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4. Asthma

Author

Finegan, Barry A., Dillane, Derek, editor, and Finegan, Barry A., editor

Published
2021
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28. Abstract

Author

Chhina, Tejinder S., Lapierre, Harry, Jackson, Zane S., Leong-Poi, Howard M., Teitel, Jerome M., Latter, David A., Strauss, Bradley H., Gross, Peter L., Mazer, C. David, Brull, Richard, McCartney, Colin J. L., Abbas, Sherif, Nova, Hugo, Rawson, Regan, Chan, Vincent W. S., Katz, Joel, Graham, Brent, Anastakis, Dimitri, von Schroeder, Herbert, Lallo, Alexandre, Drolet, Pierre, Lacroix, Mélanie, You-Ten, Kong E., Caraiscos, Valerie B., Elliot, Erin M., Cheng, Victor Y., MacDonald, John F., Orser, Beverley A., Lau, Bertrand, Tsui, Ban C. H., Mollison, Heather L., McKay, William P. S., Patel, Rajesh, Chow, Vance, Negraeff, October, Karam, Rana, Yang, H., Raymer, K., Butler, R., Parlow, J., Roberts, R., Campbell, David C., Breen, Terrance W., Halpern, Stephen, Muir, Holly, Nunn, Robert, Katznelson, Rita, Karkouti, Keyvan, Ghannam, Mohammed, Abdelnaem, Esam, Carroll, Jo, McCluskey, Stuart, Yau, Terrence M., Karski, Jacek, Hare, Gregory M. T., Li, Xiamao, Qu, Rong, Cheung, May S. M., Coackley, Carla, Baker, Andrew J., Ronayne, Michael, Song, Dajun, Chung, Frances, Ward, Barnaby, Yogendran, Suntheralingam, Sibbick, Carolyn, Silcox, Lisa C., Ashbury, Ted L., Milne, Brian, VanDen Kerkhof, Elizabeth G., Morgan, Pamela J., Cleave-Hogg, Doreen, DeSousa, Susan, Wang, Louie, Lujic, Jelka, Donnelly, Niamh I., Torok-Both, Clint J., Finegan, Barry, Jacka, Michael J., Finegan, Barry A., Chawla, Rajiv, Agrawal, Ravi K., Kumar, Mahendra, Goldstein, David H., Paul, James E., Sawhney, Monakshi, Beattie, W. Scott, McLean, Richard F., Parlow, Joel L., Tod, Deborah A., Souzdalnitski, Dmitri, Sourovtseva, Elena, Livingstone, Donald, Faclier, Gil, Sawyer, Jason, Kay, Joseph, Avila, Arsenio, Balki, Mrinalini, Manninen, Pirjo H., Lukitto, Karolinah, Lukins, Michael B., Quader, Keya, Agarwal, Munisha, Kawatra, Rakhi, Dali, J. S., Mak, Peter H., Jose, Geraldine, Hall, Sean R., Wang, Louie, Hong, Murray, Rapchuk, Ivan L., Loo, Karen, Deschamps, Alain, Suzuki, Asaha, Kanai, Akifumi, Hoka, Sumio, Ho, Anthony M. -H., Karmakar, Manoj K., Lee, Anna, Samy, Winnie, Yi, Jie, Lai, Paul B. S., Cho, Amy, Stockton, E., Gowrie-Mohan, S., Ramanayake, P. U., Jothilingam, S., Mirmansouri, Ali, Wagner, Alese M., Cunningham, Kirsten, Perry, Shirley, Desai, Sunil, Torok-Both, Clint, DeKoven, Kathryn, Deschamps, Alain, Brousseau, Paul, Hung, Orlando, Law, Adam, Levangie, Derek, Cheverie, Ronald, Caputo, Karen M., Byrick, Robert, Chapman, Martin, Vicente, Kim, Atlas, Glen, Léveillé, Josiane, Côté, Dany, Soucy, Julie, Bussières, Jean S., Wijeysundera, Duminda N., Djaiani, George, Rao, Vivek, Borger, Michael A., Cusimano, Robert J., Moghadam, Anoush D., Heydarzadeh, Abtin, Fayad, Ashraf A., Yang, Homer, Ling, Elizabeth, Tenenbein, Paul K., Maguire, Doug, Debrouwere, Roland, Duke, Peter C., Kowalski, Stephen E., Chakravarty, Devashish, Dupuis, Jean-Yves, Nathan, Howard, Rubens, Fraser, Masters, Roy, Hendry, Paul, Mesana, Thierry, Jung, Hyun Ju, Chamchad, Dmitri, Arkoosh, Valerie, Wijeysundera, Duminda, Chan, Chris, Datillo, Kathleen, Ivano, Joan, Clark, Cantwell, Quinn, Reed D., Braxton, John H., Taenzer, Andreas H., Sullivan, Kristen M., Al-Abdulhadi, Osama A., Biehl, Diane R., Ong, Bill Y., Boker, Abdulaziz, Stewart, Kristine I., Shaw, Susan A., Hong, Jeong-Yeon, Morgan, Pamela J., Palmer, Susan K., Kung, Rose, Halpern, Stephen H., Yee, Jennifer A., Goldszmidt, Eric, Chettle, Crystal, Kern, Ralph, Downey, Kristi, Devito, Isabella, Macarthur, Alison, Purdie, Niall L., Angle, Pamela J., Kurtz-Landy, Christine, Streiner, David, Charles, Cathy, Kung, Rose, MacDonnell, Jo Watson, Lam, Desmond, Lie, Lie Ming, Kronberg, Jean E., Thompson, Dorothy E., Dong, Haiheng, Hyder, Ayman, Wang, Qinghua, Lu, Wei-Yang, Imasogie, Ngozi N., Chung, Frances, Prahbu, Atul, Curti, Bruna, Potyomkina, Zoya, Belmont, Matthew R., Tjan, Joseph, Lien, Cynthia A., Patel, Sanjay, Imarengiaye, Charles, Peirovy, Javad, Edward, Reginald, Chung, Frances F., Kayumov, Leonid, Sinclair, David R., Moller, Henry J., Shapiro, Colin M., Michaud, Guillaume, Trager, Guillaume, Deschamps, Stephane, Hemmerling, Thomas M., Hsu, Janet, Cheng, Patrick, Granton, John T., Baxter, Alan D., Kanji, Salmaan, Oxner, Adam D., Buth, Karen J., Hirsch, Gregory M., DiQuinzio, Claudio, Hirsch, Kristine A., Denault, A., Couture, P., Carrier, M., Fortier, A., Babin, D., Tardif, J. C., Olivier, Jean-François, Basile, Fadi, Prieto, Ignatio, Lê, Nhiên, Hirasaki, Yuji, Murphy, Patricia, McRae, Karen, Waddell, Thomas, Keshavjee, Shaf, Slinger, Peter, Van Rensburg, Adriaan, Yau, Terry M., Yeo, Eric, Sutton, David, Borger, Michael, Blaise, Gilbert, Gangal, Marius D., Gao, Lan, McCluskey, Stuart A., Cheung, Wing, Metha, Bobby, Poonwala, Humara, Fedorko, Ludwik, Symon, Johnson R., Peterson, Mark D., Leipoldt, Carl C. P., Clunie, Michelle, Mckay, William P. S., Miller, Grant, Guay, Joanne, Lortie, Louise, Son, Soochang, Kim, Yunhee, Arai, Toshimi, Yamashita, Masao, Rohan, Denise, Barlow, Ross, Barbour, Sean J., Ansermino, J. Mark, Vandebeek, Christine A., Raman, Rangamani K., Nakatsuka, Nao, Montgomery, Carolyne J., Skarsgard, Erik D., Court, Colleen A., Galton, James S., Crawford, Mark W., Naser, Basem, Carter, Clifford, Liu, Richard, Usher, Andrew G., Cave, Dominic A., Tang, Cathy, Hayes, Jason A., and Tan, Juliana M.

Published
2004
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29. Abstract

Author

Snider, Douglas D., Finucane, Brendan T., Clarke, Donna, Loiselle, J., Doak, G., Anderson, B., Wood, Dan, Shysh, Sandy, Perrault, Michel-Antoine, Chouinard, Philippe, Fugère, François, Girard, François, Ruel, Monique, Hall, Wendy C. E., Hrazdil, Jiri, Jolly, Donald T., Galbraith, John C., Greacen, Maria C., Clanachan, Alexander S., Wijeysundera, Duminda N., Beattie, W. Scott, Chochinov, Michelle, Halpern, Stephen, McKeen, Dolores M., Nunn, Robert T., Barrett, Brendan S., Finley, G. Allen, Buffett-Jerrott, Susan, Stewart, Sherry, Millington, Donna, Karkouti, Keyvan, Rampersaud, Yoga R., McCluskey, Stuart A., Evans, Lucia, Ghannam, Mohammed M., Mahomed, Nizar N., Singh, Sudha, Morley-Forster, Patricia, Shamsah, Mohammed, Thomson, Ian R., Brown, Aaron D., Freedman, Jeffrey I., Hudson, Robert J., Hong, Murray, Hall, Sean, Milne, Brian, Wang, Louie, Loomis, Chris, Gilron, Ian, Tod, Debbie, Bell, Allan, Orr, Elizabeth, Dobson, Gary, Karamanoglu, Mustafa, Tyberg, John V., Chung, Frances, Imarengiaye, Charles, Rocchi, Angela, Forte, Lindy, Van Den Kerkhof, Elizabeth G., Goldstein, David H., Rimmer, Mike, Lee, Hoi Kwan, Charest, Isabelle, Martin, René, Plante, François, Shaikh, Shaheen, Yung, Damian, Bernstein, Mark, Imasogie, Ngozi, Wong, David, Luk, Ken, Yogendran, Suntheralingam, Prabhu, Atul, Hendy, Ayman, McGuire, Glenn, Wong, Jean, Daley, M. Denise, Norman, Peter H., Srejic, Una, Dougherty, Thomas, Hogervorst, Sarah, Hemmerling, Thomas M., Schmidt, Joachim, Beaulieu, Pierre, Jacobi, Klaus E., Lennox, Pamela H., Mayson, Kelly V., Arai, Toshimi, Saito, Kaori, Yamashita, Masao, Gauthier, Alain, Boudreault, Daniel, Girard, Dominique C., Wang, Louie T. S., Sikich, Nancy, Petroz, Guy, Lerman, Jerrold, Hare, Gregory M. T., Baker, Andrew J., Hum, Kathryn M., Kim, Steve Y., Barr, Aiala, Mazer, C. David, Yemen, Terrance A., Howlett, Thomas, Baker, Andrew, Phan, Nicholas, Persaud, N., Zhao, Min, Liu, Elaine, Fehlings, Michael, Withington, Davinia E., AlAyed, T., Davis, G. Michael, Ansley, David M., Dhaliwal, B. S., Xia, Zhengyuan, Ho, Anthony M. -H., Lee, Anna, Ling, Elizabeth, Daly, Alan, Teoh, Kevin, Warkentin, Theodore E., Godin, David V., Chang, Thomas K. H., Fortier, Joanne D., Basile, Fadi, Prieto, Ignacio, Mazer, David, Duke, Peter, Finegan, Barry, Cheng, Davy, Hall, Richard, Lim, B., Shannon, J., Ho, C. M., Tsai, S. K., Srebrnjak, Margaret, Walsh, Vivien, Joseph, Geena, Gomez, Teresa Valois, Fuenmayor, Carmen Rivero, Bach, Paul S., Kamani, Allaudin, Douglas, Joanne, Esler, Mark, Gunka, Vit, Angle, Pamela, Tang, Sam, Thompson, Dorothy, Kronberg, Jean, Morgan, Anwar, Leicht, Craig H., Velickovic, Ivan A., Raphael, David T., Benbassat, Maxim, Arnaudov, Dimiter, Bohorquez, Alex, Nasseri, Bita, Doyle, D. John, Kolman, Jacelyn, Keith, Ian, Morgan, Pamela J., Cleave-Hogg, Doreen, Eveleigh, Susan, Tarshis, Jordan, Davies, Sharon, Doyle, John, Loughlin, Heather Lee, O’Connor, J. Patrick, Dolman, John F., Mikelberg, Fred S., Dulovic, Gordana, Feagan, Brian G., Wong, Cindy J., Kirkley, Alexandra, Johnston, D. W. C., Smith, Frank C., Whitsitt, Paul, Shanl, William Li Pi, Backman, Steven B., Barkun, Jeffrey, Metrakos, Peter, Tchervenkov, John, Salpeter, Mary Jane, Jamnicky, Leah, Jewett, Michael, Arellano, Ramiro, Muirhead, Brian, Rashiq, Saifundin, Shah, Meera, Wilkinson, Vikki, Woolsey, Samantha J., Finegan, Barry A., Fiorilli, Carol, Parlow, Joel L., Avery, Nicole D., Dhaliwal, B. S., Djaiani, George N., Muhkerji, Jayanta, Karski, Jacek M., Carroll, Jo A., McCluskey, Stuart, Harris, Linda, Paton, Jan, Kanetos, Emmanuel, Williams, William G., Kerkhof, Elizabeth G. Van Den, Milne, Brian, Beattie, W. Scott, Hurtado, Cristina, Finegan, Barry A., Gandhi, Manoj, Clanachan, Sandy, Shin, Woo Jong, Winegar, Bruce, D., Shim, Jae Hang, Jeon, Woo Jae, Kim, Kyung Hun, Wang, Bing, Archer, David P., Samanani, Naaznin, Roth, Sheldon H., Coimbra, Claudia, Choinière, Manon, Babin, Denis, Donati, Francois, Lennox, Pamela H., Henderson, Cynthia L., Friesen, John H. P., Jacka, Michael, Cheng, Alan, McAlister, Finlay, Leak, Jessie A., Bui, Tao, Nguyen, Dy, Kowalski, Alicia, Popat, Keruyi, Kuerer, Henry, Serowka, Paul, Turner, Kim, Milne, Brian, Ashbury, Ted, Wang, Tianlong, El-Kebir, Driss, Hubert, Bernard, Veldhuizen, Ruud A. W., Gauvin, Dominique, Blaise, Gilbert, Yang, Fan, and Amicone, Patricia

Published
2002
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33. Role of glucose metabolism in the recovery of postischemic LV mechanical function: effects of insulin and other metabolic modulators

Author

Gandhi, Manoj, Finegan, Barry A., and Clanachan, Alexander S.

Subjects
Glucose metabolism -- Influence, Reperfusion injury -- Physiological aspects, Heart -- Properties, Biological sciences
Abstract

The role of proton ([H.sup.+]) production from glucose metabolism in the recovery of myocardial function during postischemic reperfusion and its alteration by insulin and other metabolic modulators were examined. Rat hearts were perfused in vitro with Krebs-Henseleit solution containing palmitate (1.2 retool/l) and glucose (11 retool/l) under nonischemic conditions or during reperfusion following no-flow ischemia. Perfusate contained normal insulin (n-Ins, 50 mU/l), zero insulin (0-1ns), or supplemental insulin (s-Ins, 1,000 mU/l) or other metabolic modulators [dichloroacetate (DCA) at 3 retool/l, oxfenicine at 1 retool/l, and [N.sup.6]-cyclohexyladenosine (CHA) at 0.5 [micro]mol/1]. Relative to n-Ins, 0-Ins depressed rates of glycolysis and glucose oxidation in nonischemic hearts and impaired recovery of postischemic function. Relative to n-lns, s-Ins did not affect aerobic glucose metabolism and did not improve recovery when present during reperfusion. When present during ischemia and reperfusion, s-Ins impaired recovery. Combinations of metabolic modulators with s-Ins stimulated glucose oxidation ~2.5-fold in nonischemic hearts and reduced [H.sup.+] production. DCA and CHA, in combination with s-Ins, improved recovery of function, but addition of oxfenicine to this combination provided no further benefit. Although DCA and CHA were each partially protective in hearts perfused with n-Ins, optimal protection was achieved with DCA + CHA; recovery of function was inversely proportional to [H.sup.+] production during reperfusion. Although supplemental insulin is not beneficial, elimination of [H.sup.+] production from glucose metabolism by simultaneous inhibition of glycolysis and stimulation of glucose oxidation optimizes recovery of postischemic mechanical function. proton production; ischemia-reperfusion; contractile function; rat hearts

Published
2008

36. Inhibition of p38 MAPK and AMPK restores adenosine-induced cardioprotection in hearts stressed by antecedent ischemia by altering glucose utilization

Author

Jaswal, Jagdip S., Gandhi, Manoj, Finegan, Barry A., Dyck, Jason R.B., and Clanachan, Alexander S.

Subjects
Protein kinases -- Properties, Glucose metabolism -- Evaluation, Ischemia -- Physiological aspects, Heart -- Physiological aspects, Biological sciences
Abstract

p38 mitugen-activated protein kinase (MAPK) and 5'-AMP-activated protein kinase (AMPK) are activated by metabolic stresses and are implicated in the regulation of glucose utilization and ischemia-reperfusion (IR) injury. This study tested the hypothesis that inhibition of p38 MAPK restores the cardioprotective effects of adenosine in stressed hearts by preventing activation of AMPK and the uncoupling of glycolysis from glucose oxidation. Working rat hearts were perfused with Krebs solution (1.2 mM palmitate, 11 mM [[sup.3]H/[sup.14]C]glucose, and 100 mU/l insulin). Hearts were stressed by transient antecedent IR (2 X 10 rain I/5 min R) before severe IR (30 min I/30 min R). Hearts were treated with vehicle, p38 MAPK inhibitor (SB-202190, 10 [micro]M), adenosine (500 [micro]M), or their combination before severe IR. After severe IR, the phosphorylation (arbitrary density units) of p38 MAPK and AMPK, rates of glucose metabolism ([micro]mol*g dry [wt.sup.-1]*[min.sup.-1), and recovery of left ventricular (LV) work (Joules) were similar in vehicle-, SB-202190- and adenosine-treated hearts. Treatment with SB202190 + adenosine versus adenosine alone decreased p38 MAPK (0.03 [+ or -] 0.01, n = 3 vs. 0.48 [+ or -] 0.10, n = 3, P < 0.05) and AMPK (0.00 [+ or -] 0.00, n = 3 vs. 0.26 [+ or -] 0.08, n = 3 P < 0.05) phosphorylation. This was accompanied by attenuated rates of glycolysis (1.51 [+ or -] 0.40, n = 7 vs. 3.95 [+ or -] 0.65, n = 7, P < 0.05) and [H.sup.+] production (2.12 [+ or -] 0.76, n = 7 vs. 6.96 [+ or -] 1.48, n = 7, P < 0.05), and increased glycogen synthesis (1.91 [+ or -] 0.25, n = 6 vs. 0.27 [+ or -] 0.28, n = 6, P < 0.05) and improved recovery of LV work (0.81 [+ or -] 0.08, n = 7 vs. 0.30 [+ or -] 0.15, n = 8, P < 0.05). These data indicate that inhibition of p38 MAPK abolishes subsequent phosphorylation of AMPK and improves the coupling of glucose metabolism, thereby restoring adenosine-induced cardioprotection. p38 mitogen-activated protein kinase; 5'-adenosine monophosphateactivated protein kinase; glucose metabolism

Published
2007

37. p38 mitogen-activated protein kinase mediates adenosine-induced alterations in myocardial glucose utilization via 5'-AMP-activated protein kinase

Author

Jaswal, Jagdip S., Gandhi, Manoj, Finegan, Barry A., Dyck, Jason R.B., and Clanachan, Alexander S.

Subjects
Adenosine -- Physiological aspects, Adenosine -- Research, Protein kinases -- Research, Glycogen -- Synthesis, Glycogen -- Research, Biological sciences
Abstract

Adenosineinduced acceleration of glycolysis in hearts stressed by transient ischemia is accompanied by suppression of glycogen synthesis and by increases in activity of adenosine 5'-monophosphate-activated protein kinase (AMPK). Because p38 mitogen-activated protein kinase (MAPK) may regulate glucose metabolism and may be activated downstream of AMPK, this study determined the effects of the p38 MAPK inhibitors SB202190 and SB203580 on adenosine-induced alterations in glucose utilization and AMPK activity. Studies were performed in working rat hearts perfused aerobically following stressing by transient ischemia (2 x 10-min ischemia followed by 5-min reperfusion). Phosphorylation of AMPK and p38 MAPK each were increased fourfold by adenosine, and these effects were inhibited by either SB202190 or SB203580. Neither of these inhibitors directly affected AMPK activity. Attenuation of the adenosine-induced increase in AMPK and p38 MAPK phosphorylation by SB202190 and SB203580 occurred independently of any change in tissue ATP-to-AMP ratio and did not alter glucose uptake, but it was accompanied by an increase in glycogen synthesis and glycogen content and by inhibition of glycolysis and proton production. There was a significant inverse correlation between the rate of glycogen synthesis and AMPK activity and between AMPK activity and glycogen content. These data demonstrate that AMPK is likely downstream of p38 MAPK in mediating the effects of adenosine on glucose utilization in hearts stressed by transient ischemia. The ability of p38 MAPK inhibitors to relieve the inhibition of glycogen synthesis and to inhibit glycolysis and proton production suggests that these agents may restore adenosine -induced cardioprotection in stressed hearts. adenosine; adenosine 5'-monophosphate-activated protein kinase; glycogen metabolism

Published
2007

40. Effects of adenosine on myocardial glucose and palmitate metabolism after transient ischemia: role of 5'-AMP-activated protein kinase

Author

Jaswal, Jagdip S., Gandhi, Manoj, Finegan, Barry A., Dyck, Jason R.B., and Clanachan, Alexander S.

Subjects
Protein kinases -- Research, Cerebral ischemia -- Risk factors, Cerebral ischemia -- Physiological aspects, Cerebral ischemia -- Health aspects, Glucose metabolism -- Research, Biological sciences
Abstract

Loss of cardioprotection by adenosine in hearts stressed by transient ischemia may be due to its effects on glucose metabolism. In the absence of transient ischemia, adenosine inhibits glycolysis, whereas it accelerates glycolysis after transient ischemia. Inasmuch as 5'-AMP-activated protein kinase (AMPK) is implicated as a regulator of glucose and fatty acid utilization, this study determined whether a differential alteration of AMPK activity contributes to acceleration of glycolysis by adenosine in hearts stressed by transient ischemia. Studies were performed in working rat hearts perfused aerobically under normal conditions or after transient ischemia (two 10-min periods of ischemia followed by 5 min of reperfusion). LV work was not affected by adenosine. AMPK phosphorylation was not affected by transient ischemia; however, phosphorylation and activity were increased nine-and threefold, respectively, by adenosine in stressed hearts. Phosphorylation of acetyl-CoA carboxylase and rates of palmitate oxidation were unaltered. Glycolysis and calculated proton production were increased 1.8-and 1.7-fold, respectively, in hearts with elevated AMPK activity. Elevated AMPK activity was associated with inhibition of glycogen synthesis and unchanged rates of glucose uptake and glycogenolysis. Phentolamine, an [alpha]-adrenoceptor antagonist, which prevents adenosine-induced activation of glycolysis in stressed hearts, prevented AMPK phosphorylation. These data demonstrate that adenosine-induced activation of AMPK after transient ischemia is not sufficient to alter palmitate oxidation or glucose uptake. Rather, activation of AMPK alters partitioning of glucose away from glycogen synthesis; the increase in glycolysis may in part contribute to loss of adenosine-induced cardioprotection in hearts subjected to transient ischemia. glucose metabolism; glucose uptake; glycogen

Published
2006

43. Learning fiberoptic intubation on a simple model transfers to the O.R.

Author

Naik, Viren, Matsumoto, Edward, Houston, Patricia, Hamstra, Stanley, Yeung, Raymond, Mallon, Joseph, Martire, Terry, Zwack, Rhonda M., Campbell, David C., Breen, Terrance W., Yip, Ray W., Roy, Jean-Denis, Girard, Michel, Drolet, Pierre, Guay, Joanne, Bolis, Rafik S., LeDez, Kenneth, Balatbat, J. T., Mukherji, J., Ali, M. J., Carroll, J., Karski, J. M., Sui, S., Cheng, D. C. H., Banner, Robert, Yip, Raymond, Zondervan, James, Chow, Vance, McMillan, Dean, Fisher, Judy, Lattermann, Ralph, Carli, Franco, Wykes, Linda, Schricker, Thomas, Mazza, Louise, Carli, Franco, Danjoux, Gerard, Thomas, David, Lennox, Pamela H., Henderson, Cynthia, Martin, Lynn, Mitchell, G W E, Vaghadia, Himat, Jassal, Rajive, Thomson, Ian R., Hudson, Robert J., McGuire, Glenn, Manninen, Pirjo, El-Beheiry, Hossam, Lozano, Andres, Wennberg, Richard, Archer, David P., Tang, Tim K. K., Staveley, Ian R., Goldstein, David H., VanDenKerkhof, Elizabeth G., Hall, Richard I., Rocker, Graeme M., O’Connor, J. P., Dunham, Jacquelyn I., Mikelberg, Frederick S., Dulovic, Gordana, Jenkins, Kathryn L., Correa, Robin, Wong, David T., McGuire, Glenn P., Fayad, A. A., Paul, J., Yang, H., Sawchuk, C., Brown, Karen A., Bates, Jason H. T., Edington, Robert, Pridham, Jeremy, Mukherji, Jayanta, Karski, Jacek M., Balatbat, Joselito, Carroll, Jo, Chun, Rosa, Cheng, Davy CH, Karski, J., DeBrouwere, R., Mathieu, M., Carroll, J., Feindel, C., Cheng, D., Clairoux, Michel, Coutu, Stéphane, McCluskey, Stuart A., Karkouti, Keyvan, Ghannam, Mohammed, Jewett, Michael, Rampersaud, Raja, Yau, Terry, Quirt, Ian, Carver, Edmund D., Kim, P., Crawford, Mark W., Finley, G. Allen, Breau, Lynn M., McGrath, Patrick J., Camfield, Carol, Mak, Peter H. K., Hui, Theresa W. C., Irwin, Michael G., Carli, Franco, Trudel, Judith, Belliveau, Paul, Mayo, Nancy, Clunie, Michelle L., Crone, Lesley-Ann L., Klassen, Linda J., Yip, Raymond W., Hubert, Bernard, Radomski, Marek, Blaise, Gilbert, Renzi, Paolo M., Paradis, Marie-Claude, Martin, René, Parent, Michel, Parent, Pierre, Gagnon, Daniel, Tétrault, Jean-Pierre, Prabhu, Atul J., Philip, Beverly K., Higgins, Patrick P., Blanshard, Hannah J., van Rensselaer, Stéphanie, Chung, Frances F., Caraiscos, Valerie B., MacDonald, John F., Orser, Beverley A., Schreiber, Markus, Georgieff, Michael, Jin, Fengling, Chung, Frances, Tong, Doris, Reiz, Joseph L., Harsanyi, Zoltan, Miceli, Paula C., Darke, Andrew C., Roy, Jean-Sébastien, St-Pierre, Jacques, Norman, Peter H., Daley, M. Denise, Turner, Kim E., Parlow, Joel L., Tod, Deborah A., Avery, Nicole D., Nicole, Pierre C., Trépanier, Claude A., Lessard, Martin R., Marcoux, Sylvie, Cowie, Dean A., Gelb, Adrian W., Shoemaker, J. Kevin, Baskett, Roger, Lim, Ben C., Dangor, Ayoub, Morgan, Pamela J., Cleave-Hogg, Doreen, Doyle, D. John, Byrick, Robert, Filipovi, Dusanka, Cashin, Fred, Chiu, Michelle, Kemp, Toby J., Bryson, Gregory L., Cleland, Mark J., Crosby, Edward T., Harioka, Tokuya, Nomura, Koichiro, Ando, Norioki, Ikegami, Naoyuki, Aoki, Toshiki, Maltby, J. Roger, Beriault, Michael T., Watson, Neil C., Liepert, David J., Fick, Gordon H., Maltby, J. Roger, Liepert, David, Prabhu, Atul J., Correa, Robin K., Wong, David T., Chung, Frances, Goyagi, Tom, Bhardwaj, Anish, Hum, Patricia D., Traystman, Richard D., Kirsch, Jeffrey R., Bainbridge, Daniel T., Swaminathan, Madhav, McCreath, Brian J., Djaiani, George, Grocott, Hilary P., Day, Fergal, Karski, Jacek, Djaiani, George, Tan, Jens, Cheng, Davy, Wake, Pamela J., Ali, Mohamed, Karski, Jacek, Sui, Sam, Guenther, Craig, Mullen, John, Bentley, Michael, Koshal, Arvind, Finegan, Barry, Murtha, William, Fredrickson, Michael J., Luginbuehl, Igor A., Bissonnette, Bruno, Granton, Jeff T., Platt, Hugh, and Craen, R. A.

Published
2001
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48. Abstracts

Author

Kavanagh, Brian P., Ngo, Cuong, Raymer, Karen, Yang, Homer, Alhashemi, Jamal A., Lui, Anne C. P., Reid, Dennis, Cicutti, Nicholas, Krepski, Barbra, Wood, Gordon, Heyland, Daren K., Badner, Neal H., Murkin, John M., Mohr, Jim, McKenzie, F. Neil, van der Starre, Peter J. A., van Rooyen-Butijn, W. T., Wilson-Yang, Kristine, Teoh, Kevin, Lee, R. M. K. W., Hossain, Imtiaz, Cheng, Davy, Karski, Jacek, Asokumar, Buvanendran, Sandier, Alan, St-Amand, Marc A., Murkin, John M., Menkis, Alan H., Downey, Donal B., Nantau, William, Adams, Sandy, Dowd, Noreen, Cheng, Davy, Wong, David, Carroll-Munro, Jo, Trachuk, Clare, Cregg, N., Cheng, D. C. H., Williams, W. G., Karski, J. M., Siu, S., Webb, G., Cheng, Davy C. H., Wong, David T., Kustra, Rafal, Karski, Jacek, Tibshirani, Robert J., Côté, Dany L., Lacey, David E., LeDez, Kenneth M., Smith, Julia A., Crosby, Edward T., Orkin, Fredrick K., Fisher, A., Volgyesi, G., Silverman, J., Edelstein, S., Rucker, J., Sommer, L., Dunington, S., Roy, L., Crochetière, C., Arsenault, M. -Y., Villeneuve, E., Lortie, L., Grange, Caroline S., Douglas, M. Joanne, Adams, Timothy J., Merrick, Pamela M., Lucas, S. Brian, Morgan, Pamela J., Halpern, Stephen, Lo, Jason, Giesinger, Carolyn L., Halpern, Stephen H., Breen, Terrance W., Vishnubala, Srigowri, Shetty, Geeta R., De Kock, M., Lagmiche, A., Scholtes, J. L., Grodecki, Wlodzimierz, Duffy, Peter J., Hull, Kathryn A., Hawboldt, Geoffrey S., Clark, Alexander J., Smith, J. Bruce, Norman, Richard W., Beattie, W. Scott, Sandier, A., Jewett, M., Valiquette, L., Katz, J., Fradet, Y., Redelmeier, D., Sampson, H., Cole, Jeff, Chedore, Todd, Snedden, Walter, Green, Robert G., Sosis, Mitchel B., Robles, Philip I., Lazar, Edward R., Jolly, Donald T., Tarn, Yun K., Tawfik, Soheir R., Clanachan, Alexander S., Milne, Avaleigh, Beamish, Travis, Cuillerier, D. J., Sharpe, M. D., Lee, J. K., Basta, M., Krahn, A. D., Klein, G. J., Yee, R., Vakharia, Narendra, Francis, Heather, Scheepers, Louis, Vaghadia, Himat, Carrier, Joanne, Martin, René, Pirlet, Marline, Claprood, Yves, Tétrault, Jean-Pierre, Wong, T. D., Ryner, L., Kozlowski, P., Scarth, G., Warrian, R. K., Lefevre, G., Thiessen, D., Girling, L., Doiron, L., McCudden, C., Saunders, J., Mutch, W. A. C., Duffy, Peter J., Langevin, Stéphan, Lessard, Martin R., Trépanier, Claude A., Hare, Gregory M. T., Ngan, Johnson C. 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Published
1997
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50. Antecedent ischemia reverses effects of adenosine on glycolysis and mechanical function of working hearts

Author

Finegan, Barry A., Gandhi, Manoj, Lopaschuk, Gary D., and Clanachan, Alexander S.

Subjects
Adenosine -- Physiological aspects, Reperfusion injury -- Physiological aspects, Heart -- Physiological aspects, Rats -- Physiological aspects, Biological sciences
Abstract

Previous studies have demonstrated the cardioprotective effects of adenosine (Ado) during ischemia and reperfusion, but the mechanisms underlying the role of Ado have not been resolved. Herein, experiments on isolated rabbit hearts showed that Ado influences the coupling of glycolysis to glucose oxidation following ischemia and that the effect of Ado was determined by the preischemic glycogen content of the heart.

Published
1996

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