184 results on '"Farina, Antonietta"'
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2. A Prospective Study of Intraarterial Infusion Chemotherapy in Advanced Wild-Type BRAF Melanoma Patients
3. A Study of Alternative TrkA Splicing Identifies TrkAIII as a Novel Potentially Targetable Participant in PitNET Progression
4. Molecular Characterization and Inhibition of a Novel Stress-Induced Mitochondrial Protecting Role for Misfolded TrkAIII in Human SH-SY5Y Neuroblastoma Cells.
5. Hypoxia-induced alternative splicing: the 11th Hallmark of Cancer
6. Circulating tumour cell liquid biopsy in selecting therapy for recurrent cutaneous melanoma with locoregional pelvic metastases: a pilot study
7. Discovery, characterization and potential roles of a novel NF-YAx splice variant in human neuroblastoma
8. A pilot study of alternative TrkAIII splicing in Merkel cell carcinoma: a potential oncogenic mechanism and novel therapeutic target
9. The Alternative TrkAIII Splice Variant, a Targetable Oncogenic Participant in Human Cutaneous Malignant Melanoma
10. Nutraceutical Preventative and Therapeutic Potential in Neuroblastoma: From Pregnancy to Early Childhood
11. The oncogenic neurotrophin receptor tropomyosin-related kinase variant, TrkAIII
12. Doxorubicin-Induced TrkAIII Activation: A Selection Mechanism for Resistant Dormant Neuroblastoma Cells
13. Mechanisms involved in selecting and maintaining neuroblastoma cancer stem cell populations, and perspectives for therapeutic targeting
14. Additional file 1 of Circulating tumour cell liquid biopsy in selecting therapy for recurrent cutaneous melanoma with locoregional pelvic metastases: a pilot study
15. TrkAIII expression in the thymus
16. Multidisciplinary Treatment, Including Locoregional Chemotherapy, for Merkel-Polyomavirus-Positive Merkel Cell Carcinomas: Perspectives for Patients Exhibiting Oncogenic Alternative Δ exon 6–7 TrkAIII Splicing of Neurotrophin Receptor Tropomyosin-Related Kinase A
17. A Pilot Study of the Predictive Potential of Chemosensitivity and Gene Expression Assays Using Circulating Tumour Cells from Patients with Recurrent Ovarian Cancer
18. TrkA alternative splicing: A regulated tumor-promoting switch in human neuroblastoma
19. Thioredoxin inhibits microvascular endothelial capillary tubule formation
20. Reconstitution of TIMP-2 expression in SH-SY5Y neuroblastoma cells by 5-azacytidine is mediated transcriptionally by NF-Y through an inverted CCAAT site
21. Inhibition of human MDA-MB-231 breast cancer cell invasion by matrix metalloproteinase 3 involves degradation of plasminogen
22. Thioredoxin alters the matrix metalloproteinase/tissue inhibitors of metalloproteinase balance and stimulates human SK-N-SH neuroblastoma cell invasion
23. TRAIL induces pro-apoptotic crosstalk between the TRAIL-receptor signaling pathway and TrkAIII in SH-SY5Y cells
24. The enemy from within: Miss-localization of a compromised receptor as a mechanism for TrkAIII oncogenic activity
25. The Alternative TrkAIII Splice Variant Targets the Centrosome and Promotes Genetic Instability
26. TrkAIII signals endoplasmic reticulum stress to the mitochondria in neuroblastoma cells, resulting in glycolytic metabolic adaptation
27. NGF sensitizes TrKA expressing SH-SY5Y nuroblastoma cells to TRAIL-induced apoptosisi
28. TRAIL induces pro-apoptotic crosstalk between the TRAIL-receptor signaling pathway and TrkAIII in SH-SY5Y cells, unveiling a potential therapeutic “Achilles heel” for the TrkAIII oncoprotein in neuroblastoma
29. Neurotrophin and Neurotrophin Receptor Involvement in Human Neuroblastoma
30. Constitutive autotaxin transcription by Nmyc-amplified and non-amplified neuroblastoma cells is regulated by a novel AP-1 and SP-mediated mechanism and abrogated by curcumin
31. Alendronate promotes plasmin-mediated MMP-9 inactivation by exposing cryptic plasmin degradation sites within the MMP-9 catalytic domain
32. Alternative TrkA Splicing and Neuroblastoma
33. Alternative TrkAIII splicing and Cancer
34. Retrograde TrkAIII transport from ERGIC to ER: a re-localisation mechanism for oncogenic activity
35. The Alternative TrkAIII splice variant by neuroblastomas and glioblastomas targets the centrosome and promotes genetic instability
36. Thioredoxin stimulates MMP-9 expression, de-regulates the MMP-9/TIMP-1 equilibrium and promotes MMP-9 dependent invasion in human MDA-MB-231 breast cancer cells
37. Cis Golgi network-associated TrkAIII targets the interphase centrosome and promotes genetic instability
38. A NOVEL HYPOXIA-REGULATED ALTERNATIVE TRKA SPLICE VARIANT AF POTENTIAL PHYSIOLOGICAL AND PATHOLOGICAL IMPORTANCE
39. The TrkAIII Oncoprotein Inhibits Mitochondrial Free Radical ROS-Induced Death of SH-SY5Y Neuroblastoma Cells by Augmenting SOD2 Expression and Activity at the Mitochondria, within the Context of a Tumour Stem Cell-like Phenotype
40. Gelatinase B/MMP-9 in Tumour Pathogenesis and Progression
41. RECONSTITUTION OF TIMP-2 EXPRESSION IN SH-SY5Y NEUROBLASTOMA CELL BY 5-AZACYTIDINA IS MEDIATED TRANSCRIPTIONALLY BY NF-Y THROUGH AN INVERTED CAAT SITE
42. TIOREDOXIN INHIBITS MICROVASCULAR ENDHOTHELIAL CAPILLARY TUBULE FORMATION
43. ALL-TRANS-RETINOIC INDUCES NUCLEAR FACTOR-B ACTIVATION AND MATRIX METALLOPROTEINASE-9 EXPRESSION AND ENHANCES BASEMENT MEMBRANE INVASIVITY OF DIFFERENTIATION-RESISTANT HUMAN SK-N-BE 9N NEUROBLASTOMA CELL
44. Thioredoxin alters the matrix metalloproteinase/tissue inhibitors of metalloproteinase balance and stimulates human SK-N-SH neuroblastoma cell invasion
45. TRASCRIPTIONAL UP-REGULATION OF MATRIX METALLOPROTEINASE-9 EXPRESSION DURING S TO N PHENOTYPE CONVERSION BY N-TYPE INVASION, DEPEND UPON GT-BOX NFKB ELEMENTS
46. TISSUE INHIBITOR -2 PROTECTION OF MATRIX METALLOPROTEINASE-2 FROM DEGRADATION BY PLASMIN IS REVERSED BY DIVALENT CATION CHELATOR EDTA AND THE BISPHOSPHONATE ALENDRONATE
47. TRASFORMING GROWTH FACTOR-BETA 1 ENHANCES THE INVASIVITY OF HUMAN MDBA-MB-231 BREAST CANCER CELLS BY UP REGULATING UROKINASE ACTIVITY
48. Two gamma-interferon-activation sites (GAS) on the promoter of the human intercellular adhesion molecule 1 (ICAM-1) gene are required for induction of transcription by IFN-gamma
49. TrkAIII Promotes Microtubule Nucleation and Assembly at the Centrosome in SH-SY5Y Neuroblastoma Cells, Contributing to an Undifferentiated Anaplastic Phenotype
50. GLUCORTICOID RECEPTOR-MEDIATED SUPPRESSION OF THE INTERLEUKIN 2 GENE EXPRESSION THROUGH IMPAIRMENT OF THE COOPERATIVITY BETWEEN NUCLEAR FACTOR OF ACTIVATED T CELLS AND AP-1 ENHANCER ELEMENTS
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