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2. Che-1/AATF-induced transcriptionally active chromatin promotes cell proliferation in multiple myeloma

3. Che‐1‐induced inhibition of mTOR pathway enables stress‐induced autophagy

5. Che-1/AATF, a multivalent adaptor connecting transcriptional regulation, checkpoint control, and apoptosis (1)

6. Che-1 phosphorylation by ATM/ATR and Chk2 kinases activates p53 transcription and the G 2/M checkpoint

10. Delayed internalization and lack of recycling in a beta2-adrenergic receptor fused to the G protein alpha-subunit

11. Che-1/aatf-induced transcriptionally active chromatin promotes cell growth in multiple myeloma

12. Abstract 350: Che-1/aatf-induced transcriptionally active chromatin promotes cell growth in multiple myeloma

14. Che-1 sustains hypoxic response of colorectal cancer cells by affecting Hif-1α stabilization

19. Centrosomal Che-1 Protein Is Involved in the Regulation of Mitosis and DNA Damage Response by Mediating Pericentrin (PCNT)-dependent Chk1 Protein Localization

20. Abstract 2983: Che-1 promotes tumor cell survival by sustaining mutant p53 transcription and inhibiting DNA damage response activation

21. The artificial gene Jazz, a transcriptional regulator of utrophin, corrects the dystrophic pathology in mdx mice

24. Utrophin Up-Regulation by an Artificial Transcription Factor in Transgenic Mice

27. NRAGE associates with the anti-apoptotic factor Che-1 and regulates its degradation to induce cell death

29. Che-1 phosphorylation by ATM/ATR and Chk2 kinases activates p53 transcription and the G2/M checkpoint

30. RNA Polymerase II subunit 3 is retained in the cytoplasm by its interaction with HCR, the psoriasis vulgaris candidate gene product

31. Che-1 Arrests Human Colon Carcinoma Cell Proliferation by Displacing HDAC1 from the p21 Promoter

33. The α‐like RNA polymerase II core subunit 3 (RPB3) is involved in tissue‐specific transcription and muscle differentiation via interaction with the myogenic factor myogenin

48. Delayed internalization and lack of recycling in a beta2-adrenergic receptor fused to the G protein alpha-subunit.

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