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1. Detection of Cancer Stem Cells in Normal and Dysplastic/Leukemic Human Blood

2. Molecular taxonomy of myelodysplastic syndromes and its clinical implications

4. A miRNA screening identifies miR-192-5p as associated with response to azacitidine and lenalidomide therapy in myelodysplastic syndromes

5. Deciphering signaling pathways in hematopoietic stem cells: the molecular complexity of Myelodysplastic Syndromes (MDS) and leukemic progression

6. Implications of TP53 allelic state for genome stability, clinical presentation and outcomes in myelodysplastic syndromes

9. Molecular and clinical presentation of UBA1-mutated myelodysplastic syndromes

11. DNA methylation profiling of myelodysplastic syndromes and clinical response to azacitidine: A multicentre retrospective study

12. Inositide-Dependent Nuclear Signalling in Health and Disease

14. Abilities of β-Estradiol to interact with chemotherapeutic drugs, signal transduction inhibitors and nutraceuticals and alter the proliferation of pancreatic cancer cells

15. Nuclear Phospholipids and Signaling: An Update of the Story.

16. Metformin influences drug sensitivity in pancreatic cancer cells

18. Response of high-risk MDS to azacitidine and lenalidomide is impacted by baseline and acquired mutations in a cluster of three inositide-specific genes

19. Nuclear Phosphoinositides as Key Determinants of Nuclear Functions

21. Inositide-Dependent Nuclear Signalling in Health and Disease

22. Abstract 6168: Implementation and adoption of a web tool to support precision diagnostic and treatment decisions for patient with myelodysplastic syndromes

23. Supplementary Table S1 from Prospective Phase II Study on 5-Days Azacitidine for Treatment of Symptomatic and/or Erythropoietin Unresponsive Patients with Low/INT-1–Risk Myelodysplastic Syndromes

24. Supplementary Figure S2 from Prospective Phase II Study on 5-Days Azacitidine for Treatment of Symptomatic and/or Erythropoietin Unresponsive Patients with Low/INT-1–Risk Myelodysplastic Syndromes

26. Author Correction: Implications of TP53 allelic state for genome stability, clinical presentation and outcomes in myelodysplastic syndromes

27. TiMMing: developing an innovative suite of bioinformatic tools to harmonize and track the origin of copy number alterations in the evolutive history of multiple myeloma

28. Nuclear shape instability caused by lamin A deregulation promotes invasiveness in pediatric bone sarcomas: from nucleo-cytoskeleton dynamics to novel therapeutic opportunities

30. DNA Methylation Profiles Capture Clinical and Molecular Heterogeneity of MDS and Can be Harnessed for the Development of Robust Biomarkers Predictive of Response to Azacitidine

31. Incidence, Clinical Associations, and Co-Mutation Patterns of UBA1 Mutations in MDS

32. Venetoclax Rapidly and Strongly Enhances the Phospholipase C Response to Azacitidine Therapy in Myelodysplastic Syndromes

37. Molecular International Prognostic Scoring System for Myelodysplastic Syndromes

38. Demethylation and Up-Regulation of an Oncogene after Hypomethylating Therapy

41. Effects of the Mutant TP53 Reactivator APR-246 on Therapeutic Sensitivity of Pancreatic Cancer Cells in the Presence and Absence of WT-TP53

43. Role of PLCγ1 in the modulation of cell migration and cell invasion in glioblastoma

44. Effects of the MDM2 inhibitor Nutlin-3a on sensitivity of pancreatic cancer cells to berberine and modified berberines in the presence and absence of WT-TP53

45. Role of Mir-192-5p during Response to Azacitidine and Lenalidomide Therapy in Myelodysplastic Syndromes

46. “Modulating Phosphoinositide Profiles as a Roadmap for Treatment in Acute Myeloid Leukemia”

47. Implications of TP53 allelic state for genome stability, clinical presentation and outcomes in myelodysplastic syndromes (vol 26, pg 1549, 2020)

48. Clinical and Molecular Insights in Erythropoiesis Regulation of Signal Transduction Pathways in Myelodysplastic Syndromes and β-Thalassemia

49. Unravelling Heterogeneity of Amplified Human Amniotic Fluid Stem Cells Sub-Populations

50. Location-dependent role of phospholipase C signaling in the brain: Physiology and pathology

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