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2. Assortative mating and parental genetic relatedness contribute to the pathogenicity of variably expressive variants

3. Rare deleterious mutations of HNRNP genes result in shared neurodevelopmental disorders.

4. Rare variants in the genetic background modulate cognitive and developmental phenotypes in individuals carrying disease-associated variants

7. Assortative mating and parental genetic relatedness drive the pathogenicity of variably expressive variants

8. Descripción y evolución del primer caso de síndrome de Jacobsen diagnosticado en Argentina, su analogía con anemia de Fancon

9. Heterozygous Deletion of Long Noncoding RNA AK127244 Is a Susceptibility Factor for Neurodevelopmental Delay

14. Bilateral Periventricular Nodular Heterotopia and Lissencephaly in an Infant with Unbalanced t(12;17)(q24.31; p13.3) Translocation

15. Additional file 3 of Rare deleterious mutations of HNRNP genes result in shared neurodevelopmental disorders

16. Additional file 2 of Rare deleterious mutations of HNRNP genes result in shared neurodevelopmental disorders

19. Heterozygous Deletion of Long Noncoding RNA AK127244 Is a Susceptibility Factor for Neurodevelopmental Delay.

20. 8p23.2-pter Microdeletions: Seven New Cases Narrowing the Candidate Region and Review of the Literature

21. Identification of pathogenic gene variants in small families with intellectually disabled siblings by exome sequencing

26. Cryptic telomeric rearrangements in subjects with mental retardation associated with dysmorphism and congenital malformations

28. Disruptive variants of CSDE1 associate with autism and interfere with neuronal development and synaptic transmission

30. Rare variants in the genetic background modulate the expressivity of neurodevelopmental disorders

31. Biallelic intragenic duplication in ADGRB3(BAI3) gene associated with intellectual disability, cerebellar atrophy, and behavioral disorder

32. Definition of minimal duplicated region encompassing theXIAPandSTAG2genes in the Xq25 microduplication syndrome

33. Molecular Mechanisms Generating and Stabilizing Terminal 22q13 Deletions in 44 Subjects with Phelan/McDermid Syndrome

35. Screening of subtelomeric rearrangements in autistic disorder: Identification of a partial trisomy of 13q34 in a patient bearing a 13q;21p translocation

38. Genetic modifiers and ascertainment drive variable expressivity of complex disorders.

39. Assortative mating and parental genetic relatedness drive the pathogenicity of variably expressive variants.

40. Partial monosomy Xq(Xq23 --> qter) and trisomy 4p(4p15.33 --> pter) in a woman with intractable focal epilepsy, borderline intellectual functioning, and dysmorphic features.

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