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179 results on '"Fulciniti M"'

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3. P881: CDK7 CONTRIBUTES TO METABOLIC REPROGRAMMING IN MM CELLS THROUGH C-MYC MEDIATED TRANSCRIPTIONAL CONTROL OF GLYCOLYTIC GENES

12. Targeting Myeloma Bone Disease via Activin A Inhibition: B357

16. Favorable Progression-Free Survival Associated with Immune Bio-Markers Modulated By Pomalidomide in Relapsed/Refractory Multiple Myeloma: An Analysis of Phase III Study

17. PF588 SIGNIFICANT POM EFFECT ON THE IMMUNE-CELL PROFILES IN B, T AND NK SUBSETS IN A LARGE COHORT CLINICAL TRIAL IN RELAPSED/REFRACTORY MYELOMA

18. S119 THE ROLE OF RECURRENT SOMATIC ALTERATIONS IN THE NON-CODING GENOME WITH FUNCTIONAL IMPLICATIONS IN MM

21. Protein arginine methyltransferase 5 has prognostic relevance and is a druggable target in multiple myeloma

22. Nucleotide excision repair is a potential therapeutic target in multiple myeloma

25. miR-23b/SP1/c-myc forms a feed-forward loop supporting multiple myeloma cell growth

27. Targeting IL-17A in multiple myeloma: a potential novel therapeutic approach in myeloma

29. Protein arginine methyltransferase 5 has prognostic relevance and is a druggable target in multiple myeloma

32. miR-29b sensitizes multiple myeloma cells to bortezomib-induced apoptosis through the activation of a feedback loop with the transcription factor Sp1

33. B026 CS1 Promotes Multiple Myeloma Cell Adhesion, Clonogenic Growth, and Tumorigenicity via c-Maf Mediation

34. B203 A New Anti-Dkk-1 Antibody Targeting Myeloma Bone Disease

37. B037 CAL-101: A Selective Inhibitor of PI3K p110D for the Treatment of Multiple Myeloma

39. B357 Targeting Myeloma Bone Disease via Activin A Inhibition

41. Targeting PI3K and RAD51 in Barrett's adenocarcinoma: Impact on DNA damage checkpoints, expression profile and tumor growth

42. The genomic landscape of structural variations and complex events in multiple myeloma

43. Analysis of mutations and structural variants to redefine the genomic landscape of multiple myeloma and its clinical implications

44. Mechanism of action of immunomodulatory agents in multiple myeloma.

45. miR-29b sensitizes multiple myeloma cells to bortezomib-induced apoptosis through the activation of a feedback loop with the transcription factor Sp1

46. CS1 promotes multiple myeloma cell adhesion, clonogenic growth, and tumorigenicity via c-maf–mediated interactions with bone marrow stromal cells

47. HLA class I, NKG2D and natural cytotoxicity receptors regulate multiple myeloma cell recognition by natural killer cells

48. Development of hyperdiploidy starts at an early age and takes a decade to complete.

49. Dual therapeutic targeting of MYC and JUNB transcriptional programs for enhanced anti-myeloma activity.

50. Venetoclax resistance leads to broad resistance to standard-of-care anti-MM agents, but not to immunotherapies.

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