143 results on '"Fasullo, Michael"'
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2. Alcoholic-Hepatitis, Links to Brain and Microbiome: Mechanisms, Clinical and Experimental Research.
3. Checkpoint Control of DNA Repair in Yeast
4. Genotoxic Assays for Measuring P450 Activation of Chemical Mutagens
5. High-throughput screening of the Saccharomyces cerevisiae genome for 2-amino-3-methylimidazo [4,5-f] quinoline resistance identifies colon cancer-associated genes
6. Cellular Responses to Aflatoxin-Associated DNA Adducts
7. High-Throughput Screening of theSaccharomyces cerevisiaeGenome for 2-Amino-3-Methylimidazo [4,5-f] Quinoline Resistance Identifies Colon Cancer-Associated Genes
8. The continuing evolution of barcode applications: Functional toxicology to cell lineage
9. Editor’s Highlight: High-Throughput Functional Genomics Identifies Modulators of TCE Metabolite Genotoxicity and Candidate Susceptibility Genes
10. CYP1B1 converts procarcinogens into genotoxins in Saccharomyces cerevisiae
11. CYP1B1 converts procarcinogens into genotoxins in Saccharomyces cerevisiae
12. Recombinational Substrates Designed to Study Recombination between Unique and Repetitive Sequences in vivo
13. Genomic profiling of budding yeast resistance to food carcinogens underscore the importance of DNA damage tolerance pathways in avoiding mutations
14. Genome Profiling for Aflatoxin B1 Resistance in Saccharomyces cerevisiae Reveals a Role for the CSM2/SHU Complex in Tolerance of Aflatoxin B1-Associated DNA Damage
15. Metallized Carbon Nanotubes for Radiation Therapy Enhancement
16. Elevated dNTP levels suppress hyper-recombination in Saccharomyces cerevisiae S-phase checkpoint mutants
17. Saccharomyces cerevisiae RAD53 (CHK2) but not CHK1 is required for double-strand break-initiated SCE and DNA damage-associated SCE after exposure to X rays and chemical agents
18. The Saccharomyces cerevisiae PDS1 and RAD9 checkpoint genes control different DNA double-strand break repair pathways
19. Stimulation of sister chromatid exchanges and mutation by aflatoxin B1-DNA adducts in Saccharomyces cerevisiae requires MEC1 (ATR), RAD53, and DUN1
20. Cellular Responses to Aflatoxin-Associated DNA Adducts
21. Checkpoint Control of DNA Repair in Yeast
22. Mating type regulates the radiation-associated stimulation of reciprocal translocation events in Saccharomyces cerevisiae
23. Inverted repeat-stimulated sister-chromatid exchange events are RAD1-independent but reduced in a msh2 mutant
24. Multiple recombination pathways for sister chromatid exchange in Saccharomyces cerevisiae: role of RAD1 and the RAD52 epistasis group genes
25. Genotoxic Assays for Measuring P450 Activation of Chemical Mutagens
26. Genome Profiling for Aflatoxin B1 Resistance inSaccharomyces cerevisiaeReveals a Role for the CSM2/SHU Complex in Tolerance of Aflatoxin B1-associated DNA Damage
27. tRNA modification and cancer: potential for therapeutic prevention and intervention
28. DNA damaging agents trigger the expression of the HML silent mating type locus in Saccharomyces cerevisiae
29. An in vitro system for measuring genotoxicity mediated by human CYP3A4 in Saccharomyces cerevisiae
30. An in vitro system for measuring genotoxicity mediated by human CYP3A4 in Saccharomyces cerevisiae
31. Selective Oncolytic Therapy for Hypoxic Breast Cancer Cells
32. Expression of Saccharomyces cerevisiaeMATa and MATα enhances the HO endonuclease-stimulation of chromosomal rearrangements directed by his3 recombinational substrates
33. Both RAD5-dependent and independent pathways are involved in DNA damage-associated sister chromatid exchange in budding yeast
34. An in vitro system for measuring genotoxicity mediated by human CYP3A4 in Saccharomyces cerevisiae
35. CYP1A1 I462V polymorphism is associated with reduced genotoxicity in yeast despite positive association with increased cancer risk
36. Saccharomyces cerevisiae rad51 Mutants Are Defective in DNA Damage-Associated Sister Chromatid Exchanges but Exhibit Increased Rates of Homology-Directed Translocations
37. High-Throughput Functional Genomics Identifies Modulators of TCE Metabolite Genotoxicity and Candidate Susceptibility Genes.
38. Nucleotide Salvage Deficiencies, DNA Damage and Neurodegeneration
39. Activation of aflatoxin B1 by expression of human CYP1A2 polymorphisms in Saccharomyces cerevisiae
40. Genotoxicity of Aflatoxin B(1): Evidence for a Recombination-mediated Mechanism in Saccharomyces cerevisiae
41. Aflatoxin B1-Associated DNA Adducts Stall S Phase and Stimulate Rad51 foci inSaccharomyces cerevisiae
42. Elevated dNTP levels suppress hyper-recombination in Saccharomyces cerevisiae S-phase checkpoint mutants
43. Hypoxia enhances the replication of oncolytic herpes simplex virus in p53- breast cancer cells
44. Saccharomyces cerevisiae G2 checkpoint and double‐strand break (DSB) repair genes synergistically suppress the formation of homology‐directed translocations
45. UV but not X rays stimulate homologous recombination between sister chromatids and homologs in a Saccharomyces cerevisiae mec1 (ATR) hypomorphic mutant
46. Stimulation of sister chromatid exchanges and mutation by aflatoxin B1‐DNA adducts inSaccharomyces cerevisiaerequiresMEC1(ATR),RAD53, andDUN1
47. TheSaccharomyces cerevisiaecheckpoint genes RAD9, CHK1 and PDS1 are required for elevated homologous recombination in a mec1 (ATR) hypomorphic mutant
48. Activation of the Budding Yeast Securin Pds1 But Not Rad53 Correlates with Double-Strand Break-Associated G2/M Cell Cycle Arrest in amec1Hypomorphic Mutant
49. Transcriptional Response of Yeast to Aflatoxin B1: Recombinational Repair InvolvingRAD51andRAD1
50. Activation of aflatoxin B1 by expression of human CYP1A2 polymorphisms in Saccharomyces cerevisiae.
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