119 results on '"Ftouhi, B."'
Search Results
2. Évaluation de l’équilibre tensionnel par la mesure ambulatoire de la pression artérielle et étude des facteurs associés à un mauvais contrôle tensionnel chez 300 diabétiques de type 2 hypertendus traités
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Ben-Hamouda-Chihaoui, M., Kanoun, F., Ftouhi, B., Lamine-Chtioui, F., Kamoun, M., and Slimane, H.
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- 2011
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Catalog
3. Influence plasma levels of matrix metalloproteinase among patients obese with or without metabolic syndrome
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Zidi, W., primary, Benkhalifa, M., additional, Zayani, Y., additional, Aloui, S., additional, Fourti, N., additional, Guizani, I., additional, Boussairi, S., additional, Sanhaji, H., additional, Ftouhi, B., additional, Feki, M., additional, and Allal-Elasmi, M., additional more...
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- 2022
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4. Platelet PMCA‐ and SERCA‐type Ca2+‐ATPase expression in diabetes: a novel signature of abnormal megakaryocytopoiesis
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CHAABANE, C., DALLY, S., CORVAZIER, E., BREDOUX, R., BOBE, R., FTOUHI, B., RAIES, A., and ENOUF, J.
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- 2007
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5. Syndrome de McCune-Albright associé à un diabète sucré
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Chihaoui, M., Hamza, N., Lamine, F., Jabeur, S., Yazidi, M., Ftouhi, B., and Slimane, H.
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- 2012
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6. P2-17: Collagenase-1 and arterial hypertension in patients with metabolic syndrome
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Benkhalifa, M., primary, Zayani, Y., additional, Zidi, W., additional, Aloui, S., additional, Ftouhi, B., additional, Slimen, H., additional, Allal Elasmi, M., additional, and Kaabachi, N., additional more...
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- 2015
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7. Profil épidémiologique, clinique et métabolique de 200 patients obèses
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Rojbi, I., primary, Yazidi, M., additional, Sahli, N., additional, Chaker, F., additional, Gharbi, R., additional, Ftouhi, B., additional, and Slimane, H., additional
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- 2015
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8. Étiologies de l’hyperthyroïdie au cours de la grossesse
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Gharbi, R., primary, Hbaili, N., additional, Chihaoui, M., additional, Yazidi, M., additional, Chaker, F., additional, Kenoun, F., additional, Ftouhi, B., additional, and Slimane, H., additional
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- 2014
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9. Place de la dysrégulation nocturne de la pression artérielle dans le développement des complications vasculaires chez le diabétique de type 2 hypertendu
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Chihaoui, M., primary, Yazidi, M., additional, Brahim, M. Ben, additional, Chaker, F., additional, Kanoun, F., additional, Ftouhi, B., additional, and Slimane, H., additional
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- 2014
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10. La dysrégulation nocturne de la pression artérielle chez le diabétique de type 2 hypertendu ; prévalence et facteurs associés
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Chihaoui, M., primary, Yazidi, M., additional, Ben Brahim, M., additional, Chaker, F., additional, Kanoun, F., additional, Ftouhi, B., additional, and Slimane, H., additional
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- 2014
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11. Predictive risk factors for deterioration from normoglycemic state to type 2 diabetes mellitus or impaired glucose tolerance in a Tunisian urban population
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Melika Chihaoui, Kanoun F, Ben Rehaiem B, Ben Brahim S, Ftouhi B, Mekaouar A, Fekih M, Mbazâd A, Zouari B, and Ben Khalifa F
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Adult ,Blood Glucose ,Glycated Hemoglobin ,Male ,Analysis of Variance ,Time Factors ,Tunisia ,Urban Population ,Incidence ,Blood Pressure ,Glucose Tolerance Test ,Middle Aged ,Body Mass Index ,Diabetes Mellitus, Type 2 ,Predictive Value of Tests ,Reference Values ,Risk Factors ,Glucose Intolerance ,Multivariate Analysis ,Humans ,Female ,Prospective Studies ,Follow-Up Studies - Abstract
To determine the predictive risk factors for the development of type 2 diabetes mellitus (DM) or impaired glucose tolerance (IGT) in a normoglycemic population.This is a ten-year prospective study in a randomly selected urban population including 1835 subjects aged=30 years living in Tunis, 1460 were normoglycemic according to 2 hours blood glucose WHO criteria, and 701 among them attended the follow-up assessment ten years later. Subjects with impaired glucose tolerance (IGT) were excluded. Subjects underwent a physical examination including weight/height, iliac circumference (IC) and blood pressure measurements. Fasting and 2-hour venous blood sampling, after a 75 g oral glucose load, were performed for the measurement of plasma glucose (G(0), G(2h)), insulin (I(0), I(2h)), total cholesterol (TC) and glycated hemoglobin (HbA(1c)) levels.Out of the 701 normoglycemic subjects in 1985, 77 developed diabetes and 130 impaired glucose tolerance after 10 years, giving a mean annual incidence rate of 1.1% for diabetes and 1.85% for IGT. Univariate analysis showed that risk factors for diabetes were age, BMI, IC, SBP, G(0) and total cholesterol in both sexes, I(0) and I(2h) in men only and DBP G(2h) and HbA(1c) in women only. Risk factors for IGT were BMI, IC and G(2h) in both sexes, I(2h) in men only and G(0) in women only. Multivariate analysis revealed that BMI, G(0) and G(2h) were independent risk factors for conversion to diabetes or IGT in both sexes, but HbA(1c) and IC were risk factors only in men.Early screening and prevention of diabetes must focus on obese subjects, especially those with central fat distribution, and those with moderate increase in fasting and/or two-hour blood glucose levels within the normal range. more...
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- 2001
12. Tumeur stromale de l’intestin grêle révélée par des hypoglycémies sévères
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Ben Said, R., primary, Ftouhi, B., additional, Bousselmi, J., additional, Ben Nacef, I., additional, Chihaoui, M., additional, and Slimene, H., additional
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- 2013
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13. Un phéochromocytome malin révélé par une pancréatite aiguë
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Bousselmi, J., primary, Ftouhi, B., additional, Bensaid, R., additional, Bennacef, I., additional, Hbeili, N., additional, Chihaoui, M., additional, and Slimene, H., additional
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- 2013
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14. Les réhospitalisations au cours du diabète de type 1
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Meriem, Y., primary, Chihaoui, M., additional, Ftouhi, B., additional, Ben Said, R., additional, and Slimane, H., additional
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- 2012
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15. Place de la surrénalectomie bilatérale dans le traitement de la maladie de Cushing
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Ben Nacef, I., primary, Chihaoui, M., additional, Ben Said, R., additional, Ftouhi, B., additional, Lamine, F., additional, Kanoun, F., additional, and Sliméne, H., additional
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- 2012
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16. Un microadénome hypophysaire peut-il être responsable d’une baisse de l’acuité visuelle ?
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Ben Said, R., primary, Chihaoui, M., additional, Ben Nacef, I., additional, Ftouhi, B., additional, and Lamine, F., additional
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- 2012
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17. P279 Effet du polymorphisme G3057A du gène du récepteur de la leptine sur le risque de l’obésité et ses désordres métaboliques chez une population Tunisienne
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Ftouhi, B., primary, Ben Ali, S., additional, Kallel, A., additional, Sediri, Y., additional, Haj-Taïeb, S., additional, Omar, S., additional, Sanhaji, H., additional, Feki, M., additional, Elasmi, M., additional, Slimane, H., additional, Jemaa, R., additional, and Kaabachi, N., additional more...
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- 2010
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18. P263 Taux de C-Reactive Proteine et Syndrome métabolique
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Ftouhi, B., primary, Zidi, W., additional, Elasmi, M., additional, Zayani, Y., additional, Ounifi, S., additional, Slimen, H., additional, and Kaabachi, N., additional
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- 2010
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19. P268 Uricémie et syndrome métabolique dans la population du grand Tunis
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Zayani, Y., primary, Elasmi, M., additional, Feki, M., additional, Hadj Taieb, S., additional, Omar, S., additional, Sanhagi, H., additional, Jemaa, R., additional, Ftouhi, B., additional, Hsairi, M., additional, Mbazaa, A., additional, and Kaabachi, N., additional more...
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- 2010
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20. P283 Association du polymorphisme Pro12Ala du gène PPARgamma 2 avec l’obésité et l’IMC chez une population tunisienne
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Ftouhi, B., primary, Ben Ali, S., additional, Sediri, Y., additional, Kallel, A., additional, Feki, M., additional, Elasmi, M., additional, Haj-Taïeb, S., additional, Omar, S., additional, Sanhaji, H., additional, Slimane, H., additional, Jemaa, R., additional, and Kaabachi, N., additional more...
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- 2010
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21. P294 Association entre l’indice de masse corporel et la syndrome métabolique
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Ftouhi, B., primary, Zidi, W., additional, Elasmi, M., additional, Zayani, Y., additional, Ounifi, S., additional, Slimen, H., additional, and Kaabachi, N., additional
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- 2010
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22. The ‐2548G/A LEP polymorphism is associated with blood pressure in Tunisian obese patients
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Ben Ali, S., primary, Kallel, A., additional, Ftouhi, B., additional, Sediri, Y., additional, Feki, M., additional, Slimane, H., additional, Jemaa, R., additional, and Kaabachi, N., additional
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- 2008
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23. P1-103 - Impact de la pratique du football sur la masse osseuse et les marqueurs biochimiques à différents stades pubertaires
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Nebigh, A., primary, Rebai, H., additional, Tabka, Z., additional, Kamoun, M., additional, Chihaoui, M., additional, Ftouhi, B., additional, Slimane, H., additional, Sahli, H., additional, Sellami, S., additional, Kalai, E., additional, and Abdelmoula, J., additional more...
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- 2006
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24. P202 - Déficit enzymatique en 11β-hydroxylase : à propos de 8 cas
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Chihaoui, M., primary, Ftouhi, B., additional, Kamoun, M., additional, Slimane, H., additional, and Morel, Y., additional
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- 2005
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25. P142 - Traitement médical des adénomes à prolactine : à propos de 108 cas
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Ftouhi, B., primary, Chihaoui, M., additional, Maazoun, I., additional, Chraga, L., additional, Kamoun, M., additional, and Slimane, H., additional
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- 2005
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26. P069 - Carcinome parathyroïdien intrathyroïdien : à propos d’un cas
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Chihaoui, M., primary, Hamdani, L., additional, Ftouhi, B., additional, Kamoun, M., additional, Sahtout, S., additional, Hachicha, S., additional, Haouet, S., additional, and Slimane, H., additional
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- 2005
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27. P189 - L’hypertension artérielle surrénalienne : à propos de 41 cas
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Ftouhi, B., primary, Chihaoui, M., additional, Kamoun, M., additional, Fazaa, S., additional, and Slimane, H., additional
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- 2005
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28. P137 - Dysrégulation nocturne de la pression artérielle (DNP) et risque de microangiopathie chez le diabétique de type 2
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Chihaoui, M., primary, Kanoun, F., additional, Ftouhi, B., additional, Kamoun, M., additional, and Slimane, H., additional
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- 2004
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29. P232 - Circonstances diagnostiqués et apport de l’exploration morphologique dans l’hyperparathyroïdie primaire
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Ftouhi, B., primary, Chihaoui, M., additional, Sahtout, S., additional, Lahiani, R., additional, Kamoun, M., additional, Hachicha, S., additional, and Slimane, H., additional
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- 2004
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30. P061 - Hyperparathyroïdie primaire et cancer papillaire de la thyroïde. à propos d’un cas
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Bouzid, C., primary, Ftouhi, B., additional, Daoud, I., additional, Kamoun, M., additional, Chihaoui, M., additional, and Slimane, H., additional
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- 2004
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31. P092 - Épidémiologie de la pathologie thyroïdienne dans un service d’endocrinologie à Tunis, Tunisie
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Kamoun, M., primary, Ftouhi, B., additional, Ben Brahim, A., additional, Chihaoui, M., additional, Smadhi, H., additional, Bouzid, C., additional, Daoued, I., additional, and Slimane, H., additional
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- 2004
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32. P066 - Les nodules thyroïdiens de l’enfant
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Ftouhi, B., primary, Sahtout, S., additional, Kharrat, S., additional, Chihaoui, M., additional, Kamoun, M., additional, Hachicha, S., additional, and Slimane, H., additional
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- 2004
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33. P136 - Variations nycthémérales de la frequence cardiaque chez le diabetique de type 2
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Chihaoui, M., primary, Kanoun, F., additional, Ftouhi, B., additional, Kamoun, M., additional, and Slimane, H., additional
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- 2004
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34. P078 - Diagnostic, traitement et évolution de la maladie de basedow. À propos de 52 cas
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Bouzid, C., primary, Kamoun, M., additional, Chihaoui, M., additional, Ftouhi, B., additional, Daoued, I., additional, Ben Brahim, A., additional, Smadhi, H., additional, and Slimane, H., additional
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- 2004
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35. P134 - L’élévation de la pression artérielle évaluée par la mesure ambulatoire de la pression artérielle (MAPA) est un facteur de risque majeur de la rétinopathie diabétique
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Chihaoui, M., primary, Kamoun, M., additional, Ftouhi, B., additional, Berhaiem, B., additional, and Slimane, H., additional
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- 2004
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36. P060 - Circonstances diagnostiques du cancer différencié de la thyroïde. à propos de 70 cas
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Ftouhi, B., primary, Sahtout, S., additional, Lahiani, R., additional, Chihaoui, M., additional, Kamoun, M., additional, Hachicha, S., additional, and Slimane, H., additional
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- 2004
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37. P075 - Thyroïdite d’hashimoto, caractéristiques épidémiologiques et cliniques
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Daoued, I., primary, Kamoun, M., additional, Bouzid, C., additional, Ftouhi, B., additional, Chihaoui, M., additional, Ben Brahim, A., additional, Smadhi, H., additional, and Slimane, H., additional
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- 2004
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38. P135 - Intérêt de la mesure ambulatoire de la pression artérielle (MAPA) dans le diagnostic et le contrôle de l’hypertension artérielle chez le diabétique de type 2
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Chihaoui, M., primary, Ftouhi, B., additional, Sayem, N., additional, Kanoun, F., additional, and Slimane, H., additional
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- 2004
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39. 35th Annual Meeting of the European Association for the Study of Diabetes
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Melander, A., Olsson, J., Lindberg, G., Salzman, A., Howard, T., Stang, P., Lydick, E., Emslie-Smith, A., Boyle, D. I. R., Evans, J. M. M., Macdonald, T. M., Bain, J., Sullivan, F., Juhl, C., Pørksen, N., Sturis, J., Hollingdal, M., Pincus, S., Veldhuis, J., Dejgaard, A., Schmitz, O., Kristensen, J. S., Frandsen, K. B., Bayer, Th., Müller, P., Dunning, B. E., Paladini, S., Gutierrez, C., Deacon, R., Valentin, M., Grunberger, G., Weston, W. M., Patwardhan, R., Rappaport, E. B., Sargeant, L. A., Wareham, N. J., Khaw, K. T., Zethelius, Björn, Lithell, Hans, Hales, C. Nicholas, Berne, Christian, Lakka, H.-M., Oksanen, L., Tuomainen, T.-P., Kontula, K., Salonen, J. T., Dekker, J. M., de Boks, P., de Vegt, F., Stehouwer, C. D. A., Nijpels, G., Bouter, L. M., Heine, R. J., Bruno, G., Cavallo-Perin, P., Bargero, G., D’Errico, N., Borra, M., Macchia, G., Pagano, G., Newton, R. W., Ruta, D. A., New, J. P., Wallace, C., Roxburgh, M. A., Young, R. J., Vaughan, N. J. A., Elliott, P., Brennan, G., Devers, M., MacAlpine, R., Steinke, D., Lawson, D. H., Decallonne, B., Casteels, K., Gysemans, C., Bouillon, R., Mathieu, C., Linn, Thomas, Strate, Christine, Schneider, Kerstin, Funda, D. P., Jirsa, M., Kozáková, H., Kaas, A., Kofronová, O., Tlaskalová-Hogenová, H., Buschard, K., Wanka, H., Hartmann, A., Kuttler, B., Rasmussen, S. B., Sørensen, T. S., Markholst, H., Petersen, J. S., Karounos, D., Dyrberg, T., Mabley, J. G., Haskó, G., Szabó, C., Seissler, J., Nguyen, T. B. T., Steinbrenner, H., Scherbaum, W. A., Cipriani, R., Gabriele, A., Sensi, M., Guidobaldi, L., Pantellini, F., Cerrito, M. G., Scarpa, S., Di Mario, U., Morano, S., Ceolotto, G., Iori, E., Baritono, E., Del Prato, S., Semplicini, A., Trevisan, R., Zerbini, G., Meregalli, G., Asnaghi, V., Tentori, F., Maestroni, A., Mangili, R., Marescotti, C., Vedovato, M., Tiengo, A., Tadjieva, J., Mankovsky, B. N., Van Aken, S., Raes, A., Vande Walle, J., Matthys, D., Craen, M., Hansen, H. P., Lund, S. S., Rossing, P., Jensen, T., Parving, H.-H., Andersen, S., Tarnow, L., Hansen, B. V., Trautner, C., Haastert, B., Ennenbach, N., Willich, S., Tabák, Á. Gy., Orchard, T. J., Spranger, J., Preissner, K. T., Schatz, H., Pfeiffer, A., Cantón, A., Burgos, R., Hernández, C., Lecube, A., Mesa, J., Segura, R. M., Mateo, C., Simó, R., Fathallah, L., Greene, D. A., Obrosova, I., Gilbert, R. E., Kelly, D. J., Cox, A. J., Berka-Wilkinson, J. L., Taylor, H. R., Panagiotopoulos, S., Lee, V., Jerums, G., Cooper, M. E., Hitman, G. A., Aganna, E., Ogunkolade, W. B., Rema, M., Deepa, R., Shanthi-Rani, C. S., Barakat, K., Kumarajeewa, T. R., Cassell, P. G., McDermott, M. F., Mohan, V., Ways, K., Bursell, S., Devries, T., Woodworth, J., Alatorre, C., King, G., Aiello, L. P., Karisen, A. E., Pavlovic, D., Nielsen, K., Jensen, J., Andersen, H. U., Pociot, F., Mandrup-Poulsen, T., Eizirik, D. L., Nerup, J., Lortz, S., Tiedge, M., Lenzen, S., Lally, F. J., Bone, A. J., Darville, M. I., Ho, Y.-S., Sternesjö, J., Sandler, S., Chen, M.-C., Schuit, F., Pipeleers, D. G., Merezak, S., Hardikar, A., Hoet, J. J., Remacle, C., Reusens, B., Bréant, B., Garofano, A., Czernichow, P., Kubota, N., Terauchi, Y., Miki, H., Tamemoto, H., Yamauchi, T., Nakano, R., Komeda, K., Eto, K., Tobe, K., Kimura, S., Kadowaki, T., Ide, T., Murakami, K., Tsunoda, M., Mochizuki, T., Ozanne, S. E., Nave, B. T., Wang, C. L., Dorling, M. W., Petry, C. J., Koopmans, S. J., van der Bent, C., Que, I., Radder, J. K., Sebokova, E., Sana, A. K., Klimes, I., Ruderman, N., Morviducci, L., Pastore, L., Morelli, S., Sagratella, E., Zorretta, D., Buongiomo, A., Tamburrano, G., Giaccari, A., Martinenghi, Sabina, De Angelis, Gabriella Cusella, Ravasi, Flavio, Bifari, Francesco, Bordignon, Claudio, Falqui, Luca, Kessler, A., Dransfeld, O., Sasson, S., Tomas, E., Zorzano, A., Eckel, J., Thorsby, P., Rosenfalck, A. M., Kjems, L., Hanssen, K. F., Madsbad, S., Birkeland, K. I., Hamilton-Wessler, M., Markussen, J., Bergman, R. N., Melki, V., Hanaire-Broutin, H., Bessières-Lacombe, S., Tauber, J.-P., Home, P. D., Lindholm, A., Riis, A., Rosenstock, J., Schwartz, S., Clark, C., Edwards, M., Donley, D., Swift, P., Mortensen, H. B., Lynggaard, H., Hougaard, P., Cull, C. A., Neil, H. A. W., Frighi, V., Manley, S. E., Holman, R. R., Turner, R. C., Steiner, G., Davis, W. A., Weeraratna, T., Bruce, D. G., Davis, T. M. E., Vergès, B., Duvillard, L., Pont, F., Florentin, E., Gambert, Ph., Benko, B., Ljubić, S., Turk, Z., Granić, M., März, W., Wollschläger, H., Klein, G., Neiss, A., Wehling, M., Huxtable, S. J., Saker, P. J., Walker, M., Frayling, T. M., Levy, J. C., O’Rahilly, S., Hattersley, A. T., McCarthy, M. I., Orecchio, A., Giacchini, A., Dominici, R., Canettieri, G., Trinti, B., Zani, M., Andreoli, M., Sciacchitano, S., de Silva, A. M., Whitecross, K., Pasco, J., Kotowicz, M., Nicholson, G., Zimmet, P., Boyko, E. J., Collier, G. R., Frittitta, L., Pizzuti, A., Argiolas, A., Graci, S., Goldfine, I. D., Bozzali, M., Ercolino, T., Costanzo, B., Iacoviello, L., Tassi, V., Trischitta, V., Wauters, M., Rankinen, T., Mertens, I., Chagnon, M., Bouchard, C., Van Gaal, L., Sivenius, K., Valve, R., Hakkarainen, V., Niskanen, L., Laakso, M., Uusitupa, M., Beridze, N., Japaridze, M., Kurashvili, R., Dundua, M., Kebuladze, G., Kazakhashvili, N., Offley-Shore, B., Thomas, B., Ghebremeskel, K., Crawford, M., Lowy, C., Eriksson, Ulf J., Martin Simán, C., Wisse, Bert, Gittenberger-de Groot, Adriana C., Wentzel, P., Eriksson, U. J., Wender-Ożegowska, E., Drews, K., Biczysko, R., Bronisz, A., Rość, D., Graczykowska-Koczorowska, A., Kotschy, M., Sokup, A., Kohnert, K. D., Besch, W., Strese, J., Frick, U., Zander, E., Kemer, W., Škrha, J., Kvasnička, J., Kalvodová, B., Hilgertová, J., Schatteman, K., Goossens, F., Scharpé, S., De Leeuw, I., Hendriks, D., Legakis, I. N., Panayiotou, D., Mountokalakis, Th. D., Enderle, M. D., Beckmann, P., Balletshofer, B., Rittig, K., Maerker, E., Volk, A., Meisner, C., Jacob, S., Matthaei, S., Häring, H. U., Rett, K., Ueda, K., Nakagawa, T., Shimajiri, Y., Kokawa, M., Matsumoto, E., Sasaki, H., Sanke, T., Nanjo, K., McKinnon, Caroline M., Macfarlane, Wendy M., Docherty, Kevin, Furukawa, N., Shirotani, T., Kishikawa, H., Kaneko, K., Araki, E., Shichiri, M., Prentki, M., Roduit, R., Susini, S., Buteau, J., Ejrnæs, A. M., Andersen, N. Aa., Osterhoff, M., Möhlig, M., Ortmann, J., Bikashaghi, F., Mayer, C., Bikashagi, F., Ackermans, M. T., Pereira Arias, A. M., Bisschop, P. H. L. T., Endert, E., Sauerwein, H. P., Romijn, J. A., Gastaldelli, A., Baldi, S., Pettiti, M., Natali, A., Frascerra, S., Camastra, S., Toschi, E., Ferrannini, E., Stingl, H., Krssak, M., Bischof, M. G., Krebs, M., Fürnsinn, C., Nowotny, P., Waldhäusl, W., Roden, M., Neeft, M., Meijer, A. 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40. 35th Annual Meeting of the European Association for the Study of Diabetes : Brussels, Belgium, 28 September-2 October 1999
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Melander, A., Olsson, J., Lindberg, G., Salzman, A., Howard, T., Stang, P., Lydick, E., Emslie-Smith, A., Boyle, D. I. R., Evans, J. M. M., Macdonald, T. M., Bain, J., Sullivan, F., Ad Darts Memo, Morris For The Collaboration, Juhl, C., Porksen, N., Sturis, J., Hollingdal, M., Pincus, S., Veldhuis, J., Dejgaard, A., Schmitz, O., Kristensen, J. S., Frandsen, K. B., Bayer, Th, Muller, P., Dunning, B. E., Paladini, S., Gutierrez, C., Deacon, R., Valentin, M., Grunberger, G., Weston, W. M., Patwardhan, R., Rappaport, E. B., Sargeant, L. A., Wareham, N. J., Khaw, K. T., Zethelius, Bjorn, Lithell, Hans, Hales, C. Nicholas, Berne, Christian, Lakka, H-M, Oksanen, L., Tuomainen, T-P, Kontula, K., Salonen, J. T., Dekker, J. M., Boks, P., Vegt, F., Stehouwer, C. D. A., Nijpels, G., Bouter, L. M., Heine, R. J., Bruno, G., Cavallo-Perin, P., Bargero, G., D Errico, N., Borra, M., Macchia, G., Pagano, G., Newton, R. W., Ruta, D. A., New, J. P., Wallace, C., Roxburgh, M. A., Young, R. J., Vaughan, N. J. A., Elliott, P., Brennan, G., Devers, M., Macalpine, R., Steinke, D., Lawson, D. H., Decallonne, B., Casteels, K., Gysemans, C., Bouillon, R., Mathieu, C., Linn, Thomas, Strate, Christine, Schneider, Kerstin, Funda, D. P., Jirsa, M. Jr, Kozakova, H., Kaas, A., Kofronova, O., Tlaskalova-Hogenova, H., Buschard, K., Wanka, H., Hartmann, A., Kuttler, B., Rasmussen, S. B., Sorensen, T. S., Markholst, H., Petersen, J. S., Karounos, D., Dyrberg, T., Mabley, J. G., Hasko, G., Szabo, C., Seissler, J., Nguyen, T. B. T., Steinbrenner, H., Scherbaum, W. A., Cipriani, R., Gabriele, A., Sensi, M., Guidobaldi, L., Pantellini, F., Cerrito, M. G., Scarpa, S., Di Mario, U., Morano, S., Ceolotto, G., Iori, E., Baritono, E., Del Prato, S., Semplicini, A., Trevisan, R., Zerbini, G., Meregalli, G., Asnaghi, V., Tentori, F., Maestroni, A., Mangili, R., Marescotti, C., Vedovato, M., Tiengo, A., Tadjieva, J., Mankovsky, B. N., Aken, S., Raes, A., Vande Walle, J., Matthys, D., Craen, M., Hansen, H. P., Lund, S. S., Rossing, P., Jensen, T., Parving, H-H, Genediab, Study Group, Andersen, S., Tarnow, L., Hansen, B. V., Trautner, C., Haastert, B., Ennenbach, N., Willich, S., Tabak, A. Gy, Orchard, T. J., Spranger, J., Preissner, K. T., Schatz, H., Pfeiffer, A., Canton, A., Burgos, R., Hernandez, C., Lecube, A., Mesa, J., Segura, R. M., Mateo, C., Simo, R., Fathallah, L., Greene, D. A., Obrosova, I., Gilbert, R. E., Kelly, D. J., Cox, A. J., Berka-Wilkinson, J. L., Taylor, H. R., Panagiotopoulos, S., Lee, V., Jerums, G., Cooper, M. E., Hitman, G. A., Aganna, E., Ogunkolade, W. B., Rema, M., Deepa, R., Shanthi-Rani, C. S., Barakat, K., Kumarajeewa, T. R., Cassell, P. G., Mcdermott, M. F., Mohan, V., Ways, K., Bursell, S., Devries, T., Woodworth, J., Alatorre, C., King, G., Aiello, L. P., Karisen, A. E., Pavlovic, D., Nielsen, K., Jensen, J., Andersen, H. U., Pociot, F., Mandrup-Poulsen, T., Eizirik, D. L., Nerup, J., Lortz, S., Tiedge, M., Lenzen, S., Lally, F. J., Bone, A. J., Darville, M. I., Ho, Y-S, Sternesjo, J., Sandler, S., Chen, M-C, Schuit, F., Pipeleers, D. G., Merezak, S., Hardikar, A., Hoet, J. J., Remacle, C., Reusens, B., Breant, B., Garofano, A., Czernichow, P., Kubota, N., Terauchi, Y., Miki, H., Tamemoto, H., Yamauchi, T., Nakano, R., Komeda, K., Eto, K., Tobe, K., Kimura, S., Kadowaki, T., Ide, T., Murakami, K., Tsunoda, M., Mochizuki, T., Ozanne, S. E., Nave, B. T., Wang, C. L., Dorling, M. W., Petry, C. J., Koopmans, S. J., Bent, C., Que, I., Radder, J. K., Sebokova, E., Sana, A. K., Klimes, I., Ruderman, N., Morviducci, L., Pastore, L., Morelli, S., Sagratella, E., Zorretta, D., Buongiomo, A., Tamburrano, G., Giaccari, A., Martinenghi, Sabina, Angelis, Gabriella Cusella, Ravasi, Flavio, Bifari, Francesco, Bordignon, Claudio, Falqui, Luca, Kessler, A., Dransfeld, O., Sasson, S., Tomas, E., Zorzano, A., Eckel, J., Thorsby, P., Rosenfalck, A. M., Kjems, L., Hanssen, K. F., Madsbad, S., Birkeland, K. I., Hamilton-Wessler, M., Markussen, J., Bergman, R. N., Melki, V., Hanaire-Broutin, H., Bessieres-Lacombe, S., Gedec, Study Group, Tauber, J-P, Home, P. D., Lindholm, A., Riis, A., European Insulin Aspart Study Group, Rosenstock, J., Schwartz, S., Clark, C., Edwards, M., Donley, D., Us Dm, Study Group Of Insulin Glargine In Type, Swift, P., Mortensen, H. B., Lynggaard, H., Hougaard, P., Hvidore Study group on Childhood Diabetes, Cull, C. A., Neil, H. A. W., Frighi, V., Manley, S. E., Holman, R. R., Turner, R. C., Ukpds, Group, Steiner, G., Dais, Project Group, Davis, W. A., Weeraratna, T., Bruce, D. G., Davis, T. M. E., Verges, B., Duvillard, L., Pont, F., Florentin, E., Gambert, Ph, Benko, B., Ljubic, S., Turk, Z., Granic, M., Marz, W., Wollschlager, H., Klein, G., Neiss, A., Wehling, M., Huxtable, S. J., Saker, P. J., Walker, M., Frayling, T. M., Levy, J. C., O Rahilly, S., Hattersley, A. T., Mccarthy, M. 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T., Kardos, A., Gingl, Z., Kempler, P., Rudas, L., Lonovics, J., Marchand, M., Stevens, L. K., Tarnas, Gy, Eurodiab Iddm, Study Group, Estrella, F., Christensen, N. J., Keresztes, K., Barna, I., Hermanyi, Zs, Vargha, P., Bonnevie, L., Chanudet, X., Larroque, P., Tutuncu, N. Bascil, Deger, A., Batur, M. K., Yildirir, A., Onalan, O., Aksoyek, S., Kabakciota, G., Erbas, T., Galicka-Latala, D., Surdacki, A., Gerritsen, J., Tenvoorde, B. J., Heethaar, R. M., Tagawa, T. S., Kodama, M., Yoshioka, R., Yamasaki, Y., Didangelos, T., Athyros, V., Kontopoulos, A., Papageorgiou, A., Karamitsos, D., Lacigova, S., Rusavy, Z., Karova, R., Perrild, H., Kay, L., Jorgensen, T., Bien, A. I., Witek, P., Geraldes, Elizabete, Rodrigues, D., Pereira, L., Domenech, A., Leitao, P., Anagnostopoulos, D., Foster, A. V. M., Nag, S., Barsoum, M., Lewis, G., Dunlop, N., Connolly, V., Bilous, R., Kelly, W., Chantelau, E., Gede, A., Sharman, D., O Halloran, D., Best, C., Abbas, Z. G., Lutale, J., Gill, G. V., Jarvis, W. R., Archibald, L. K., Corcoran, S., Mansell, J., Pibworth, L., Terada, H., Shiba, T., Utugi, N., Utugi, T., Blum, M., Strobel, J., Hoffken, K., Razvi, F. M., Kritzinger, E. E., Taylor, K., Jones, S., Illahi, W., Grubetaer, M., Hartmann, P., Hoffstadt, K., Leiden, H. A., Moll, A. C., Polak, B. C. P., Pietragalla, G. B., Maurino, M., Montanaro, M., Karadeniz, S., Tommasini, P., Quadrini, C., Demiraj, V., Rispoli, E., Ota, A., Takama, H., Saito, N., Hemandez, C., Lepore, D., Antico, L., Giardina, B., Franconi, F., Michoud, E., Chamot, S., Riva, Ch, Hammes, H-P, Renner, O., Breier, G., Lin, J., Alt, A., Betzholtz, C., Bretzel, R. G. Rd, Manti, R., Gallo, M., Molinar Hin, A., Brignardello, E., Boccuzzi, G., Li, Shanfang, Xiang, Kunsan, Zhang, Rugeng, Shangguan, Xinhong, Wu, Jianrong, Donnan, P. T., Broomhall, J., Hunter, K., Morris, A. D., Darts Memo, Collaboration, Ioannidis, G., Peppa, M., Rontogianni, E., Kallifronas, M., Lekatsas, I., Chrysanthopoulou, G., Anthopoulos, L., Kesse, M., Thalassinos, N., Neves, C., Medina, J. L., Lopes, F., Guvener, N., Guvener, M., Kocagoz, T., Boke, E., Pasaoglu, I., Bascil Tutuncu, N., Oto, A., Karvonen, M. K., Koulu, M., Pesonen, U., Mercuri, M., Rauramaa, R., Pittsburgh Epidemiology of Diabetes Complications (EDC) Study, Rutter, M. K., Kestevan, P., Mccomb, J. M., Marshall, S. M., Sobieska, M., Wiktorowicz, K., Kanters, S. D. J. M., Banga, J. D., Algra, A., Frijns, C. J. M., Beutler, J. J., Fijnheer, R., Nicoloff, G., Baydanoff, S., Stanimirova, N., Petrova, Ch, Lario, S., Campistol, J. M., Cases, A., Claria, J., Inigo, P., Esmatjcs, E., Sarman, B., Toth, M., Kocsis, I., Somogyi, A., Bumbure, A., Jachimowicz, K., Samson, J., Tomasiak, M., Sobol, A., Stanczyk, L., Watala, C., Stradina, P., Wisniewska-Jarosinska, M., Marciniak, D., Wieclawska, B., Golanski, J., Zinnat, R., Mahmud, I., Buyukasik, Yahya, Demiroglu, H., Szczepanik, A., Skowronski, M., Murawska, A., Meeking, D. R., Allard, S., Munday, J., Chowienczyk, P., Shaw, K. M., Cummings, M. H., Simkova, R., Jirsa, M., Hadoke, P. W. F., Mcintyre, C. A., Jones, G. C., Williams, B. C., Elliott, A. I., Mcknight, J. A., Pernow, J., Bombonato, G. C., Finucci, G. F., Zotta, L., Senses, V., Ozyazgan, S., Ince, E., Tuncdemir, M., Sultuybek, G., Akkan, A. G., Unlucerci, Y., Bekpinar, S., Meyer, M. F., Lee, B. C., Shore, A. C., Humphreys, J. M., Tooke, J. E., Omo, G., Giovannitti, G., Caricato, F., Mariani, M., Pedrinelli, R., Kiviet-Boehm, C., Schwelling, V., Pfohl, M., Mcinerney, D., Itoh, H., Ohno, T., Katoh, N., Baumgartner-Parzer, S., Artwohl, M., Graier, W., Ludwig, C., Tachi, Y., Bannai, C., Shinohara, M., Shimpuku, H., Ohura, K., Bertacca, A., Sasvari, M., Szaleczki, E., Pusztai, P., Boes, U., Klaus, E., Dittrich, P., Wagner, Z., Wittmann, I., Poto, L., Wagner, L., Mazak, I., Nagy, J., Feletto, F., Taboga, C., Tonutti, L., Lizzio, S., Russo, A., Selmo, V., Ceriello, A., Lekakis, J., Papamichael, C. M., Stamatelopoulos, K., Stamatelopoulos, S., Yillar, D. O., Gay, M., Lillaz, E., Passaro, A., Vanini, A., Calzoni, F., D Elia, K., Carantoni, M., Zuliani, G., Fellin, R., Solini, A., Chwatko, G., Bald, E., Dramais, A-S, Wallemacq, P. E., Vandeleene, B., Ciaria, M. V., Ariano, M., Strom, R., Gibney, J., Weiss, U., Turner, B., O Gorman, P., Watts, G., Powrie, J., Crook, M., Shaw, K., and Cummings, M. more...
41. Abnormal circulating levels of matrix metalloproteinases and their inhibitors in diabetes mellitus
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Zayani Y, Monia Allal-Elasmi, Mp, Jacob, Zidi W, Ftouhi B, Feki M, Slimane H, and Kaabachi N
42. Bone Mineral Density in Sheehan's Syndrome; Prevalence of Low Bone Mass and Associated Factors.
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Chihaoui M, Yazidi M, Chaker F, Belouidhnine M, Kanoun F, Lamine F, Ftouhi B, Sahli H, and Slimane H
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- Absorptiometry, Photon methods, Adult, Aged, Bone Density, Bone Diseases, Metabolic diagnostic imaging, Female, Femur Neck diagnostic imaging, Humans, Lumbar Vertebrae diagnostic imaging, Middle Aged, Osteoporosis diagnostic imaging, Retrospective Studies, Risk Factors, Bone Diseases, Metabolic etiology, Hypopituitarism complications, Osteoporosis etiology
- Abstract
Hypopituitarism is a known cause of bone mineral loss. This study aimed to evaluate the frequency of osteopenia and osteoporosis in patients with Sheehan's syndrome (SS) and to determine the risk factors. This is a retrospective study of 60 cases of SS that have had a bone mineral density (BMD) measurement. Clinical, biological, and therapeutic data were collected. The parameters of osteodensitometry at the femoral neck and the lumbar spine of 60 patients with SS were compared with those of 60 age-, height-, and weight-matched control women. The mean age at BMD measurement was 49.4 ± 9.9 yr (range: 25-76 yr). The mean duration of SS was 19.3 ± 8.5 yr (range: 3-41 yr). All patients had corticotropin deficiency and were treated with hydrocortisone at a mean daily dose of 26.3 ± 4.1 mg. Fifty-seven patients (95%) had thyrotropin deficiency and were treated with thyroxine at a mean daily dose of 124.3 ± 47.4 µg. Thirty-five of the 49 patients, aged less than 50 yr at diagnosis and having gonadotropin deficiency (71.4%), had estrogen-progesterone substitution. Osteopenia was present in 25 patients (41.7%) and osteoporosis in 21 (35.0%). The BMD was significantly lower in the group with SS than in the control group (p < 0.001). The odds ratio of osteopenia-osteoporosis was 3.1 (95% confidence interval: 1.4-6.8) at the femoral neck and 3.7 (95% confidence interval: 1.7-7.8) at the lumbar spine. The lumbar spine was more frequently affected by low bone mineral mass (p < 0.05). The duration of the disease and the daily dose of hydrocortisone were independently and inversely associated with BMD at the femoral neck. The daily dose of thyroxine was independently and inversely associated with BMD at the lumbar spine. Estrogen-progesterone replacement therapy was not associated with BMD. Low bone mineral mass was very common in patients with SS. The lumbar spine was more frequently affected. The duration of the disease and the doses of hydrocortisone and thyroxine were involved in bone mineral loss., (Copyright © 2016 International Society for Clinical Densitometry. Published by Elsevier Inc. All rights reserved.) more...
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- 2016
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43. Inflammations mediators and circulating levels of matrix metalloproteinases: Biomarkers of diabetes in Tunisians metabolic syndrome patients.
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Zayani Y, El Golli N, Zidi W, Guizani I, Boussairi S, Aloui S, Ayadi I, Ftouhi B, Feki M, Ben Romdhane N, Ben Sliman H, Allal-Elasmi M, and Kaabachi N
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- Adult, Aged, C-Reactive Protein metabolism, Cardiovascular Diseases blood, Cardiovascular Diseases diagnosis, Case-Control Studies, Diabetes Mellitus, Type 2 blood, Diabetes Mellitus, Type 2 ethnology, Female, Humans, Inflammation blood, Male, Matrix Metalloproteinase 9 blood, Metabolic Syndrome ethnology, Middle Aged, ROC Curve, Tissue Inhibitor of Metalloproteinase-1 blood, Tissue Inhibitor of Metalloproteinase-2 blood, Tumor Necrosis Factor-alpha blood, Biomarkers blood, Diabetes Mellitus, Type 2 diagnosis, Inflammation Mediators blood, Matrix Metalloproteinases blood, Metabolic Syndrome blood
- Abstract
Aims: This study investigates the relationships between matrix metalloproteinases, inflammations mediators and type 2 diabetes mellitus in Tunisians metabolic syndrome (Mets) patients., Methods: The study has included 239 MetS patients and 247 controls. Mets was defined according to the NCEP-ATPIII report. Mets patients were also divided into two categories: 29 MetS non-diabetics and 210 MetS diabetics. Dysglycemia markers, matrix metalloproteinase-9 (MMP-9), Tissue inhibitors of metalloproteinases (TIMP-1 and TIMP-2), tumor necrosis factor α (TNF-α), C-reactive protein (CRP) levels and White Blood Cells (WBC) counts were determined in patients and controls., Results: In our study, the level of inflammatory markers WBC, TNF-α and matrix metalloproteinases (MMP-8 and MMP-9) were significantly higher in diabetic patients with MetS, as compared with non-diabetic MetS patients. Inflammation mediators and MMP-9 were significantly associated with many clinical characteristics of MetS. The use of ROC "Receiver Operating Characteristic" analysis revealed the impact of TNF alpha on diabetes patients with MetS. In fact TNF alpha was found as a sensitive parameter in these patients with a sensitivity of 85%., Conclusion: Inflammation, matrix metalloproteinases and dysglycemia markers are not expressed in isolation but rather concurrently and are continuously interacting with each other, in MetS and diabetics patients. These markers fit with an early stage of cardiovascular disease (CVD); and measuring them could improve the risk evaluation, an early diagnosis, and the prognosis of CVD., (Copyright © 2016 Elsevier Ltd. All rights reserved.) more...
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- 2016
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44. Lack of association between FokI polymorphism in vitamin D receptor gene ( VDR ) & type 2 diabetes mellitus in the Tunisian population.
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Mahjoubi I, Kallel A, Sbaï MH, Ftouhi B, Ben Halima M, Jemaa Z, Feki M, Slimane H, Jemaa R, and Kaabachi N
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- Adult, Diabetes Mellitus, Type 2 pathology, Female, Gene Frequency genetics, Genetic Predisposition to Disease, Genotype, Humans, Male, Middle Aged, Polymorphism, Single Nucleotide, Tunisia, Diabetes Mellitus, Type 2 genetics, Genetic Association Studies, Receptors, Calcitriol genetics
- Abstract
Background & Objectives: The impact of several environmental and genetic factors on diabetes is well documented. Though the association between the vitamin D receptor (VDR) gene polymorphisms and type 2 diabetes mellitus (T2DM) has been analyzed in different ethnic groups, the results have been inconsistent. The aim of this study was to evaluate the possible association between VDR FokI polymorphism and genetic susceptibility to T2DM in Tunisian population., Methods: A total of 439 unrelated patients with T2DM and 302 healthy controls were included in the study. Genomic DNA was extracted from blood and genotyped for the single nucleotide polymorphism (SNP) of FokI (T/C: (rs2228570) by polymerase chain reaction and restriction fragment length polymorphism (PCR-RFLP) analysis., Results: The genotype distribution and the relative allelic frequencies for the FokI polymorphism were not significantly different between T2DM and controls: in T2DM patients the frequencies of the CC, CT, and TT genotypes were 52.6, 41.0, and 6.1 per cent, respectively, and in controls the genotype frequencies were 55.6, 38.7, and 5.6 per cent, respectively. In our study, the TT genotype of the FokI polymorphism was not associated with T2DM (OR =1.19, 95% CI 0.63 - 2.25, P=0.577)., Interpretation & Conclusions: Our study showed no significant association of the FokI polymorphism in the vitamin D receptor gene with type 2 diabetes mellitus in Tunisian population., Competing Interests: None. more...
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- 2016
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45. The Gly482Ser polymorphism of the peroxisome proliferator-activated receptor-γ coactivator-1α (PGC-1α) is associated with type 2 diabetes in Tunisian population.
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Jemaa Z, Kallel A, Sleimi C, Mahjoubi I, Feki M, Ftouhi B, Slimane H, Jemaa R, and Kaabachi N
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- Adult, Case-Control Studies, Female, Follow-Up Studies, Genotype, Humans, Male, Middle Aged, Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha, Polymerase Chain Reaction, Polymorphism, Restriction Fragment Length, Prognosis, Risk Factors, Tunisia epidemiology, Biomarkers metabolism, Diabetes Mellitus, Type 2 epidemiology, Diabetes Mellitus, Type 2 genetics, Polymorphism, Single Nucleotide genetics, Transcription Factors genetics
- Abstract
Background and Aims: Peroxisome proliferator-activated receptor-γ co-activator-1α (PGC-1α) is a transcriptional co-activator involved in adaptive thermogenesis, skeletal muscle metabolism, fatty acid oxidation, and gluconeogenesis. Several studies have suggested that the common PGC-1α polymorphism Gly482Ser (rs8192678) may be associated with risk of type 2 diabetes (T2D), with conflicting results. The aim of this study was to analyze whether the Gly482Ser variant is a risk factor for development of T2D in Tunisian population., Methods: In a case-control study 487 unrelated patients with type 2 diabetes and 402 apparently healthy controls were recruited from January 2008 to August 2010. The Gly482Ser polymorphism was determined by PCR-RFLP analysis., Results: A significant difference in genotypes distribution was observed between patients (Gly/Gly: 34.1%; Gly/Ser: 47.1%; Ser/Ser: 18.5%) and controls (Gly/Gly: 43.8%; Gly/Ser: 42.3%; Ser/Ser: 13.9%) (χ(2)=9.44, p=0.009). The T2D patient group showed a significant higher frequency of the Ser allele compared to the controls (43% vs. 34%; OR: 1.35, 95% [CI]: 1.11-1.65, p=0.002). The association between the Gly482Ser polymorphism and T2D remained significant after adjustment for other well-established cardiovascular risk factors., Conclusions: In the current study, a significant and independent association between the Gly482Ser polymorphism (rs8192678) of the PGC-1α gene and T2D in the Tunisian population was found., (Copyright © 2013 Diabetes India. Published by Elsevier Ltd. All rights reserved.) more...
- Published
- 2015
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46. Tumor necrosis factor-α (TNF-α) -863C/A promoter polymorphism is associated with type 2 diabetes in Tunisian population.
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Kallel A, Ftouhi B, Jemaa Z, Mahjoubi I, Feki M, Slimane H, Jemaa R, and Kaabachi N
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- Adult, Aged, Case-Control Studies, Diabetes Mellitus, Type 2 epidemiology, Female, Gene Frequency, Genotype, Humans, Insulin Resistance genetics, Logistic Models, Male, Middle Aged, Polymerase Chain Reaction, Polymorphism, Restriction Fragment Length, Tumor Necrosis Factor-alpha metabolism, Tunisia epidemiology, Diabetes Mellitus, Type 2 genetics, Polymorphism, Genetic, Promoter Regions, Genetic genetics, Tumor Necrosis Factor-alpha genetics
- Abstract
Aims: Tumor necrosis factor α (TNFα) plays a key role in orchestrating the complex events involved in inflammation and immunity. Accordingly, TNF α has been implicated in a wide range of autoimmune and infectious diseases, but also in conditions such as obesity and insulin resistance. The aim of the present study was to investigate the association between the -863C/A polymorphism in the promoter of the TNFα gene and type 2 diabetes in the Tunisian population., Methods: The polymorphism -863C/A in the TNFα gene was determined in 211 type 2 diabetes patients and 345 healthy controls using the Polymerase Chain Reaction-Restriction Fragment Length Polymorphism (PCR-RFLP) analysis., Results: A significant difference in genotype distribution and allele frequency was observed between patients and controls. Patients with type 2 diabetes had significantly higher frequency of the CA+AA genotypes compared to controls [35.5% vs. 22.3%; OR (95%CI), 1.91 (1.31-2.8); p=0.001]. The type 2 diabetes patient group showed a significant higher frequency of the A allele compared to the controls (0.19 vs. 0.11; p=0.001). After adjustment by a stepwise logistic regression method, hypertension, dyslipidemia, and CA+AA genotype were found to be significantly associated with T2D., Conclusion: The present study showed a significant and independent association between the -863C/A polymorphism of the TNFα gene and type 2 diabetes in the Tunisian population., (Copyright © 2013 Elsevier Ireland Ltd. All rights reserved.) more...
- Published
- 2013
- Full Text
- View/download PDF
47. Adiponectin and metabolic syndrome in a Tunisian population.
- Author
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Ben Ali S, Jemaa R, Ftouhi B, Kallel A, Feki M, Slimene H, and Kaabachi N
- Subjects
- Adult, Aged, Body Height, Body Mass Index, Body Weight, Female, Humans, Insulin Resistance, Male, Metabolic Syndrome diagnosis, Middle Aged, Tunisia, Young Adult, Adiponectin blood, Metabolic Syndrome blood
- Abstract
The aim of this study was to investigate the relationship between the adiponectin levels and various characteristics of the metabolic syndrome (MS) in a sample of the Tunisian population. Three hundred and fifty-four individuals were included in this study. Body mass index, blood pressure, HDL-cholesterol, triglycerides, glucose, insulin, and adiponectin concentrations were measured. Insulin resistance was assessed by homeostasis model assessment of insulin resistance (HOMA-IR). MS was identified with the NCEP-ATP III criteria. Subjects with MS showed significantly lower adiponectin levels compared to those without MS. For both genders, the prevalence and the number of MS components increased significantly as the adiponectin concentrations decreased. Subjects with the lowest adiponectin quartile had an increased risk of MS adjusted for age, gender, and HOMA-IR. Our findings suggest that hypoadiponectinemia is strongly associated with the risk of MS independent of insulin resistance. more...
- Published
- 2012
- Full Text
- View/download PDF
48. Abnormal circulating levels of matrix metalloproteinases and their inhibitors in diabetes mellitus.
- Author
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Zayani Y, Allal-Elasmi M, Jacob MP, Zidi W, Ftouhi B, Feki M, Slimane H, and Kaabachi N
- Subjects
- Adult, Case-Control Studies, Diabetes Mellitus, Type 2 blood, Enzyme-Linked Immunosorbent Assay, Female, Humans, Male, Middle Aged, Diabetes Mellitus, Type 2 enzymology, Matrix Metalloproteinases blood, Tissue Inhibitor of Metalloproteinase-1 blood, Tissue Inhibitor of Metalloproteinase-2 blood
- Abstract
Background: The aim of this study was to determine plasma levels of matrix metalloproteinases (MMPs) 2, 3, and 9 and their tissue inhibitors (TIMPs) 1 and 2 in type 2 diabetic patients (T2DM) compared to healthy subjects., Methods: The study involved 54 patients with T2DM and 57 age and gender matched healthy adults as controls. MMPs 2 and 9 were analyzed by gelatin zymography and MMP-3 and TIMPs 1 and 2 by ELISA., Results: For technical feasibility, MMPs 2 and 9 were expressed in pro forms. Pro-MMP-9 was significantly higher (p < 0.05), whereas TIMP-1 and TIMP-2 levels were significantly decreased (p < 0.01) in patients with T2DM compared to controls. The MMP-3/TIMP-1 and the MMP-3/TMP-2 ratios were significantly higher in T2DM patients than controls (p < 0.05). Fasting plasma glucose was inversely correlated with TIMP-1 (r = -0.412, p < 0.01) and TIMP-2 (r = -0.315, p < 0.001), but was not associated with MMPs., Conclusions: The present study identified abnormalities in plasma markers for extracellular matrix metabolism in T2DM. The new parameters would constitute an effective approach to explore the complications of uncontrolled diabetes. more...
- Published
- 2012
49. Relationship of plasma leptin and adiponectin concentrations with menopausal status in Tunisian women.
- Author
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Ben Ali S, Jemaa R, Ftouhi B, Kallel A, Feki M, Slimene H, and Kaabachi N
- Subjects
- Adult, Body Mass Index, Case-Control Studies, Female, Humans, Middle Aged, Tunisia, Adiponectin blood, Leptin blood, Menopause
- Abstract
To evaluate the effect of menopausal status and body mass index (BMI) on circulating leptin and adiponectin concentrations and investigate whether there is an influence of menopausal transition on the relationships of these adipokines and leptin to adiponectin (L/A) ratio with lipid profile and insulin resistance in a sample of Tunisian women. One hundred ninety-six premenopausal (mean age 35.3±7.6 years) and 180 postmenopausal women (mean age 53.4±6.2 years) were included in the study. Participants were stratified into obese and normal weight groups based upon their baseline BMI. Fasting glucose, HDL-cholesterol (HDL-C), triglycerides (TG), total cholesterol (TC), insulin, leptin, and adiponectin concentrations were measured. Homeostasis model assessment of insulin resistance (HOMA-IR) was calculated. Premenopausal women had significantly higher leptin and L/A ratio and lower adiponectin levels than postmenopausal women. Menopause had no effect on the mean values of BMI, insulin or HOMA-IR, HDL-C, and TG. Using a multiple linear regression model, menopausal status was identified, as significant independent predictor for leptin and adiponectin levels. Irrespective of the menopausal status, obese women exhibited higher leptin and L/A ratio and lower adiponectin levels compared to those with normal weight. Comparison between the two menopausal stages in obese and normal weight groups showed that leptin and L/A ratio decreased, while adiponectin increased from pre- to postmenopausal stage only in obese group. The L/A ratio correlated better with lipid profile and HOMA-IR in postmenopausal stage. The present study showed a significant interaction between menopause and BMI on leptin and adiponectin secretion. Menopausal transition affects the relationships of these adipokines with lipids and insulin resistance., (Copyright © 2011 Elsevier Ltd. All rights reserved.) more...
- Published
- 2011
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50. Gender-specific effect of Pro12Ala polymorphism in peroxisome proliferator-activated receptor gamma-2 gene on obesity risk and leptin levels in a Tunisian population.
- Author
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Ben Ali S, Ben Yahia F, Sediri Y, Kallel A, Ftouhi B, Feki M, Elasmi M, Haj-Taieb S, Souheil O, Sanhagi H, Slimane H, Jemaa R, and Kaabachi N
- Subjects
- Case-Control Studies, Female, Humans, Male, Risk Factors, Tunisia, Alanine genetics, Leptin blood, Obesity genetics, PPAR gamma genetics, Proline genetics, Sex Factors
- Abstract
Objectives: This study was undertaken to investigate the impact of the Pro12Ala (rs1801282) polymorphism of the peroxisome proliferator-activated receptor gamma-2 (PPARgamma-2) gene on obesity or body mass index (BMI) and plasma leptin, insulin, adiponectin and lipid levels in a sample of the Tunisian population., Design and Methods: The study included 387 obese patients and 288 control subjects. The Pro12Ala genotype was determined by polymerase chain reaction followed by a digestion with the restriction of endonuclease BstUI., Results: In the whole population, there is no significant difference in genotype frequencies of the Pro12Ala polymorphism between obese patients and controls. However, separate analysis by gender revealed that obese men (but not women) had significantly higher frequency of Pro/Ala genotypes compared to controls (12.2% vs. 4.1%; chi(2)=6.76, p=0.009). In comparison to Pro/Pro homozygotes, Ala-allele bearers had a significantly higher risk of obesity [OR (95% CI)=3.26 (1.28-8.33)]. When obese subjects were stratified according to type 2 diabetes status, the association with obesity was only significant in obese non-diabetic patients [OR (95% CI)=3.74 (1.43-9.74), p=0.007]. Additionally, obese male patients carrying the Ala-allele had significantly higher body mass index (p=0.007) and plasma leptin levels (p=0.023) compared to those homozygous for Pro-allele. The significant effect of Pro12Ala polymorphism on plasma leptin levels disappeared after adjustment for age and BMI., Conclusion: The present study provides evidence that the Pro12Ala polymorphism of the PPARgamma-2 gene is associated with obesity in non-diabetic men from Tunisian origin. more...
- Published
- 2009
- Full Text
- View/download PDF
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