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2. Large-scale genomic investigation of pediatric cholestasis reveals a novel hepatorenal ciliopathy caused by PSKH1 mutations

3. Clinical exome sequencing data from patients with inborn errors of immunity: Cohort level diagnostic yield and the benefit of systematic reanalysis

4. Clinical and Molecular Characteristics of Neuronal Ceroid Lipofuscinosis in Saudi Arabia

5. Beyond the exome: utility of long-read whole genome sequencing in exome-negative autosomal recessive diseases

6. Gain and loss of function variants in EZH1 disrupt neurogenesis and cause dominant and recessive neurodevelopmental disorders

7. Bi-allelic variants in CEP295 cause Seckel-like syndrome presenting with primary microcephaly, developmental delay, intellectual disability, short stature, craniofacial and digital abnormalities

8. Biallelic variants in CRIPT cause a Rothmund-Thomson-like syndrome with increased cellular senescence

9. Biallelic variants in HECT E3 paralogs, HECTD4 and UBE3C, encoding ubiquitin ligases cause neurodevelopmental disorders that overlap with Angelman syndrome

10. Loss-of-function mutations in UDP-Glucose 6-Dehydrogenase cause recessive developmental epileptic encephalopathy.

11. Loss of FOCAD, operating via the SKI messenger RNA surveillance pathway, causes a pediatric syndrome with liver cirrhosis

13. Biallelic loss of human CTNNA2, encoding αN-catenin, leads to ARP2/3 complex overactivity and disordered cortical neuronal migration.

14. Clinical and Molecular Characteristics of Neuronal Ceroid Lipofuscinosis in Saudi Arabia

15. Biallelic Mutations in FUT8 Cause a Congenital Disorder of Glycosylation with Defective Fucosylation

16. Mutations in TP73 cause impaired mucociliary clearance and lissencephaly

20. Kinesin family member 12‐related hepatopathy: A generally indolent disorder with elevated gamma‐glutamyl‐transferase activity.

21. The phenotypic spectrum of PTCD3 deficiency.

22. Clinical, molecular, and biochemical delineation of asparagine synthetase deficiency in Saudi cohort

23. The phenotypic spectrum of PTCD3 deficiency

24. Large-scale genomic investigation of pediatric cholestasis reveals a novel hepatorenal ciliopathy caused by PSKH1 mutations

25. Long-term effectiveness of carglumic acid in patients with propionic acidemia (PA) and methylmalonic acidemia (MMA): a randomized clinical trial

26. Loss of Oxidation Resistance 1, OXR1, Is Associated with an Autosomal-Recessive Neurological Disease with Cerebellar Atrophy and Lysosomal Dysfunction

27. Bi-allelic Variants in IQSEC1 Cause Intellectual Disability, Developmental Delay, and Short Stature

29. Genomic and phenotypic delineation of congenital microcephaly

31. Expanding the phenotype of PPP1R21‐related neurodevelopmental disorder

33. Clinical, Biochemical, and Molecular Features in 37 Saudi Patients with Very Long Chain Acyl CoA Dehydrogenase Deficiency

34. Expanding the phenome and variome of skeletal dysplasia

35. FARS2 deficiency; new cases, review of clinical, biochemical, and molecular spectra, and variants interpretation based on structural, functional, and evolutionary significance

36. Variants in EXOSC9 Disrupt the RNA Exosome and Result in Cerebellar Atrophy with Spinal Motor Neuronopathy

37. Molecular autopsy in maternal–fetal medicine

39. Biallelic MAD2L1BP (p31comet) mutation is associated with mosaic aneuploidy and juvenile granulosa cell tumors

43. PLAA Mutations Cause a Lethal Infantile Epileptic Encephalopathy by Disrupting Ubiquitin-Mediated Endolysosomal Degradation of Synaptic Proteins

45. NEMF mutations that impair ribosome-associated quality control are associated with neuromuscular disease

46. Author Correction: NEMF mutations that impair ribosome-associated quality control are associated with neuromuscular disease

48. Spectrum of Mutations in 60 Saudi Patients with Mut Methylmalonic Acidemia

49. Bi-allelic genetic variants in the translational GTPases GTPBP1 and GTPBP2 cause a distinct identical neurodevelopmental syndrome

50. Unravelling 5-oxoprolinuria (pyroglutamic aciduria) due to bi-allelic OPLAH mutations: 20 new mutations in 14 families

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