122 results on '"Folgiero, Valentina"'
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2. GMP-manufactured CRISPR/Cas9 technology as an advantageous tool to support cancer immunotherapy
3. Manipulating the tumor immune microenvironment to improve cancer immunotherapy: IGF1R, a promising target
4. AATF/Che-1 RNA polymerase II binding protein overexpression reduces the anti-tumor NK-cell cytotoxicity through activating receptors modulation
5. Data from Negative Regulation of β4 Integrin Transcription by Homeodomain-Interacting Protein Kinase 2 and p53 Impairs Tumor Progression
6. Data from Transcription Factor NF-Y Induces Apoptosis in Cells Expressing Wild-Type p53 through E2F1 Upregulation and p53 Activation
7. Supplementary Methods from Transcription Factor NF-Y Induces Apoptosis in Cells Expressing Wild-Type p53 through E2F1 Upregulation and p53 Activation
8. Supplementary Figures 1-2 from Transcription Factor NF-Y Induces Apoptosis in Cells Expressing Wild-Type p53 through E2F1 Upregulation and p53 Activation
9. Supplementary Figures 3-4 from Transcription Factor NF-Y Induces Apoptosis in Cells Expressing Wild-Type p53 through E2F1 Upregulation and p53 Activation
10. Supplementary Figures 5-6 from Transcription Factor NF-Y Induces Apoptosis in Cells Expressing Wild-Type p53 through E2F1 Upregulation and p53 Activation
11. Supplementary Table 1 from Negative Regulation of β4 Integrin Transcription by Homeodomain-Interacting Protein Kinase 2 and p53 Impairs Tumor Progression
12. Recent advances in searching c-Myc transcriptional cofactors during tumorigenesis
13. Abstract 3749: Mapping the DNA accessibility landscape of BCP-ALL patients revealed principles of tumor evolution
14. Enhancer plasticity sustains oncogenic transformation and progression of B-Cell Acute Lymphoblastic leukemia.
15. CAPE and its synthetic derivative VP961 restore BACH1/NRF2 axis in Down Syndrome
16. Metastatic Group 3 Medulloblastoma in a Patient With Tuberous Sclerosis Complex: Case Description and Molecular Characterization of the Tumor
17. Enhancement of Neuroblastoma NK-Cell-Mediated Lysis through NF-kB p65 Subunit-Induced Expression of FAS and PVR, the Loss of Which Is Associated with Poor Patient Outcome
18. PI3K/Akt Pathway: The Indestructible Role of a Vintage Target as a Support to the Most Recent Immunotherapeutic Approaches
19. GD2 redirected CAR T and activated NK-cell-mediated secretion of IFNγ overcomes MYCN-dependent IDO1 inhibition, contributing to neuroblastoma cell immune escape
20. Advanced Non-animal Models in Biomedical Research: Breast Cancer
21. Chronic PERK induction promotes Alzheimer-like neuropathology in Down syndrome: Insights for therapeutic intervention
22. Upfront treatment with mTOR inhibitor everolimus in pediatric low‐grade gliomas: A single‐center experience
23. Proteomics Study of Peripheral Blood Mononuclear Cells in Down Syndrome Children
24. Indoleamine 2,3-dioxygenase 1 (IDO1) activity correlates with immune system abnormalities in multiple myeloma
25. Upfront treatment with mTOR inhibitor everolimus in pediatric low-grade gliomas: A single-center experience.
26. A new baby in the c-Myc-directed transcriptional machinery: Che-1/AATF
27. Che‐1 is targeted by c‐Myc to sustain proliferation in pre‐B‐cell acute lymphoblastic leukemia
28. A New Insight into Pediatric Leukemia: Che-1 Involvement in Oncogenic c-Myc Signaling
29. IDO1 involvement in mTOR pathway: a molecular mechanism of resistance to mTOR targeting in medulloblastoma
30. Metastatic Group 3 Medulloblastoma in a Patient With Tuberous Sclerosis Complex: Case Description and Molecular Characterization of the Tumor
31. Indoleamine 2,3-dioxygenase-1 (IDO1) expression by childhood acute myeloid leukemia restrains IFN-{gamma} production by T cells and may portend an unfavorable prognosis
32. Indoleamine 2, 3-Dioxygenase-1 (IDO1) Is Expressed by a Subset of Childhood Acute Myeloid Leukemias and Restrains IFN-gamma Production by T Cells
33. TIM-3/Gal-9 interaction induces IFNγ-dependent IDO1 expression in acute myeloid leukemia blast cells
34. Indoleamine 2,3-dioxygenase 1 (IDO1) activity in leukemia blasts correlates with poor outcome in childhood acute myeloid leukemia
35. A new babyin the c-Myc-directed transcriptional machinery: Che-1/AATF
36. Indoleamine 2,3-dioxygenase-1 (IDO1) expression by childhood acute myeloid leukemias inhibits T-cell production of IFN-γ and confers an unfavorable prognosis
37. Indoleamine 2,3-dioxygenase 1 (IDO1) activity in leukemia blasts correlates with poor outcome in childhood acute myeloid leukemia
38. Indoleamine 2,3-dioxygenase-1 (IDO1) expression by childhood acute myeloid leukemia restrains IFN-γ production by T cells and may portend an unfavorable prognosis (P2157)
39. Indoleamine 2,3-Dioxygenase-1 (IDO1) Is Expressed by a Subset of Childhood Acute Myeloid Leukemias and Restrains IFN-γ Production by T Cells
40. HIPK2 downregulates vimentin and inhibits breast cancer cell invasion
41. Transcription Factor NF-Y Induces Apoptosis in Cells Expressing Wild-Type p53 through E2F1 Upregulation and p53 Activation
42. Purification and Characterization of Adipose-Derived Stem Cells from Patients with Lipoaspirate Transplant
43. Negative Regulation of β4 Integrin Transcription by Homeodomain-Interacting Protein Kinase 2 and p53 Impairs Tumor Progression
44. Induction of ErbB-3 Expression by α6β4 Integrin Contributes to Tamoxifen Resistance in ERβ1-Negative Breast Carcinomas
45. The α6β4 Integrin Can Regulate ErbB-3 Expression: Implications for α6β4 Signaling and Function
46. Loss of β4 Integrin Subunit Reduces the Tumorigenicity of MCF7 Mammary Cells and Causes Apoptosis upon Hormone Deprivation
47. Involvement of α6β4 integrin in the mechanisms that regulate breast cancer progression
48. Involvement of α6β4integrin in the mechanisms that regulate breast cancer progression
49. Indoleamine 2,3-dioxygenase 1 (IDO1) activity in leukemia blasts correlates with poor outcome in childhood acute myeloid leukemia.
50. Negative regulation of beta4 integrin transcription by homeodomain-interacting protein kinase 2 and p53 impairs tumor progression.
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