIA, BELLIDO, P. GOLDENZWEIG, Y. GOTRA, J. HAN, A. HAREL, D.C. MINER, D. ORBAKER, G. PETRILLO, D. VISHNEVSKIY, M. ZIELINSKI, A. BHATTI, L. DEMORTIER, K. GOULIANOS, G. LUNGU, C. MESROPIAN, M. YAN, O. ATRAMENTOV, A. BARKER, D. DUGGAN, Y. GERSHTEIN, R. GRAY, E. HALKIADAKIS, D. HIDAS, D. HITS, A. LATH, S. PANWALKAR, R. PATEL, A. RICHARDS, K. ROSE, S. SCHNETZER, S. SOMALWAR, R. STONE, S. THOMAS, G. CERIZZA, M. HOLLINGSWORTH, S. SPANIER, Z.C. YANG, A. YORK, J. ASAADI, R. EUSEBI, J. GILMORE, A. GURROLA, T. KAMON, V. KHOTILOVICH, R. MONTALVO, C.N. NGUYEN, J. PIVARSKI, A. SAFONOV, S. SENGUPTA, A. TATARINOV, D. TOBACK, M.WEINBERGER, N. AKCHURIN, C. BARDAK, J. DAMGOV, C. JEONG, K. KOVITANGGOON, S.W. LEE, P. MANE, Y. ROH, A. SILL, I. VOLOBOUEV, R.WIGMANS, E. YAZGAN, E. APPELT, E. BROWNSON, D. ENGH, C. FLOREZ, W. GABELLA, W. JOHNS, P. KURT, C. MAGUIRE, A. MELO, P. SHELDON, J. VELKOVSKA, M.W. ARENTON, M. BALAZS, S. BOUTLE, M. BUEHLER, S. CONETTI, B. COX, B. FRANCIS, R. HIROSKY, A. LEDOVSKOY, C. LIN, C. NEU, T. PATEL, R. YOHAY, S. GOLLAPINNI, R. HARR, P.E. KARCHIN, V. LOGGINS, M. MATTSON, C. MILST`ENE, A. SAKHAROV, M. ANDERSON, M. BACHTIS, J.N. BELLINGER, D. CARLSMITH, S. DASU, J. EFRON, L. GRAY, K.S. GROGG, M. GROTHE, R. HALL-WILTON, M. HERNDON, P. KLABBERS, J. KLUKAS, A. LANARO, C. LAZARIDIS, J. LEONARD, J. LIU, D. LOMIDZE, R. LOVELESS, A. MOHAPATRA, W. PARKER, D. REEDER, I. ROSS, A. SAVIN, W.H. SMITH, J. SWANSON, M.WEINBERG, S.CHATRCHYAN, F. PRIMAVERA, L. BRIGLIADORI, Université de Strasbourg (UNISTRA)-Université de Haute-Alsace (UHA) Mulhouse - Colmar (Université de Haute-Alsace (UHA))-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS)-Université de Strasbourg (UNISTRA)-Université de Haute-Alsace (UHA) Mulhouse - Colmar (Université de Haute-Alsace (UHA))-Institut National de Physique Nucléaire et de Physique des Particules du CNRS (IN2P3)-Centre National de la Recherche Scientifique (CNRS), Yerevan Phys Inst, Inst Hochenergiephys OeAW, Natl Ctr Particle & High Energy Phys, Univ Antwerp, Vrije Univ Brussel, Univ Libre Brussels, Univ Ghent, Catholic Univ Louvain, Univ Mons, Centro Brasileiro de Pesquisas Físicas (CBPF), Universidade do Estado do Rio de Janeiro (UERJ), Universidade Estadual Paulista (Unesp), Bulgarian Acad Sci, Univ Sofia, Inst High Energy Phys, Peking Univ, Universidad de los Andes, Tech Univ Split, Univ Split, Rudjer Boskovic Inst, 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- Subjects
Particle physics ,Nuclear and High Energy Physics ,Astrophysics::High Energy Astrophysical Phenomena ,Monte Carlo method ,Hadron ,HERA ,FOS: Physical sciences ,ComputingMilieux_LEGALASPECTSOFCOMPUTING ,Hadron-hadron scattering ,7. Clean energy ,01 natural sciences ,PARTICLE PHYSICS ,LARGE HADRON COLLIDER ,CMS ,High Energy Physics - Experiment ,Nuclear physics ,DEEP-INELASTIC SCATTERING ,High Energy Physics - Experiment (hep-ex) ,0103 physical sciences ,[PHYS.HEXP]Physics [physics]/High Energy Physics - Experiment [hep-ex] ,DISTRIBUTIONS ,ddc:530 ,Multiplicity (chemistry) ,010306 general physics ,Nuclear Experiment ,deep-inelastic scattering ,hadron-hadron scattering ,hera ,distributions ,Quantum chromodynamics ,Physics ,high energy collider ,Large Hadron Collider ,010308 nuclear & particles physics ,Física ,Deep inelastic scattering ,HEP ,Transverse plane ,FIS/01 - FISICA SPERIMENTALE ,Physique des particules élémentaires ,High Energy Physics::Experiment ,LHC ,Particle Physics - Experiment - Abstract
Open Access: This article is distributed under the terms of the Creative Commons Attribution License.-- CMS Collaboration: et al., Measurements of jet characteristics from inclusive jet production in proton-proton collisions at a centre-of-mass energy of 7 TeV are presented. The data sample was collected with the CMS detector at the LHC during 2010 and corresponds to an integrated luminosity of 36 inverse picobarns. The mean charged hadron multiplicity, the differential and integral jet shape distributions, and two independent moments of the shape distributions are measured as functions of the jet transverse momentum for jets reconstructed with the anti-kT algorithm. The measured observables are corrected to the particle level and compared with predictions from various QCD Monte Carlo generators., European Commission; Federal Ministry of Science, Research and Economy (Austria); National Fund for Scientific Research (Belgium); Research Foundation – Flanders; Conselho Nacional de Desenvolvimento Científico e Tecnológico (Brasil); Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (Brasil); Fundação Carlos Chagas Filho de Amparo à Pesquisa do Estado do Rio de Janeiro; Fundação de Amparo à Pesquisa do Estado de São Paulo; Bulgarian Ministry of Education and Science; Centre National de la Recherche Scientifique (France); Commissariat à l'énergie atomique et aux énergies alternatives (France); Ministry of Science and Technology of the People's Republic of China; Chinese Academy of Sciences; National Natural Science Foundation of China; Colciencias (Colombia); Croatian Ministry of Science, Education and Sport; Academy of Finland; Helsinki Institute of Physics; Bundesministerium für Bildung und Forschung (Deutschland); Deutsche Forschungsgemeinschaft; Helmholtz Association; General Secretariat of Research and Technology (Greece); Hungarian Scientific Research Fund; National Office for Research and Technology (Hungary); Department of Atomic Energy (India); Department of Science and Technology (India); Council of Science and Industrial Research (India); Institute for Research in Fundamental Sciences (Iran); Science Foundation Ireland; Istituto Nazionale di Fisica Nucleare (Italia); Korean Ministry of Education, Science and Technology; Lithuanian Academy of Sciences; Centro de Investigación y de Estudios Avanzados del Instituto Politécnico Nacional (México); Consejo Nacional de Ciencia y Tecnología (México); Secretaría de Educación Pública (México); Universidad Autónoma de San Luis Potosí; Ministry of Science and Innovation (New Zealand); Ministry of Science and Higher Education and the National Science Centre (Poland); Fundação para a Ciência e a Tecnologia (Portugal); Joint Institute for Nuclear Research (Russia); Russian Academy of Sciences; Russian Foundation for Basic Research; Ministry of Education, Science and Technological Development (Serbia); Ministerio de Ciencia e Innovación (España); Swiss Funding Agencies; Swiss National Science Foundation; National Science Council (Taiwan); The Scientific and Technological Research Council of Turkey; Turkish Atomic Energy Authority; Science and Technology Facilities Council (UK); Department of Energy (US); National Science Foundation (US); A. G. Leventis Foundation; Alfred P. Sloan Foundation; Alexander von Humboldt Foundation; Foundation for Polish Science
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- 2012
5. Study of W boson production in PbPb and pp collisions at √sNN=2.76 TeV
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- Subjects
Heavy-ions ,Nuclear and High Energy Physics ,Heavy Ion collision ,CMS experiment ,ROOT-S=7 TEV ,Physics::Instrumentation and Detectors ,CMS ,Physics ,ATLAS DETECTOR ,W bosons ,HEAVY-ION COLLISIONS ,PRODUCTION CROSS-SECTION ,Physics and Astronomy ,High Energy Physics::Experiment ,LHC ,Nuclear Experiment - Abstract
A measurement is presented of W-boson production in PbPb collisions carried out at a nucleon-nucleon (NN) centre-of-mass energy root S-NN of 2.76 TeV at the LHC using the CMS detector. In data corresponding to an integrated luminosity of 7.3 mu b(-1), the number of W -> mu v(mu) decays is extracted in the region of muon pseudorapidity vertical bar eta mu vertical bar < 2.1 and transverse momentum p(T)(mu) > 25 GeV/c. Yields of muons found per unit of pseudorapidity correspond to (159 +/- 10(stat.) +/- 12(syst.)) x 10(-8) W and (154 +/- 10(stat.) +/- 12(syst.)) x 10(-8) W- bosons per minimum-bias PbPb collision. The dependence of W production on the centrality of PbPb collisions is consistent with a scaling of the yield by the number of incoherent NN collisions. The yield of W bosons is also studied in a sample of pp interactions at root S = 2.76 TeV corresponding to an integrated luminosity of 231 nb(-1). The individual W+ and W- yields in PbPb and pp collisions are found to agree, once the neutron and proton content in Pb nuclei is taken into account. Likewise, the difference observed in the dependence of the positive and negative muon production on pseudorapidity is consistent with next-to-leading-order perturbative QCD calculations.
- Published
- 2012
6. IS THERE ANY CARDIOPROTECTIVE ROLE OF TAURINE DURING COLD ISCHEMIC PERIOD FOLLOWING GLOBAL MYOCARDIAL ISCHEMIA?
- Author
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Demirkilic, U., Sahin, M., Yucel, O., Guler, A., Arslan, S., Gamsizkan, M., Cingoz, F., Doganci, S., and Yaman, H.
- Published
- 2011
7. Poster presentations
- Author
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Aksu F, Topacoglu H, Arman C, Atac A, Tetik S, Hasanovic A, Kulenovic A, Mornjakovic Z, Pikula B, Sarac-Hadzihalilovic A, Voljevica A, Bamac B, Colak T, Alemdar M, Dundar G, Selekler M, Dincer O, Colak E, Ozbek A, Kilic C, Kamburoglu K, Ozen T, Kavak V, Kirici Y, Oztas E, Soysal HA, Unur E, Ekinci N, Karaca O, Malakhova O, Kocaoglu M, Toker S, Taser F, Kilincoglu V, Yurtgun MF, Dalcik C, Zeybek A, Baroncini M, Peltier J, Jissendi P, Pruvo JP, Francke JP, Prevot V, Kosif R, Arifoglu Y, Diramali M, Sarsilmaz M, Kose E, Ogeturk M, Akpinar B, Kus I, Meydan S, Kara A, Kurtoglu Z, Tekdemir I, Elhan A, Bas O, Odaci E, Mollaoglu H, Ucok K, Kaplan S, Senoglu M, Nacitarhan V, Kurutas EB, Senoglu N, Altun I, Atli Y, Ozbag D, Karakas S, Bilgin MD, Tellioglu AM, Ozlem S, Akcanal B, Yildiz Y, Gunes H, Kose H, Uzum I, Gundogmus UN, Caglayan C, Pavlova V, Dimitrova M, Georgieva L, Nikolova E, Uzmansel D, Ozturk NC, Saylam CY, Ozgiray E, Orhan M, Cagli S, Zileli M, Ozkan D, Akkaya T, Comert A, Balikci N, Ozdemir E, Gumus H, Ergul Z, Kaya O, Altun S, Unlu RE, Orbay H, Kim DI, Han SH, Kim YS, Kim HJ, Lee KS, Elcioglu O, Ozden H, Guven G, Imre N, Yalcin B, Ozan H, Akyer P, Guvencer M, Karatosun V, Sagoo MG, Aland RC, Ustuner D, Ustuner MC, Ai J, Ghazi SR, Mansouri SH, Tuncer MC, Aluclu MU, Karabulut O, Hatipoglu ES, Nazaroglu H, Icke C, Akbay E, Gunay T, Icke S, Yildiz S, Yazar F, Barlas BO, Zahoi DE, Kavakli A, Tas U, Dabak DO, Sapmaz HI, Kocabiyik N, Ozer CM, Ozcan A, Elevli L, Desdicioglu K, Alanbay I, Govsa F, Akdogan I, Kiroglu Y, Onur S, Evcil EH, Cankara N, Malas MA, Kalcioglu MT, Duman S, Ulcay T, Uzun A, Karabulut Z, Barut C, Sevinc O, Yurdakan G, Kacar D, Erdogan AR, Kurt H, Demir B, Saltan M, Burukoglu D, Degirmenci I, Erdogan A, Damar O, Is M, Bayramoglu G, Kabay S, Uysal O, Senturk H, Bayramoglu A, Ozbayar C, Kutlu A, Canbek M, Cevli SC, Hancerlioglu O, Koplay M, Aksakalli E, Dikici F, Kale A, Gayretli O, Gurses IA, Ozdemir ST, Ercan I, Baskan EB, Yilmaz M, Ozkaya G, Saricaoglu H, Erturk M, Kayalioglu G, Uzel M, Kahraman G, Tanyeli E, Soyluoglu AI, Tacar O, Demirant A, Bilgin M, Karadede A, Aktas A, Koyuncu E, Sulak O, Albay S, Ozguner G, Ozbek E, Ozturk AH, Demirci T, Ciftcioglu E, Demir MT, Kopuz C, Eroglu E, Gedikli S, Ozyurek H, Nural MS, Incesu L, Ogur G, Kara E, Celebi B, Yildiz A, Altunkaynak BZ, Kuvat SV, Tagil SM, Ertekin C, Uysal H, Bademkiran F, Albayrak N, Esmer AF, Coskun NK, Sindel M, Kizilay F, Yalin S, Karapinar N, Tokdemir M, Karakurt L, Tumkaya L, Korkmaz A, Ayas B, Ciftci N, Terzi Y, Baran O, Nergiz Y, Akkus M, Aluclu U, Topal AE, Yuksel D, Acar HI, Kendir S, Hekimoglu E, Basman D, Ozener B, Pelin C, Zagyapan R, Kurkcuoglu A, Koc M, Erdinc M, Erdinc L, Kelle I, Sancakdar E, Cetin N, Tunik S, Yildirim A, Kaplanoglu I, Ayaz E, Ilhan N, Okumus M, Yuksel KZ, Ciralik H, Yilmaz Z, Gumusalan Y, Gamsizkan M, Kazkayasi M, Unver Dogan N, Uysal II, Karalezli A, Fazliogullari Z, Buyukmumcu M, Bozkurt MC, Cicekcibasi AE, Demiryurek D, Ozsoy MH, Tuccar E, Baran OP, Soker S, Bahceci S, Nasir Y, Yilmaz MT, Cicekcibasi EA, Ulusoy M, Gunaslan P, Bilge N, Akkaya M, Genc A, Akcer S, Gonul Y, Cosar E, Koken G, Ari I, Bakirci S, Kafa IM, Uysal M, Karabulut AK, Keles B, Emlik D, Uyar Y, Ozturk K, Yilmaz NA, Salbacak A, Kacira BK, Arazi M, Demirci S, Kiresi D, Gumus S, Seker M, Uyar M, Astaneh ME, Khorshid A, Uygur R, Songur A, Sonmez OF, Dogan KH, Kolcu G, Iliescu M, Bordei P, Iliescu D, Ciobotaru C, Lucescu V, Covaleov A, Ionescu C, Guirao M, Páramo E, Mutuberria R, Sánchez-Montesinos I, Roda O, Girón F, Lopez-Soler M, Campos-López R, Guirao-Piñeiro M, Pascual-Morenilla MT, Sanchez-Montesinos I, Pascual MT, Garzon I, Serrato D, Nieto-Aguilar R, Sanchez-Quevedo M, Ozdemir MB, Ozean RH, Bagdatli D, Adiguzel E, Dogan Z, Aycan O, Vardi N, Erkal HS, Ozturk H, Mocanu S, Stefanescu C, Ionescu A, Talpes R, Sapte E, Dina C, Surdu L, Bulbuc I, Medina MT, Medina J, López-Soler M, Martin-Oviedo C, Lowy-Benoliel A, Maranillo E, Martinez-Guirado T, Sañudo J, Scola B, Vazquez T, Arráez-Aybar LA, Conejo-Menor JL, Gonzáles-Gómez CC, Torres-García AJ, Nasu H, Chiba S, Gutierrez-Semillera M, Paksoy Y, Kalaycioglu A, Yildirim M, Ozyasar A, Ozdogmus O, Cakmak YO, Verimli U, Cavdar S, Yildizhan B, Aktan Ikiz ZA, Ucerler H, Ozgur Z, Yilmaz S, Demirtas A, Mavili E, Hacialiogullari M, Susar H, Arslan S, Aycan K, Ozkaya V, Pilmane M, Boka S, Ortug G, Ramirez C, Pascual-Font A, Valderrama-Canales F, Kucukalic A, Kapur E, Talovic E, Baca V, Grill R, Horak Z, Kachlik D, Dzupa V, Konarik M, Knize J, Veleminsky P, Smrzova T, Otcenasek M, Chmelova J, Kheck M, Cupka T, Hnatek L, van der Meijs F, Cech P, Musil V, Ozkan HM, Muratli SK, Tayefi H, Ergur I, Kiray A, Toktas M, Alkoc O, Acar T, Uzun I, Ozen OA, Aycicek A, Alkoc OA, Unlu M, Corumlu U, Ikiz IC, Oygucu IH, Sendemir E, Kaner T, Caglar V, Eser O, Iyigun O, Pirzirenli G, Kaya AH, Aydin ME, Celik F, True H, Ozkaya S, Ergur BU, Zeybek G, Bacakoglu K, Tadjalli M, Poostpasand A, Mansouiri SH, Allahvaisi O, Soleimanirad J, Nikkhoo B, Nagato Y, Haruki Y, Yazawa K, Okazaki T, Haida M, Imai Y, Peirouvi T, Mahzad-Sadaghiani M, Noroozinia F, Siamak S, Farjah G, Mola S, Biegaj E, Skadorwa T, Pawlewicz K, Kapolka R, Chachulska A, Zabicka J, Krasowska A, Prusik A, Jaczewski G, Kolesnik A, Taghavi MM, Alavi SH, Moallem SA, Safikhani Z, Panahi M, Dabiri S, Shekaari MA, Latorre R, Soria F, Lopez-Albors O, Sarria R, Ayala I, Serrano I, Perez-Cuadrado E, Musienko V, Tkachenko D, Colakoglu N, Kus MA, Jalali M, Nikravesh MR, Moeen AA, Karimfar MH, Rafighdoost H, Mohammadi S, Korneeva M, Rafighdoust H, Lovasova K, Bolekova A, Kluchova D, Sulla I, Kapitonova MY, Syed Ahmad Fuad SB, Jayakaran F, Shams AR, Aghaee F, Baqer Z, Faroki M, Das S, Kassim N, Latiff A, Suhaimi F, Ghafar N, Hlaing KP, Maatoq I, Othman F, Kiray M, Bagriyanik HA, Pekcetin C, Ozogul C, Fidan M, Sun F, Sanchez-Margallo F, Gil F, Crisostomo V, Uson J, Ramirez G, Turamanlar O, Kirpiko O, Haktanir A, Climent S, Losilla S, Climent M, Sarikcioglu L, Senol Y, Yildirim FB, Utuk A, Kunicki J, Pasbakhsh P, Omidi N, Omidi H, Nazhvani FD, Ghalebi SR, Javan N, Mohagery A, Bideskan AR, Taheri MM, Fazel AR, Tiengo C, Macchi V, Stecco C, Porzionato A, Mazzoleni F, De Caro R, Clemente A, Morra A, Greco P, Pavan P, Natali A, Demir M, Dokur M, Acer N, Mavi A, Matveeva N, Lazarova D, Korneti K, Jovevska S, Jurkovik D, Papazova M, Havasi M, Alboghobeish N, Savari A, Salamat N, Sharifi M, Kwak HH, Hu KS, Kim GC, Park BS, Sinav A, Gulati AK, Gulati NK, Alshammary H, Nazhvani SD, Vafafar A, Esmaeilpour T, Bahmanpour S, Elyasi L, Monabbati A, Ghanadi M, Paryani MR, Gilanpour H, Amirsam B, Omaña RE, López SG, De la Garza Castro O, Vega EU, Lopez SG, Talebpour F, Golmohammadi R, Dashti G, Atlasi MA, Mehdizadeh M, Bahadori MH, Joghataei MT, Hatami L, Boroujeni MB, Estakhr J, Esfandiary E, Marzban M, Bakhtiary M, Modiry N, Jafarpur M, Mofidpur H, Mahmoudian A, Jafarpour M, Mahmoudian AR, Sanjarmousavi N, Doassans I, Sorrenti N, Decuadro G, Saibene A, Poumayrac M, Laza S, Almiron C, Vergara ME, Soria V, Lasa S, Perez A, Castro G, Maria AS, Soleimani M, Katebi M, Bakhshayesh M, Oner M, Halici M, Yikilmaz A, Guney A, Turk Y, Edizer M, Beden U, Icten N, Afshar M, Hasanzadeh Taheri MM, Moalem A, Golalipour MJ, Tamizi A, Ahi M, Mohammadpour S, Maiery A, Acikel C, Ulkur E, Karagoz H, Celikoz B, Bedi K, Ginus P, Golalipoor MJ, Mohammadi MR, Jhand P, Mansourian AR, Hosseinpoor K, Keshtkar AA, Alsaffar R, Balajadeh BK, Ghafari S, Azarhosh R, Fazeli SA, Jahanshahi M, Gharravi AM, Alicioglu B, Karakas HM, Harma A, Yang HM, Won SY, Lee JG, Lee JY, Kim YR, Song WC, Koh KS, Hwang EN, Choi HG, Kim SH, Kim SY, Hur MS, Ulucam E, Celbis O, Kim DH, Hong HS, Choi JH, Park JT, Kim HC, Abbasi H, Hosseinipanah SM, Hosseini M, Amani A, Ashrafi HR, Sadeghimehr M, Sheverdin V, Amani Z, Ashrafi A, Ashrafi AR, Javad H, Kachap MJ, Poumayrac MC, Almirón C, Rivara A, Sirilo A, Freire D, Cirillo A, Veragara ME, Krmek V, Krmek N, Jo-Osvatic A, Nikolic V, Radic R, Tubbs RS, Loukas M, Fogg Q, Ashwood N, Cilingiroglu S, Ozbakir C, Mazoochi T, Sabanciogullari V, Gumus C, Erdil FH, Cimen M, Moodi H, Ghiasi F, Akbari A, Hami J, Khazei M, Haghparast E, Mitsakis I, Anastasiou A, Mitsakis M, Sianou K, Hainoglou R, Francisco M, Mitsaki C, Konstantinidi M, Prapa S, Leksan I, Mrcela T, Selthofer R, Kermanian F, Ahmadpoor ME, Dalili N, Elian AH, Moaiery A, Jamalpour Z, Nourani MR, Asgari A, Hassanzadeh Taheri MM, Ebrahimzadeh A, Eftekharvaghefi SH, Mohammadi A, Sheibani V, Nematollahi-Mahani SN, Latifpour M, Deilami M, Soroure-Azimzadeh B, Nabipour F, Najafipour H, Nakhaee N, Yaghoobi M, Eftekharvaghefi R, Salehinejad P, Azizi H, Riasi HR, Nobakht M, Asalgoo S, Rahbar R, Najafzadeh N, Moosavizadeh K, Ezzatabadypour M, Majidi M, Malekpor-Afshar R, Karimzade F, Hoseini M, Bayat M, Gorgi A, Nezhadi A, Bakhtiari M, Jazi HR, Jafaryan M, Haghir H, Rahimi S, Rassouli FB, Gorji A, Habibi A, Pouya F, Mousavi A, Rajabalian S, Abolidokht A, Khanlarkhani N, Naderian H, Berjis N, Namavar MR, Talaei T, Mazaheri Z, Monabati A, Kosar MI, Karacan K, Chegini H, Nikzad H, Ayhan E, Ustundag S, Akkin SM, Ogut T, Rayegan P, Meibodi MA, Ghaem RM, Zargarpoor R, Eftekhar Vaghefi SH, Moshkdanian G, Poya F, Kohestani H, Abarghoeai RR, Abarghoeai PR, Mahmodi AA, Poraboli A, Kohestani HR, Vaghefi RE, Eftekhar Vaghefy SH, Vaghefy RE, Saba M, Javadnia F, Zhaleh M, Nezhad DB, Gholami MR, Piagkou M, Aikaterini VK, Piagkos G, Douvetzemis S, Skandalakis P, Anagnostopoulou S, Papadopoulos N, Celik HH, Tatar I, Tatar EC, Mocan BO, Sargon MF, Denk CC, Rasoolijazi H, Joghataie MT, Roghani M, Dinc G, Kurklu M, Ozboluk S, Komurcu M, Koebke J, Balioglu MB, Kaygusuz MA, Bozkus FS, Korkmaz O, Bayram SB, Can MA, Nasiri E, Jafar-Kazemi K, Maghoul S, Amini A, Hassanzade MM, Davari MH, Van Hoof T, Gomes GT, Audenaert E, Verstraete K, Kerckaert I, D'Herde K, Benninger B, Hedley G, Filipoiu FM, Tarta E, Enyedi M, Pantu C, Stanciulescu R, Skobowiat C, Calka J, Majewski M, Rezaian M, Yaghoobfar A, Hamedi S, and Shomali T
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- 2009
8. Intralesional treatments for hypertrophic scars: comparison among corticosteroid, 5-fluorouracil and botulinum toxin in rabbit ear hypertrophic scar model.
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ÇALISKAN, E., GAMSIZKAN, M., AÇIKGÖZ, G., DURMUş, M., TOKLU, S., DOğRUL, A., KURT, A., and TUNCA, M.
- Abstract
OBJECTIVE: Different treatment modalities have been used either alone, or in combination to achieve an optimum improvement for hypertrophic scars. Intralesional injections of corticosteroids and 5-fluorouracil are among the most commonly used treatments. Recently, botulinum toxin is proposed as a new treatment option. In this study, it is aimed to compare the efficacies of intralesional triamcinolone acetonide, 5-fluorouracil and botulinum toxin-A for hypertrophic scars. In order to minimize the variables affecting scar formation, standardized wounds in rabbit ear hypertrophic scar model was used. MATERIALS AND METHODS: Four surgical wounds were created on both ears of eight rabbits. Injections to be compared (triamcinolone acetonide, 5-fluorouracil, botulinum toxin-A and control) are administered intralesionally to established scars on day 30. Scars were harvested on day 60 for morphometric analysis including hypertrophic index, fibroblast density, and relative collagen density. RESULTS: Triamcinolone acetonide and 5-fluorouracil injections decreased hypertrophic indexes significantly compared to botulinum toxin-A and control group. However, only 5-fluorouracil was effective to reduce fibroblast counts significantly. No statistically significant differences were found between the treatment groups in terms of collagen index. CONCLUSIONS: According to the results of our study, triamcinolone acetonide and 5-fluorouracil are comparatively effective as monotherapy, but botulinum toxin-A was not effective on established hypertrophic scars. [ABSTRACT FROM AUTHOR]
- Published
- 2016
9. Effects of Human Amniotic Fluid on Costal Cartilage Regeneration (an Experimental Study)
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Kavakli, K., primary, Gurkok, S., additional, Caylak, H., additional, Genc, O., additional, Gamsizkan, M., additional, Yucel, O., additional, Karasahin, E., additional, Gozubuyuk, A., additional, and Tasci, C., additional
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- 2011
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10. OP-281: IS THERE ANY CARDIOPROTECTIVE ROLE OF TAURINE DURING COLD ISCHEMIC PERIOD FOLLOWING GLOBAL MYOCARDIAL ISCHEMIA?
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Sahin, M., primary, Yucel, O., additional, Guler, A., additional, Doganci, S., additional, Cingoz, F., additional, Arslan, S., additional, Gamsizkan, M., additional, Yaman, H., additional, and Demirkilic, U., additional
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- 2011
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11. Muscle herniation of the extremities coexistent with piezogenic pedal papules: A case report
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Uyar Belkiz, Özel Mehmet Ali, Gamsızkan Mehmet, Özcan Yunus, and Acar Emine Müge
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Dermatology ,RL1-803 - Published
- 2022
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12. Is there any cardioprotective role of Taurine during cold ischemic period following global myocardial ischemia?
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Gamsizkan Mehmet, Arslan Sıddık, Cingoz Faruk, Jahollari Artan, Doganci Suat, Guler Adem, Yucel Orhan, Sahin Mehmet A, Yaman Halil, and Demirkilic Ufuk
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Surgery ,RD1-811 ,Anesthesiology ,RD78.3-87.3 - Abstract
Abstract Background The aim of the present study was to investigate the cardioprotective effect of Taurine on the donor hearts during cold ischemic period. Methods 32 rats were divided into four groups (sham, taurine, ischemia, treatment group, 8 rats in each). All rats were fed with rat food for three weeks. Taurine and treatment groups were given a 200 mg/kg/day dose of Taurine by oral gavage besides rat feed. Cardiectomy was performed in all rats after three weeks. In ischemia and treatment groups, harvested hearts were kept in 0.9% sodium chloride at +4 degrees C for 5 hours. Tissue samples were taken from left ventricle in all groups. These samples were evaluated by histopathologic and biochemical examination. Results In the present study results of the biochemical and histopathological examination reveals the protective effects of Taurine. As a marker of lipid peroxidation, Malondialdehyde (MDA) levels in ischemia group were significantly higher than both Sham and Taurine groups. MDA values were recorded; 3.62 ± 0.197 in the sham group, 2.07 ± 0.751 in the Taurine group, 9.71 ± 1.439 in the ischemia group and 7.68 ± 1.365 in the treatment group. MDA levels decreased in treatment group. (p < 0.05) In accordance with MDA findings, while superoxide dismutase and glutathione peroxidase levels decreased in ischemia group, they increased in treatment group. (p < 0.05) There was no differences in Catalase (CAT) enzyme level between treatment and ischemia group (p = 1.000). CAT level results were recorded; 7.08 ± 0.609 in the sham group, 6.15 ± 0.119 in the Taurine group, 5.02 ± 0.62 in the ischemia group, and 5.36 ± 0.384 in the treatment group. Less intracellular edema and inflammatory cell reaction were observed in histologic examination in favor of treatment group. (p < 0.01) Conclusion Taurine decreased myocardial damage during cold ischemic period following global myocardial ischemia.
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- 2011
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13. Intraosseous tophus deposits in the os trigonum.
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Ercin E, Gamsizkan M, Avsar S., Ercin, Ersin, Gamsizkan, Mehmet, and Avsar, Serdar
- Abstract
High levels of uric acid cause accumulation of monosodium urate crystals. This formation of masses is called tophus. Intraosseous tophus deposits are rare, even for patients with gout. We report an unusual case of intraosseous tophus deposits in the os trigonum. The patient presented with ankle pain with no previous history of gout. On examination, tenderness on the posterior aspect of his ankle and limitation of plantarflexion was noted. Laboratory values were normal, except for an elevated serum uric acid value. Radiographs of the right ankle showed the presence of a large os trigonum with osteosclerotic changes, whereas magnetic resonance imaging showed intraosseous tophus deposits in the os trigonum. Conservative therapy failed, and the patient was admitted for an endoscopic resection of the os trigonum.Intraosseous chalky crystals were detected during endoscopic resection of the os trigonum. The histological diagnosis was tophaceous gout. The underlying pathological mechanism of intraosseous tophi is uncertain. Penetration of urate crystals from the joint due to hyperuricemia may be the mechanism of deposition in this patient.When a patient with hyperuricemia presents with posterior ankle impingement symptoms, intraosseous tophus deposits should be included in the differential diagnosis. Posterior endoscopic excision may be an option for treating intraosseous lesions of the os trigonum because of good visualization, satisfactory excision, and rapid recovery time. [ABSTRACT FROM AUTHOR]
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- 2012
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14. Giant solitary osteochondroma arising from the fifth metatarsal bone: a case report.
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Yildirim C, Rodop O, Kuskucu M, Sahin O, and Gamsizkan M
- Abstract
Accounting for 20% to 50% of all benign forms, solitary osteochondroma is the most common bone tumor. The long bones of the lower extremity are most frequently affected, whereas the small bones of the hands, feet, pelvis, scapula, and spine are less common locations. Osteochondromas are benign osseous neoplasms with a distinct hyaline cartilage cap originating from the physis, and they cease to grow with skeletal maturity. Treatment of osteochondroma is usually conservative, unless symptoms, usually pain, are progressive or the lesion demonstrates rapid or new growth, or if enlargement after skeletal maturation is noted or malignant transformation is suspected. In this report, we describe the case of an adult with a giant, symptomatic osteochondroma localized to the fifth metatarsal. The lesion was treated with excision, and after more than 2 years of follow-up, no evidence of recurrence was noted. This case demonstrated that, despite the benign nature of the lesion, a large osteochondroma could localize to a metatarsal. [ABSTRACT FROM AUTHOR]
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- 2010
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15. Dermatoscopic and Dermatopathologic Features of a 3-Year-Old Buried Polypropylene Suture.
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Özcan Y and Gamsizkan M
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- Humans, Child, Preschool, Sutures adverse effects, Skin, Dermatologic Surgical Procedures, Polypropylenes adverse effects, Fistula
- Abstract
Abstract: Cutaneous foreign bodies are a well-known cause of delayed wound healing and complications such as abscesses, fistula formation, and secondary infections. Polypropylene sutures are widely utilized in cutaneous surgery because they easily travel through tissues while eliciting minimal tissue reactions. Despite these advantages, retained polypropylene sutures can cause complications. The authors report a case of a retained polypropylene suture that remained buried after a total excision 3 years prior. It started to cause cutaneous symptoms when the patient began exercising 1 week prior to presentation. The authors also examine the dermatoscopic and dermatopathologic features and other complications related to retained polypropylene sutures that have been reported in the literature., (Copyright © 2023 Wolters Kluwer Health, Inc. All rights reserved.)
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- 2023
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16. Clinicopathologic Features and Prognostic Significance of Immunohistochemistry and In Situ Hybridization Based Molecular Classification in Gastric Carcinoma.
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Issin G, Sayar İ, Demir F, Bakkaloğlu İG, Gamsizkan M, Yildiz Z, Yilmaz I, Özmen SA, Çağatay DV, Zemheri IE, Demiriz M, and Günal A
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- Humans, Herpesvirus 4, Human genetics, Herpesvirus 4, Human metabolism, B7-H1 Antigen genetics, B7-H1 Antigen metabolism, Prognosis, Immunohistochemistry, Tumor Suppressor Protein p53 genetics, Microsatellite Instability, In Situ Hybridization, Epstein-Barr Virus Infections diagnosis, Epstein-Barr Virus Infections genetics, Epstein-Barr Virus Infections complications, Stomach Neoplasms diagnosis, Stomach Neoplasms genetics, Carcinoma complications
- Abstract
Background/aim: Gastric carcinoma (GC) is a highly heterogeneous disease with many subtypes that have different morphologic and molecular characteristics. In the current study, we analyzed immunohistochemical (IHC) and in situ hybridization (ISH) features of GCs and evaluated their association with prognosis and clinicopathological features., Materials and Methods: Three hundred cases analyzed by IHC and ISH for microsatellite stability, p53, e-cadherin, HER2, PD-L1 expression, and Epstein-Barr virus (EBV) status. Cases were classified into five subgroups based on expression profile. The relationships between subgroups, clinicopathological features, and survival were determined., Results: Ten (3.3%) cases were classified as EBV-associated, 45 (15%) as microsatellite instable (MSI), 73 (24.3%) as EBV-/microsatellite-stable (MSS)/epithelial-mesenchymal-transformation (EMT)-like, 75 (25%) as EBV-/MSS/ non-EMT-like/p53+, and 97 (32.3%) as EBV-/MSS/non-EMT-like/p53-. The MSI subtype had the best overall survival (OS). In contrast, the EBV-/MSS/EMT-like subtype had the poorest OS. The MSI subtype was also related with old age of the patient and antrum-corpus localized tumors, whereas the EBV-/MSS/EMT-like was associated with young age, larger tumor size, and advanced stage presentation. PD-L1 positivity is highly correlated with MSI and EBV-associated subtypes., Conclusion: Our data demonstrated a link between IHC/ISH characteristics of GC and clinical outcomes. IHC/ISH based molecular classification may be helpful in predicting the survival.
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- 2023
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17. Receptor Tyrosine Kinase Pathway and Infiltrating Urothelial Carcinoma.
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Buyucek S, Coskun SK, Onal B, Gamsizkan M, Cangur S, and Esbah O
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- Humans, Proto-Oncogene Proteins p21(ras) genetics, Mutation, ErbB Receptors genetics, Carcinoma, Transitional Cell genetics, Urinary Bladder Neoplasms genetics
- Abstract
Receptor tyrosine kinase pathway is frequently searched for cancer causing mutations in tumors. Emerging targeted therapies are gleam of hope for them. Infiltrating urothelial carcinoma can have many morphological aspects according to their differentiation/variants. To evaluate KRAS, BRAF, and PIK3CA mutations and HER2, EGFR, and p16 expression, we divided urothelial carcinomas into two groups: differentiated/variants (n = 12) and conventional (n = 12). We compared results with clinical, demographic, histopathologic features and survival rates. No statistically significant results could be obtained in the comparison of histopathologic properties/survival rates with mutation analysis and EGFR, HER2, and p16 status. Differentiated/variants urothelial carcinoma showed higher EGFR expression (P < 0.001). Glandular differentiation was the most frequent type, followed by squamous and sarcomatoid differentiation. We observed the most common mutation at KRAS with a propensity for urothelial carcinoma with glandular differentiation. More than one mutation/high protein expression was seen in some tumors. Targeted therapies for KRAS mutation can be effective at urothelial carcinoma with glandular differentiation. Heterologous expression of relevant proteins and genes can be a cause for targeted treatment obstacle. The determination of the molecular characters of tumors is a guide in creating targeted treatment algorithms and in choosing the patient.
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- 2023
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18. Rifaximine spacer application is not superior to local teicoplanin treatment in a rat model of osteomyelitis.
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Yucel MO, Turhan Y, Arican M, Karaduman ZO, Saglam S, Tekce Y, and Gamsizkan M
- Abstract
Objective: Acute and chronic osteomyelitis generally require long-term antibiotic therapy and surgical debridement. Implant-associated osteomyelitis, particularly from methicillin-resistant Staphylococcus aureus (MRSA) strains, is difficult to treat. Rifaximin is an antibiotic derived from rifamycin which may be effective in the treatment of osteomyelitis in terms of its wide spectrum of action and pharmacological properties. The aim of this experimental study was to investigate the local efficacy of rifaximin in rat models with MRSA and implant associated osteomyelitis., Methods: This study was carried out with 40 adult Wistar albino rats. The rats were randomly divided into 4 equal groups with 10 rats in each. An implant related MRSA osteomyelitis was created in the right tibia metaphysis of each rat by Norden's experimental osteomyelitis model. After 4 weeks, the implants of each tibia were removed and debridement was applied. Group 1 was designed as control group and no other treatment was applied other than debridement. Bone cement without any antibiotic was applied to Group 2, bone cement with teicoplanin was applied to Group 3 and bone cement with rifaximin was applied to Group 4. After 4 weeks from the second surgery, euthanasia was performed to the rats and the clinical, histopathological and microbiological results were compared., Results: There was no statistically significant difference between the groups in clinical scoring. A statistically significant difference was found between the histopathological scores of Group 1 and Group 2 and the histopathological scores of Groups 3 and 4; the histopathological scores of Group 1 and Group 2 were found to be higher than Group 3 and Group 4. When the pre-and post-treatment colony numbers were compared, although there was a statistically significant difference between Group 3 and Group 2, no statistically significant difference was found between Group 4 and Group 1 results., Conclusion: In spite of its wide spectrum, the local efficacy of rifaximin in the treatment of osteomyelitis could not be demonstrated. This study shows the ability to shed light on some future comprehensive studies with the inclusion of infection markers., Competing Interests: No conflict of interest was declared by the authors., (© Copyright 2022 by Istanbul Provincial Directorate of Health.)
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- 2022
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19. Dermatopathological Correlation of Clinically Challenging Cutaneous Lesions: a Single Center Experience of 2184 Cases.
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Ozcan Y, Ozlu E, Karagun E, Uyar B, and Gamsizkan M
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Introduction: Although a trained eye can easily identify typical skin lesions, histopathological examination and clinicopathological correlation are critical in challenging cases., Objectives: The primary objective is to organize the final diagnoses reached following clinicopathological consensus in clinically challenging cutaneous lesions, identifying the most common diagnostic scenarios encountered by dermatopathologists and discussing their diverse differentials submitted by clinicians. The secondary objective is to investigate how the case profile and clinician decision-making processes evolved during the COVID-19 pandemic., Methods: Skin and mucosa samples collected by the dermatology department between 2016 and 2020 were classified based on pathology reports. For frequent diagnoses, preliminary diagnoses stated by clinicians on pathology requisition forms were reviewed. The years preceding and following the first nationally reported COVID-19 case were compared to investigate the pandemic's impact on the distribution of dermatology and dermatopathology cases., Results: One thousand nine hundred and eighty-nine reports were classified into 4 major categories: inflammatory (49.8%), neoplastic (30.1%), other diseases (7.1%), and non-diagnostic (12.8%). We further classified inflammatory diseases based on major tissue reaction patterns and neoplasms based on cell origin. We analyzed the leading diagnoses in each category, discussed their differential diagnoses, and provided clinicians with clues to reduce errors in practice. Following the pandemic, the overall number of pathology reports and patient admissions dropped dramatically, with significant changes in case profiles., Conclusions: We presented and discussed the frequently encountered confounding cases to sketch the diagnostic landscape. In the authors' experience, clinicopathological correlation can increase the rate of reaching the diagnosis by up to 75.3%., Competing Interests: Competing interests: None., (©2022 Ozcan et al.)
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- 2022
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20. Diffuse-type giant cell tumor: Pigmented villonodular synovitis of patellar fat pad.
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Degirmenci E, Sahin AA, Bulum YE, Gamsizkan M, and Orhan Z
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- Adipose Tissue pathology, Humans, Knee Joint diagnostic imaging, Knee Joint pathology, Knee Joint surgery, Synovial Membrane pathology, Giant Cell Tumors pathology, Synovitis, Pigmented Villonodular diagnosis, Synovitis, Pigmented Villonodular surgery
- Abstract
Pigmented villonodular synovitis (PVNS) is a rare, relatively benign intra-articular lesion characterized by slowly progressing proliferation of the synovial tissue. It is most commonly observed in the knee joint. Localized and diffuse types are two types of PVNS depending on the synovial involvement. Arthroscopic and excisional resections are recommended as the treatment methods for the PVNS. Radiotherapy or chemotherapy can be adjuvant therapeutic options for the widespread masses. In this study, we presented a case of diffuse PVNS originating from the patellar fat pad., Competing Interests: None
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- 2022
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21. STAT3, VEGF, and PSMA Expression Patterns in Malignant Peripheral Nerve Sheath Tumors, Malignant Melanomas, and Glioblastomas: Does Staining Percentage and Intensity Have an Effect on Survival?
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Coskun SK, Gamsizkan M, Yalcınkaya U, and Sungur MA
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- Antigens, Surface, Glutamate Carboxypeptidase II metabolism, Humans, Male, STAT3 Transcription Factor metabolism, Staining and Labeling, Transducers, Vascular Endothelial Growth Factor A metabolism, Vascular Endothelial Growth Factors metabolism, Glioblastoma, Melanoma, Neurofibrosarcoma
- Abstract
Malignant peripheral nerve sheath tumors (MPNSTs), glioblastomas (GBMs), and malignant melanomas (MMs) are neural crest-originating aggressive tumors with a poor prognosis. Signal transducer and transcription activator 3 (STAT3) plays a role in many biological processes, including cell life and proliferation, the acute phase response, chronic inflammation, autoimmunity, metabolism, and cancer progression, It is also known to be a prooncogenic transcription factor. Vascular endothelial growth factor (VEGF) is one of the most potent proangiogenic stimuli ever identified. It mediates tumor neovascularization, and is associated with angiogenesis and lymphangiogenesis. The prostate-specific membrane antigen (PSMA) folate hydrolase I, despite its name, has been found in tissues other than the prostate. It is overexpressed in prostate cancer cells and several other cancers, and has the potential to be a target for radioligand therapy. We investigated the value of STAT3, VEGF and PSMA immunohistochemical expression patterns and their effects on survival in MPNSTs, GBMs, and MMs. Their expression patterns were evaluated in 25 MPNSTs, 27 GBMs, and 25 MM cases. All GBM cases stained positively for STAT3 and VEGF. In the other groups, the staining patterns were heterogeneous. None of the cases showed positive staining with PSMA. There was no statistically significant difference in survival between cases with differing VEGF and STAT3 staining patterns in the MPSNT and MM groups, but there was an increase in mortality as the VEGF score increased in the GBM group. The suppression of VEGF and STAT3 may be a promising avenue for treatment of MPNSTs, GBMs, and MMs, although further research is needed.
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- 2022
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22. CD123 immunoexpression in cutaneous lupus erythematosus, polymorphous light eruption, pityriasis rosea, and mycosis fungoides.
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Karagun E, Gamsizkan M, Buyucek S, and Coskun S
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Introduction: CD123-positive plasmacytoid dendrocytes are prominent in the infiltrate of cutaneous lupus erythematous., Aim: To determine the significance of the CD123 immunostain, which labels plasmacytoid dendritic cells (PDC), in cutaneous lupus erythematous (CLE), polymorphous light eruption (PLE), pityriasis rosea (PR) and mycosis fungoides (MF)., Material and Methods: A total of 76 cases, including MF ( n = 27), CLE ( n = 19), PR ( n = 19), and PLE ( n = 11), were included in the study after reviewing their diagnostic clinical features and pathologic findings. The primary antibody against CD123 was performed in all cases., Results: CD123+ immunostaining in PDCs was positive in all cases. The highest mean percentage was noted in CLE (15.2%), followed by PLE (15%), PR (8.8%), and MF (2%). Besides, the clustering of CD123-positive cells was significant in CLE and PLE compared to MF and PR., Conclusions: PDC may have an important role in the aetiology of PLE and CLE cases. CD123 is a useful marker for differentiating CLE and PLE from MF and PR., Competing Interests: The authors declare no conflict of interest., (Copyright: © 2021 Termedia Sp. z o. o.)
- Published
- 2021
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23. KRAS, BRAF, PIK3CA mutation frequency of radical prostatectomy samples and review of the literature.
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Bahcivan A, Gamsizkan M, Kantarcioglu Coskun S, Cangur S, Yuksel A, Ceyhan A, and Onal B
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- Class I Phosphatidylinositol 3-Kinases genetics, Humans, Male, Mutation Rate, Phosphatidylinositol 3-Kinases genetics, Prostatectomy, Proto-Oncogene Proteins genetics, Proto-Oncogene Proteins p21(ras) genetics, Colorectal Neoplasms, Proto-Oncogene Proteins B-raf genetics
- Abstract
Objective: The molecular basis of prostate cancer is highly heterogeneous. Our study aimed to perform the mutation analysis of KRAS, BRAF, PIK3CA , and immunohistochemical (IHC) evaluation of EGFR, HER2, p16, and PTEN to demonstrate new areas for targeted therapies., Methods: A total of 24 prostatectomy samples diagnosed with adenocarcinoma were analyzed by microarray hybridization. Also, these samples were IHC stained for EGFR, HER2, P16, and PTEN. The cases were divided into two groups based on low and high Gleason scores. All findings were compared with the clinicopathological parameters of the patients., Results: While KRAS mutation was in 3/24 (12.5%) of our cases, BRAF and PIK3CA mutations were not detected. There was no significant difference between the groups in terms of KRAS mutation frequency. HER2 was immunohistochemically negative in all samples. There was no correlation between EGFR, P16 immunopositivity, and clinicopathological features., Conclusion: KRAS mutation frequency is similar to those in Asian populations. BRAF and PIK3CA mutation frequencies have been reported in the literature in the range of 0-15% and 0-10.4%, respectively, consistent with our study findings. HER2 immunoexpression is a controversial issue in the literature. EGFR and p16 expressions may not correlate with the stage.
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- 2020
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24. BRAF mutation, TERT promoter mutation, and HER2 amplification in sporadic or neurofibromatosis-related neurofibromas and malignant peripheral nerve sheath tumors: do these molecules have a signature in malignant transformation?
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Coskun S, Gamsizkan M, Yilmaz I, Yalcinkaya U, Sungur MA, Buyucek S, and Onal B
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- Adolescent, Adult, Aged, Child, Female, Gene Amplification, Humans, Male, Middle Aged, Mutation, Neurofibroma genetics, Promoter Regions, Genetic, Young Adult, Neurofibromatosis 1 genetics, Neurofibrosarcoma genetics, Proto-Oncogene Proteins B-raf genetics, Receptor, ErbB-2 genetics, Telomerase genetics
- Abstract
Peripheral nerve sheath tumors may occur sporadically or related to neurofibromatosis (NF). Unless the mechanisms of tumorigenesis in NF related malignant peripheral nerve sheath tumors (MPNST) are better understood, it remained unclear in sporadic cases. We aimed to investigate the genetic route for malignancy in both individuals with NF-1 and sporadic ones to open a way for targeted therapies in the future. We investigated the role of HER2 with Dual ISH DNA Probe Cocktail test, BRAF mutation (exon 15) and TERT promoter mutation frequency with Sanger sequencing method in respectively 25 sporadic neurofibromas, 25 NF-1 related neurofibromas and 25 MPNST cases from two institutes. Categorical data were analyzed and summarized as frequency and percentage. Statistical analysis was done with SPSS v.22 statistical package, and the statistical significance level was considered as 0.05. We identified TERT promoter mutation only in one sporadic MPNST (4%) and no BRAF mutation in any case. HER2 amplification is found in 10/25 (40%) MPNST cases. No mutations or gene amplification detected in neurofibromas (p < 0.001). MPNSTs are sarcomas with poor prognosis and limited treatment options. TERT promoter mutations and HER2 amplification may play a putative role in therapeutic purposes., (© 2020 APMIS. Published by John Wiley & Sons Ltd.)
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- 2020
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25. Effects of acidic and nonacidic reflux on the eustachian tube: An animal experiment.
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Kilic E, Gerek M, Karakoc O, and Gamsizkan M
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We investigated the effects of pepsin/hydrochloric acid and bile acids on eustachian tube function and eustachian tube mucosa in 40 Sprague-Dawley rats. The animals were randomly assigned to groups of 10: one group received pepsin/hydrochloric acid (pepsin/HCl group), another received human bile (human bile group), a third received a mixture of pepsin/HCl and human bile (combination group), and the fourth received isotonic saline solution (control group). Test solutions were applied transnasally three times a day for 10 days. Passive opening pressures and passive closing pressures were measured digitally at baseline and then on days 3, 5, and 10. After 10 days, the rats were sacrificed and histologic changes in the eustachian tube mucosa were analyzed. At study's end, we observed that the increases in passive opening pressures and passive closing pressures in all three experimental groups were significantly greater than those of the control group. Moreover, the increases in passive opening and closing pressures were significantly greater in the combination group than in both the pepsin/HCl and the human bile groups. In the tympanic orifice, the degree of lymphocyte and polymorphonuclear leukocyte infiltration was significantly higher in all three experimental groups than in the control group. In the nasopharyngeal orifice, lymphoid follicle formation was significantly more common in the human bile group than in the control group; also, the presence of subepithelial vasodilation and subepithelial edema was significantly more common in the pepsin/HCl and combination groups than in the controls. Of the three experimental solutions tested, the combination of pepsin/HCl and human bile was the most injurious to eustachian tube function. Reflux of bile acids causes eustachian tube dysfunction, and this damage worsens with the introduction of an acidic compound.
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- 2018
26. Mutation analysis of metastatic melanomas in the central nervous system: results of a panel of 5 genes in 48 cases.
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Gamsizkan M, Yilmaz I, Simsek HA, Onguru O, Griffin A, and Tihan T
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- Adolescent, Adult, Aged, Aged, 80 and over, DNA Mutational Analysis, Female, Humans, Male, Middle Aged, Polymerase Chain Reaction, Young Adult, Brain Neoplasms genetics, Brain Neoplasms secondary, Melanoma genetics, Melanoma secondary, Mutation
- Abstract
Melanocytic lesions in the central nervous system (CNS) may be primary to the site but are more commonly metastases from cutaneous primaries. In fact, melanomas are one of the most common malignancies that can metastasize to the brain, and some patients may not have a diagnosis of melanoma prior to the discovery of the CNS lesion. In such cases, identifying the primary site may be challenging. We reviewed the archives of a large referral center for melanocytic tumors involving the CNS and selected 48 patients for this study based on our inclusion criteria. We used sequencing to identify mutation status of these tumors and compared these with clinicopathological features. Mutations in exon 9, 11, 13, 17, and 18 of KIT gene, exon 15 of BRAF gene, exon 2 and 3 of NRAS gene, exon 4 and 5 of GNAQ and GNA11 genes were analyzed. Mutations in BRAF-exon 15 were the most common among tumors (58.3%). NRAS-exon 2 and NRAS-exon 3 mutations were detected in 3 and 7 cases, respectively. GNAQ-exon 4, GNAQ-exon 5 and GNA11-exon 5 mutation were present in 1 tumor each. Eight tumors were wild type for all 5 genes, and 6 of these were not known primary despite a work-up and clinical follow-up. Only 1 of these tumors showed a mutation in exon 11 of KIT gene. When compared to primary melanocytic lesions of the CNS, metastatic melanomas were characterized by BRAF gene mutations and wild-type GNAQ and GNA11 genes.
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- 2016
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27. Talar Osteochondroma Fracture Presenting as Posterior Ankle Impingement.
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Ercin E, Bilgili MG, Gamsizkan M, and Avsar S
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- Bone Neoplasms pathology, Bone Neoplasms surgery, Diagnosis, Differential, Humans, Male, Osteochondroma pathology, Osteochondroma surgery, Radiography, Young Adult, Ankle Joint diagnostic imaging, Bone Neoplasms diagnostic imaging, Joint Diseases diagnostic imaging, Osteochondroma diagnostic imaging, Talus injuries
- Abstract
Osteochondromas are the most common benign bone tumors. They are usually asymptomatic and found incidentally. When symptomatic, the symptoms are usually due to its location and size. Fracture of an osteochondroma presenting as posterior ankle impingement is a rare condition. We describe a 22-year-old man with solitary exostosis who presented with a posterior ankle mass and posterior ankle impingement with 2 years of follow-up. Surgical intervention was the treatment of choice in this patient, and histologic examination revealed a benign osteochondroma. Osteochondromas found in the posterior aspect of the talus can be complicated by fracture due to persistent motion of the ankle. Talar osteochondroma should be included in the differential diagnosis of posterior ankle impingement causes. Posterior talar osteochondromas, especially when a stalk is present, should be treated surgically before it is more complicated by a fracture and posterior ankle impingement.
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- 2016
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28. Molecular alterations in malignant blue nevi and related blue lesions.
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Yilmaz I, Gamsizkan M, Sari SO, Yaman B, Demirkesen C, Heper A, Calli AO, Narli G, Kucukodaci Z, Berber U, Demirel D, Akalin T, Demiriz M, and Buyukbabani N
- Abstract
Malignant blue nevi (MBN) are extremely rare dermal melanocytic tumors that arise in association with atypical cellular blue nevi (ACBN), cellular blue nevi (CBN), common blue nevi (BN), or de novo. The frequency of BRAF, NRAS, and KIT mutations in malignant melanoma varies according to histological subtype and localization. These mutations are rarely observed in blue nevi, which have recently been shown to carry activating mutations in GNAQ/GNA11 genes. Only few small molecular studies of MBN have been published. The aim of the present study was to analyze in MBN and related blue lesions such as ACBN, CBN, and BN the prevalence of BRAF, NRAS, KIT, GNAQ, and GNA11 gene mutations and their association with clinicopathological features. We included in our study 12 MBN, 6 ACBN, 29 CBN, and 35 common BN diagnosed between 1996 and 2014. Sanger sequencing method was used for mutation analysis. Overall, GNAQ exon 5 mutation was the most frequent alteration (46 %), in 2 of 12 (17 %) MBN, 1 of 6 (17 %) ACBN, 22 of 29 (76 %) CBN, and 13 of 35 (37 %) common BN. BRAF V600E and GNA11 exon 5 mutations were respectively detected in 3 of 12 (25 %) and in 2 of 12 (17 %) MBN while none in ACBN, CBN, and common BN. None of the cases harbored NRAS exon 2/3, KIT exon 9/11/13/17/18, or GNAQ/GNA11 exon 4 mutations. GNAQ gene exon 5 mutations are rare in MBN and ACBN but frequent in CBN and common BN. Remarkably, BRAF V600E and GNA11 exon 5 mutations were only detected in MBN, whereas none were found in ACBN, CBN, or common BN. Our data contribute new elements to the limited data on molecular alterations in MBN.
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- 2015
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29. Nevus sebaceus with basal cell carcinoma, poroma, and verruca vulgaris.
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Cicek AF, Aykan A, Yapici A, Gamsizkan M, Ozturk S, and Demiriz M
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- Adult, Diagnosis, Differential, Hair Diseases pathology, Humans, Male, Scalp pathology, Skin pathology, Warts pathology, Carcinoma, Basal Cell pathology, Hamartoma pathology, Nevus pathology, Poroma pathology, Skin Neoplasms pathology, Sweat Gland Neoplasms pathology
- Abstract
Nevus sebaceus (NS) is a congenital, benign, hamartomatous lesion and it is possible to see several benign or malignant tumors accompanying it. One of these is the poroma, which is very rare, and has only been reported twice before, in the English literature. In this paper, we presented two new cases of NS. One of them was a 40-year-old male who presented with a congenital skin lesion on his temporoparietal region. This lesion was composed of four different lesions, including NS, poroma, basal cell carcinoma (BCC), and verruca vulgaris. The second patient was a 41-year-old male presenting with a yellow-brown patch on the scalp. This lesion was comprised of NS and BCC. In addition to these presentations, we discussed the differential diagnosis between BCC and trichoblastoma, both of which are likely to be seen with NS. For this purpose, we recommended an immunohistological panel, which may be useful for differentiating these two morphologically similar lesions.
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- 2015
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30. BRAF, KIT, NRAS, GNAQ and GNA11 mutation analysis in cutaneous melanomas in Turkish population.
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Yilmaz I, Gamsizkan M, Kucukodaci Z, Berber U, Demirel D, Haholu A, and Narli G
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- Adult, Aged, Aged, 80 and over, Female, Gene Frequency, Humans, Male, Middle Aged, Mutation, Retrospective Studies, Sequence Analysis, DNA, Turkey, Young Adult, GTP-Binding Proteins genetics, Melanoma genetics, Protein Kinases genetics, Proto-Oncogene Proteins genetics, Skin Neoplasms genetics
- Abstract
Background: KIT and mitogen-activated protein kinase cascade are important for melanomagenesis. In the present study, we analyzed the frequency of BRAF, NRAS, KIT, GNAQ and GNA11 gene mutations and investigated their association with clinicopathological features of melanomas in Turkish population., Materials and Methods: Forty-seven primary cutaneous melanomas were included in our study. Sanger sequencing method was used for mutation analysis in all cases., Results: Mean age was 62.1 (29-101) years. Female:male ratio was 17:30. Among 47 melanomas, 14 (29.8%) BRAF, 10 (21.3%) NRAS, 4 (8.5%) KIT and 1(2.1%) GNAQ gene mutations were detected. Two of the KIT mutations were found in acral lentiginous melanoma (ALM). In the head and neck region, mutation frequency was significantly lower than in other locations (P = 0.035). The only GNAQ gene mutation (p.Q209L) was detected in a melanoma arising from blue nevus located on the scalp. None of the melanomas harbored NRAS exon 2, KIT exon 13/17/18, GNAQ exon 4 and GNA11 exon 4/5 mutations. Overall mutation frequency did not show significant difference between metastatic (8/14, 57.1%) and nonmetastatic (18/33, 54.5%) patients. We did not observe any significant association between mutation status and gender or age of various patients., Conclusions: Our results support that BRAF and NRAS gene mutations are common in cutaneous melanomas. The activating mutations of KIT gene are rare and especially seen in ALM. GNAQ and GNA11 mutations are infrequent in cutaneous melanomas and may be associated only with melanomas arising from blue nevus.
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- 2015
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31. Calcifying fibrous tumor: a case report.
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Gamsizkan M, Yildirim C, Daş K, and Günhan Ö
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- Biomarkers, Tumor analysis, Biopsy, Calcinosis metabolism, Calcinosis surgery, Collagen analysis, Fibroblasts chemistry, Humans, Immunohistochemistry, Magnetic Resonance Imaging, Male, Neoplasms, Fibrous Tissue chemistry, Neoplasms, Fibrous Tissue surgery, Soft Tissue Neoplasms chemistry, Soft Tissue Neoplasms surgery, Young Adult, Calcinosis pathology, Fibroblasts pathology, Neoplasms, Fibrous Tissue pathology, Soft Tissue Neoplasms pathology
- Abstract
Calcifying fibrous tumors are rarely seen and affect mostly children and young adults. A 21-year-old man presented with multiple palpable masses in the area from the right inguinal region to the anteromedial thigh. We performed a diagnostic excisional biopsy. Histopathologically, it was composed of fibroblasts, psammoma bodies, dystrophic calcifications and foci of mononuclear inflammatory cell infiltration in a collagenous dense stroma. We herein reported a case of calcifying fibrous tumor and discussed its clinical and morphological features with regard to the literature.
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- 2015
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32. An unusual infiltrative basal cell carcinoma with osteoclastic stromal changes mimicking carcinosarcoma: a case report.
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Gamsizkan M, Naujokas A, Simsek HA, and McCalmont TH
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- Aged, 80 and over, Biomarkers, Tumor analysis, Biopsy, Carcinoma, Basal Cell chemistry, Carcinoma, Basal Cell surgery, Cell Proliferation, Diagnosis, Differential, Eyelid Neoplasms chemistry, Eyelid Neoplasms surgery, Humans, Immunohistochemistry, Male, Mitotic Index, Osteoclasts chemistry, Predictive Value of Tests, Skin Neoplasms chemistry, Skin Neoplasms surgery, Stromal Cells chemistry, Carcinoma, Basal Cell pathology, Carcinosarcoma pathology, Eyelid Neoplasms pathology, Osteoclasts pathology, Skin Neoplasms pathology, Stromal Cells pathology
- Abstract
A 91-year-old man presented with an ulcerated nodule on his left lower eyelid. The tumor showed an epithelial component composed of basaloid and clear cells and a stroma that contained many osteoclastic giant cells. Strong, diffuse expression for cytokeratin 17 and p63 was noted in the epithelial component, whereas no staining was present in the sarcomatoid stroma, suggesting that the osteoclast-rich stromal component represented an unusual benign stromal reaction to the carcinoma rather than a manifestation of carcinosarcoma. Further supporting this interpretation was the absence of mitotic figures and low Ki-67 proliferation index (of approximately 1%) in the stromal cells. We herein reported a case of unusual infiltrative basal cell carcinoma, accompanied by a clear cell carcinomatous features and concurrent benign osteoclastic stromal changes.
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- 2015
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33. Taurine attenuates lung ischemia-reperfusion injury after lung transplantation in rats.
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Guler L, Tavlasoglu M, Yucel O, Guler A, Sahin MA, Kurkluoglu M, Sirin Y, Eken A, Gamsizkan M, Dakak M, Gurkok S, and Genc O
- Subjects
- Animals, Disease Models, Animal, Hypertonic Solutions, Lung blood supply, Lung metabolism, Lung Transplantation methods, Male, Malondialdehyde analysis, Malondialdehyde metabolism, Rats, Rats, Sprague-Dawley, Reperfusion Injury metabolism, Superoxide Dismutase analysis, Superoxide Dismutase metabolism, Antioxidants therapeutic use, Lung drug effects, Lung pathology, Reperfusion Injury drug therapy, Reperfusion Injury pathology, Taurine therapeutic use
- Abstract
Purpose: Taurine, the major intracellular free amino acid found in high concentrations in mammalian cells, is known to be an endogenous antioxidant and a membrane-stabilizing agent. It was hypothesized that taurine may be effective in reducing ischemia-reperfusion injury after lung transplantation and an experimental study was conducted in a rat model., Methods: The number of Sprague-Dawley rats used in the study was 35. Animals were randomized into five groups of 7 rats each, including control, donor I, donor II, ischemia-reperfusion injury, and treatment groups. All animals were exposed to the same experimental conditions in the preoperative period. Rats were fixed in a supine position after the induction. After the rats were shaved, a left pneumonectomy was performed following sternotomy in control, donor I, and donor II groups. The harvested grafts in donor I and donor II groups were transplanted to the rats of the ischemia-reperfusion group and treatment group, respectively. However, taurine was administered intraperitoneally for 3 days before the harvesting procedure in donor II. All harvested lungs were kept in a Euro-Collins solution at +4 °C for 24 h in a half-inflated manner. After harvesting and transplantation, lungs were sampled for histopathological and biochemical analysis., Results: Malondialdehyde and superoxide dismutase, glutathione peroxidase, and catalase levels were lower in the treatment group than the other groups (p < 0.05). Histopathological findings were better in treatment group than the ischemia-reperfusion group (p < 0.05)., Conclusion: It was demonstrated that donor treatment with taurine resulted in preservation of transplanted lung tissue in respect to histopathological and biochemical findings.
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- 2014
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34. Minor salivary gland neoplasms.
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Dalgic A, Karakoc O, Aydin U, Hidir Y, Gamsizkan M, Karahatay S, and Gerek M
- Subjects
- Adenoma, Pleomorphic epidemiology, Adolescent, Adult, Aged, Carcinoma, Adenoid Cystic epidemiology, Carcinoma, Mucoepidermoid epidemiology, Epidemiologic Studies, Female, Follow-Up Studies, Humans, Male, Middle Aged, Neoplasm Recurrence, Local epidemiology, Palatal Neoplasms epidemiology, Radiotherapy, Adjuvant statistics & numerical data, Retrospective Studies, Sex Factors, Turkey epidemiology, Young Adult, Salivary Gland Neoplasms epidemiology, Salivary Glands, Minor pathology
- Abstract
Objective: This study aimed to investigate the clinical presentation, histopathologic and epidemiological aspects, as well as the treatment modalities and outcomes of patients with minor salivary gland tumors (MSGTs)., Subjects and Methods: A series of 23 patients with MSGTs were reviewed retrospectively., Results: This study included 11 (48%) benign and 12 (52%) malignant tumors of minor salivary glands. Minor salivary gland tumors were more common in men (70%) than in women (30%). The mean age was 31.3 years for benign tumors and 46.3 years for malignant tumors. Pleomorphic adenoma was the most common benign tumor, followed by myoepithelioma. Mucoepidermoid carcinoma and adenoid cystic carcinoma were the most common malignant tumors. The most common symptom was a painless mass of the palate. Surgical treatment was performed in all patients. Adjuvant radiotherapy was used in 3 malignant tumors. Twenty-three patients were followed-up for a median of 5 years. Two patients with malignant tumors underwent a second surgery for postoperative local recurrence. They were successfully treated with the second surgery., Conclusions: Minor salivary gland tumors are relatively uncommon neoplasms of the head and neck region. There is limited literature on MSGTs. This study provides a versatile approach for MSGTs from demographic data and clinical presentations to treatment modalities and treatment outcomes.
- Published
- 2014
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35. A case of atypical scleromyxedema without gammopathy treated with cyclosporine.
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Açikgöz G, Özmen I, Hüseynov S, Gamsizkan M, Çaliskan E, Arca E, and Koç E
- Subjects
- Dermatologic Agents therapeutic use, Facial Dermatoses drug therapy, Facial Dermatoses pathology, Humans, Male, Paraproteinemias, Young Adult, Cyclosporine therapeutic use, Scleromyxedema drug therapy, Scleromyxedema pathology
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- 2014
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36. A retrospective multicenter evaluation of cutaneous melanomas in Turkey.
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Gamsizkan M, Yilmaz I, Buyukbabani N, Demirkesen C, Demiriz M, Cetin ED, Ince U, Akalin T, Demirkan NC, Lebe B, Erdem O, Gokoz O, Sakiz D, Demireli PT, Astarci HM, Adim SB, Zemheri IE, Acikalin A, Yaman B, Aydin O, and Bassorgun CI
- Subjects
- Adolescent, Adult, Aged, Aged, 80 and over, Child, Child, Preschool, Female, Humans, Male, Melanoma pathology, Middle Aged, Multivariate Analysis, Neoplasm Staging, Prognosis, Retrospective Studies, Sentinel Lymph Node Biopsy, Sex Factors, Skin Neoplasms pathology, Tumor Burden, Turkey epidemiology, Young Adult, Lymph Nodes pathology, Melanoma epidemiology, Skin Neoplasms epidemiology
- Abstract
Background: We defined melanoma distribution in a large series of Turkish patients and evaluated the prognostic parameters of melanomas., Materials and Methods: A total of 1574 patients' data was retrospectively collected at 18 centers in Turkey. Demographic characteristics were questioned and noted. Prognostic parametres were evaluated based on sentinel lymph node involvement., Results: Mean age was 56.7 (4-99) years. While 844 (53.6%) cases were male, 730 (46.4%) cases were female. One thousand four hundred forty-seven (92%) cases were invasive melanoma and 127 (8%) cases were in-situ melanoma. The most common histopathological form was the superficial spreading melanoma (SSM) which was found in 549 patients (37.9%). It was followed by nodular melanoma in 379 (26.2%), acral lentiginous melanoma (ALM) in 191 (13.2%) and lentigo maligna melanoma in 132 (9.1%), respectively. On univariate analysis, lymphovascular invasion (p<0.001), tumor thickness (p<0.001), histopathological subtype (p<0.001), Clark level (p=0.001), ulceration (p<0.001), ≥6/mm2 mitosis (p=0.005), satellite formation (p=0.001) and gender (p=0.03) were found to be associated with sentinel lymph node positivity. Regression was associated with sentinel lymph node negativity (p=0.017). According to multivariate analysis, lymphovascular invasion and tumor thickness were significant independent predictive factors of SLN positivity. Patient age, tumor localization, precursor lesions, lymphocytic infiltration and neurotropism were not related with sentinel lymph node involvement., Conclusions: In this retrospective analysis, it was found that the prevalence of SSM is at a lower rate while the prevalence of ALM is at a higher rate when compared to western countries. According to Breslow index; most of the melanoma lesions' thickness were greater than 2 mm, corresponding Clark IV. Vascular invasion and tumor thickness are the most important factors for sentinel lymph node involvement.
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- 2014
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37. Submandibular triangle masses.
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Dalgic A, Karakoc O, Karahatay S, Hidir Y, Gamsizkan M, Birkent H, and Gerek M
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- Adolescent, Adult, Aged, Aged, 80 and over, Biopsy, Fine-Needle, Child, Diagnosis, Differential, Diagnostic Imaging, Female, Humans, Lymphadenitis pathology, Lymphadenitis surgery, Male, Middle Aged, Sialadenitis pathology, Sialadenitis surgery, Submandibular Gland Neoplasms pathology, Submandibular Gland Neoplasms surgery, Lymphadenitis diagnosis, Sialadenitis diagnosis, Submandibular Gland Neoplasms diagnosis
- Abstract
Objective: The study aims to analyze the demographic data of a large case series operated on because of submandibular triangle mass for more than 10 years and presents a review of the literature., Materials and Methods: The charts of patients who underwent surgical intervention for submandibular triangle mass between January 2000 and November 2010 were reviewed. The medical history, age, sex, duration of symptoms, clinical presentation, preoperative investigations, and histopathologic diagnosis were reviewed., Results: The study included 66 subjects; 12 patients (18.2%) with submandibular sialolithiasis, 18 patients (27.2%) with sialadenitis, 10 patients with lymphadenitis (15.1%), and 26 patients (39.3%) with tumors. Of the tumors, 23% was malignant and 77% was benign. Benign tumors of submandibular gland comprised 22.7% and malign tumors of submandibular gland comprised 3% of all submandibular mass. The most common benign tumor was pleomorphic adenoma. The most frequent histopathologic diagnoses of submandibular masses originated from the submandibular gland, and these comprised 71.2% of all submandibular mass pathologies. The main symptom was a painless mass. Ultrasonography was the most common preoperative diagnostic procedure. Fine-needle aspiration biopsy was performed in 26 patients. A clear diagnosis could not be provided in 3 (12%) patients., Conclusion: Infectious conditions and benign tumors are more frequent than malign tumors in the submandibular region. The histopathologic diagnoses mainly consisted of submandibular sialadenitis, sialolithiasis, pleomorphic adenoma, and lymphadenitis. Ultrasonography is the first option of radiologic evaluation. Fine-needle aspiration biopsy is a very useful and usually sufficient diagnostic procedure for histopathologic diagnosis. Excisional biopsy can be performed when the fine-needle aspiration biopsy failed.
- Published
- 2013
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38. The effects of hyperbaric oxygen therapy on experimental colon anastomosis after preoperative chemoradiotherapy.
- Author
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Yildiz R, Can MF, Yagci G, Ozgurtas T, Guden M, Gamsizkan M, Ozturk E, and Cetiner S
- Subjects
- Anastomosis, Surgical, Animals, Colectomy, Colon pathology, Female, Random Allocation, Rats, Rats, Wistar, Anastomotic Leak prevention & control, Chemoradiotherapy, Adjuvant adverse effects, Colon surgery, Hyperbaric Oxygenation, Neoadjuvant Therapy adverse effects, Postoperative Care methods, Wound Healing drug effects, Wound Healing radiation effects
- Abstract
The aim of the present study was to investigate the effect of hyperbaric oxygen therapy (HBOT) on colon anastomosis after chemoradiotherapy (CRT). Sixty female Wistar-Albino rats were divided into 5 groups and underwent left colon resection and end-to-end anastomosis. CRT simulation was performed on 2 sham groups before the anastomosis, and 1 of these groups was administered additional postoperative HBOT. Two groups were administered CRT before the anastomosis, and 1 of them received additional postoperative HBOT. On postoperative day 5, all groups underwent relaparotomy; burst pressure was measured and samples were obtained for histopathologic and biochemical analysis. There was a significant weight loss in the CRT groups and postoperative HBOT had an improving effect. Significantly decreased burst pressure values increased up to the levels of the controls after HBOT. Hydroxyproline levels were elevated in all groups compared to the control group. Hydroxyproline levels decreased with HBOT after CRT. No significant difference was observed between the groups regarding fibrosis formation at the anastomosis site. However, regression was observed in fibrosis in the group receiving HBOT after CRT. Preoperative CRT affected anastomosis and wound healing unfavorably. These unfavorable effects were alleviated by postoperative HBOT. HBOT improved the mechanical and biochemical parameters of colon anastomosis in rats.
- Published
- 2013
- Full Text
- View/download PDF
39. Proanthocyanidin prevents myocardial ischemic injury in adult rats.
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Guler A, Sahin MA, Yucel O, Yokusoglu M, Gamsizkan M, Ozal E, Demirkilic U, and Arslan M
- Subjects
- Animals, Catalase metabolism, Glutathione Peroxidase metabolism, Heart Ventricles pathology, Malondialdehyde metabolism, Oxidative Stress physiology, Rats, Statistics, Nonparametric, Superoxide Dismutase metabolism, Grape Seed Extract pharmacology, Myocardial Reperfusion Injury prevention & control, Oxidative Stress drug effects, Proanthocyanidins pharmacology
- Abstract
Background: Proanthocyanidin is a bioflavonoid known to have protective effect against oxidative injury. We investigated the cardioprotective effect of proanthocyanidin., Material/methods: Thirty-two Rattus Norvegicus rats were categorized equally as the control group (CG), proanthocyanidin group (PCG), ischemia group (IG) and proanthocyanidin-treated group (PCT). Rats in CG and IG were fed standard rat food and PCG and PCT were fed standard rat food plus proanthocyanidin (100 mg/kg/day twice a day by oral gavage) for 3 weeks. In CG and PCG the myocardial samples were prepared immediately, and in IG and PCT hearts were placed in transport solution and kept at 4°C for 5 hours, then prepared for evaluation. Malondialdehyde (MDA) level, superoxide dismutase (SOD), catalase (CAT) and glutathione peroxidase (GPx) activities were measured., Results: MDA levels were significantly higher in IG and PCT than in CG and PCG. The activity of SOD was significantly lower in IG and higher in PCG than in the other groups. The activity of GPx was significantly lower in IG than in the other groups. The activities of CAT were significantly lower in IG and PCT than in the other groups and were significantly lower in IG than PCT. Histopathologic evaluation revealed normal findings in CG and PCG. While ischemic injury was observed in IG, the content of muscle fibers was better preserved in PCT., Conclusions: Proanthocyanidin may have a protective effect on myocardial ischemic injury.
- Published
- 2011
- Full Text
- View/download PDF
40. Periosteal chondroma that presented as a subcutaneous mass in the ring finger.
- Author
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Yildirim C, Unay K, Rodop O, and Gamsizkan M
- Subjects
- Biopsy, Needle, Bone Neoplasms diagnosis, Bone Neoplasms surgery, Chondroma diagnosis, Chondroma surgery, Diagnosis, Differential, Edema diagnosis, Edema etiology, Fingers pathology, Fingers surgery, Follow-Up Studies, Humans, Immunohistochemistry, Magnetic Resonance Imaging methods, Male, Pain diagnosis, Pain etiology, Photomicrography, Skin Neoplasms diagnosis, Tomography, X-Ray Computed methods, Treatment Outcome, Young Adult, Bone Neoplasms pathology, Chondroma pathology, Periosteum pathology, Skin Neoplasms pathology
- Abstract
We present an unusual case of periosteal chondroma of the middle phalanx in the right ring finger that presented as a subcutaneous mass in a 21-year-old man. It was treated by marginal excision, with no postoperative local recurrence.
- Published
- 2011
- Full Text
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41. Is there any cardioprotective role of Taurine during cold ischemic period following global myocardial ischemia?
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Sahin MA, Yucel O, Guler A, Doganci S, Jahollari A, Cingoz F, Arslan S, Gamsizkan M, Yaman H, and Demirkilic U
- Subjects
- Animals, Disease Models, Animal, Dose-Response Relationship, Drug, Ischemic Preconditioning, Myocardial, Lipid Peroxidation, Male, Myocardial Reperfusion Injury metabolism, Myocardial Reperfusion Injury physiopathology, Myocardium pathology, Rats, Spectrophotometry, Taurine administration & dosage, Treatment Outcome, Catalase metabolism, Glutathione Peroxidase metabolism, Myocardial Reperfusion Injury prevention & control, Myocardium enzymology, Superoxide Dismutase metabolism, Taurine therapeutic use
- Abstract
Background: The aim of the present study was to investigate the cardioprotective effect of Taurine on the donor hearts during cold ischemic period., Methods: 32 rats were divided into four groups (sham, taurine, ischemia, treatment group, 8 rats in each). All rats were fed with rat food for three weeks. Taurine and treatment groups were given a 200 mg/kg/day dose of Taurine by oral gavage besides rat feed. Cardiectomy was performed in all rats after three weeks. In ischemia and treatment groups, harvested hearts were kept in 0.9% sodium chloride at +4 degrees C for 5 hours. Tissue samples were taken from left ventricle in all groups. These samples were evaluated by histopathologic and biochemical examination., Results: In the present study results of the biochemical and histopathological examination reveals the protective effects of Taurine. As a marker of lipid peroxidation, Malondialdehyde (MDA) levels in ischemia group were significantly higher than both Sham and Taurine groups. MDA values were recorded; 3.62 ± 0.197 in the sham group, 2.07 ± 0.751 in the Taurine group, 9.71 ± 1.439 in the ischemia group and 7.68 ± 1.365 in the treatment group. MDA levels decreased in treatment group. (p < 0.05) In accordance with MDA findings, while superoxide dismutase and glutathione peroxidase levels decreased in ischemia group, they increased in treatment group. (p < 0.05) There was no differences in Catalase (CAT) enzyme level between treatment and ischemia group (p = 1.000). CAT level results were recorded; 7.08 ± 0.609 in the sham group, 6.15 ± 0.119 in the Taurine group, 5.02 ± 0.62 in the ischemia group, and 5.36 ± 0.384 in the treatment group. Less intracellular edema and inflammatory cell reaction were observed in histologic examination in favor of treatment group. (p < 0.01), Conclusion: Taurine decreased myocardial damage during cold ischemic period following global myocardial ischemia.
- Published
- 2011
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42. The role of oxidative stress and effect of alpha-lipoic acid in reexpansion pulmonary edema - an experimental study.
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Gumus S, Yucel O, Gamsizkan M, Eken A, Deniz O, Tozkoparan E, Genc O, and Bilgic H
- Abstract
Introduction: We investigated the role of oxidative stress in the pathogenesis of reexpansion pulmonary edema (RPE) and effect of alpha-lipoic acid (ALA) in the prevention of RPE., Material and Methods: There were 4 groups consisting of 10 rats in each group; control group (CG), α-lipoic acid group (ALAG), reexpansion pulmonary edema group (RPEG), reexpansion pulmonary edema plus α-lipoic acid group (RPE + ALAG). In all the groups, all rats were sacrificed 2 hours after the reexpansion of lungs. To indicate oxidative stress malondialdehyde (MDA), and to indicate antioxidant status superoxide dismutase (SOD), catalase (CAT) and glutathione peroxides (GPx) were measured in the lungs of rats., Results: Mean MDA value was lower in CG (7.02 ±0.14) and in ALAG (6.95 ±0.11) than the other groups (p = 0.001). It was highest in RPEG (8.89 ±0.21) (p = 0.001). It was lower in RPE + ALA G (7.21 ±0.32) than RPEG (p = 0.001). Antioxidant levels: GPx (37.21 ±3.01), CAT (2.87 ±0.14) and SOD (100.12 ±12.39) were lowest in RPEG among all groups (p = 0.001). These values were GPx (45.21 ±3.54), CAT (3.24 ±0.21) and SOD (172.36 ±15.48) in RPE + ALA G and were greater than those of RPEG (p = 0.001). While normal pulmonary parenchyma was seen in 2 rats in RPE + ALAG, it was not seen in RPEG. Pulmonary edema was seen in 1 rat in RPE + ALAG; however, it was seen in 3 in RPEG., Conclusions: Oxidative stress might have an important role in the pathogenesis of RPE. In addition, ALA treatment might contribute in preventing RPE.
- Published
- 2010
- Full Text
- View/download PDF
43. Primary dural lymphoma mimicking a subdural hematoma.
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Gocmen S, Gamsizkan M, Onguru O, Sefali M, and Erdogan E
- Subjects
- Female, Humans, Magnetic Resonance Imaging methods, Middle Aged, Dura Mater pathology, Hematoma, Subdural physiopathology, Lymphoma, Non-Hodgkin diagnosis, Meningeal Neoplasms diagnosis
- Abstract
Intracranial marginal zone B-cell lymphoma presenting as a dural-based mass is rare. A 45-year-old woman who had generalized tonic-clonic seizures and speech disturbance for 6 months was referred to our hospital. Radiology suggested a subdural hematoma (SDH). No improvement in the radiological findings or symptoms occurred with conservative follow-up. Therefore, she underwent a craniotomy for drainage of the suspected SDH. Intraoperatively, dural plaque-like thickening was observed, with no SDH, and a biopsy was performed. After histopathological and immunohistochemical studies, a mucosa-associated lymphoid tissue (MALT) lymphoma was diagnosed. The patient underwent radiotherapy with no postoperative complications or recurrence. Early diagnosis and treatment of primary dural lymphoma is important. Histopathological evaluation is necessary for diagnosis. MRI cannot reliably differentiate between SDH and some dural lesions that present as diffuse infiltration. Therefore, these differential diagnoses should be considered., (Copyright 2009 Elsevier Ltd. All rights reserved.)
- Published
- 2010
- Full Text
- View/download PDF
44. Do zinc and selenium prevent the antioxidant, hepatic and renal system impairment caused by aspirin in rats?
- Author
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Kesik V, Lenk MK, Kurekci AE, Acikel CH, Akgul EO, Aydin A, Erdem O, and Gamsizkan M
- Subjects
- Animals, Glutathione Peroxidase metabolism, Kidney metabolism, Kidney physiopathology, Liver enzymology, Liver metabolism, Liver physiopathology, Male, Malondialdehyde metabolism, Rats, Rats, Wistar, Superoxide Dismutase metabolism, Antioxidants metabolism, Aspirin adverse effects, Kidney drug effects, Liver drug effects, Selenium administration & dosage, Zinc administration & dosage
- Abstract
Aspirin is widely used as an antiinflammatory drug especially in children with rheumatic fever arthritis. The diminishing effects of aspirin on antioxidant enzymes and hepato-renal systems at high doses are well-known. It is now evident that the damage at antioxidant system worsens the clinical picture of the disease and prolongs the treatment time. Thus, we investigated the effect of antioxidant enzyme cofactors-zinc and selenium-supplementation on superoxide dismutase (SOD), glutathione peroxidase (GSH-Px), and malondialdehyde (MDA) levels (erythrocyte and liver) and hepato-renal toxicity during aspirin treatment at therapeutic doses. The rats were divided into five groups. The first and second groups were given aspirin 75 mg/kg/day and aspirin plus selenium (Selenium 200, selenium 200 mg tablet as selenium yeast, GNC) and zinc (Zinc 100, zinc 100 mg tablet as zinc gluconate, GNC), respectively, the third and fourth take 50 mg/kg/day aspirin and aspirin plus selenium and zinc twice a day, respectively. The fifth group was control. The rats were treated with aspirin for 5 weeks as in the treatment of rheumatic fever arthritis in children. Erythrocyte SOD and MDA levels were preserved with supplementation, whereas there was no change for GSH-Px levels. Liver SOD, GSH-Px, and MDA levels were not changed. In zinc- and selenium-supplemented groups, the levels of serum alanine aminotransferase, uric acid, and direct bilirubin levels were found statistically decreased compared with nonsupplemented groups. There was no significant histopathologic change in specimens of hepatic and renal tissues. Trace element supplementation may prevent free radical damage and shorten treatment time in children using long-term aspirin treatment.
- Published
- 2008
- Full Text
- View/download PDF
45. Cyclooxygenase-2 (Cox-2) expression and angiogenesis in glioblastoma.
- Author
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Onguru O, Gamsizkan M, Ulutin C, and Gunhan O
- Subjects
- Adult, Aged, Brain Neoplasms blood supply, Female, Glioblastoma blood supply, Glioblastoma pathology, Humans, Immunohistochemistry, Kaplan-Meier Estimate, Male, Middle Aged, Prognosis, Brain Neoplasms enzymology, Brain Neoplasms pathology, Cyclooxygenase 2 biosynthesis, Glioblastoma enzymology, Neovascularization, Pathologic pathology
- Abstract
Cyclooxygenase-2 (Cox-2), the key enzyme that catalyzes the first steps in the biosynthesis of the prostaglandins from arachidonic acid, appears to play a role in the regulation of progression, invasiveness and angiogenesis of various neoplasms. We analyzed the immunohistochemical expression of Cox-2 and angiogenic parameters (microvessel density (MVD) and vascular patterns) in 54 glioblastomas. We also examined their relation with prognosis. Cox-2 immunohistochemical expression was observed in 48 tumors (89%). There was no staining in six tumors (11%). On univariate analysis, MVD was correlated with a poor outcome (MVD > 70; hazard ratio, 0.441; 95% confidence interval, 0.200-0.975, P = 0.041). But MVD showed no prognostic impact on multivariate analysis. Neither Cox-2 expression nor vascular pattern showed prognostic value. The difference in Cox-2 expression between the classical and bizarre vascular pattern in glioblastomas was statistically significant (P = 0.047). However, no correlation was found between Cox-2 expression and MVD. These findings suggest that Cox-2 is heterogeneously expressed in glioblastomas without a significant association with MVD. However, Cox-2 expression may be related to vascular pattern in glioblastomas.
- Published
- 2008
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46. Comparative morphometric analysis of intraepidermal and dermal lymphocytes in early mycosis fungoides and some inflammatory dermatoses.
- Author
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Gamsizkan M, Demiriz M, Karslioğlu Y, and Günhan O
- Subjects
- Adult, Aged, Dermis anatomy & histology, Epidermis anatomy & histology, Female, Humans, Inflammation, Male, Middle Aged, Dermis pathology, Epidermis pathology, Lymphocytes pathology, Mycosis Fungoides pathology, Skin Diseases pathology, Skin Neoplasms pathology
- Abstract
Objective: Early stage mycosis fungoides (MF) may be indistinguishable from some inflammatory dermatoses (IDs) histopathologically. Various morphologic features of lymphocytes in epidermis and dermis may help to differentiate early-stage MF from other IDs., Study Design: We studied 25 cases of early-stage MF and 30 cases of various IDs with prominent exocytosis. The following nuclear variables were measured on dermal and epidermal lymphocytes in MF and IDs (control) groups: nuclear area and perimeter, area convex hull, perimeter convex hull, diameter equivalent circle, minimum feret, maximum feret and form factor. All measurements were done using routinely prepared and hematoxylin-eosin-stained slides., Results: There were statistically significant differences (p < 0.01) for all of the morphometric variables between early-stage MF and IDs., Conclusion: Our results revealed that the nuclear contour irregularities of intraepidermal lymphocytes are higher than those of dermal lymphocytes in MF. Their nuclear geometry is also different. Comparison of the nuclear features of intraepidermal with dermal lymphocytes may provide additional support in the diagnosis of early-stage MF.
- Published
- 2007
47. Nephrogenic adenoma of the bladder in a girl with Turner's syndrome: an unusual association.
- Author
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Kibar Y, Sümer F, Yildirim I, Gamsizkan M, Avci A, and Dayanç M
- Subjects
- Adenoma pathology, Adolescent, Female, Humans, Medical Records, Metaplasia, Urinary Bladder Neoplasms pathology, Vesico-Ureteral Reflux surgery, Adenoma complications, Turner Syndrome complications, Urinary Bladder Neoplasms complications
- Abstract
We describe a nephrogenic adenoma of the bladder in a 14-year-old girl with Turner's syndrome. The patient also had a past history of urological surgery for vesicoureteral reflux. In this case, the multifocal lesions were successfully treated by transurethral resection.
- Published
- 2004
- Full Text
- View/download PDF
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