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2. Modeling the progression of neuropsychiatric symptoms in Alzheimer’s disease with PET-based Braak staging

3. Vascular risk burden is a key player in the early progression of Alzheimer’s disease

5. Sex modulates the role of astrocyte reactivity in preclinical Alzheimer’s disease

6. Synaptic dysfunction, Tau pathology and Neurodegeneration

7. CSF total tau is more closely associated with synaptic dysfunction than overt neurodegeneration

8. Association of reactive astrogliosis and microglial activation with tau pathology in Alzheimer’s disease

9. Increased regional brain inflammation predicts longitudinal brain atrophy in individuals in the Alzheimer’s disease continuum

10. Plasma p‐tau and brain‐derived total tau biomarkers predict longitudinal changes in Aβ, tau, and cognition across the AD continuum

11. Neuroinflammation Exacerbates Irritability and Agitation in Alzheimer’s Disease

12. Association between plasma inflammatory biomarkers and [18F]FDG‐PET signal in a transgenic amyloid rat model

13. Amyloid β‐dependent tau phosphorylation is triggered by reactive astrocytes in preclinical Alzheimer’s disease

14. Association between glial and synaptic fluid biomarkers and brain [18F]FDG‐PET signal

15. APOEε4 potentiates the effects of Aβ pathology on the deposition of neurofibrillary tangles via tau phosphorylation

16. Amyloid and Tau Predominance Subtyping Identifies CI Patients With Different Clinical Phenotypes

17. Association between brain [18F]FDG‐PET signal and genetic cell markers in the brain tissue

18. Microglial activation interacts with amyloid‐β to drive longitudinal tau tangle accumulation and cognitive decline

19. Astrocyte reactivity is associated with synaptic dysfunction across the aging and Alzheimer’s disease spectrum, whereas microglial reactivity is specifically associated with synaptic dysfunction related to cognitive impairment

20. Potential utility of using both APOEε4 and Aβ positivity to enrich clinical trials of tau‐targeting therapies

21. Sex impacts the association of plasma Glial Fibrillary Acidic Protein with neurodegeneration in Alzheimer’s disease

22. Increased Genetic Risk for ADHD Potentiates Cognitive Impairment and Brain Hypometabolism in Alzheimer’s Disease Patients

23. Microglial Activation Contributes to Neuropsychiatric Dysfunction in Alzheimer’s Disease

24. Astrocyte reactivity potentiates longitudinal tau tangle accumulation in cognitively unimpaired individuals

26. Synapse dysfunction and astrocyte reactivity are associated independently of amyloid‐β and tau pathologies

28. Plasma p‐tau181 and nfl as surrogates biomarkers in clinical trials targeting preclinical stage of Alzheimer’s disease

29. Employing transfer learning to optimize deep learning models for Alzheimer’s disease classification using two tau PET tracers

30. Associations between brain TSPO and [18F]FDG‐PET signals in neurodegenerative disorders

32. Synaptic dysfunction in the presence of tau tangles is not a strong predictor of atrophy

33. Plasma p‐tau231 and p‐tau217 provides information on tau tangle deposition in symptomatic Alzheimer’s disease individuals

35. Amyloid and tau‐driven signatures in hippocampal GFAP‐positive astrocytes

36. The impact of hypertension on longitudinal cognitive decline in cognitively unimpaired individuals

38. Comparison of plasma amyloid, tau, and astrocyte biomarkers to identify AD pathophysiology

40. Amyloid and tau pathologies associate with distinct aspects of the inflammatory cascade

41. Impact of meningeal and age‐related off‐target binding on longitudinal [18F]MK6240 quantification

43. Impact of meningeal and age‐related off‐target binding on longitudinal [ 18 F]MK6240 quantification.

44. The impact of individual vascular risk factors on longitudinal neurodegeneration in cognitively unimpaired individuals

45. Genetic risk for attention‐deficit/hyperactivity disorder is associated with amyloid‐dependent cognitive decline in older adults

46. Longitudinal changes in plasma p‐tau181 as a surrogate variable to populational interventions

48. Apolipoprotein E ε4 associates with microglial activation in early Braak regions independently of amyloid‐β and tau

49. Temporal evaluation of imaging and biofluid biomarkers, spatial neuropathology and glial function in the TgF344‐AD rat model.

50. Potential Utility of Plasma P-Tau and Neurofilament Light Chain as Surrogate Biomarkers for Preventive Clinical Trials.

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