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1. Analogy-Forming Transformers for Few-Shot 3D Parsing

3. TIDEE: Tidying Up Novel Rooms using Visuo-Semantic Commonsense Priors

4. Simple-BEV: What Really Matters for Multi-Sensor BEV Perception?

5. Particle Video Revisited: Tracking Through Occlusions Using Point Trajectories

7. Review of Iris Presentation Attack Detection Competitions

8. Adeno-to-squamous transition drives resistance to KRAS inhibition in LKB1 mutant lung cancer

9. Move to See Better: Self-Improving Embodied Object Detection

10. Iris Liveness Detection Competition (LivDet-Iris) -- The 2020 Edition

11. Open Source Iris Recognition Hardware and Software with Presentation Attack Detection

12. Iris Presentation Attack Detection: Where Are We Now?

13. Robust Iris Presentation Attack Detection Fusing 2D and 3D Information

14. AlignNet: A Unifying Approach to Audio-Visual Alignment

15. Review of Iris Presentation Attack Detection Competitions

16. Iris Presentation Attack Detection Based on Photometric Stereo Features

18. ZFP57 dictates allelic expression switch of target imprinted genes

22. GSIR: Generalizable 3D Shape Interpretation and Reconstruction

29. A vitamin-C-derived DNA modification catalysed by an algal TET homologue

31. Figure S2 from In Vivo Epigenetic CRISPR Screen Identifies Asf1a as an Immunotherapeutic Target in Kras-Mutant Lung Adenocarcinoma

32. Data from In Vivo Epigenetic CRISPR Screen Identifies Asf1a as an Immunotherapeutic Target in Kras-Mutant Lung Adenocarcinoma

33. Table S1 from In Vivo Epigenetic CRISPR Screen Identifies Asf1a as an Immunotherapeutic Target in Kras-Mutant Lung Adenocarcinoma

35. Supplementary Figure Legend from The Use of Quantitative Real-Time Reverse Transcriptase PCR for 5′ and 3′ Portions of ALK Transcripts to Detect ALK Rearrangements in Lung Cancers

36. Table S1 from Epigenetic CRISPR Screens Identify Npm1 as a Therapeutic Vulnerability in Non–Small Cell Lung Cancer

37. Supplementary Table 1 from The Use of Quantitative Real-Time Reverse Transcriptase PCR for 5′ and 3′ Portions of ALK Transcripts to Detect ALK Rearrangements in Lung Cancers

38. Supplementary Figure 2 from The Use of Quantitative Real-Time Reverse Transcriptase PCR for 5′ and 3′ Portions of ALK Transcripts to Detect ALK Rearrangements in Lung Cancers

39. Supplementary Figure 4 from The Use of Quantitative Real-Time Reverse Transcriptase PCR for 5′ and 3′ Portions of ALK Transcripts to Detect ALK Rearrangements in Lung Cancers

40. Supplementary Table 2 from The Use of Quantitative Real-Time Reverse Transcriptase PCR for 5′ and 3′ Portions of ALK Transcripts to Detect ALK Rearrangements in Lung Cancers

41. Supplementary Figure 1 from The Use of Quantitative Real-Time Reverse Transcriptase PCR for 5′ and 3′ Portions of ALK Transcripts to Detect ALK Rearrangements in Lung Cancers

42. Data from Epigenetic CRISPR Screens Identify Npm1 as a Therapeutic Vulnerability in Non–Small Cell Lung Cancer

43. Supplementary Table 3 from The Use of Quantitative Real-Time Reverse Transcriptase PCR for 5′ and 3′ Portions of ALK Transcripts to Detect ALK Rearrangements in Lung Cancers

44. Supplementary Data from Epigenetic CRISPR Screens Identify Npm1 as a Therapeutic Vulnerability in Non–Small Cell Lung Cancer

45. Supplementary Figure 3 from The Use of Quantitative Real-Time Reverse Transcriptase PCR for 5′ and 3′ Portions of ALK Transcripts to Detect ALK Rearrangements in Lung Cancers

46. Data from The CRTC1-NEDD9 Signaling Axis Mediates Lung Cancer Progression Caused by LKB1 Loss

47. Supplementary Figure Legends 1-12 from The CRTC1-NEDD9 Signaling Axis Mediates Lung Cancer Progression Caused by LKB1 Loss

48. Supplementary Figures 1-12 from The CRTC1-NEDD9 Signaling Axis Mediates Lung Cancer Progression Caused by LKB1 Loss

49. Supplementary Methods from The CRTC1-NEDD9 Signaling Axis Mediates Lung Cancer Progression Caused by LKB1 Loss

50. Supplementary Table 1 from The CRTC1-NEDD9 Signaling Axis Mediates Lung Cancer Progression Caused by LKB1 Loss

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