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1,200 results on '"Gammon, Marilie D."'

1. Prediagnosis aspirin use, DNA methylation, and mortality after breast cancer: A population‐based study

Author

Wang, Tengteng, McCullough, Lauren E, White, Alexandra J, Bradshaw, Patrick T, Xu, Xinran, Cho, Yoon Hee, Terry, Mary Beth, Teitelbaum, Susan L, Neugut, Alfred I, Santella, Regina M, Chen, Jia, and Gammon, Marilie D

Subjects
Health Services and Systems, Biomedical and Clinical Sciences, Health Sciences, Oncology and Carcinogenesis, Genetics, Cancer, Breast Cancer, Prevention, Good Health and Well Being, Adult, Aged, Anti-Inflammatory Agents, Non-Steroidal, Aspirin, BRCA1 Protein, Breast Neoplasms, Cause of Death, Cohort Studies, DNA Methylation, Epigenesis, Genetic, Female, Genetic Predisposition to Disease, Humans, Middle Aged, Population Surveillance, Prognosis, Promoter Regions, Genetic, aspirin, breast cancer, DNA methylation, epigenetic, mortality, Public Health and Health Services, Oncology & Carcinogenesis, Oncology and carcinogenesis, Public health
Abstract

BackgroundThe authors hypothesized that epigenetic changes may help to clarify the underlying biologic mechanism linking aspirin use to breast cancer prognosis. To the authors' knowledge, this is the first epidemiologic study to examine whether global methylation and/or tumor promoter methylation of breast cancer-related genes interact with aspirin use to impact mortality after breast cancer.MethodsPrediagnosis aspirin use was assessed through in-person interviews within a population-based cohort of 1508 women diagnosed with a first primary breast cancer in 1996 and 1997. Global methylation in peripheral blood was assessed by long interspersed elements-1 (LINE-1) and the luminometric methylation assay. Promoter methylation of 13 breast cancer-related genes was measured in tumor by methylation-specific polymerase chain reaction and the MethyLight assay. Vital status was determined by the National Death Index through December 31, 2014 (N = 202/476 breast cancer-specific/all-cause deaths identified among 1266 women with any methylation assessment and complete aspirin data). Cox proportional hazards regression was used to estimate hazard ratios (HRs) and 95% CIs, and the likelihood ratio test was used to evaluate multiplicative interactions.ResultsAll-cause mortality was elevated among aspirin users who had methylated promotor of BRCA1 (HR, 1.67; 95% CI, 1.26-2.22), but not among those with unmethylated promoter of BRCA1 (HR, 0.99; 95% CI, 0.67-1.45; P for interaction ≤.05). Decreased breast cancer-specific mortality was observed among aspirin users who had unmethylated promotor of BRCA1 and PR and global hypermethylation of LINE-1 (HR, 0.60, 0.78, and 0.63, respectively; P for interaction ≤.05), although the 95% CIs included the null.ConclusionsThe current study suggests that the LINE-1 global methylation and promoter methylation of BRCA1 and PR in tumor may interact with aspirin use to influence mortality after breast cancer.

Published
2019

2. Genetic polymorphisms of diabetes‐related genes, their interaction with diabetes status, and breast cancer incidence and mortality: The Long Island Breast Cancer Study Project

Author

Parada, Humberto, Cleveland, Rebecca J, North, Kari E, Stevens, June, Teitelbaum, Susan L, Neugut, Alfred I, Santella, Regina M, Martinez, Maria E, and Gammon, Marilie D

Subjects
Aging, Diabetes, Human Genome, Cancer, Genetics, Prevention, Breast Cancer, Aetiology, 2.1 Biological and endogenous factors, Good Health and Well Being, Adult, Aged, Aged, 80 and over, Biomarkers, Breast Neoplasms, Case-Control Studies, Diabetes Mellitus, Female, Follow-Up Studies, Genetic Predisposition to Disease, Genome-Wide Association Study, Genotype, Humans, Incidence, Middle Aged, Polymorphism, Single Nucleotide, Prognosis, Survival Rate, Young Adult, breast cancer, diabetes, genetics, incidence, mortality, single nucleotide polymorphisms, survival, Oncology and Carcinogenesis, Oncology & Carcinogenesis
Abstract

To examine 143 diabetes risk single nucleotide polymorphisms (SNPs), identified from genome-wide association studies, in association with breast cancer (BC) incidence and subsequent mortality. A population-based sample of Caucasian women with first primary invasive BC (n = 817) and controls (n = 1021) were interviewed to assess diabetes status. Using the National Death Index, women with BC were followed for >18 years during which 340 deaths occurred (139 BC deaths). Genotyping was done using DNA extracted from blood samples. We used unconditional logistic regression to estimate age-adjusted odds ratios and 95% confidence intervals (CIs) for BC incidence, and Cox regression to estimate age-adjusted hazard ratios and CIs for all-cause and BC-specific mortality. Twelve SNPs were associated with BC risk in additive genotype models, at α = 0.05. The top three significant SNPs included SLC30A8-rs4876369 (P = 0.0034), HHEX-rs11187146 (P = 0.0086), and CDKN2A/CDKN2B-rs1333049 (P = 0.0094). Diabetes status modified the associations between rs4876369 and rs2241745 and BC incidence, on the multiplicative interaction scale. Six SNPs were associated with all-cause (CDKAL1-rs981042, P = 0.0032; HHEX-rs1111875, P = 0.0361; and INSR-rs919275, P = 0.0488) or BC-specific (CDKN2A/CDKN2B-rs3218020, P = 0.0225; CDKAL1-rs981042, P = 0.0246; and TCF2/HNF1B-rs3094508, P = 0.0344) mortality in additive genotype models, at α = 0.05. Genetic polymorphisms that increase the risk of developing diabetes may also increase the risk of developing and dying from BC.

Published
2019

5. Grilled, Barbecued, and Smoked Meat Intake and Survival Following Breast Cancer.

Author

Parada, Humberto, Steck, Susan E, Bradshaw, Patrick T, Engel, Lawrence S, Conway, Kathleen, Teitelbaum, Susan L, Neugut, Alfred I, Santella, Regina M, and Gammon, Marilie D

Subjects
Biomedical and Clinical Sciences, Oncology and Carcinogenesis, Cancer, Breast Cancer, Good Health and Well Being, Aged, Animals, Breast Neoplasms, Cattle, Cause of Death, Cooking, Diet, Female, Follow-Up Studies, Humans, Meat, Middle Aged, New York, Polycyclic Aromatic Hydrocarbons, Poultry, Seafood, Sheep, Survival Rate, Swine, Oncology & Carcinogenesis, Oncology and carcinogenesis
Abstract

Grilled, barbecued, and smoked meat intake, a prevalent dietary source of polycyclic aromatic hydrocarbon (PAH) carcinogens, may increase the risk of incident breast cancer. However, no studies have examined whether intake of this PAH source influences survival after breast cancer. We interviewed a population-based cohort of 1508 women diagnosed with first primary invasive or in situ breast cancer in 1996 and 1997 at baseline and again approximately five years later to assess grilled/barbecued and smoked meat intake. After a median of 17.6 years of follow-up, 597 deaths, of which 237 were breast cancer related, were identified. Multivariable Cox regression was used to estimate adjusted hazard ratios (HRs) and 95% confidence intervals (CIs) for mortality as related to prediagnosis intake, comparing high (above the median) to low intake, as well as postdiagnosis changes in intake, comparing every combination of pre-/postdiagnosis intake to low pre-/postdiagnosis intake. All statistical tests were two-sided. High prediagnosis grilled/barbecued and smoked meat intake was associated with increased risk of all-cause mortality (HR = 1.23, 95% CI = 1.03 to 1.46). Other associations were noted, but estimates were not statistically significant. These include high prediagnosis smoked beef/lamb/pork intake and increased all-cause (HR = 1.17, 95% CI = 0.99 to 1.38, Ptrend = .10) and breast cancer-specific (HR = 1.23, 95% CI = 0.95 to 1.60, Ptrend = .09) mortality. Also, among women with continued high grilled/barbecued and smoked meat intake after diagnosis, all-cause mortality risk was elevated 31% (HR = 1.31, 95% CI = 0.96 to 1.78). Further, breast cancer-specific mortality was decreased among women with any pre- and postdiagnosis intake of smoked poultry/fish (HR = 0.55, 95% CI = 0.31 to 0.97). High intake of grilled/barbecued and smoked meat may increase mortality after breast cancer.

Published
2017

6. Association Between Levels of Sex Hormones and Risk of Esophageal Adenocarcinoma and Barrett’s Esophagus

Author

Xie, Shao-Hua, Fang, Rui, Huang, Mingtao, Dai, Juncheng, Thrift, Aaron P., Anderson, Lesley A., Chow, Wong-Ho, Bernstein, Leslie, Gammon, Marilie D., Risch, Harvey A., Shaheen, Nicholas J., Reid, Brian J., Wu, Anna H., Iyer, Prasad G., Liu, Geoffrey, Corley, Douglas A., Whiteman, David C., Caldas, Carlos, Pharoah, Paul D., Hardie, Laura J., Fitzgerald, Rebecca C., Shen, Hongbing, Vaughan, Thomas L., and Lagergren, Jesper

Published
2020
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7. Identification of four novel susceptibility loci for oestrogen receptor negative breast cancer.

Author

Couch, Fergus J, Kuchenbaecker, Karoline B, Michailidou, Kyriaki, Mendoza-Fandino, Gustavo A, Nord, Silje, Lilyquist, Janna, Olswold, Curtis, Hallberg, Emily, Agata, Simona, Ahsan, Habibul, Aittomäki, Kristiina, Ambrosone, Christine, Andrulis, Irene L, Anton-Culver, Hoda, Arndt, Volker, Arun, Banu K, Arver, Brita, Barile, Monica, Barkardottir, Rosa B, Barrowdale, Daniel, Beckmann, Lars, Beckmann, Matthias W, Benitez, Javier, Blank, Stephanie V, Blomqvist, Carl, Bogdanova, Natalia V, Bojesen, Stig E, Bolla, Manjeet K, Bonanni, Bernardo, Brauch, Hiltrud, Brenner, Hermann, Burwinkel, Barbara, Buys, Saundra S, Caldes, Trinidad, Caligo, Maria A, Canzian, Federico, Carpenter, Jane, Chang-Claude, Jenny, Chanock, Stephen J, Chung, Wendy K, Claes, Kathleen BM, Cox, Angela, Cross, Simon S, Cunningham, Julie M, Czene, Kamila, Daly, Mary B, Damiola, Francesca, Darabi, Hatef, de la Hoya, Miguel, Devilee, Peter, Diez, Orland, Ding, Yuan C, Dolcetti, Riccardo, Domchek, Susan M, Dorfling, Cecilia M, Dos-Santos-Silva, Isabel, Dumont, Martine, Dunning, Alison M, Eccles, Diana M, Ehrencrona, Hans, Ekici, Arif B, Eliassen, Heather, Ellis, Steve, Fasching, Peter A, Figueroa, Jonine, Flesch-Janys, Dieter, Försti, Asta, Fostira, Florentia, Foulkes, William D, Friebel, Tara, Friedman, Eitan, Frost, Debra, Gabrielson, Marike, Gammon, Marilie D, Ganz, Patricia A, Gapstur, Susan M, Garber, Judy, Gaudet, Mia M, Gayther, Simon A, Gerdes, Anne-Marie, Ghoussaini, Maya, Giles, Graham G, Glendon, Gord, Godwin, Andrew K, Goldberg, Mark S, Goldgar, David E, González-Neira, Anna, Greene, Mark H, Gronwald, Jacek, Guénel, Pascal, Gunter, Marc, Haeberle, Lothar, Haiman, Christopher A, Hamann, Ute, Hansen, Thomas VO, Hart, Steven, Healey, Sue, Heikkinen, Tuomas, Henderson, Brian E, and Herzog, Josef

Subjects
Chromosomes, Human, Pair 2, Humans, Breast Neoplasms, Genetic Predisposition to Disease, Cyclophilins, tRNA Methyltransferases, BRCA1 Protein, Receptors, Estrogen, Risk Factors, Genotype, Heterozygote, Mutation, Polymorphism, Single Nucleotide, Female, Genome-Wide Association Study, Chromosomes, Human, Pair 2, Receptors, Estrogen, Polymorphism, Single Nucleotide, Breast Cancer, Cancer, Prevention, Genetics, Human Genome, 2.1 Biological and endogenous factors
Abstract

Common variants in 94 loci have been associated with breast cancer including 15 loci with genome-wide significant associations (P

Published
2016

8. DNA methylation modifies the association between obesity and survival after breast cancer diagnosis

Author

McCullough, Lauren E, Chen, Jia, Cho, Yoon Hee, Khankari, Nikhil K, Bradshaw, Patrick T, White, Alexandra J, Garbowski, Gail, Teitelbaum, Susan L, Terry, Mary Beth, Neugut, Alfred I, Hibshoosh, Hanina, Santella, Regina M, and Gammon, Marilie D

Subjects
Biomedical and Clinical Sciences, Oncology and Carcinogenesis, Breast Cancer, Prevention, Obesity, Cancer, Nutrition, Clinical Research, Genetics, Detection, screening and diagnosis, 4.1 Discovery and preclinical testing of markers and technologies, Good Health and Well Being, Adenomatous Polyposis Coli Protein, Body Mass Index, Breast Neoplasms, DNA Methylation, Female, Humans, Nuclear Proteins, Prognosis, Promoter Regions, Genetic, Proportional Hazards Models, Survival Analysis, Twist-Related Protein 1, Body mass index, Epigenetics, Methylation, Breast cancer, Survival, Clinical Sciences, Oncology & Carcinogenesis, Clinical sciences, Oncology and carcinogenesis
Abstract

Mechanisms underlying the poor breast cancer prognosis among obese women are unresolved. DNA methylation levels are linked to obesity and to breast cancer survival. We hypothesized that obesity may work in conjunction with the epigenome to alter prognosis. Using a population-based sample of women diagnosed with first primary breast cancer, we examined modification of the obesity-mortality association by DNA methylation. In-person interviews were conducted approximately 3 months after diagnosis. Weight and height were assessed [to estimate body mass index (BMI)], and blood samples collected. Promoter methylation of 13 breast cancer-related genes was assessed in archived tumor by methylation-specific PCR and Methyl Light. Global methylation in white blood cell DNA was assessed by analysis of long interspersed elements-1 (LINE-1) and with the luminometric methylation assay (LUMA). Vital status among 1308 patients (with any methylation biomarker and complete BMI assessment) was determined after approximately 15 years of follow-up (N = 194/441 deaths due to breast cancer-specific/all-cause mortality). We used Cox proportional hazards regression to estimate hazard ratios (HRs) and 95 % confidence intervals (CIs) using two-sided p values of 0.05. Breast cancer-specific mortality was higher among obese (BMI ≥ 30) patients with promoter methylation in APC (HR = 2.47; 95 % CI = 1.43-4.27) and TWIST1 (HR = 4.25; 95 % CI = 1.43-12.70) in breast cancer tissue. Estimates were similar, but less pronounced, for all-cause mortality. Increased all-cause (HR = 1.81; 95 % CI = 1.19-2.74) and breast cancer-specific (HR = 2.61; 95 % CI = 1.45-4.69) mortality was observed among obese patients with the lowest LUMA levels. The poor breast cancer prognosis associated with obesity may depend on methylation profiles, which warrants further investigation.

Published
2016

9. Cardiovascular Disease Mortality Among Breast Cancer Survivors

Author

Bradshaw, Patrick T, Stevens, June, Khankari, Nikhil, Teitelbaum, Susan L, Neugut, Alfred I, and Gammon, Marilie D

Subjects
Prevention, Aging, Cancer, Breast Cancer, Heart Disease, Rehabilitation, Cardiovascular, Good Health and Well Being, Adult, Aged, Aged, 80 and over, Breast Neoplasms, Cardiovascular Diseases, Case-Control Studies, Female, Follow-Up Studies, Humans, Kaplan-Meier Estimate, Middle Aged, New York, Proportional Hazards Models, Risk Factors, Survivors, Statistics, Public Health and Health Services, Epidemiology
Abstract

BackgroundCardiovascular disease (CVD) is of increasing concern among breast cancer survivors. However, the burden of this comorbidity in this group relative to the general population, and its temporal pattern, remains unknown.MethodsWe compared deaths due to CVD in a population-based sample of 1,413 women with incident breast cancer diagnosed in 1996-1997, and 1,411 age-matched women without breast cancer. Date and cause of death through December 31, 2009 were assessed through the national death index and covariate data was gathered through structured interviews and medical record abstraction. Hazard ratios (HR) and 95% confidence intervals were calculated using Cox regression for overall mortality (HR) and CVD-specific death (cause-specific HR). Subdistribution HRs for CVD death were estimated from the Fine-Gray model.ResultsRisk of death was greater among breast cancer survivors compared with women without breast cancer (HR: 1.8 [1.5, 2.1]). An increase in CVD-related death among breast cancer survivors was evident only 7 years after diagnosis (years 0-7, cause-specific HR: 0.80 [0.53, 1.2], subdistribution HR: 0.59 [0.40, 0.87]); years 7+, cause-specific HR: 1.8 [1.3, 2.5], subdistribution HR: 1.9 [1.4, 2.7]; P interaction: 0.001). An increase in CVD-related mortality was observed among breast cancer survivors receiving chemotherapy.ConclusionsBreast cancer survivors are at greater risk for CVD-related mortality compared with women without breast cancer and this increase in risk is manifested approximately 7 years after diagnosis. Efforts should be made to identify risk factors and interventions that can be employed during this brief window to reduce the excess burden of CVD in this vulnerable population.

Published
2016

10. Latent class analysis suggests four distinct classes of complementary medicine users among women with breast cancer

Author

Strizich, Garrett, Gammon, Marilie D, Jacobson, Judith S, Wall, Melanie, Abrahamson, Page, Bradshaw, Patrick T, Terry, Mary Beth, Teitelbaum, Susan, Neugut, Alfred I, and Greenlee, Heather

Subjects
Health Sciences, Traditional, Complementary and Integrative Medicine, Cancer, Clinical Trials and Supportive Activities, Mind and Body, Prevention, Nutrition, Breast Cancer, Complementary and Integrative Health, Clinical Research, Adult, Age Factors, Aged, Breast Neoplasms, Complementary Therapies, Dietary Supplements, Educational Status, Female, Humans, Middle Aged, Mind-Body Therapies, New York, Patients, Reproducibility of Results, Treatment Outcome, Alternative medicine, Breast cancer, Latent class analysis, Epidemiology, Complementary and Alternative Medicine, Complementary & Alternative Medicine, Traditional, complementary and integrative medicine
Abstract

BackgroundBreast cancer patients commonly report using >1 form of complementary and alternative medicine (CAM). However, few studies have attempted to analyze predictors and outcomes of multiple CAM modalities. We sought to group breast cancer patients by clusters of type and intensity of complementary and alternative medicine (CAM) use following diagnosis.MethodsDetailed CAM use following breast cancer diagnosis was assessed in 2002-2003 among 764 female residents of Long Island, New York diagnosed with breast cancer in 1996-1997. Latent class analysis (LCA) was applied to CAM modalities while taking into account frequency and intensities.ResultsFour distinct latent classes of CAM use emerged: 1) "Low-dose supplement users" (40%), who used only common nutritional supplements; 2) "Vitamin/mineral supplement users" (39%), using an abundance of supplements in addition to other practices; 3) "Mind-body medicine users" (12%), with near-universal use of supplements, mind-body medicine techniques, and massage; and 4) "Multi-modality high-dose users" (9%), who were highly likely to use nearly all types of CAM. Predictors of membership in classes with substantial CAM use included younger age, more education, higher income, Jewish religion, ideal body mass index, higher fruit and vegetable intake, higher levels of physical activity, receipt of adjuvant chemotherapy, and prior use of oral contraceptives.ConclusionsLCA identified important subgroups of breast cancer patients characterized by varying degrees of complementary therapy use. Further research should explore the reproducibility of these classes and investigate the association between latent class membership and breast cancer outcomes.

Published
2015

11. No Association Between Vitamin D Status and Risk of Barrett's Esophagus or Esophageal Adenocarcinoma: A Mendelian Randomization Study

Author

Dong, Jing, Gharahkhani, Puya, Chow, Wong-Ho, Gammon, Marilie D., Liu, Geoffrey, Caldas, Carlos, Wu, Anna H., Ye, Weimin, Onstad, Lynn, Anderson, Lesley A., Bernstein, Leslie, Pharoah, Paul D., Risch, Harvey A., Corley, Douglas A., Fitzgerald, Rebecca C., Iyer, Prasad G., Reid, Brian J., Lagergren, Jesper, Shaheen, Nicholas J., Vaughan, Thomas L., MacGregor, Stuart, Love, Sharon, Palles, Claire, Tomlinson, Ian, Gockel, Ines, May, Andrea, Gerges, Christian, Anders, Mario, Böhmer, Anne C., Becker, Jessica, Kreuser, Nicole, Thieme, Rene, Noder, Tania, Venerito, Marino, Veits, Lothar, Schmidt, Thomas, Schmidt, Claudia, Izbicki, Jakob R., Hölscher, Arnulf H., Lang, Hauke, Lorenz, Dietmar, Schumacher, Brigitte, Mayershofer, Rupert, Vashist, Yogesh, Ott, Katja, Vieth, Michael, Weismüller, Josef, Nöthen, Markus M., Moebus, Susanne, Knapp, Michael, Peters, Wilbert H.M., Neuhaus, Horst, Rösch, Thomas, Ell, Christian, Jankowski, Janusz, Schumacher, Johannes, Neale, Rachel E., Whiteman, David C., and Thrift, Aaron P.

Published
2019
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14. Interactions Between Genetic Variants and Environmental Factors Affect Risk of Esophageal Adenocarcinoma and Barrett’s Esophagus

Author

Dong, Jing, Levine, David M., Buas, Matthew F., Zhang, Rui, Onstad, Lynn, Fitzgerald, Rebecca C., Corley, Douglas A., Shaheen, Nicholas J., Lagergren, Jesper, Hardie, Laura J., Reid, Brian J., Iyer, Prasad G., Risch, Harvey A., Caldas, Carlos, Caldas, Isabel, Pharoah, Paul D., Liu, Geoffrey, Gammon, Marilie D., Chow, Wong-Ho, Bernstein, Leslie, Bird, Nigel C., Ye, Weimin, Wu, Anna H., Anderson, Lesley A., MacGregor, Stuart, Whiteman, David C., Vaughan, Thomas L., and Thrift, Aaron P.

Published
2018
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15. Determining Risk of Barrett’s Esophagus and Esophageal Adenocarcinoma Based on Epidemiologic Factors and Genetic Variants

Author

Dong, Jing, Buas, Matthew F., Gharahkhani, Puya, Kendall, Bradley J., Onstad, Lynn, Zhao, Shanshan, Anderson, Lesley A., Wu, Anna H., Ye, Weimin, Bird, Nigel C., Bernstein, Leslie, Chow, Wong-Ho, Gammon, Marilie D., Liu, Geoffrey, Caldas, Carlos, Pharoah, Paul D., Risch, Harvey A., Iyer, Prasad G., Reid, Brian J., Hardie, Laura J., Lagergren, Jesper, Shaheen, Nicholas J., Corley, Douglas A., Fitzgerald, Rebecca C., Whiteman, David C., Vaughan, Thomas L., and Thrift, Aaron P.

Published
2018
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17. Electric Blanket Use and Breast Cancer on Long Island

Author

Kabat, Geoffrey C., O'Leary, Erin S., Schoenfeld, Elinor Randi, Greene, Judith M., Grimson, Roger, Henderson, Kevin, Kaune, William T., Gammon, Marilie D., Britton, Julie A., Teitelbaum, Susan L., Neugut, Alfred I., and Leske, M. Cristina

Published
2003

29. Pregnancy Characteristics and Maternal Risk of Breast Cancer

Author

Troisi, Rebecca, Weiss, Helen A., Hoover, Robert N., Potischman, Nancy, Swanson, Christine A., Brogan, Donna R., Coates, Ralph J., Gammon, Marilie D., Malone, Kathleen E., Daling, Janet R., and Brinton, Louise A.

Published
1998

32. Table S2 from Novel Predictors of Breast Cancer Survival Derived from miRNA Activity Analysis

Author

Aushev, Vasily N., primary, Lee, Eunjee, primary, Zhu, Jun, primary, Gopalakrishnan, Kalpana, primary, Li, Qian, primary, Teitelbaum, Susan L., primary, Wetmur, James, primary, Degli Esposti, Davide, primary, Hernandez-Vargas, Hector, primary, Herceg, Zdenko, primary, Parada, Humberto, primary, Santella, Regina M., primary, Gammon, Marilie D., primary, and Chen, Jia, primary

Published
2023
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33. Figure S7 from Novel Predictors of Breast Cancer Survival Derived from miRNA Activity Analysis

Author

Aushev, Vasily N., primary, Lee, Eunjee, primary, Zhu, Jun, primary, Gopalakrishnan, Kalpana, primary, Li, Qian, primary, Teitelbaum, Susan L., primary, Wetmur, James, primary, Degli Esposti, Davide, primary, Hernandez-Vargas, Hector, primary, Herceg, Zdenko, primary, Parada, Humberto, primary, Santella, Regina M., primary, Gammon, Marilie D., primary, and Chen, Jia, primary

Published
2023
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34. Supplementary Methods, Tables 1 - 6 from A Genome-wide Association Study of Early-Onset Breast Cancer Identifies PFKM as a Novel Breast Cancer Gene and Supports a Common Genetic Spectrum for Breast Cancer at Any Age

Author

Ahsan, Habibul, primary, Halpern, Jerry, primary, Kibriya, Muhammad G., primary, Pierce, Brandon L., primary, Tong, Lin, primary, Gamazon, Eric, primary, McGuire, Valerie, primary, Felberg, Anna, primary, Shi, Jianxin, primary, Jasmine, Farzana, primary, Roy, Shantanu, primary, Brutus, Rachelle, primary, Argos, Maria, primary, Melkonian, Stephanie, primary, Chang-Claude, Jenny, primary, Andrulis, Irene, primary, Hopper, John L., primary, John, Esther M., primary, Malone, Kathi, primary, Ursin, Giske, primary, Gammon, Marilie D., primary, Thomas, Duncan C., primary, Seminara, Daniela, primary, Casey, Graham, primary, Knight, Julia A., primary, Southey, Melissa C., primary, Giles, Graham G., primary, Santella, Regina M., primary, Lee, Eunjung, primary, Conti, David, primary, Duggan, David, primary, Gallinger, Steve, primary, Haile, Robert, primary, Jenkins, Mark, primary, Lindor, Noralane M., primary, Newcomb, Polly, primary, Michailidou, Kyriaki, primary, Apicella, Carmel, primary, Park, Daniel J., primary, Peto, Julian, primary, Fletcher, Olivia, primary, dos Santos Silva, Isabel, primary, Lathrop, Mark, primary, Hunter, David J., primary, Chanock, Stephen J., primary, Meindl, Alfons, primary, Schmutzler, Rita K., primary, Müller-Myhsok, Bertram, primary, Lochmann, Magdalena, primary, Beckmann, Lars, primary, Hein, Rebecca, primary, Makalic, Enes, primary, Schmidt, Daniel F., primary, Bui, Quang Minh, primary, Stone, Jennifer, primary, Flesch-Janys, Dieter, primary, Dahmen, Norbert, primary, Nevanlinna, Heli, primary, Aittomäki, Kristiina, primary, Blomqvist, Carl, primary, Hall, Per, primary, Czene, Kamila, primary, Irwanto, Astrid, primary, Liu, Jianjun, primary, Rahman, Nazneen, primary, Turnbull, Clare, primary, Dunning, Alison M., primary, Pharoah, Paul, primary, Waisfisz, Quinten, primary, Meijers-Heijboer, Hanne, primary, Uitterlinden, Andre G., primary, Rivadeneira, Fernando, primary, Nicolae, Dan, primary, Easton, Douglas F., primary, Cox, Nancy J., primary, and Whittemore, Alice S., primary

Published
2023
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35. Legends for Supplemental Figures from Novel Predictors of Breast Cancer Survival Derived from miRNA Activity Analysis

Author

Aushev, Vasily N., primary, Lee, Eunjee, primary, Zhu, Jun, primary, Gopalakrishnan, Kalpana, primary, Li, Qian, primary, Teitelbaum, Susan L., primary, Wetmur, James, primary, Degli Esposti, Davide, primary, Hernandez-Vargas, Hector, primary, Herceg, Zdenko, primary, Parada, Humberto, primary, Santella, Regina M., primary, Gammon, Marilie D., primary, and Chen, Jia, primary

Published
2023
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36. Data from Pleiotropic Analysis of Cancer Risk Loci on Esophageal Adenocarcinoma Risk

Author

Lee, Eunjung, primary, Stram, Daniel O., primary, Ek, Weronica E., primary, Onstad, Lynn E., primary, MacGregor, Stuart, primary, Gharahkhani, Puya, primary, Ye, Weimin, primary, Lagergren, Jesper, primary, Shaheen, Nicholas J., primary, Murray, Liam J., primary, Hardie, Laura J., primary, Gammon, Marilie D., primary, Chow, Wong-Ho, primary, Risch, Harvey A., primary, Corley, Douglas A., primary, Levine, David M., primary, Whiteman, David C., primary, Bernstein, Leslie, primary, Bird, Nigel C., primary, Vaughan, Thomas L., primary, and Wu, Anna H., primary

Published
2023
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37. Legends for Supplemental Tables from Novel Predictors of Breast Cancer Survival Derived from miRNA Activity Analysis

Author

Aushev, Vasily N., primary, Lee, Eunjee, primary, Zhu, Jun, primary, Gopalakrishnan, Kalpana, primary, Li, Qian, primary, Teitelbaum, Susan L., primary, Wetmur, James, primary, Degli Esposti, Davide, primary, Hernandez-Vargas, Hector, primary, Herceg, Zdenko, primary, Parada, Humberto, primary, Santella, Regina M., primary, Gammon, Marilie D., primary, and Chen, Jia, primary

Published
2023
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38. Supplemental Methods from Novel Predictors of Breast Cancer Survival Derived from miRNA Activity Analysis

Author

Aushev, Vasily N., primary, Lee, Eunjee, primary, Zhu, Jun, primary, Gopalakrishnan, Kalpana, primary, Li, Qian, primary, Teitelbaum, Susan L., primary, Wetmur, James, primary, Degli Esposti, Davide, primary, Hernandez-Vargas, Hector, primary, Herceg, Zdenko, primary, Parada, Humberto, primary, Santella, Regina M., primary, Gammon, Marilie D., primary, and Chen, Jia, primary

Published
2023
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41. Data from A Genome-wide Association Study of Early-Onset Breast Cancer Identifies PFKM as a Novel Breast Cancer Gene and Supports a Common Genetic Spectrum for Breast Cancer at Any Age

Author

Ahsan, Habibul, primary, Halpern, Jerry, primary, Kibriya, Muhammad G., primary, Pierce, Brandon L., primary, Tong, Lin, primary, Gamazon, Eric, primary, McGuire, Valerie, primary, Felberg, Anna, primary, Shi, Jianxin, primary, Jasmine, Farzana, primary, Roy, Shantanu, primary, Brutus, Rachelle, primary, Argos, Maria, primary, Melkonian, Stephanie, primary, Chang-Claude, Jenny, primary, Andrulis, Irene, primary, Hopper, John L., primary, John, Esther M., primary, Malone, Kathi, primary, Ursin, Giske, primary, Gammon, Marilie D., primary, Thomas, Duncan C., primary, Seminara, Daniela, primary, Casey, Graham, primary, Knight, Julia A., primary, Southey, Melissa C., primary, Giles, Graham G., primary, Santella, Regina M., primary, Lee, Eunjung, primary, Conti, David, primary, Duggan, David, primary, Gallinger, Steve, primary, Haile, Robert, primary, Jenkins, Mark, primary, Lindor, Noralane M., primary, Newcomb, Polly, primary, Michailidou, Kyriaki, primary, Apicella, Carmel, primary, Park, Daniel J., primary, Peto, Julian, primary, Fletcher, Olivia, primary, dos Santos Silva, Isabel, primary, Lathrop, Mark, primary, Hunter, David J., primary, Chanock, Stephen J., primary, Meindl, Alfons, primary, Schmutzler, Rita K., primary, Müller-Myhsok, Bertram, primary, Lochmann, Magdalena, primary, Beckmann, Lars, primary, Hein, Rebecca, primary, Makalic, Enes, primary, Schmidt, Daniel F., primary, Bui, Quang Minh, primary, Stone, Jennifer, primary, Flesch-Janys, Dieter, primary, Dahmen, Norbert, primary, Nevanlinna, Heli, primary, Aittomäki, Kristiina, primary, Blomqvist, Carl, primary, Hall, Per, primary, Czene, Kamila, primary, Irwanto, Astrid, primary, Liu, Jianjun, primary, Rahman, Nazneen, primary, Turnbull, Clare, primary, Dunning, Alison M., primary, Pharoah, Paul, primary, Waisfisz, Quinten, primary, Meijers-Heijboer, Hanne, primary, Uitterlinden, Andre G., primary, Rivadeneira, Fernando, primary, Nicolae, Dan, primary, Easton, Douglas F., primary, Cox, Nancy J., primary, and Whittemore, Alice S., primary

Published
2023
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42. Data from Novel Predictors of Breast Cancer Survival Derived from miRNA Activity Analysis

Author

Aushev, Vasily N., primary, Lee, Eunjee, primary, Zhu, Jun, primary, Gopalakrishnan, Kalpana, primary, Li, Qian, primary, Teitelbaum, Susan L., primary, Wetmur, James, primary, Degli Esposti, Davide, primary, Hernandez-Vargas, Hector, primary, Herceg, Zdenko, primary, Parada, Humberto, primary, Santella, Regina M., primary, Gammon, Marilie D., primary, and Chen, Jia, primary

Published
2023
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43. Data from Replication and Functional Genomic Analyses of the Breast Cancer Susceptibility Locus at 6q25.1 Generalize Its Importance in Women of Chinese, Japanese, and European Ancestry

Author

Cai, Qiuyin, primary, Wen, Wanqing, primary, Qu, Shimian, primary, Li, Guoliang, primary, Egan, Kathleen M., primary, Chen, Kexin, primary, Deming, Sandra L., primary, Shen, Hongbing, primary, Shen, Chen-Yang, primary, Gammon, Marilie D., primary, Blot, William J., primary, Matsuo, Keitaro, primary, Haiman, Christopher A., primary, Khoo, Ui Soon, primary, Iwasaki, Motoki, primary, Santella, Regina M., primary, Zhang, Lina, primary, Fair, Alecia Malin, primary, Hu, Zhibin, primary, Wu, Pei-Ei, primary, Signorello, Lisa B., primary, Titus-Ernstoff, Linda, primary, Tajima, Kazuo, primary, Henderson, Brian E., primary, Chan, Kelvin Y.K., primary, Kasuga, Yoshio, primary, Newcomb, Polly A., primary, Zheng, Hong, primary, Cui, Yong, primary, Wang, Furu, primary, Shieh, Ya-Lan, primary, Iwata, Hiroji, primary, Le Marchand, Loic, primary, Chan, Sum Yin, primary, Shrubsole, Martha J., primary, Trentham-Dietz, Amy, primary, Tsugane, Shoichiro, primary, Garcia-Closas, Montserrat, primary, Long, Jirong, primary, Li, Chun, primary, Shi, Jiajun, primary, Huang, Bo, primary, Xiang, Yong-Bing, primary, Gao, Yu-Tang, primary, Lu, Wei, primary, Shu, Xiao-Ou, primary, and Zheng, Wei, primary

Published
2023
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44. Supplementary Methods and Materials from Replication and Functional Genomic Analyses of the Breast Cancer Susceptibility Locus at 6q25.1 Generalize Its Importance in Women of Chinese, Japanese, and European Ancestry

Author

Cai, Qiuyin, primary, Wen, Wanqing, primary, Qu, Shimian, primary, Li, Guoliang, primary, Egan, Kathleen M., primary, Chen, Kexin, primary, Deming, Sandra L., primary, Shen, Hongbing, primary, Shen, Chen-Yang, primary, Gammon, Marilie D., primary, Blot, William J., primary, Matsuo, Keitaro, primary, Haiman, Christopher A., primary, Khoo, Ui Soon, primary, Iwasaki, Motoki, primary, Santella, Regina M., primary, Zhang, Lina, primary, Fair, Alecia Malin, primary, Hu, Zhibin, primary, Wu, Pei-Ei, primary, Signorello, Lisa B., primary, Titus-Ernstoff, Linda, primary, Tajima, Kazuo, primary, Henderson, Brian E., primary, Chan, Kelvin Y.K., primary, Kasuga, Yoshio, primary, Newcomb, Polly A., primary, Zheng, Hong, primary, Cui, Yong, primary, Wang, Furu, primary, Shieh, Ya-Lan, primary, Iwata, Hiroji, primary, Le Marchand, Loic, primary, Chan, Sum Yin, primary, Shrubsole, Martha J., primary, Trentham-Dietz, Amy, primary, Tsugane, Shoichiro, primary, Garcia-Closas, Montserrat, primary, Long, Jirong, primary, Li, Chun, primary, Shi, Jiajun, primary, Huang, Bo, primary, Xiang, Yong-Bing, primary, Gao, Yu-Tang, primary, Lu, Wei, primary, Shu, Xiao-Ou, primary, and Zheng, Wei, primary

Published
2023
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47. Genome-wide association studies in oesophageal adenocarcinoma and Barrett's oesophagus: a large-scale meta-analysis

Author

Gharahkhani, Puya, Fitzgerald, Rebecca C, Vaughan, Thomas L, Palles, Claire, Gockel, Ines, Tomlinson, Ian, Buas, Matthew F, May, Andrea, Gerges, Christian, Anders, Mario, Becker, Jessica, Kreuser, Nicole, Noder, Tania, Venerito, Marino, Veits, Lothar, Schmidt, Thomas, Manner, Hendrik, Schmidt, Claudia, Hess, Timo, Böhmer, Anne C, Izbicki, Jakob R, Hölscher, Arnulf H, Lang, Hauke, Lorenz, Dietmar, Schumacher, Brigitte, Hackelsberger, Andreas, Mayershofer, Rupert, Pech, Oliver, Vashist, Yogesh, Ott, Katja, Vieth, Michael, Weismüller, Josef, Nöthen, Markus M, Attwood, Stephen, Barr, Hugh, Chegwidden, Laura, de Caestecker, John, Harrison, Rebecca, Love, Sharon B, MacDonald, David, Moayyedi, Paul, Prenen, Hans, Watson, R G Peter, Iyer, Prasad G, Anderson, Lesley A, Bernstein, Leslie, Chow, Wong-Ho, Hardie, Laura J, Lagergren, Jesper, Liu, Geoffrey, Risch, Harvey A, Wu, Anna H, Ye, Weimin, Bird, Nigel C, Shaheen, Nicholas J, Gammon, Marilie D, Corley, Douglas A, Caldas, Carlos, Moebus, Susanne, Knapp, Michael, Peters, Wilbert H M, Neuhaus, Horst, Rösch, Thomas, Ell, Christian, MacGregor, Stuart, Pharoah, Paul, Whiteman, David C, Jankowski, Janusz, and Schumacher, Johannes

Published
2016
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