74 results on '"Franze, E"'
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2. OC.11.2 RAFOXANIDE INHIBITS EPITHELIAL STAT3 ACTIVATION AND STAT3-DRIVEN ONCOGENIC IMMUNE RESPONSE IN THE COLORECTAL CANCER MICROENVIRONMENT
3. T.02.4 THE FOOD ADDITIVE MALTODEXTRIN INDUCES ENDOPLASMIC RETICULUM STRESS AND APOPTOSIS IN COLORECTAL CANCER CELLS
4. OC.06.6 HEPCIDIN UPREGULATION IN COLORECTAL CANCER ASSOCIATES WITH ACCUMULATION OF REGULATORY MACROPHAGES AND EPITHELIAL–MESENCHYMAL TRANSITION AND CORRELATES WITH PROGRESSION OF THE DISEASE
5. Defective expression of SIRT1 contributes to sustain inflammatory pathways in the gut
6. IL-15 positively regulates IL-21 production in celiac disease mucosa
7. Interleukin-25 production is differently regulated by TNF-α and TGF-β1 in the human gut
8. OC.13.1 SMAD7 REGULATES IMMUNOGENIC DEATH PATHWAY IN COLORECTAL CANCER CELLS
9. OC.13.3 SMAD7 KNOCKDOWN REDUCES THE ACTIVATION OF STAT3 IN COLORECTAL CANCER
10. RECIPROCAL REGULATION BETWEEN SMAD7 AND SIRT1 IN THE GUT
11. T.07.3 HEPCIDIN SUSTAINS COLORECTAL CANCER CELL SURVIVAL
12. PC.01.3 BROMODOMAIN-CONTAINING PROTEIN 4 (BRD4) ENHANCES INTERLEUKIN-34 EXPRESSION IN INFLAMED GUT OF PATIENTS WITH INFLAMMATORY BOWEL DISEASE
13. OC.07.4 RAFOXANIDE INDUCES IMMUNOGENIC DEATH OF COLORECTAL CANCER CELLS
14. OC.06.5 GATA6 DEFICIENCY LEADS TO EPITHELIAL BARRIER DYSFUNCTION AND ENHANCES SUSCEPTIBILITY TO GUT INFLAMMATION
15. INTERLEUKIN-34 INDUCES CC-CHEMOKINE LIGAND 20 IN GUT EPITHELIAL CELLS
16. OC.16.3 THE FRAGILE X MENTAL RETARDATION PROTEIN REGULATES RIP1K AND COLORECTAL CANCER RESISTANCE TO NECROPTOSIS
17. OC.16.2 PROGRANULIN SUSTAINS STAT3 HYPER-ACTIVATION AND ONCOGENIC FUNCTION IN COLORECTAL CANCER CELLS
18. OC.11.3 THE DEUBIQUITINASE OTUD5 ENHANCES INFLAMMATORY CYTOKINE PRODUCTION IN THE GUT OF INFLAMMATORY BOWEL DISEASE PATIENTS
19. OC.11.2 INTERLEUKIN-34 STIMULATES GUT FIBROBLASTS TO PRODUCE COLLAGEN SYNTHESIS
20. PLASMA CELLS IN THE MUCOSA OF PATIENTS WITH INFLAMMATORY BOWEL DISEASE PRODUCE GRANZYME B AND POSSESS CYTOTOXIC ACTIVITIES
21. A FUNCTIONAL ROLE FOR IL-34 IN SUSTAINING INFLAMMATORY PATHWAYS IN IBD
22. TH17-RELATED CYTOKINES AND EXCESSIVE ACTIVATION OF STAT3 SUSTAIN COLORECTAL CANCER CELL GROWTH
23. OC.03.1 REGULATION OF COLON CARCINOGENESIS BY THE ANTI-HELMINTH AGENT RAFOXANIDE
24. LACTOBACILLI CASEI (LC) REDUCE TRYPSIN EXPRESSION IN INTESTINAL CELLS THEREBY LEADING TO INACTIVATION OF PAR2 AND UP-REGULATION OF ZO-1
25. High Expression of ADAM19, a Sheddase for TNF-Alpha, in the Mucosa of Patients With Inflammatory Bowel Disease (IBD)
26. Interleukin-25 production is differently regulatd bt TNF-alpha and TGF beta 1 in the human gut
27. OC.10.1 RECIPROCAL REGULATION BETWEEN SMAD7 AND SIRT1 IN THE GUT
28. OC.04.3 EXPRESSION OF TBET AND RORGAMMA-T IN REGULATORY T CELLS INFLUENCE THE INCIDENCE AND SIZE OF TUMOR MASSES IN A CAC MODEL
29. A functional role for Smad7 in sustaining colon cancer cell growth and survival
30. P.05.1: Interferon Regulatory Factor 5 Expressing Cells Infiltrate Lamina Propria of IBD Patients and Produce Inflammatory Cytokines
31. OC.14.3: PAD4 and Neutrophil Extracellular Traps are Increased in the Inflamed Colon Mucosa of Patients with Ulcerative Colitis
32. OC.01.4: Interleukin-34 Sustains Pro-Tumorigenic Signals in Colon Cancer Tissue
33. OC.01.2: Rorgamma-T-Expressing Regulatory T Cells Sustain Colorectal Cancer Growth Through a Foxo3-Dependent Suppression of Dendritic Cell-Derived IL-6 Production
34. OC.09.8 INTERLEUKIN-34 INDUCES CC-CHEMOKINE LIGAND 20 IN GUT EPITHELIAL CELLS
35. OC.06.1: INHIBITION OF COLITIS BY IL-25 ASSOCIATES WITH INDUCTION OF ALTERNATIVELY ACTIVATED MACROPHAGES
36. P.06.6 PLASMA CELLS IN THE MUCOSA OF PATIENTS WITH INFLAMMATORY BOWEL DISEASE PRODUCE GRANZYME B AND POSSESS CYTOTOXIC ACTIVITIES
37. P.06.5 A FUNCTIONAL ROLE FOR IL-34 IN SUSTAINING INFLAMMATORY PATHWAYS IN IBD
38. OC.05.2 TH17-RELATED CYTOKINES AND EXCESSIVE ACTIVATION OF STAT3 SUSTAIN COLORECTAL CANCER CELL GROWTH
39. OC.03.1 SMAD7 EXPRESSION AND REGULATION BY SIRT1 IN INFLAMMATORY BOWEL DISEASES
40. PC.01.2 SMAD7 SUSTAINS COLON CANCER CELL GROWTH BY MODULATING PP1 AND EIF2-ALPHA TO REGULATE CDC25A
41. P.18.8 IL-25 PREVENTS AND CURES FULMINANT HEPATITIS THROUGH A MYELOID-DERIVED SUPPRESSOR CELL-DEPENDENT MECHANISM
42. P.05.2 DIMINISHED EXPRESSION OF SCAVENGER RECEPTORS ASSOCIATES WITH ACCUMULATION OF APOPTOTIC BODIES IN CELIAC DISEASE MUCOSA
43. P.03.6 CD163-EXPRESSING MACROPHAGES WITH INFLAMMATORY PROPERTIES INFILTRATE HEAVILY THE GUT MUCOSA OF PATIENTS WITH INFLAMMATORY BOWEL DISEASE
44. OC.12.6 CHARACTERIZATION OF INFLAMMATORY CYTOKINE RESPONSE IN HUMAN SPORADIC COLORECTAL CANCER
45. OC.10.5 IN VITRO AND IN VIVO KNOCK-DOWN OF SMAD7 INHIBITS GROWTH AND PROMOTES APOPTOSIS OF COLON CANCER CELLS
46. P.05.11 HIGH EXPRESSION OF THE “A DISINTEGRIN AND METALLOPROTEASE” 19 (ADAM19), A SHEDDASE FOR TNF-α IN THE MUCOSA OF PATIENTS WITH INFLAMMATORY BOWEL DISEASES
47. OC.08.6 2-METHOXY-5-AMINO-N-HYDROXYBENZAMIDE, A DERIVATIVE OF MESALAMINE, INHIBITS COLON CANCER CELL GROWTH THROUGH CYCLOOXYGENASE-2-DEPENDENT AND -INDEPENDENT MECHANISMS
48. OC.03.2 DEFECTIVE EXPRESSION OF THE ANTI-INFLAMMATORY SIRT1 IN INFLAMMATORY BOWEL DISEASES
49. P.1.101: LACTOBACILLI CASEI (LC) REDUCE TRYPSIN EXPRESSION IN INTESTINAL CELLS THEREBY LEADING TO INACTIVATION OF PAR2 AND UP-REGULATION OF ZO-1
50. P.1.33: 2-METHOXY-5-AMINO-N-HYDROXYBENZAMIDE SENSITIZES COLON CANCER (CC) CELLS TO TRAIL THROUGH MODULATION OF DEATH RECEPTOR (DR)5 AND SURVIVIN
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