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1. Dietary guanidinoacetate reduces spaghetti meat myopathy risk in the breast muscle of broiler chickens

Author: J. Dayan, Z. Uni, F. Soglia, M. Zampiga, M. Bordini, M. Petracci, and F. Sirri
Subjects: Breast myopathies, Broiler chicken, Creatine, Guanidinoacetate, Meat quality, Animal culture, SF1-1100
Abstract: The global demand for white chicken meat along with the increase in the occurrence of growth-related breast muscle myopathies (BMMs) [namely white striping (WS), wooden breast (WB), and spaghetti meat (SM)] highlights the need for solutions that will improve meat quality while maintaining the high productivity of modern broilers. Guanidinoacetate (GAA), a precursor of creatine, is used as a feed additive and has previously shown the potential to affect the quality of breast meat. This study investigated growth performance, meat quality and the risk ratio for the development of BMMs in broilers assigned to two dietary treatments: control (CON) group, fed a commercial basal diet, and supplemented GAA (sGAA) group, receiving the control diet supplemented on top with 0.06% GAA. Growth performance indicators such as BW, daily weight gain, daily feed intake, feed conversion ratio and cumulative feed conversion ratio were recorded on a pen basis. As a trait affecting animal welfare, the occurrence of foot pad dermatitis was also evaluated. At day 43, birds were processed, and breasts were scored for the incidence and severity of BMMs (n = 166 and 165 in CON and sGAA groups, respectively). Quality traits (ultimate pH, colour) and technological properties (i.e., drip and cooking losses, marinade uptake, shear force, and oxidation levels of the lipid and the protein fractions) of breast meat were assessed in both treatments on samples not showing any macroscopic sign of BMMs (n = 20 breast fillets per group). Data of myopathy risk ratio were analysed as the risk for each group to develop WS, WB, and SM myopathies. Our results show that while sGAA and control groups did not differ significantly in growth performance, a remarkably beneficial effect of GAA was observed on the incidence of BMMs with significantly reduced risk of sGAA group to develop SM myopathy. The risk of sGAA group to develop SM was 30% lower compared to CON (P = 0.028). Finally, a significantly lower drip loss was observed in sGAA in comparison with CON (1.78 vs 2.48%, P = 0.020). Together, our results show that the inclusion of 0.06% GAA in feed can improve the water-holding capacity of meat and reduce the risk to develop SM myopathy without compromising the performance of broilers.
Published: 2024
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2. Factors affecting space use by laying hens in a cage-free aviary system: effect of nest lighting at pullet housing and of curtain nest color during laying

Author: G. Pillan, G. Xiccato, C. Ciarelli, F. Bordignon, A. Concollato, A. Pascual, M. Birolo, F. Pirrone, F. Sirri, X. Averόs, I. Estevez, and A. Trocino
Subjects: laying, hen distribution, aviary, nest lighting, color preference, Animal culture, SF1-1100
Abstract: ABSTRACT: At 17 wk of age, 1,800 Lohman brown hens were housed in 8 pens of an experimental aviary system, specifically set up for the purposes of the present study, and kept until 26 wk without or with nest lighting (lights inside the nest 1.5 h before the lighting of the installation) for training in the nest use. Then, at 27 wk, 4 combinations of nest curtains were adopted to evaluate the effects on hens’ distribution, that is, nests with red (RR) or yellow (YY) curtains at all tiers; nests with red and yellow curtains at the first and second tier, respectively (RY); or nests with yellow and red curtains at the first and second tier, respectively (YR). The use of enlightened compared to dark nests at housing increased the oviposition rate (P < 0.001) and decreased the rate of broken (P < 0.001) and dirty eggs (P < 0.05) from 27 to 45 wk, while increasing the rate of eggs laid inside the nests (P < 0.001). The presence of yellow nest curtains increased the rate of hens on the floor in pens YY and YR compared to pens RR and RY (35.3 and 35.5% vs. 34.1 and 33.3%, respectively; P = 0.05) and the rate of floor eggs in pens YR (2.23% vs. 1.63 and 1.65% in pens RR and RY; P < 0.05). In pens RY, a higher rate of eggs was always found on the second tier compared to the first one with the most inhomogeneous distribution compared to pens RR, YY, and YR (+10.8 vs. +3.4, +1.9, and +4.6 percentage points of eggs laid on the second tier compared to the first one, respectively). In conclusion, nest lighting at housing trained hens to the use of nests while improving egg production in terms of quantity and quality. The use of yellow curtains on nests moved hens between the different levels of the aviary but this was not associated with an increased nest use for laying.
Published: 2023
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3. Corrigendum to Performance and meat quality traits of slow-growing chickens stimulated in ovo with galactooligosaccharides and exposed to heat stress [Poultry Science Volume 101, Issue 8, August 2022, 101972]

Author: S. Tavaniello, A. Slawinska, F. Sirri, M. Wu, D. De Marzo, M. Siwek, and G. Maiorano
Subjects: Animal culture, SF1-1100
Published: 2022
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4. Performance and meat quality traits of slow-growing chickens stimulated in ovo with galactooligosaccharides and exposed to heat stress

Author: S. Tavaniello, A. Slawinska, F. Sirri, M. Wu, D. De Marzo, M. Siwek, and G. Maiorano
Subjects: slow-growing broiler, in ovo stimulation, heat stress, performance, meat quality, Animal culture, SF1-1100
Abstract: ABSTRACT: In vivo performance, carcass and meat quality traits of slow-growing chickens stimulated in ovo with trans galactooligosaccharides (GOS) and exposed to heat stress were evaluated. On d 12 of egg incubation, 3,000 fertilized eggs (Hubbard JA57) were divided into prebiotic group (GOS) injected with 3.5 mg GOS/egg, saline group (S) injected with physiological saline (only to assess the hatchability rate) and an uninjected control group (C). After hatching, 600 male chicks (300 from GOS and 300 from C) were housed on floor pens (6 pens/treatment, 25 birds/pen) and reared under neutral (TN) or heat stress conditions (HS, 30°C from 36 to 50 d). BW, daily feed intake (DFI), daily weight gain (DWG), feed conversion rate (FCR), and mortality were measured. At 50 d of age, 15 randomly selected birds/treatment/environmental conditions were slaughtered and the pectoral muscle (PM) was collected for analyses. Hatchability was similar among groups. BW of the newly hatched chicks was lower (P < 0.01) in GOS compared to C. Final BW, DWG, DFI, and FCR were not affected (P > 0.05) by GOS. HS reduced final BW (−12.93%, P < 0.001). During finisher phase, DFI and DWG were lower (P < 0.001) and FCR was higher (P < 0.01) in HS compared to TN. Mortality was not affected (P > 0.05) by GOS and HS. Meat from GOS chickens had a higher (P < 0.01) pH and was darker (P < 0.05) compared to C. Proximate composition, cholesterol content, fatty acid profile, and intramuscular collagen properties of PM were not affected by GOS. The HS group showed a lower (P < 0.05) content of both collagen and monounsaturated fatty acids than TN group. Significant interactions between GOS and temperature were found for FA composition. In conclusion, the differences in performance have had an impact on the responses to HS in Hubbard chickens, but not on mortality rate. GOS did not relieve the negative effect of HS on chickens’ performance.
Published: 2022
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5. Effect of chronic heat stress on gastrointestinal histology and expression of feed intake-regulatory hormones in broiler chickens

Author: M. Mazzoni, M. Zampiga, P. Clavenzani, G. Lattanzio, C. Tagliavia, and F. Sirri
Subjects: Appetite regulation, Climate change, Enteroendocrine cell, Poultry, Thermal stress, Animal culture, SF1-1100
Abstract: Heat stress (HS) dramatically impairs the growth performance of broiler chickens, mainly as a consequence of reduced feed intake due to the loss of appetite. This study was aimed at evaluating the alterations induced by chronic HS conditions on the morphological and morphometric features of the gastrointestinal (GI) tract and on the expression of some enteroendocrine cells (EECs) involved in the regulation of feed intake in chickens. Three hundred male chickens (Ross 308) were divided into two experimental groups and raised either in thermoneutral environment for the whole fattening period (0–41 days) (TNT group) or subjected to chronic HS conditions (30 °C for 24 h/day) from 35 to 41 days (HS group). Samples of proventriculus, duodenum, jejunum and cecum were collected from 24 broilers (12/group). Haematoxylin-eosin was used for the morphometric evaluations, while immunohistochemistry was applied for the evaluation of EECs expressing ghrelin (GHR), cholecystokinin (CCK), neuropeptide Y (NPY), glucagon-like peptide-1 (GLP-1), and serotonin (5-HT). In the proventriculus, HS reduced total wall thickness and mucous layer height (P ≤ 0.01) as well as mean diameter, circumference, and area of the compound tubular glands (P ≤ 0.001) with respect to TNT. The small intestine of HS birds was characterised by decreased villous height and total thickness (duodenum, P ≤ 0.01; jejunum, P ≤ 0.001), whereas crypt depth and width were reduced only in the jejunum (P ≤ 0.01). HS had negligible effects on the morphological aspects of the cecum. In the proventriculus, an increase in GHR and NPY EECs was observed in response to HS (P ≤ 0.001). Similarly, the small intestine villi of the HS group showed greater GLP-1 (P ≤ 0.05), 5-HT (P ≤ 0.001) and CCK (P ≤ 0.01) EECs. Moreover, the expression of 5-HT EECs was higher in the duodenal (P ≤ 0.01) and jejunal (P ≤ 0.01) crypts of HS birds, whereas GLP-1 and CCK EECs increased only in jejunal crypts (P ≤ 0.05). Finally, 5-HT EEC expression was increased in the cecum of HS group (P ≤ 0.01). In conclusion, these outcomes demonstrate that chronic HS induces morphometric alterations not only in the small intestine but also in a key organ such as the proventriculus. Furthermore, HS conditions affect the presence and distribution of EECs, suggesting that some GI peptides and biogenic amine may be implicated in the regulation of appetite and voluntary feed intake in heat-stressed broiler chickens.
Published: 2022
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6. Effects of genotype and age on eggshell cuticle coverage and color profile in modern laying hen strains

Author: F. Sirri, M. Zampiga, and A. Berardinelli
Subjects: hen genotype, age, eggshell cuticle, color, egg, Animal culture, SF1-1100
Abstract: ABSTRACT: The aim of this research was to investigate the effects of laying hen genotype and age on eggshell cuticle deposition. A total of 4,320 brown eggs were obtained from 3 modern hen strains (A, B, and C), currently used worldwide for commercial egg production, at different intervals of age (20–30, 40–50, and 60–70 wk). Four samplings of 120 randomly collected eggs were carried out for each genotype/interval of age. Eggs were individually weighed and cuticle blue staining was used to assess quality and degree of cuticle coverage. On each egg, the eggshell color profile was assessed before and after staining using the CIE L*a*b* system and these values were used to calculate ΔE*ab. A 4-point scale visual score (VS) system was also applied to estimate the degree of cuticle coverage after staining (0 = no coverage, 1 = partial coverage, 2 = total coverage - low degree, 3 = total coverage - high degree). The effects of genotype and age and their interaction on eggshell color attributes were assessed by means of factorial ANOVA, while omnibus Chi-Square and Chi-squared Automatic Interaction Detector algorithm were applied for the analysis of VS data. Overall, both genotype and age affected the eggshell color profile as well as the degree of cuticle coverage. Hen strain A showed better cuticle deposition in comparison with B and particularly C one, being ΔE*ab values significantly higher. The VS evaluation revealed that eggs with impaired cuticle coverage degree increased with the hen age (23, 34, and 37%, respectively for 20–30, 40–50, and 60–70 wk; P < 0.05). However, a significant interaction between genotype and age was observed: transition from early to late hen age resulted in a significantly different pattern of ΔE*ab changes in each genotype. The classification tree analysis confirmed that the hen genotype has a greater effect than the age on cuticle deposition. In conclusion, considering the importance of the cuticle in table egg production, these results highlight the crucial role exerted by the genotype on eggshell cuticle coverage.
Published: 2022
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7. Impact of galactooligosaccharides delivered in ovo on mitigating negative effects of heat stress on performance and welfare of broilers

Author: A. Slawinska, M. Zampiga, F. Sirri, A. Meluzzi, M. Bertocchi, S. Tavaniello, and G. Maiorano
Subjects: broiler performance, food-pad dermatitis, galactooligosaccharides, heat stress, in ovo stimulation, Animal culture, SF1-1100
Abstract: ABSTRACT: Galactooligosaccharides (GOS) delivered in ovo improve intestinal health of broiler chickens. This study aimed to demonstrate the impact of in ovo stimulation with GOS prebiotic on day 12 of egg incubation on performance and welfare traits in broiler chickens. The incubating eggs were divided into 3 groups, based on the substance injected in ovo: 3.5 mg of GOS dissolved in 0.2 mL physiological saline (GOS), 0.2 mL physiological saline (S), or uninjected controls (C). Constant heat stress (HS) was induced on days 32 to 42 post-hatch by increasing environmental temperature to 30°C. Thermoneutral (TN) animals were kept at 25°C. The performance (body weight [BW], daily feed intake [DFI], daily weight gain [DWG], and feed conversion rate [FCR]) were measured and mortality was scored for starter (days 0 to 13), grower (days 14 to 27), and finisher (days 28 to 42) feeding phases. Rectal temperature was scored on days 32 to 42. Food-pad dermatitis (FPD) was scored post-mortem (day 42). GOS increased (P < 0.01) BW on day 42 (2.892 kg in GOS vs. 2.758 kg in C). Heat stress significantly reduced (P < 0.01) final BW (2.516 kg in TN vs. 3.110 kg in HS). During finisher phase, DFI was significantly higher in GOS vs. C (173.2 g vs. 165.7 g; P < 0.05). FCR calculated for the entire rearing period (days 0 to 42) ranged from 1.701 in C to 1.653 in GOS (P < 0.05). GOS improved FCR in HS animals during finisher phase (P < 0.05). Rectal temperature of GOS chickens under HS reached 42.5°C 1 day earlier than C and S (P < 0.05), which suggests that those birds recovered earlier from the high environmental temperature. Heat stress increased (P < 0.05) mortality about 5 times compared to TN during finisher phase (from 1.59% in TN to 7.69% in HS). GOS decreased FPD in TN conditions by 20% (no lesions in 81% in GOS vs. 60% in C). GOS delivered in ovo mitigated negative effects of HS on performance and welfare in broiler chickens.
Published: 2020
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8. Breast muscle and plasma metabolomics profile of broiler chickens exposed to chronic heat stress conditions

Author: M. Zampiga, L. Laghi, C. Zhu, A. Cartoni Mancinelli, S. Mattioli, and F. Sirri
Subjects: Energy homeostasis, Environmental condition, Metabolism, Metabolite, Omics technology, Animal culture, SF1-1100
Abstract: Understanding the variations of muscle and plasma metabolites in response to high environmental temperature can provide important information on the molecular mechanisms related to body energy homeostasis in heat-stressed broiler chickens. In this study, we investigated the effect of chronic heat stress conditions on the breast muscle (Pectoralis major) and plasma metabolomics profile of broiler chickens by means of an innovative, high-throughput analytical approach such as the proton nuclear magnetic resonance (1H NMR) spectrometry. A total of 300 Ross 308 male chicks were split into two experimental groups and raised in either thermoneutral conditions for the entire rearing cycle (0–41 days) (TNT group; six replicates of 25 birds/each) or exposed to chronic heat stress conditions (30 °C for 24 h/day) from 35 to 41 days (CHS group; six replicates of 25 birds/each). At processing (41 days), plasma and breast muscle samples were obtained from 12 birds/experimental group and then subjected to 1H NMR analysis. The reduction of BW and feed intake as well as the increase in rectal temperature and heterophil: lymphocyte ratio confirmed that our experimental model was able to stimulate a thermal stress response without significantly affecting mortality. The 1H NMR analysis revealed that a total of 26 and 19 molecules, mostly related to energy and protein metabolism as well as antioxidant response, showed significantly different concentrations respectively in the breast muscle and plasma in response to the thermal challenge. In conclusion, the results obtained in this study indicated that chronic heat stress significantly modulates the breast muscle and plasma metabolome in fast-growing broiler chickens, allowing to delineate potential metabolic changes that can have important implications in terms of body energy homeostasis, growth performance and product quality.
Published: 2021
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9. Black soldier fly defatted meal as a dietary protein source for broiler chickens: effects on carcass traits, breast meat quality and safety

Author: A. Schiavone, S. Dabbou, M. Petracci, M. Zampiga, F. Sirri, I. Biasato, F. Gai, and L. Gasco
Subjects: Hermetia illucens, broiler chickens, fatty acids, insect meal, heavy metals, Animal culture, SF1-1100
Abstract: Finding insect meals as alternative sources of poultry feedstuffs is a recent research topic; therefore, the present study aimed to evaluate the effects of defatted black soldier fly (Hermetia illucens L., HI) larvae meal in broiler chicken diets on the carcass characteristics and meat quality parameters, proximate composition, fatty acid profile and the heavy metal content of the breast meat. Four dietary treatments were designed: a control diet (HI0) and three experimental diets (HI5, HI10 and HI15), corresponding to 50, 100 and 150 g/kg HI inclusion levels, respectively. The inclusion of 50, 100 and 150 g HI meal per kg feed supply 16.56%, 33.01% and 49.63% of required crude protein. The broilers were slaughtered at day 35, the carcasses were weighed and the breast muscles were excised from 16 birds per each feeding group (two birds per replicate pens) and used for meat quality evaluation. Linear and quadratic responses were observed, for increasing HI meal levels, in the live and carcass weights (maximum for HI10). As far as the colour of the breast meat is concerned, redness (a*) showed a linear response, while yellowness (b*) linearly decreased with increasing HI meal levels (minimum for HI15). As the HI larvae meal increased in the diets, the moisture content linearly decreased and the protein content increased. The total saturated fatty acid and total monounsaturated fatty acid proportions rose to the detriment of the polyunsaturated fatty acid fraction. The HI larvae meal, used in the current study, represents a valuable protein source for broiler chickens when included by up to 100 g/kg in their diets, as an improved slaughtering performance was observed without any detrimental effects on meat quality parameters or heavy metal residues in the meat.
Published: 2019
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10. Implications of white striping and spaghetti meat abnormalities on meat quality and histological features in broilers

Author: G. Baldi, F. Soglia, M. Mazzoni, F. Sirri, L. Canonico, E. Babini, L. Laghi, C. Cavani, and M. Petracci
Subjects: chicken breast, abnormalities, white striping, spaghetti meat, meat quality, Animal culture, SF1-1100
Abstract: During the past few years, there has been an increasing prevalence of broiler breast muscle abnormalities, such as white striping (WS) and wooden breast conditions. More recently, a new muscular abnormality termed as spaghetti meat (SM) because of the altered structural integrity of the Pectoralis major muscle often associated with WS has emerged. Thus, this study aimed at evaluating the effects of WS and SM conditions, occurring alone or combined within the same P. major muscle, on meat quality traits and muscle histology. In two replications, 96 P. major muscles were classified into four classes: normal (N), WS, SM and WS/SM. The whole fillet was used for weight assessment and morphometric measurements, then each sample was cut in order to separate the superficial layer from the deep one and used to evaluate proximate composition, histological features, nuclear magnetic resonance relaxation times, functional properties and both myofibrillar and sarcoplasmic proteins profile. Fillets affected by WS and SM abnormalities exhibited higher weights and increased thickness and length. SM condition was associated with a relevant decrease in protein content coupled with a significant increase in moisture level, whereas fat content was affected only by the simultaneous presence of WS. Histological evaluations revealed that abnormal samples were characterized by several degenerative aspects that almost completely concerned the superficial layer of the fillets. White striped fillets exhibited necrosis and lysis of fibers, fibrosis, lipidosis, loss of cross striation and vacuolar degeneration. Moreover, SM samples were characterized by poor fiber uniformity and a progressive rarefaction of the endo- and peri-mysial connective tissue, whereas WS/SM fillets showed intermediate histological features. Nuclear magnetic resonance relaxation analysis revealed a higher proportion of extra-myofibrillar water in the superficial section of all the abnormal fillets, especially in SM samples, which consequently led to a reduction of the water holding capacity of meat. As for functional properties, abnormal fillets exhibited a lower protein solubility and higher ultimate pH values on both the superficial and deep sections. Although abnormal fillets exhibited higher yellowness values, no relevant effect on meat color was observed. The occurrence of WS and SM abnormalities led to increased carbonylation levels and more intense proteolytic processes. Overall, muscle abnormalities mainly affect the superficial layer of P. major muscle and particularly the occurrence of SM myopathy seems to implicate a more pronounced modification of meat quality traits than the mere presence of WS.
Published: 2018
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11. Fatty acid composition of chicken breast meat is dependent on genotype-related variation of FADS1 and FADS2 gene expression and desaturating activity

Author: E. Boschetti, A. Bordoni, A. Meluzzi, C. Castellini, A. Dal Bosco, and F. Sirri
Subjects: chicken, fatty acid desaturases, n-3/n-6 PUFA, FADS1, FADS2, Animal culture, SF1-1100
Abstract: In Western countries the dietary guidance emphasizes the need to decrease the intake of saturated fatty acids and to replace them with polyunsaturated fatty acids (PUFA), particularly long chain n-3 PUFA (LC-PUFA). The production of poultry meat having a lower fat content and healthier fatty acid (FA) profile is a hot topic for the poultry industry, and the possibility to identify genotypes able to produce meat with a higher LC-PUFA content deserves attention. The aims of the present study were to evidence in chicken (i) a genotype-related different expression of the desaturating enzymes delta-6 (Δ6, EC 1.14.99.25), delta-5 (Δ5, EC 1.14.19.) and delta-9 (Δ9, EC 1.14.19.1); (ii) the impact of the hypothesized different expression on the meat FA composition; (iii) the distribution of desaturase products in the different lipid classes. Slow (SG), medium (MG) and fast (FG) growing chickens fed the same diet were evaluated either for the relative expression of FADS1, FADS2 and SCD1 genes in liver (by q-PCR), or for the FA composition of breast meat. MG and particularly SG birds showed a greater expression of FADS2 and FADS1 genes, a higher Δ6 and Δ5 activity (estimated using desaturase indices), and consequently a higher LC-PUFA content in the breast meat than FG birds. The relationship between genotype and desaturating ability was demonstrated, with a significant impact on the PUFA content of breast meat. Due to the high consumption rate of avian meat, the identification of the best genotypes for meat production could represent an important goal not only for the food industry, but also for the improvement of human nutrition.
Published: 2016
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12. Effect of fast-, medium- and slow-growing strains on meat quality of chickens reared under the organic farming method

Author: F. Sirri, C. Castellini, M. Bianchi, M. Petracci, A. Meluzzi, and A. Franchini
Subjects: organic farming, chicken, genotype, meat quality, Animal culture, SF1-1100
Abstract: The characteristics of meat quality, chemical and fatty acid composition, from fast-growing (FG) and medium-growing (MG) meat-type and slow-growing (SG) egg-type chickens reared under organic conditions were compared. Three-hundred and sixty 1-day-old male chicks, equally divided into three experimental groups represented by strains (FG: Cobb 700, MG: Naked neck Kabir and SG: Brown Classic Lohman) were housed into three poultry houses with outdoor pasture availability of 10 m2/bird located in the same Research Centre of the University of Perugia. All the birds were fed ad libitum the same diets formulated according to the European Union (EU) Regulations by using organic raw materials. Birds from the FG and MG groups were raised until 81 days, whereas birds from the SG group were raised until 96 days in order to achieve an acceptable market live weight. SG birds showed significantly (P < 0.01) higher breast meat drip and cook losses, Allo-Kramer shear values and collagen content. In comparison with FG and SG, MG exhibited a higher breast meat pH (5.86% v. 5.79% and 5.78%, respectively; P < 0.01) and a lower lightness (54.88% v. 57.81% and 56.98%, respectively; P < 0.05). Genotype dramatically affected the lipid content as well as the fatty acid composition of both breast and thigh meat. SG exhibited the lowest content of lipid, both in breast and in thigh meat, the lowest proportions of monounsaturated fatty acids (MUFA) and the highest proportions of polyunsaturated fatty acids (PUFA). The total n-3 PUFA of SG breast meat was double that of FG meat and intermediate with respect to MG birds (8.07% v. 4.07% v. 5.14% total fatty acids; P < 0.01). The fatty acid composition of thigh meat is similar to that of breast meat, but the differences among genotypes are less pronounced. Total saturated fatty acids were not affected by the genotype. In conclusion, meat functional properties of FG and MG strains appeared much more attractive both for industry and consumer (lower drip and cook losses and higher tenderness), whereas from a nutritional point of view, meat from SG appeared healthier (less fat and higher content of n-3 PUFA) and thus might better fit with the consumer’s expectations of organic products.
Published: 2011
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13. Dietary conjugated linoleic acid supplementation of laying hens: effects on egg fatty acid composition and quality

Author: A. Franchini, G. Minelli, F. Sirri, N. Tallarico, and A. Meluzzi
Subjects: laying hen, egg, conjugated linoleic acid (CLA)., Animal culture, SF1-1100
Abstract: Conjugated linoleic acid (CLA) is a common term for a group of positional and geometric isomers of linoleic acid; the most commonly found in natural products is cis-9, trans-11-octadecanoic acid. Naturally occurring CLA is produced as an intermediate product of microbial metabolism of linoleic acid in the rumen. For this reason the CLA concentration is higher in ruminant products, such as milk, cheese and meat compared to products derived from monogastrics, particularly chicken meat and egg yolk. Recent investigations suggest that CLA have anticarcinogenic properties, antiatherosclerotic and antioxidant activity (Ip et al., 1995; Nicolosi et al., 1997; Du et al., 2001)...
Published: 2011
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14. Oxidative stability and quality traits of n-3 PUFA enriched chicken meat

Author: A. Franchini, N. Iaffaldano, N. Tallarico, G. Minelli, and F. Sirri
Subjects: chicken, n-3 PUFA, oxidative stability, organoleptic properties, Animal culture, SF1-1100
Abstract: Considering the beneficial effects of n-3 polyunsaturated fatty acids (PUFA) for human health (Leskanich and Noble, 1997) several studies have been carried out to enrich animal products with these compounds. Both eggs and poultry meat enriched with n-3 PUFA may be considered as valid n-3 PUFA alternative sources to natural occurring fish products (Meluzzi et al., 2001; Sirri et al., 2002)...
Published: 2011
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15. Foot dermatitis and productive traits in broiler chickens kept with different stocking densities, litter types and light regimen

Author: A. Meluzzi, S. Lolli, E. Folegatti, G. Minelli, and F. Sirri
Subjects: Stocking density, Litter type, Light regimen, Broiler chicken., Animal culture, SF1-1100
Abstract: The purpose of the research was to study the effects of high and low stocking density of broiler chickens, different types of litter and the adoption of short and long lighting regimen on broiler welfare through the evaluation of their productivity and incidence of foot pad dermatitis. 2,400 male Ross 508 were divided into 8 groups of four replicates each and reared in 32 pens according to a 3 factors experimental design of two levels each: low (LD) or high (HD) stocking density (11 and 14 birds/m2 for LD and HD respectively), chopped straw (CS) or wood shaving (WS) litters and short (SL) or long (LL) light regimens (16 h light and 8 h dark or 23 h light and 1 h dark respectively for SL and LL). Broiler growth rate and feed efficiency were positively influenced by a stocking density lower than 30 kg of live weight per m2. The adoption of a short light regimen reduced feed intake and improved feed conversion rate without modifying body live weight. Chickens reared on wood shaving litter showed a lower incidence of foot pad dermatitis.
Published: 2010
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16. Egg quality traits of laying hens reared in organic and conventional systems

Author: A. Franchini, A. Meluzzi, E. Folegatti, F. Sirri, and G. Minelli
Subjects: Laying hen, Organic and conventional systems, Egg, Physico-chemical properties., Animal culture, SF1-1100
Abstract: This study aims to compare the physico-chemical properties of eggs (weight, eggshell breaking strength, Haugh index, yolk colour, lipid, cholesterol, protein, ash and dry matter) laid either by hens reared according to the organic method or by caged hens kept in conventional system. More than 1,400 eggs have been analysed at the beginning, in the middle and at the end of the laying cycle in organic and conventional farms. The egg obtained from the organic system were lighter (64.4 vs 66.2 g) being yolk, albumen and eggshell weights statistically lower in comparison with those produced in conventional system. The yolk/albumen ratio resulted lower in the organic eggs (0.38 vs 0.39). The percentage of eggshell was not affected by the hen rearing system while the eggshell strength resulted higher in the eggs produced in the conventional system (3.265 vs 3.135 kg). The organic yolks were paler than the conventional ones. Organic eggs showed significantly higher contents of protein (17.1% vs 16.7%) and cholesterol (1.26% vs 1.21%).
Published: 2010
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17. Avian model to mitigate gut-derived immune response and oxidative stress during heat.

Author: Anna Slawinska, Sérgio Mendes, A. Dunislawska, Maria Siwek, M. Zampiga, F. Sirri, A. Meluzzi, S. Tavaniello, and Giuseppe Maiorano
Published: 2019
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18. Chronic tinnitus and BDNF/GDNF CpG promoter methylations: a case–control study

Author: Orenay-Boyacioglu, Seda, Caliskan, Metin, Boyacioglu, Olcay, Coskunoglu, Aysun, Bozkurt, Gokay, and Cam, F. Sirri
Published: 2019
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19. Prevalence of the Angiotensin I Converting Enzyme Gene Insertion/Deletion Polymorphism in a Healthy Turkish Population

Author: Berdeli, Afig and Cam, F. Sirri
Published: 2009
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20. Volume IV The DUNE far detector single-phase technology

Author: Abi, B. Acciarri, R. Acero, M. A. Adamov, G. Adams, D. and Adinolfi, M. Ahmad, Z. Ahmed, J. Alion, T. Monsalve, S. Alonso Alt, C. Anderson, J. Andreopoulos, C. Andrews, M. Andrianala, F. Andringa, S. Ankowski, A. Antonova, M. and Antusch, S. Aranda-Fernandez, A. Ariga, A. Arnold, L. O. and Arroyave, M. A. Asaadi, J. Aurisano, A. Aushev, V and Autiero, D. Azfar, F. Back, H. Back, J. J. Backhouse, C. and Baesso, P. Bagby, L. Bajou, R. Balasubramanian, S. and Baldi, P. Bambah, B. Barao, F. Barenboim, G. Barker, G. and Barkhouse, W. Barnes, C. Barr, G. Barranco Monarca, J. and Barros, N. Barrow, J. L. Bashyal, A. Basque, V Bay, F. Bazo Alba, J. Beacom, J. F. Bechetoille, E. Behera, B. Bellantoni, L. Bellettini, G. Bellini, V Beltramello, O. Belver, D. Benekos, N. Neves, F. Bento Berger, J. and Berkman, S. Bernardini, P. Berner, R. M. Berns, H. and Bertolucci, S. Betancourt, M. Bezawada, Y. Bhattacharjee, M. and Bhuyan, B. Biagi, S. Bian, J. Biassoni, M. Biery, K. and Bilki, B. Bishai, M. Bitadze, A. Blake, A. Siffert, B. Blanco Blaszczyk, F. Blazey, G. Blucher, E. and Boissevain, J. Bolognesi, S. Bolton, T. Bonesini, M. and Bongrand, M. Bonini, F. Booth, A. Booth, C. Bordoni, S. and Borkum, A. Boschi, T. Bostan, N. Bour, P. Boyd, S. and Boyden, D. Bracinik, J. Braga, D. Brailsford, D. and Brandt, A. Bremer, J. Brew, C. Brianne, E. Brice, S. J. and Brizzolari, C. Bromberg, C. Brooijmans, G. Brooke, J. and Bross, A. Brunetti, G. Buchanan, N. Budd, H. Caiulo, D. Calafiura, P. Calcutt, J. Calin, M. Calvez, S. and Calvo, E. Camilleri, L. Caminata, A. Campanelli, M. and Caratelli, D. Carini, G. Carlus, B. Carniti, P. and Terrazas, I. Caro Carranza, H. Castillo, A. Castromonte, C. and Cattadori, C. Cavalier, F. Cavanna, F. Centro, S. and Cerati, G. Cervelli, A. Cervera Villanueva, A. Chalifour, M. and Chang, C. Chardonnet, E. Chatterjee, A. Chattopadhyay, S. Chaves, J. Chen, H. Chen, M. Chen, Y. Cherdack, D. Chi, C. Childress, S. Chiriacescu, A. Cho, K. and Choubey, S. Christensen, A. Christian, D. Christodoulou, G. and Church, E. Clarke, P. Coan, T. E. Cocco, A. G. and Coelho, J. Conley, E. Conrad, J. Convery, M. Corwin, L. and Cotte, P. Cremaldi, L. Cremonesi, L. Crespo-Anadon, I, J. Cristaldo, E. Cross, R. Cuesta, C. Cui, Y. and Cussans, D. Dabrowski, M. Da Motta, H. Peres, L. Da Silva and David, Q. Davies, G. S. Davini, S. Dawson, J. De, K. and De Almeida, R. M. Debbins, P. De Bonis, I Decowski, M. and De Gouvea, A. De Holanda, P. C. Astiz, I. L. De Icaza and Deisting, A. De Jong, P. Delbart, A. Delepine, D. and Delgado, M. Dell'Acqua, A. De Lurgio, P. De Mello Neto, J. R. DeMuth, D. M. Dennis, S. Densham, C. Deptuch, G. and De Roeck, A. De Romeri, V De Vries, J. Dharmapalan, R. and Dias, M. Diaz, F. Diaz, J. Di Domizio, S. Di Giulio, L. and Ding, P. Di Noto, L. Distefano, C. Diurba, R. Diwan, M. Djurcic, Z. Dokania, N. Dolinski, M. Domine, L. and Douglas, D. Drielsma, F. Duchesneau, D. Duffy, K. Dunne, P. Durkin, T. Duyang, H. Dvornikov, O. Dwyer, D. and Dyshkant, A. Eads, M. Edmunds, D. Eisch, J. Emery, S. and Ereditato, A. Escobar, C. Sanchez, L. Escudero Evans, J. J. Ewart, E. Ezeribe, A. C. Fahey, K. Falcone, A. and Farnese, C. Farzan, Y. Felix, J. Fernandez-Martinez, E. and Fernandez Menendez, P. Ferraro, F. Fields, L. Filkins, A. and Filthaut, F. Fitzpatrick, R. S. Flanagan, W. Fleming, B. and Flight, R. Fowler, J. Fox, W. Franc, J. Francis, K. and Franco, D. Freeman, J. Freestone, J. Fried, J. and Friedland, A. Fuess, S. Furic, I Furmanski, A. P. Gago, A. Gallagher, H. Gallego-Ros, A. Gallice, N. Galymov, V and Gamberini, E. Gamble, T. Gandhi, R. Gandrajula, R. and Gao, S. Garcia-Gamez, D. Garcia-Peris, M. A. Gardiner, S. and Gastler, D. Ge, G. Gelli, B. Gendotti, A. Gent, S. and Ghorbani-Moghaddam, Z. Gibin, D. Gil-Botella, I Girerd, C. Giri, A. Gnani, D. Gogota, O. Gold, M. and Gollapinni, S. Gollwitzer, K. Gomes, R. A. Gomez Bermeo, L. and Gomez Fajardo, L. S. Gonnella, F. Gonzalez-Cuevas, J. and Goodman, M. C. Goodwin, O. Goswami, S. Gotti, C. and Goudzovski, E. Grace, C. Graham, M. Gramellini, E. Gran, R. Granados, E. Grant, A. Grant, C. Gratieri, D. and Green, P. Green, S. Greenler, L. Greenwood, M. Greer, J. and Griffith, C. Groh, M. Grudzinski, J. Grzelak, K. Gu, W. Guarino, V Guenette, R. Guglielmi, A. Guo, B. and Guthikonda, K. Gutierrez, R. Guzowski, P. Guzzo, M. M. and Gwon, S. Habig, A. Hackenburg, A. Hadavand, H. Haenni, R. Hahn, A. Haigh, J. Haiston, J. Hamernik, T. and Hamilton, P. Han, J. Harder, K. Harris, D. A. Hartnell, J. Hasegawa, T. Hatcher, R. Hazen, E. Heavey, A. and Heeger, K. M. Hennessy, K. Henry, S. Hernandez Morquecho, M. and Herner, K. Hertel, L. Hesam, A. S. Hewes, J. and Pichardo, A. Higuera Hill, T. Hillier, S. J. Himmel, A. and Hoff, J. Hohl, C. Holin, A. Hoppe, E. Horton-Smith, G. A. Hostert, M. Hourlier, A. Howard, B. Howell, R. and Huang, J. Huang, J. Hugon, J. Iles, G. Iliescu, A. M. and Illingworth, R. Ioannisian, A. Itay, R. Izmaylov, A. and James, E. Jargowsky, B. Jediny, F. Jesus-Valls, C. Ji, X. Jiang, L. Jimenez, S. Jipa, A. Joglekar, A. and Johnson, C. Johnson, R. Jones, B. Jones, S. Jung, C. and Junk, T. Jwa, Y. Kabirnezhad, M. Kaboth, A. Kadenko, I and Kamiya, F. Karagiorgi, G. Karcher, A. Karolak, M. and Karyotakis, Y. Kasai, S. Kasetti, S. P. Kashur, L. and Kazaryan, N. Kearns, E. Keener, P. Kelly, K. J. Kemp, E. and Ketchum, W. Kettell, S. Khabibullin, M. Khotjantsev, A. and Khvedelidze, A. Kim, D. King, B. Kirby, B. Kirby, M. and Klein, J. Koehler, K. Koerner, L. W. Kohn, S. and Koller, P. P. Kordosky, M. Kosc, T. Kose, U. Kostelecky, V Kothekar, K. Krennrich, F. Kreslo, I Kudenko, Y. and Kudryavtsev, V Kulagin, S. Kumar, J. Kumar, R. Kuruppu, C. Kus, V Kutter, T. Lambert, A. Lande, K. Lane, C. E. Lang, K. Langford, T. Lasorak, P. Last, D. and Lastoria, C. Laundrie, A. Lawrence, A. Lazanu, I LaZur, R. Le, T. Learned, J. LeBrun, P. Miotto, G. Lehmann and Lehnert, R. de Oliveira, M. Leigui Leitner, M. Leyton, M. and Li, L. Li, S. Li, S. Li, T. Li, Y. Liao, H. and Lin, C. Lin, S. Lister, A. Littlejohn, B. R. Liu, J. and Lockwitz, S. Loew, T. Lokajicek, M. Lomidze, I Long, K. and Loo, K. Lorca, D. Lord, T. LoSecco, J. Louis, W. C. and Luk, K. Luo, X. Lurkin, N. Lux, T. Luzio, V. P. and MacFarland, D. Machado, A. Machado, P. Macias, C. and Macier, J. Maddalena, A. Madigan, P. Magill, S. Mahn, K. and Maio, A. Maloney, J. A. Mandrioli, G. Maneira, J. C. and Manenti, L. Manly, S. Mann, A. Manolopoulos, K. Plata, M. Manrique Marchionni, A. Marciano, W. Marfatia, D. and Mariani, C. Maricic, J. Marinho, F. Marino, A. D. and Marshak, M. Marshall, C. Marshall, J. Marteau, J. and Martin-Albo, J. Martinez, N. Caicedo, D. A. Martinez and Martynenko, S. Mason, K. Mastbaum, A. Masud, M. Matsuno, S. Matthews, J. Mauger, C. Mauri, N. Mavrokoridis, K. and Mazza, R. Mazzacane, A. Mazzucato, E. McCluskey, E. and McConkey, N. McFarland, K. S. McGrew, C. McNab, A. and Mefodiev, A. Mehta, P. Melas, P. Mellinato, M. Mena, O. and Menary, S. Mendez, H. Menegolli, A. Meng, G. and Messier, M. Metcalf, W. Mewes, M. Meyer, H. Miao, T. and Michna, G. Miedema, T. Migenda, J. Milincic, R. Miller, W. Mills, J. Milne, C. Mineev, O. Miranda, O. G. and Miryala, S. Mishra, C. Mishra, S. Mislivec, A. Mladenov, D. Mocioiu, I Moffat, K. Moggi, N. Mohanta, R. and Mohayai, T. A. Mokhov, N. Molina, J. A. Bueno, L. Molina and Montanari, A. Montanari, C. Montanari, D. Montano Zetina, L. M. Moon, J. Mooney, M. Moor, A. Moreno, D. Morgan, B. Morris, C. Mossey, C. Motuk, E. Moura, C. A. and Mousseau, J. Mu, W. Mualem, L. Mueller, J. Muether, M. and Mufson, S. Muheim, F. Muir, A. Mulhearn, M. and Muramatsu, H. Murphy, S. Musser, J. Nachtman, J. Nagu, S. Nalbandyan, M. Nandakumar, R. Naples, D. Narita, S. and Navas-Nicolas, D. Nayak, N. Nebot-Guinot, M. Necib, L. and Negishi, K. Nelson, J. K. Nesbit, J. Nessi, M. and Newbold, D. Newcomer, M. Newhart, D. Nichol, R. Niner, E. Nishimura, K. Norman, A. Northrop, R. Novella, P. and Nowak, J. A. Oberling, M. Del Campo, A. Olivares Olivier, A. and Onel, Y. Onishchuk, Y. Ott, J. Pagani, L. Pakvasa, S. Palamara, O. Palestini, S. Paley, J. M. Pallavicini, M. Palomares, C. Pantic, E. Paolone, V Papadimitriou, V and Papaleo, R. Papanestis, A. Paramesvaran, S. Parke, S. and Parsa, Z. Parvu, M. Pascoli, S. Pasqualini, L. and Pasternak, J. Pater, J. Patrick, C. Patrizii, L. and Patterson, R. B. Patton, S. Patzak, T. Paudel, A. and Paulos, B. Paulucci, L. Pavlovic, Z. Pawloski, G. Payne, D. Pec, V Peeters, S. J. Penichot, Y. Pennacchio, E. and Penzo, A. Peres, O. L. Perry, J. Pershey, D. Pessina, G. and Petrillo, G. Petta, C. Petti, R. Piastra, F. and Pickering, L. Pietropaolo, F. Pillow, J. Plunkett, R. and Poling, R. Pons, X. Poonthottathil, N. Pordes, S. and Potekhin, M. Potenza, R. Potukuchi, V, B. Pozimski, J. and Pozzato, M. Prakash, S. Prakash, T. Prince, S. Prior, G. and Pugnere, D. Qi, K. Qian, X. Raaf, J. Raboanary, R. and Radeka, V Rademacker, J. Radics, B. Rafique, A. and Raguzin, E. Rai, M. Rajaoalisoa, M. Rakhno, I and Rakotondramanana, H. Rakotondravohitra, L. Ramachers, Y. and Rameika, R. Ramirez Delgado, M. Ramson, B. Rappoldi, A. and Raselli, G. Ratoff, P. Ravat, S. Razafinime, H. Real, J. and Rebel, B. Redondo, D. Reggiani-Guzzo, M. Rehak, T. and Reichenbacher, J. Reitzner, S. D. Renshaw, A. Rescia, S. and Resnati, F. Reynolds, A. Riccobene, G. Rice, L. C. and Rielage, K. Rigaut, Y. Rivera, D. Rochester, L. Roda, M. and Rodrigues, P. Alonso, M. Rodriguez Rondon, J. Rodriguez and Roeth, A. Rogers, H. Rosauro-Alcaraz, S. Rossella, M. and Rout, J. Roy, S. Rubbia, A. Rubbia, C. Russell, B. and Russell, J. Ruterbories, D. Saakyan, R. Sacerdoti, S. and Safford, T. Sahu, N. Sala, P. Samios, N. Sanchez, M. and Sanders, D. A. Sankey, D. Santana, S. Santos-Maldonado, M. and Saoulidou, N. Sapienza, P. Sarasty, C. Sarcevic, I and Savage, G. Savinov, V Scaramelli, A. Scarff, A. and Scarpelli, A. Schaffer, T. Schellman, H. Schlabach, P. and Schmitz, D. Scholberg, K. Schukraft, A. Segreto, E. and Sensenig, J. Seong, I Sergi, A. Sergiampietri, F. and Sgalaberna, D. Shaevitz, M. Shafaq, S. Shamma, M. and Sharma, H. R. Sharma, R. Shaw, T. Shepherd-Themistocleous, C. Shin, S. Shooltz, D. Shrock, R. Simard, L. Simos, N. Sinclair, J. Sinev, G. Singh, J. Singh, J. Singh, V Sipos, R. Sippach, F. Sirri, G. Sitraka, A. and Siyeon, K. Smargianaki, D. Smith, A. Smith, A. Smith, E. and Smith, P. Smolik, J. Smy, M. Snopok, P. Nunes, M. Soares Sobel, H. Soderberg, M. Solano Salinas, C. J. and Soldner-Rembold, S. Solomey, N. Solovov, V Sondheim, W. E. and Sorel, M. Soto-Oton, J. Sousa, A. Soustruznik, K. and Spagliardi, F. Spanu, M. Spitz, J. Spooner, N. J. and Spurgeon, K. Staley, R. Stancari, M. Stanco, L. Steiner, H. Stewart, J. Stillwell, B. Stock, J. Stocker, F. and Stokes, T. Strait, M. Strauss, T. Striganov, S. Stuart, A. Summers, D. Surdo, A. Susic, V Suter, L. Sutera, C. Svoboda, R. Szczerbinska, B. Szelc, A. Talaga, R. and Tanaka, H. Oregui, B. Tapia Tapper, A. Tariq, S. Tatar, E. Tayloe, R. Teklu, A. Tenti, M. Terao, K. Ternes, C. A. Terranova, F. Testera, G. Thea, A. Thompson, J. L. and Thorn, C. Timm, S. Tonazzo, A. Torti, M. Tortola, M. and Tortorici, F. Totani, D. Toups, M. Touramanis, C. and Trevor, J. Trzaska, W. H. Tsai, Y. T. Tsamalaidze, Z. and Tsang, K. Tsverava, N. Tufanli, S. Tull, C. Tyley, E. and Tzanov, M. Uchida, M. A. Urheim, J. Usher, T. and Vagins, M. Vahle, P. Valdiviesso, G. Valencia, E. and Vallari, Z. Valle, J. W. Vallecorsa, S. Van Berg, R. Van de Water, R. G. Forero, D. Vanegas Varanini, F. Vargas, D. and Varner, G. Vasel, J. Vasseur, G. Vaziri, K. Ventura, S. Verdugo, A. Vergani, S. Vermeulen, M. A. Verzocchi, M. de Souza, H. Vieira Vignoli, C. Vilela, C. Viren, B. and Vrba, T. Wachala, T. Waldron, V, A. Wallbank, M. and Wang, H. Wang, J. Wang, Y. Wang, Y. Warburton, K. and Warner, D. Wascko, M. Waters, D. Watson, A. Weatherly, P. Weber, A. Weber, M. Wei, H. Weinstein, A. Wenman, D. Wetstein, M. While, M. R. White, A. Whitehead, L. H. and Whittington, D. Wilking, M. J. Wilkinson, C. Williams, Z. Wilson, F. Wilson, R. J. Wolcott, J. Wongjirad, T. and Wood, K. Wood, L. Worcester, E. Worcester, M. Wret, C. Wu, W. Wu, W. Xiao, Y. Yang, G. Yang, T. and Yershov, N. Yonehara, K. Young, T. Yu, B. Yu, J. and Zalesak, J. Zambelli, L. Zamorano, B. Zani, A. Zazueta, L. Zeller, G. Zennamo, J. Zeug, K. Zhang, C. Zhao, M. Zhivun, E. Zhu, G. Zimmerman, E. D. Zito, M. and Zucchelli, S. Zuklin, J. Zutshi, V Zwaska, R. DUNE Collaboration
Published: 2020

21. Volume I. Introduction to DUNE

Author: Abi, B. Acciarri, R. Acero, M. A. Adamov, G. Adams, D. and Adinolfi, M. Ahmad, Z. Ahmed, J. Alion, T. Monsalve, S. Alonso Alt, C. Anderson, J. Andreopoulos, C. Andrews, M. Andrianala, F. Andringa, S. Ankowski, A. Antonova, M. and Antusch, S. Aranda-Fernandez, A. Ariga, A. Arnold, L. O. and Arroyave, M. A. Asaadi, J. Aurisano, A. Aushev, V. and Autiero, D. Azfar, F. Back, H. Back, J. J. Backhouse, C. and Baesso, P. Bayby, L. Bajou, R. Balasubramanian, S. and Baldi, P. Bambah, B. Barao, F. Barenhoim, G. Barker, G. and Barkhouse, W. Barnes, C. Barr, G. Monarca, J. Barranco and Barros, N. Barrow, J. L. Bashyal, A. Basque, V. Bay, F. Alba, J. Bazo Beacom, J. F. Bechetoille, E. Behera, B. Bellantoni, L. Bellettini, G. Bellini, V. and Beltramello, O. Belver, D. Benckos, N. Neves, F. Bento and Berger, J. Berkman, S. Bernardini, P. Berner, R. M. and Berns, H. Bertolucci, S. Betancourt, M. Bezawada, Y. and Bhattacharjee, M. Bhuyan, B. Biagi, S. Bian, J. and Biassoni, M. Bierv, K. Bilki, B. Bishai, M. Bitadze, A. and Blake, A. Siffert, B. Blanco Blaszczyk, F. Blazey, G. and Blucher, E. Boissevain, J. Bolognesi, S. Bolton, T. and Bonesini, M. Bongrand, M. Bonini, F. Booth, A. Booth, C. and Bordoni, S. Borkum, A. Boschi, T. Bostan, N. Bour, P. Boyd, S. Bovden, D. Bracinik, J. Braga, D. and Brailsford, D. Brandt, A. Bremer, J. Brew, C. Brianne, E. Brice, S. J. Brizzolari, C. Bromherg, C. Brooijmans, G. Brooke, J. Bross, A. Brunetti, G. Buchanan, N. and Budd, H. Caiulo, D. Calaftura, P. Calcutt, J. Calin, M. and Calvez, S. Calvo, E. Camilleri, L. Caminata, A. and Campanelli, M. Caratelli, D. Carini, G. Carlus, B. and Carniti, P. Terrazas, I. Caro Carranza, H. Castillo, A. and Castromonte, C. Cattadori, C. Cavalier, F. Cavanna, F. and Centro, S. Cerati, G. Cervelli, A. Cervera Villanueva, A. and Chalifour, M. Chang, C. Chardonnet, E. Chatterjee, A. and Chattopadhyay, S. Chaves, J. Chen, H. Chen, M. Chen, Y. Cherdack, D. Chi, C. Childress, S. Chiriacescu, A. and Cho, K. Choubey, S. Christensen, A. Christian, D. and Christodoulou, G. Church, E. Clarke, P. Coan, T. E. and Cocco, A. G. Coelho, J. Conley, E. Conrad, J. Convery, M. Corwin, L. Cotte, P. Cremaldi, L. Cremonesi, L. and Crespo-Anadon, J. I. Cristaldo, E. Cross, R. Cuesta, C. and Cui, Y. Cussans, D. Dahrowski, M. Da Motta, H. Da Silva Peres, L. David, Q. Davies, G. S. Davini, S. Dawson, J. and De, K. De Almeida, R. M. Debbins, P. De Bonis, I. and Decowski, M. De Gouvea, A. De Holanda, P. C. Astiz, I. L. De Icaza Deistin, A. De Jong, P. Delbart, A. Delepine, D. and Delgado, M. Dell'Acqua, A. De Lurgio, P. De Mello Neto, J. R. DeMuth, D. M. Dennis, S. Densham, C. Deptuch, G. and De Roeck, A. De Romeri, V. De Vries, J. Dharmapalan, R. and Dias, M. Diaz, F. Diaz, J. Di Domizio, S. Di Giulio, L. Ding, P. Di Noto, L. Distefano, C. Diurba, R. and Diwan, M. Djurcic, Z. Dokania, N. Dolinski, M. Domine, L. Douglas, D. Drielsma, F. Duchesneau, D. Duffy, K. and Dunne, P. Durkin, T. Duyang, H. Dvornikov, O. Dwyer, D. and Dyshkant, A. Eads, M. Edmunds, D. Eisch, J. Emery, S. Ereditato, A. Escobar, C. Escudero Sanchez, L. Evans, J. J. Ewart, E. Ezeribe, A. C. Fahey, K. Falcone, A. and Farnese, C. Farzan, Y. Felix, J. Fernandez-Martinez, E. and Menendez, P. Fernandez Ferraro, F. Fields, L. Filkins, A. and Filthaut, F. Fitzpatrick, R. S. Flanagan, W. Fleming, B. and Flight, R. Fowler, J. Fox, W. Franc, J. Francis, K. and Franco, D. Freeman, J. Freestone, J. Fried, J. and Friedland, A. Fuess, S. Furic, I. Furmanski, A. P. Gago, A. Gallagher, H. Gallego-Ros, A. Gallice, N. Galymov, V. and Gamberini, E. Gamble, T. Gandhi, R. Gandrajula, R. and Gao, S. Garcia-Gamez, D. Garcia-Peris, M. A. Gardiner, S. and Gastler, D. Ge, G. Gelli, B. Gendotti, A. Gent, S. and Ghorbani-Moghaddam, Z. Gibin, D. Gil-Botella, L. Girerd, C. Giri, A. Gnani, D. Gogota, O. Gold, M. and Gollapinni, S. Gollwitzer, K. Gomes, R. A. Bermeo, L. Gomez and Fajardo, L. S. Gomez Gonnella, F. Gonzalez-Cuevas, J. and Goodman, M. C. Goodwin, O. Goswami, S. Gotti, C. and Goudzovski, E. Grace, C. Graham, M. Gramellini, E. Gran, R. Granados, E. Grant, A. Grant, C. Gratieri, D. and Green, P. Green, S. Greenler, L. Greenwood, M. Greer, J. and Griffith, C. Groh, M. Grudzinski, J. Grzelak, K. Gu, W. Guarino, V. Guenette, R. Guglielmi, A. Guo, B. and Guthikonda, K. Gutierrez, R. Guzowski, P. Guzzo, M. M. and Gwon, S. Habig, A. Hackenburg, A. Hadavand, H. Haenni, R. Hahn, A. Haigh, J. Haiston, J. Hamernik, T. and Hamilton, P. Han, J. Harder, K. Harris, D. A. Hartnell, J. Hasegawa, T. Hatcher, R. Hazen, E. Heavey, A. and Heeger, K. M. Hennessy, K. Henry, S. Morquecho, Vi. Hernandez Herner, K. Hertel, L. Hesam, A. S. Hewes, J. and Pichardo, A. Higuera Hill, T. Hillier, S. J. Himmel, A. and Hoff, J. Hohl, C. Holin, A. Hoppe, E. Horton-Smith, G. A. Hostert, M. Hourlier, A. Howard, B. Howell, R. and Huang, J. Huang, J. Hugon, J. Iles, G. Iliescu, A. M. and Illingworth, R. Ioannisian, A. Itay, R. Izmaylov, A. and James, E. Jargowsky, B. Jediny, F. Jesus-Valls, C. Ji, X. Jiang, L. Jimenez, S. Jipa, A. Joglekar, A. and Johnson, C. Johnson, R. Jones, B. Jones, S. Jung, C. and Junk, T. Jwa, Y. Kabirnezhad, M. Kaboth, A. Kadenko, I. and Kamiya, F. Karagiorgi, G. Karcher, A. Karolak, M. and Karyotakis, Y. Kasai, S. Kasetti, S. P. Kashur, L. and Kazaryan, N. Kearns, E. Keener, P. Kelly, K. J. Kemp, E. and Ketchum, W. Kettell, S. Kliabibullin, M. Khotjantsev, A. and Khvedelidze, A. Kim, D. King, B. Kirby, B. Kirby, M. and Klein, J. Koehler, K. Koerner, L. W. Kohn, S. and Koller, P. P. Kordosky, M. Kosc, T. Kose, U. Kostelecky, V. Kothekar, K. Krennrich, F. Kreslo, I. Kudenko, Y. and Kudryavtsev, V. Kulagin, S. Kumar, J. Kumar, R. Kuruppu, C. Kus, V. Kutter, T. Lamhert, A. Lande, K. Lane, C. E. Lang, K. Langford, T. Lasorak, P. Last, D. and Lastoria, C. Laundrie, A. Lawrence, A. Lazanu, I. LaZur, R. Le, T. Learned, J. LeBrun, P. Miotto, G. Lehmann and Lehnert, R. de Oliveira, M. Leigui Leitner, M. Leyton, M. and Li, L. Li, S. Li, S. Li, T. Li, Y. Liao, H. and Lin, C. Lin, S. Lister, A. Littlejohn, B. R. Liu, J. and Lockwitz, S. Loew, T. Lokajicek, M. Lomidze, I. Long, K. and Loo, K. Lorca, D. Lord, T. LoSecco, J. Louis, W. C. and Luk, K. Luo, X. Lurkin, N. Lux, T. Luzio, V. P. and MacFarland, D. Machado, A. Machado, P. Macias, C. and Macier, J. Maddalena, A. Madigan, P. Magill, S. Mahn, K. and Maio, A. Maloney, J. A. Mandrioli, G. Maneira, J. C. and Manenti, L. Manly, S. Mann, A. Manolopoulos, K. Plata, M. Manrique Marchionni, A. Marciano, W. Marfatia, D. and Mariani, C. Maricic, J. Marinho, F. Marino, A. D. and Marshak, M. Marshall, C. Marshall, J. Marteau, J. and Martin-Alho, J. Martinez, N. Caicedo, D. A. Martinez and Martynenko, S. Mason, K. Masthaum, A. Masud, M. Matsuno, S. Matthews, J. Mauger, C. Mauri, N. Mavrokoridis, K. and Mazza, R. Mazzacane, A. Mazzucato, E. McCluskey, E. and McConkey, N. McFarland, K. S. McGrew, C. McNab, A. and Mefodiev, A. Mehta, P. Melas, P. Mellinato, V. Mena, O. and Mcnary, S. Mendez, H. Menegolli, A. Meng, G. and Messier, V. Metcalf, W. Mewes, M. Meyer, H. Miao, T. and Michna, G. Miedema, T. Migenda, J. Milincic, R. Miller, W. Mills, J. Milne, C. Mineev, O. Miranda, O. G. and Miryala, S. Mishra, C. Mishra, S. Mislivec, A. Mladenov, D. Mocioiu, I. Moffat, K. Moggi, N. Mohanta, R. and Mohayai, T. A. Mokhov, N. Molina, J. A. Bueno, L. Molina and Montanari, A. Montanari, C. Montanari, D. Montano Zetina, L. M. Moon, J. Mooney, M. Moor, A. Moreno, D. Morgan, B. Morris, C. Mossey, C. Motuk, E. Moura, C. A. and Mousseau, J. Mu, W. Mualem, L. Mueller, J. Muether, M. and Mufson, S. Muheim, F. Muir, A. Mulhearn, M. and Muramatsu, H. Murphy, S. Musser, J. Nachtman, J. Nagu, S. Nalbandyan, M. Nandakumar, R. Naples, D. Narita, S. and Navas-Nicolas, D. Nayak, N. Nebot-Guinot, M. Necib, L. and Negishi, K. Nelson, J. K. Nesbit, J. Nessi, M. and Newbold, D. Newcomer, M. Newhart, D. Nichol, R. Niner, E. Nishimura, K. Norman, A. Northrop, R. Novella, P. and Nowak, J. A. Oberling, M. Del Campo, A. Olivares Olivier, A. and Onel, Y. Onishchuk, Y. Ott, J. Pagani, L. Pakvasa, S. Palamara, O. Palestini, S. Paley, J. M. Pallavicini, M. Palomares, C. Pantic, E. Paolone, V. Papadimitriou, V. Papaleo, R. Papanestis, A. Paramesvaran, S. Parke, S. and Parsa, Z. Parvu, M. Pascoli, S. Pasqualini, L. and Pasternak, J. Pater, J. Patrick, C. Patrizii, L. and Patterson, R. B. Patton, S. Patzak, T. Paudel, A. and Paulos, B. Paulucci, L. Pavlovic, Z. Pawloski, G. Payne, D. Pec, V. Peeters, S. J. Penichot, Y. Pennacchio, E. and Penzo, A. Peres, O. L. Perry, J. Pershey, D. and Pessina, G. Petrillo, G. Petta, C. Petti, R. Piastra, F. and Pickering, L. Pietropaolo, F. Pillows, J. Plunkett, R. and Poling, R. Pons, X. Poonthottathil, N. Pordes, S. and Potekhin, M. Potenza, R. Potukuchi, B. V. Pozimski, J. and Pozzato, M. Prakash, S. Prakash, T. Prince, S. Prior, G. and Pugnere, D. Qi, K. Qian, X. Raaf, J. Raboanary, R. and Radeka, V. Rademacker, J. Radics, B. Rafique, A. and Raguzin, E. Rai, M. Rajarialisoa, M. Rakhno, I. and Rakotondramanana, H. Rakotondravollitra, L. Ramachers, Y. and Rameika, R. Delgado, Vi. Ramirez Ramson, B. Rappoldi, A. and Raselli, G. Ratoff, P. Ravat, S. Razafinime, H. Real, J. and Rebel, B. Redondo, D. Reggiani-Guzzo, M. Rehak, T. and Reichenbacher, J. Reitzner, S. D. Renshaw, A. Rescia, S. and Resnati, F. Reynolds, A. Riccobene, G. Rice, L. C. and Rielage, K. Rigaut, Y. Rivera, D. Rochester, L. Roda, M. and Rodrigues, P. Alonso, M. Rodriguez Rondon, J. Rodriguez and Roeth, A. Rogers, H. Rosauro-Alcaraz, S. Rossella, M. and Rout, J. Roy, S. Rubbia, A. Rubbia, C. Russell, B. and Russell, J. Ruterbories, D. Saakyan, R. Sacerdoti, S. and Safford, T. Sahu, N. Sala, P. Samios, N. Sanchez, M. and Sanders, D. A. Sankey, D. Santana, S. Santos-Maldonado, M. and Saoulidou, N. Sapienza, P. Sarasty, C. Sarcevic, I. and Savage, G. Savinov, V. Scaramelli, A. Scarff, A. and Scarpelli, A. Schaffer, T. Schellman, H. Schlabach, P. and Schmitz, D. Scholberg, K. Schukraft, A. Segreto, E. and Sensenig, J. Seong, I. Sergi, A. Sergiampietri, F. and Sgalaberna, D. Shaevitz, M. Shafaq, S. Shamma, M. and Sharma, H. R. Sharma, R. Shaw, T. Shepherd-Themistocleous, C. Shin, S. Shooltz, D. Shrock, R. Simard, L. Simos, N. Sinclair, J. Sinev, G. Singh, J. Singh, J. Singh, V. Sipos, R. Sippach, F. Sirri, G. Sitraka, A. and Siyeon, K. Smargianaki, D. Smith, A. Smith, A. Smith, E. and Smith, P. Smolik, J. Smy, M. Snopok, P. Nunes, M. Soares Sobel, H. Soderberg, M. Salinas, C. J. Solano and Soldner-Remhold, S. Solomey, N. Solovov, V. Sondheim, W. E. and Sorel, M. Soto-Oton, J. Sousa, A. Soustruznik, K. and Spagliardi, F. Spanu, M. Spitz, J. Spooner, N. J. and Spurgeon, K. Staley, R. Stancari, M. Stanco, L. Steiner, H. Stewart, J. Stillwell, B. Stock, J. Stocker, F. and Stokes, T. Strait, M. Strauss, T. Strianov, S. Stuart, A. Summers, D. Surdo, A. Susic, V. Suter, L. Sutera, C. Svoboda, R. Szczerbinska, B. Szelc, A. Talaga, R. and Tanaka, H. Oregui, B. Tapia Tapper, A. Tariq, S. Tatar, E. Tayloe, R. Teklu, A. Tenti, M. Terao, K. Ternes, C. A. Terranova, F. Testera, G. Thea, A. Thompson, J. L. and Thorn, C. Timm, S. Tonazzo, A. Torti, M. Tortola, M. and Tortorici, F. Totani, D. Toups, M. Touramanis, C. and Trevor, J. Trzaska, Wh. Tsai, Yt. Tsamalaidze, Z. Tsang, K. Tsverava, N. Tufanli, S. Tull, C. Tyley, E. and Tzanov, Vi. Uchida, Vi. A. Urheim, J. Usher, T. Vagins, Vi. Vahle, P. Valdiviesso, G. Valencia, E. Vallari, Z. and Valle, J. W. Vallecorsa, S. Van Berg, R. Van de Water, R. G. Vanegas Forero, D. Varanini, F. Vargas, D. Varner, G. Vasel, J. Vasseur, G. Vaziri, K. Ventura, S. and Verdugo, A. Vergani, S. Vermeulen, M. A. Verzocchi, M. and Vieira de Souza, H. Vignoli, C. Vilela, C. Viren, B. and Vrba, T. Wachala, T. Waldron, A. V. Wallbank, M. Wang, H. Wang, J. Wang, Y. Wang, Y. Warburton, K. Warner, D. Wascko, M. Waters, D. Watson, A. Weatherly, P. and Weber, A. Weber, M. Wei, H. Weinstein, A. Wenman, D. and Wetstein, M. While, M. R. White, A. Whitehead, L. H. and Whittington, D. Wilking, M. J. Wilkinson, C. Williams, Z. and Wilson, F. Wilson, R. J. Wolcott, J. Wongjirad, T. and Wood, K. Wood, L. Worcester, E. Worcester, M. Wret, C. and Wu, W. Wu, W. Xiao, Y. Yang, G. Yang, T. and Yershov, N. Yonehara, K. Young, T. Yu, B. Yu, J. and Zalesak, J. Zambelli, L. Zamorano, B. Zani, A. Zazueta, L. Zeller, G. Zennamo, J. Zeug, K. Zhang, C. Zhao, M. Zhivun, E. Zhu, G. Zimmerman, E. D. Zito, M. and Zuccilelli, S. Zuklin, J. Zuishi, V. Zwaska, R. DUNE Collaboration
Published: 2020

22. EFFECT OF DIETARY SUPPLEMENTATION WITH VITAMIN E AND ORGANIC SELENIUM ON TURKEY MEAT QUALITY DURING STORAGE

Author: F. Soglia, G. Baldi, F. Sirri, C. Torres, M. Petracci, French Branch of the World’s Poultry Science Association, and F. Soglia, G.Baldi, F.Sirri, C.Torres, M.Petracci
Subjects: storage, organic selenium, turkey, Vitamin E, meat quality
Published: 2020

23. Volume III DUNE far detector technical coordination

Author: Abi, B. Acciarri, R. Acero, M. A. Adamov, G. Adams, D. and Adinolfi, M. Ahmad, Z. Ahmed, J. Alion, T. Monsalve, S. Alonso Alt, C. Anderson, J. Andreopoulos, C. Andrews, M. Andrianala, F. Andringa, S. Ankowski, A. Antonova, M. and Antusch, S. Aranda-Fernandez, A. Ariga, A. Arnold, L. O. and Arroyave, M. A. Asaadi, J. Aurisano, A. Aushev, V and Autiero, D. Azfar, F. Back, H. Back, J. J. Backhouse, C. and Baesso, P. Bagby, L. Bajou, R. Balasubramanian, S. and Baldi, P. Bambah, B. Barao, F. Barenboim, G. Barker, G. and Barkhouse, W. Barnes, C. Barr, G. Barranco Monarca, J. and Barros, N. Barrow, J. L. Bashyal, A. Basque, V Bay, F. Bazo Alba, J. Beacom, J. F. Bechetoille, E. Behera, B. Bellantoni, L. Bellettini, G. Bellini, V Beltramello, O. Belver, D. Benekos, N. Neves, F. Bento Berger, J. and Berkman, S. Bernardini, P. Berner, R. M. Berns, H. and Bertolucci, S. Betancourt, M. Bezawada, Y. Bhattacharjee, M. and Bhuyan, B. Biagi, S. Bian, J. Biassoni, M. Biery, K. and Bilki, B. Bishai, M. Bitadze, A. Blake, A. Siffert, B. Blanco Blaszczyk, F. Blazey, G. Blucher, E. and Boissevain, J. Bolognesi, S. Bolton, T. Bonesini, M. and Bongrand, M. Bonini, F. Booth, A. Booth, C. Bordoni, S. and Borkum, A. Boschi, T. Bostan, N. Bour, P. Boyd, S. and Boyden, D. Bracinik, J. Braga, D. Brailsford, D. and Brandt, A. Bremer, J. Brew, C. Brianne, E. Brice, S. J. and Brizzolari, C. Bromberg, C. Brooijmans, G. Brooke, J. and Bross, A. Brunetti, G. Buchanan, N. Budd, H. Caiulo, D. Calafiura, P. Calcutt, J. Calin, M. Calvez, S. and Calvo, E. Camilleri, L. Caminata, A. Campanelli, M. and Caratelli, D. Carini, G. Carlus, B. Carniti, P. and Terrazas, I. Caro Carranza, H. Castillo, A. Castromonte, C. and Cattadori, C. Cavalier, F. Cavanna, F. Centro, S. and Cerati, G. Cervelli, A. Cervera Villanueva, A. Chalifour, M. and Chang, C. Chardonnet, E. Chatterjee, A. Chattopadhyay, S. Chaves, J. Chen, H. Chen, M. Chen, Y. Cherdack, D. Chi, C. Childress, S. Chiriacescu, A. Cho, K. and Choubey, S. Christensen, A. Christian, D. Christodoulou, G. and Church, E. Clarke, P. Coan, T. E. Cocco, A. G. and Coelho, J. Conley, E. Conrad, J. Convery, M. Corwin, L. and Cotte, P. Cremaldi, L. Cremonesi, L. Crespo-Anadon, I, J. Cristaldo, E. Cross, R. Cuesta, C. Cui, Y. and Cussans, D. Dabrowski, M. Da Motta, H. Peres, L. Da Silva and David, Q. Davies, G. S. Davini, S. Dawson, J. De, K. and De Almeida, R. M. Debbins, P. De Bonis, I Decowski, M. and De Gouvea, A. De Holanda, P. C. Astiz, I. L. De Icaza and Deisting, A. De Jong, P. Delbart, A. Delepine, D. and Delgado, M. Dell'Acqua, A. De Lurgio, P. De Mello Neto, J. R. DeMuth, D. M. Dennis, S. Densham, C. Deptuch, G. and De Roeck, A. De Romeri, V De Vries, J. Dharmapalan, R. and Dias, M. Diaz, F. Diaz, J. Di Domizio, S. Di Giulio, L. and Ding, P. Di Noto, L. Distefano, C. Diurba, R. Diwan, M. Djurcic, Z. Dokania, N. Dolinski, M. Domine, L. and Douglas, D. Drielsma, F. Duchesneau, D. Duffy, K. Dunne, P. Durkin, T. Duyang, H. Dvornikov, O. Dwyer, D. and Dyshkant, A. Eads, M. Edmunds, D. Eisch, J. Emery, S. and Ereditato, A. Escobar, C. Sanchez, L. Escudero Evans, J. J. Ewart, E. Ezeribe, A. C. Fahey, K. Falcone, A. and Farnese, C. Farzan, Y. Felix, J. Fernandez-Martinez, E. and Fernandez Menendez, P. Ferraro, F. Fields, L. Filkins, A. and Filthaut, F. Fitzpatrick, R. S. Flanagan, W. Fleming, B. and Flight, R. Fowler, J. Fox, W. Franc, J. Francis, K. and Franco, D. Freeman, J. Freestone, J. Fried, J. and Friedland, A. Fuess, S. Furic, I Furmanski, A. P. Gago, A. Gallagher, H. Gallego-Ros, A. Gallice, N. Galymov, V and Gamberini, E. Gamble, T. Gandhi, R. Gandrajula, R. and Gao, S. Garcia-Gamez, D. Garcia-Peris, M. A. Gardiner, S. and Gastler, D. Ge, G. Gelli, B. Gendotti, A. Gent, S. and Ghorbani-Moghaddam, Z. Gibin, D. Gil-Botella, I Girerd, C. Giri, A. Gnani, D. Gogota, O. Gold, M. and Gollapinni, S. Gollwitzer, K. Gomes, R. A. Gomez Bermeo, L. and Gomez Fajardo, L. S. Gonnella, F. Gonzalez-Cuevas, J. and Goodman, M. C. Goodwin, O. Goswami, S. Gotti, C. and Goudzovski, E. Grace, C. Graham, M. Gramellini, E. Gran, R. Granados, E. Grant, A. Grant, C. Gratieri, D. and Green, P. Green, S. Greenler, L. Greenwood, M. Greer, J. and Griffith, C. Groh, M. Grudzinski, J. Grzelak, K. Gu, W. Guarino, V Guenette, R. Guglielmi, A. Guo, B. and Guthikonda, K. Gutierrez, R. Guzowski, P. Guzzo, M. M. and Gwon, S. Habig, A. Hackenburg, A. Hadavand, H. Haenni, R. Hahn, A. Haigh, J. Haiston, J. Hamernik, T. and Hamilton, P. Han, J. Harder, K. Harris, D. A. Hartnell, J. Hasegawa, T. Hatcher, R. Hazen, E. Heavey, A. and Heeger, K. M. Hennessy, K. Henry, S. Hernandez Morquecho, M. and Herner, K. Hertel, L. Hesam, A. S. Hewes, J. and Pichardo, A. Higuera Hill, T. Hillier, S. J. Himmel, A. and Hoff, J. Hohl, C. Holin, A. Hoppe, E. Horton-Smith, G. A. Hostert, M. Hourlier, A. Howard, B. Howell, R. and Huang, J. Huang, J. Hugon, J. Iles, G. Iliescu, A. M. and Illingworth, R. Ioannisian, A. Itay, R. Izmaylov, A. and James, E. Jargowsky, B. Jediny, F. Jesus-Valls, C. Ji, X. Jiang, L. Jimenez, S. Jipa, A. Joglekar, A. and Johnson, C. Johnson, R. Jones, B. Jones, S. Jung, C. and Junk, T. Jwa, Y. Kabirnezhad, M. Kaboth, A. Kadenko, I and Kamiya, F. Karagiorgi, G. Karcher, A. Karolak, M. and Karyotakis, Y. Kasai, S. Kasetti, S. P. Kashur, L. and Kazaryan, N. Kearns, E. Keener, P. Kelly, K. J. Kemp, E. and Ketchum, W. Kettell, S. Khabibullin, M. Khotjantsev, A. and Khvedelidze, A. Kim, D. King, B. Kirby, B. Kirby, M. and Klein, J. Koehler, K. Koerner, L. W. Kohn, S. and Koller, P. P. Kordosky, M. Kosc, T. Kose, U. Kostelecky, V Kothekar, K. Krennrich, F. Kreslo, I Kudenko, Y. and Kudryavtsev, V Kulagin, S. Kumar, J. Kumar, R. Kuruppu, C. Kus, V Kutter, T. Lambert, A. Lande, K. Lane, C. E. Lang, K. Langford, T. Lasorak, P. Last, D. and Lastoria, C. Laundrie, A. Lawrence, A. Lazanu, I LaZur, R. Le, T. Learned, J. LeBrun, P. Miotto, G. Lehmann and Lehnert, R. de Oliveira, M. Leigui Leitner, M. Leyton, M. and Li, L. Li, S. Li, S. Li, T. Li, Y. Liao, H. and Lin, C. Lin, S. Lister, A. Littlejohn, B. R. Liu, J. and Lockwitz, S. Loew, T. Lokajicek, M. Lomidze, I Long, K. and Loo, K. Lorca, D. Lord, T. LoSecco, J. Louis, W. C. and Luk, K. Luo, X. Lurkin, N. Lux, T. Luzio, V. P. and MacFarland, D. Machado, A. Machado, P. Macias, C. and Macier, J. Maddalena, A. Madigan, P. Magill, S. Mahn, K. and Maio, A. Maloney, J. A. Mandrioli, G. Maneira, J. C. and Manenti, L. Manly, S. Mann, A. Manolopoulos, K. Plata, M. Manrique Marchionni, A. Marciano, W. Marfatia, D. and Mariani, C. Maricic, J. Marinho, F. Marino, A. D. and Marshak, M. Marshall, C. Marshall, J. Marteau, J. and Martin-Albo, J. Martinez, N. Caicedo, D. A. Martinez and Martynenko, S. Mason, K. Mastbaum, A. Masud, M. Matsuno, S. Matthews, J. Mauger, C. Mauri, N. Mavrokoridis, K. and Mazza, R. Mazzacane, A. Mazzucato, E. McCluskey, E. and McConkey, N. McFarland, K. S. McGrew, C. McNab, A. and Mefodiev, A. Mehta, P. Melas, P. Mellinato, M. Mena, O. and Menary, S. Mendez, H. Menegolli, A. Meng, G. and Messier, M. Metcalf, W. Mewes, M. Meyer, H. Miao, T. and Michna, G. Miedema, T. Migenda, J. Milincic, R. Miller, W. Mills, J. Milne, C. Mineev, O. Miranda, O. G. and Miryala, S. Mishra, C. Mishra, S. Mislivec, A. Mladenov, D. Mocioiu, I Moffat, K. Moggi, N. Mohanta, R. and Mohayai, T. A. Mokhov, N. Molina, J. A. Bueno, L. Molina and Montanari, A. Montanari, C. Montanari, D. Montano Zetina, L. M. Moon, J. Mooney, M. Moor, A. Moreno, D. Morgan, B. Morris, C. Mossey, C. Motuk, E. Moura, C. A. and Mousseau, J. Mu, W. Mualem, L. Mueller, J. Muether, M. and Mufson, S. Muheim, F. Muir, A. Mulhearn, M. and Muramatsu, H. Murphy, S. Musser, J. Nachtman, J. Nagu, S. Nalbandyan, M. Nandakumar, R. Naples, D. Narita, S. and Navas-Nicolas, D. Nayak, N. Nebot-Guinot, M. Necib, L. and Negishi, K. Nelson, J. K. Nesbit, J. Nessi, M. and Newbold, D. Newcomer, M. Newhart, D. Nichol, R. Niner, E. Nishimura, K. Norman, A. Northrop, R. Novella, P. and Nowak, J. A. Oberling, M. Del Campo, A. Olivares Olivier, A. and Onel, Y. Onishchuk, Y. Ott, J. Pagani, L. Pakvasa, S. Palamara, O. Palestini, S. Paley, J. M. Pallavicini, M. Palomares, C. Pantic, E. Paolone, V Papadimitriou, V and Papaleo, R. Papanestis, A. Paramesvaran, S. Parke, S. and Parsa, Z. Parvu, M. Pascoli, S. Pasqualini, L. and Pasternak, J. Pater, J. Patrick, C. Patrizii, L. and Patterson, R. B. Patton, S. Patzak, T. Paudel, A. and Paulos, B. Paulucci, L. Pavlovic, Z. Pawloski, G. Payne, D. Pec, V Peeters, S. J. Penichot, Y. Pennacchio, E. and Penzo, A. Peres, O. L. Perry, J. Pershey, D. Pessina, G. and Petrillo, G. Petta, C. Petti, R. Piastra, F. and Pickering, L. Pietropaolo, F. Pillow, J. Plunkett, R. and Poling, R. Pons, X. Poonthottathil, N. Pordes, S. and Potekhin, M. Potenza, R. Potukuchi, V, B. Pozimski, J. and Pozzato, M. Prakash, S. Prakash, T. Prince, S. Prior, G. and Pugnere, D. Qi, K. Qian, X. Raaf, J. Raboanary, R. and Radeka, V Rademacker, J. Radics, B. Rafique, A. and Raguzin, E. Rai, M. Rajaoalisoa, M. Rakhno, I and Rakotondramanana, H. Rakotondravohitra, L. Ramachers, Y. and Rameika, R. Delgado, M. Ramirez Ramson, B. Rappoldi, A. and Raselli, G. Ratoff, P. Ravat, S. Razafinime, H. Real, J. and Rebel, B. Redondo, D. Reggiani-Guzzo, M. Rehak, T. and Reichenbacher, J. Reitzner, S. D. Renshaw, A. Rescia, S. and Resnati, F. Reynolds, A. Riccobene, G. Rice, L. C. and Rielage, K. Rigaut, Y. Rivera, D. Rochester, L. Roda, M. and Rodrigues, P. Alonso, M. Rodriguez Rondon, J. Rodriguez and Roeth, A. Rogers, H. Rosauro-Alcaraz, S. Rossella, M. and Rout, J. Roy, S. Rubbia, A. Rubbia, C. Russell, B. and Russell, J. Ruterbories, D. Saakyan, R. Sacerdoti, S. and Safford, T. Sahu, N. Sala, P. Samios, N. Sanchez, M. and Sanders, D. A. Sankey, D. Santana, S. Santos-Maldonado, M. and Saoulidou, N. Sapienza, P. Sarasty, C. Sarcevic, I and Savage, G. Savinov, V Scaramelli, A. Scarff, A. and Scarpelli, A. Schaffer, T. Schellman, H. Schlabach, P. and Schmitz, D. Scholberg, K. Schukraft, A. Segreto, E. and Sensenig, J. Seong, I Sergi, A. Sergiampietri, F. and Sgalaberna, D. Shaevitz, M. Shafaq, S. Shamma, M. and Sharma, H. R. Sharma, R. Shaw, T. Shepherd-Themistocleous, C. Shin, S. Shooltz, D. Shrock, R. Simard, L. Simos, N. Sinclair, J. Sinev, G. Singh, J. Singh, J. Singh, V Sipos, R. Sippach, F. Sirri, G. Sitraka, A. and Siyeon, K. Smargianaki, D. Smith, A. Smith, A. Smith, E. and Smith, P. Smolik, J. Smy, M. Snopok, P. Nunes, M. Soares Sobel, H. Soderberg, M. Solano Salinas, C. J. and Soldner-Rembold, S. Solomey, N. Solovov, V Sondheim, W. E. and Sorel, M. Soto-Oton, J. Sousa, A. Soustruznik, K. and Spagliardi, F. Spanu, M. Spitz, J. Spooner, N. J. and Spurgeon, K. Staley, R. Stancari, M. Stanco, L. Steiner, H. Stewart, J. Stillwell, B. Stock, J. Stocker, F. and Stokes, T. Strait, M. Strauss, T. Striganov, S. Stuart, A. Summers, D. Surdo, A. Susic, V Suter, L. Sutera, C. Svoboda, R. Szczerbinska, B. Szelc, A. Talaga, R. and Tanaka, H. Oregui, B. Tapia Tapper, A. Tariq, S. Tatar, E. Tayloe, R. Teklu, A. Tenti, M. Terao, K. Ternes, C. A. Terranova, F. Testera, G. Thea, A. Thompson, J. L. and Thorn, C. Timm, S. Tonazzo, A. Torti, M. Tortola, M. and Tortorici, F. Totani, D. Toups, M. Touramanis, C. and Trevor, J. Trzaska, W. H. Tsai, Y. T. Tsamalaidze, Z. and Tsang, K. Tsverava, N. Tufanli, S. Tull, C. Tyley, E. and Tzanov, M. Uchida, M. A. Urheim, J. Usher, T. and Vagins, M. Vahle, P. Valdiviesso, G. Valencia, E. and Vallari, Z. Valle, J. W. Vallecorsa, S. Van Berg, R. Van de Water, R. G. Forero, D. Vanegas Varanini, F. Vargas, D. and Varner, G. Vasel, J. Vasseur, G. Vaziri, K. Ventura, S. Verdugo, A. Vergani, S. Vermeulen, M. A. Verzocchi, M. de Souza, H. Vieira Vignoli, C. Vilela, C. Viren, B. and Vrba, T. Wachala, T. Waldron, V, A. Wallbank, M. and Wang, H. Wang, J. Wang, Y. Wang, Y. Warburton, K. and Warner, D. Wascko, M. Waters, D. Watson, A. Weatherly, P. Weber, A. Weber, M. Wei, H. Weinstein, A. Wenman, D. Wetstein, M. While, M. R. White, A. Whitehead, L. H. and Whittington, D. Wilking, M. J. Wilkinson, C. Williams, Z. Wilson, F. Wilson, R. J. Wolcott, J. Wongjirad, T. and Wood, K. Wood, L. Worcester, E. Worcester, M. Wret, C. Wu, W. Wu, W. Xiao, Y. Yang, G. Yang, T. and Yershov, N. Yonehara, K. Young, T. Yu, B. Yu, J. and Zalesak, J. Zambelli, L. Zamorano, B. Zani, A. Zazueta, L. Zeller, G. Zennamo, J. Zeug, K. Zhang, C. Zhao, M. Zhivun, E. Zhu, G. Zimmerman, E. D. Zito, M. and Zucchelli, S. Zuklin, J. Zutshi, V Zwaska, R. DUNE Collaboration
Published: 2020

24. HOW CHRONIC HEAT STRESS AFFECTS IN VIVO AND POST-MORTEM OXIDATIVE PARAMETERS OF BROILER CHICKENS?

Author: S. Mattioli, A. Mancinelli, F. Soglia, M. Zampiga, C. Castellini, A. Dal bosco, M. Petracci, F. Sirri, French Branch of the World’s Poultry Science Association, and S. Mattioli, A.Mancinelli, F.Soglia, M.Zampiga, C.Castellini, A.Dal bosco, M.Petracci, F.Sirri
Subjects: oxidative parameter, chicken, cronic heat stre, meat quality
Published: 2020

25. Chronic tinnitus and BDNF/GDNF CpG promoter methylations: a case–control study

Author: Seda Orenay-Boyacioglu, Aysun Coskunoglu, Olcay Boyacioglu, Metin Caliskan, F. Sirri Cam, Gökay Bozkurt, Department of Medical Genetics, Faculty of Medicine, Aydin Adnan Menderes University, Aydin, Turkey, Faculty of Engineering, Aydin Adnan Menderes University, Aydin, Turkey, Department of Medical Genetics, State Hospital of Sivas, Sivas, Turkey, and Department of Medical Genetics, Faculty of Medicine, Manisa Celal Bayar University, Manisa, Turkey
Subjects: Adult, Male, 0301 basic medicine, Systemic disease, medicine.medical_specialty, Adolescent, Genotype, Tinnitus, 03 medical and health sciences, 0302 clinical medicine, Neurotrophic factors, Internal medicine, Genetics, medicine, Glial cell line-derived neurotrophic factor, otorhinolaryngologic diseases, Humans, Glial Cell Line-Derived Neurotrophic Factor, Promoter Regions, Genetic, Molecular Biology, Polymorphism, Genetic, biology, business.industry, urogenital system, Brain-Derived Neurotrophic Factor, Case-control study, General Medicine, Methylation, DNA Methylation, Middle Aged, medicine.disease, Pathophysiology, 030104 developmental biology, Endocrinology, CpG site, nervous system, Case-Control Studies, 030220 oncology & carcinogenesis, biology.protein, CpG Islands, Female, medicine.symptom, business
Abstract: Brain-derived neurotrophic factor (BDNF) and Glial-derived neurotrophic factor (GDNF) are neurotrophic factors that play key roles in the auditory pathway. While the relationship between serum levels and polymorphisms of BDNF/GDNF and chronic tinnitus is emphasized in the literature, there is no study showing the link between the promoter methylations of these genes and tinnitus. For this purpose, the relationship between chronic tinnitus and peripheral blood derived BDNF/GDNF promoter methylations was investigated to identify their role in the pathophysiology of tinnitus. In this case–control study, we examined the possible effects of BDNF/GDNF methylations in the blood samples of patients with tinnitus complaints for more than 3 months. Sixty tinnitus subjects between the ages of 18–55 and 50 healthy control subjects in the same age group who were free of any otorhinolaryngology and systemic disease were selected for examination. Methylation of total 12 CpG sites in BDNF and GDNF promoter regions were determined by the bisulfite-pyrosequencing method. Statistically significant differences were detected between BDNF CpG6 and GDNF CpG3-5-6 methylation ratios in the comparison of control group and the chronic tinnitus patients (P = 0.002, 0.0005, 0.00003, and 0.0029, respectively). To our knowledge, this is the first study in the literature investigating the relationship between chronic tinnitus and peripheral blood derived BDNF/GDNF promoter methylations. It is believed that the current results might be supported by investigating the relationships between BDNF/GDNF methylations and genotypes in future research using higher sample sizes. © 2019, Springer Nature B.V.
Published: 2019

26. Allometric Models to Estimate Leaf Area for Tropical African Broadleaved Forests

Author: Vincent Medjibe, Moses Libalah, Bonaventure Sonké, Nicolas Barbier, N. F. Sirri, Pierre Ploton, S. Momo Takoudjou, Gislain Ii Mofack, Narcisse Guy Kamdem, Higher Teachers' Training College, University of Yaoundé [Cameroun], Laboratoire de Botanique systématique et d'Ecologie [ENS Yaoudé], Université de Yaoundé I-École normale supérieure [ENS] - Yaoundé 1, Botanique et Modélisation de l'Architecture des Plantes et des Végétations (UMR AMAP), Centre National de la Recherche Scientifique (CNRS)-Université de Montpellier (UM)-Institut National de la Recherche Agronomique (INRA)-Centre de Coopération Internationale en Recherche Agronomique pour le Développement (Cirad)-Institut de Recherche pour le Développement (IRD [France-Sud]), University of Yaounde I, Université de Yaoundé I [Yaoundé]-École normale supérieure [ENS] - Yaoundé 1, and Centre de Coopération Internationale en Recherche Agronomique pour le Développement (Cirad)-Institut National de la Recherche Agronomique (INRA)-Université de Montpellier (UM)-Centre National de la Recherche Scientifique (CNRS)-Institut de Recherche pour le Développement (IRD [France-Sud])
Subjects: 0106 biological sciences, Allometric models, Ecology, Allometric model, 04 agricultural and veterinary sciences, 15. Life on land, Tropical African broadleaved forests, [SDV.BV.BOT]Life Sciences [q-bio]/Vegetal Biology/Botanics, [SDV.BID.SPT]Life Sciences [q-bio]/Biodiversity/Systematics, Phylogenetics and taxonomy, 010603 evolutionary biology, 01 natural sciences, Leaf area, Geophysics, [SDV.EE.ECO]Life Sciences [q-bio]/Ecology, environment/Ecosystems, 040103 agronomy & agriculture, 0401 agriculture, forestry, and fisheries, General Earth and Planetary Sciences, Environmental science, [SDE.BE]Environmental Sciences/Biodiversity and Ecology, ComputingMilieux_MISCELLANEOUS
Abstract: Direct and semidirect estimations of leaf area (LA) and leaf area index (LAI) are scarce in dense tropical forests despite their importance in calibrating remote sensing products, forest dynamics, and biogeochemical models. We destructively sampled 61 trees belonging to 13 most abundant species in a semideciduous forest in southeastern Cameroon. For each tree, all leaves were weighed, and for a subsample of branches, leaves were counted and the LA measured. Allometric models were calibrated to allow semidirect estimation of LAI at tree and stand levels based on forest inventory data (R-2 = 0.7, bias = 21.2%, error = 39.5%) and on predictors that could be extracted from very high resolution remote sensing data (R-2 = 0.63, bias = 35.1%, error = 58.73). Using twenty-one 1-ha forest plots, stand level estimations of LAI ranged from 4.42-13.99. These values are higher than previous estimates generally obtained using indirect methods. These results may have important consequences on ecosystem exchanges and the role of tropical forest in global cycles. Plain Language Summary Leaf area (LA) and leaf area index (LAI) are useful parameters characterizing the plant-atmosphere interface where matter and energy are exchanged. However, direct or semidirect estimations are not common in dense tropical forests. In this study, we used a destructive data set of trees of varied species and sizes from the semideciduous forest of southeastern Cameroon to predict total tree LA. Based on this data, we developed operational allometric models to allow for semidirect estimation of LA and LAI at tree and stand levels. These models would be of considerable use for climate-vegetation modeling and remote sensing communities.
Published: 2019
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27. Proceedings of the 20th ASPA Congress, Bologna, June 11-13, 2013

Author: Guest Editors: A. Piva, P. Bosi, A. Bonaldo, F. Sirri, A. Badiani, G. Biagi, R. Davoli, G. Martelli, A. Meluzzi, P. Trevisi
Subjects: Zootechical Methods, lcsh:Animal culture, Zoology, lcsh:SF1-1100
Abstract: The 20th Congress of the Animal Science and Production Association (ASPA), is held in the prestigious home of the oldest university of the world: Bologna. The anniversary also celebrates the 40th year of the founding of the Society and is an occasion to remember the past and plan for the future of Animal Sciences in Italy. In this Congress, 137 oral presentations and 155 posters are presented, for a total of 292 scientific contributions. The Congress sessions with their relative number of papers are listed as follows: Animal breeding and genetics 109, Nutrition and feeding 45, Dairy production 42, Animal welfare, health and behaviour 39, Meat quality 35, Poultry and rabbit production 11 and Aquaculture 11. I would like to point out that 3 main lectures will be presented in the Animal breeding and genetics, Nutrition and feeding and Animal welfare, health and behaviour sessions, respectively. I would thank, first of all, Prof. Vincenzo Russo, Honorary President of the Congress and Emeritus of ASPA, for the contribution of the University of Bologna in organizing this event. I would also thank all the members of the Scientific Committee, Prof. Andrea Piva (Chairperson), Prof. Anna Badiani, Prof. Giacomo Biagi, Dr. Alessio Bonaldo, Prof. Paolo Bosi, Prof. Roberta Davoli, Prof. Luca Fontanesi, Prof. Giovanna Martelli, Prof. Adele Meluzzi, Dr. Attilio Mordenti, Prof. Luca Sardi, Dr. Federico Sirri, Dr. Paolo Trevisi, and of the Organizing Committee, Prof. Claudio Cavani (Chairperson), Prof. Stefania Dall’Olio, Prof. Andrea Formigoni, Prof. Pier Giorgio Monetti, Prof. Leonardo Nanni Costa, Dr. Massimiliano Petracci, Prof. Giuliano Zaghini, Dr. Paolo Zambonelli. A special thanks to the secretary of the Congress, MV Congressi. Indeed, I would like to congratulate and thank all those who have organized the meeting and have collaborated in reviewing the summaries for what they have done for ASPA and the Italian Animal Science. A special thanks to Prof. Rosanna Scipioni, Editor-in-Chief of the Italian Journal of Animal Science, for having contributed to the edition of these proceedings. Finally, I acknowledge the University of Bologna and the institutional agencies, in particular the Ministry of Agricultural, Food and Forestry Policies (MIPAAF), the Regione Emilia Romagna and the Comune di Bologna, and the main sponsors, namely Associazione Italiana Foraggi Essiccati, DSM, Vetagro and Fatro.
Published: 2013

28. Association of Angiotensin-Converting Enzyme I/D and eNOS G894T Gene Polymorphisms with Erectile Dysfunction

Author: F. Sirri Cam, Ahmet Var, Afig Berdeli, Bilali H. Gümüş, and Ege Üniversitesi
Subjects: medicine.medical_specialty, biology, business.industry, General Medicine, biology.organism_classification, medicine.disease, polymorphism, angiotensin-converting enzyme, Endocrinology, Erectile dysfunction, Angiotensin converting enzyme i, Enos, Internal medicine, medicine, business, Gene
Abstract: WOS: 000291330700023, Objective: Recent studies suggest that angiotensin II and nitric oxide (NO) may modulate penile smooth muscle tone and contractility. Because genotypes of the angiotensin converting enzyme (ACE) and endothelial nitric oxide synthase (eNOS) polymorphisms have been associated with disorders in the vascular system, in this study, we investigated an association between the ACE I/D and eNOS G894T gene polymorphisms and erectile dysfunction (ED). Material and Methods: A total of 44 patients and 45 control subjects were included in the study. Diagnosis of erectile dysfunction (< 26) was provided by International Index of Erectile Function (IIEF). The ACE I/D ye eNOS G894T gene polymorphisms were genotyped using PCR and RFLP. Results: No significant case-control difference was observed for the ACE I/D and eNOS G894T gene polymorphisms either by genotype or allele frequencies [(ACE I/D-X-2=0.930 p=0.628) and eNOS 894 G/T-X-2=2.114 p=0.348)]. In addition, there was no significant difference between ACE I/D (X-2=3.174 p=0.787) and eNOS G894T (X-2=4.320 p=0.633) and IIEF scores among the patient group. Conclusion: In this study, no association was found between ACE LID and eNOS G894T gene polymorphisms and erectile dysfunction in the Turkish population studied.
Published: 2011
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29. Analysis of the dermatoglyphics in Turkish patients with Klinefelter's syndrome

Author: Solmaz Erdem, F. Sirri Cam, Mujgan Ozdemir Erdogan, Tevhide Fistik, Mustafa Solak, Yusuf Tunca, Davut Gül, and Handan Yildiz
Subjects: medicine.medical_specialty, S syndrome, Turkish, Dermatoglyphic patterns, Epidermal Ridge, General Medicine, Ridge count, Biology, Dermatology, language.human_language, Genetics, language, medicine, Dermatoglyphics
Abstract: Cam, F. S., Gul, D., Tunca, Y., Fi´sti´k, T., Erdogan, M. O., Yi´ldi´z, H., Erdem, S. and Solak, M. 2008. Analysis ofthe dermatoglyphics in Turkish patients with Klinefelter’s syndrome. *Hereditas 145: 163 166. Lund, Sweden. eISSN1601-5223. Received December 21, 2007. Accepted May 5, 2008The word ‘‘dermatoglyphics’’ indicates study of epidermal ridge configuration on palms, soles and fingertips. Thisinvestigation was aimed to analyze dermatoglyphic patterns in Klinefelter’s syndrome (KS) patients. The study cohortconsisted of 57 cases and 25 controls. The prints were taken by using the ink method. Fingertip patterns, triradial counts, a-t-d angle and a-b ridge count were studied. There were significant differences in radial loops and whorls (pB0.05), and therewere very highly significant differences in arches (pB0.001) in KS patients as compared to controls. Dermatoglyphicpatterns at the hypothenar area (pB0.05), and areas between at the I. interdigital and thenar sites (pB0.001) have beenfound to be significantly different in KS patients compared to controls. Total ridge counts (TRC), a-b, a-t-d angels were notdifferent in the two groups (p 0.05). A definite correlation between the dermatoglyphic patterns and the KS has beenshown.Davut Gul, Department of Medical Genetics, Gulhane Military Medical Faculty, TR-06018 Etlik, Ankara, Turkey. E-mail:guld@gata.edu.tr
Published: 2008
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30. ACE Genotype May Have an Effect on Single versus Multiple Set Preferences in Strength Training

Author: Muzaffer Colakoglu, Fırat Çetinöz, Bulent Kayitken, F. Sirri Cam, Metin Vehbi Sayın, Sule Colakoglu, and Mustafa Türkmen
Subjects: Adult, Male, medicine.medical_specialty, Genotype, Weight Lifting, Physiology, Strength training, Physical Exertion, Ace genotype, Squat, Peptidyl-Dipeptidase A, Bench press, Physiology (medical), Internal medicine, medicine, Humans, Orthopedics and Sports Medicine, Exercise physiology, Set (psychology), Exercise, Genetics, Physical Education and Training, biology, business.industry, Public Health, Environmental and Occupational Health, Angiotensin-converting enzyme, General Medicine, Physical Fitness, biology.protein, business
Abstract: A polymorphic variant of the human angiotensin converting enzyme (ACE) gene was identified. The 'D' (rather than 'I') variant was associated with improvements in strength related to physical training. We set out to determine whether the response to different patterns of strength training might also differ. Ninty-nine Caucasian male non-elite athletes were randomly allocated into one of three groups: 31 non-training/control (CG: 31), single-set (SSG: 35) and multiple-set (MSG: 33). SSG and MSG trained three times a week for 6 weeks. Both training groups were underwent a strength-training program with two mesocycles (12-15 repetition maximum (RM) and 8-12 RM mesocycles). One RM loads in half squat and bench press were assessed before training and after the first and second mesocycles. ACE polymorphisms analysed by polymerase chain reaction (PCR) methods. Subjects with ACE II genotype in the MST group had improved strength development in 12-15 RM, while SST and MST groups had similar gains in 8-12 RM. Subjects with ACE DD genotype in both the SSG and the MSG had similar benefits from both 12-15 RM and 8-12 RM. Strength gains for subjects with ACE ID genotype in the SSG were similar to MSG gains in response to 8-12 RM loads but not with 12-15 RM loads. Additionally, subjects with DD genotype had superior strength gains in both strength training groups. Tailoring strength training programmes (single-set vs. multiple set) according to the athlete's ACE genotype may be advantageous.
Published: 2005
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31. Renin-angiotensin system gene polymorphisms and premature coronary heart disease

Author: F. Sirri Cam, Afig Berdeli, Ertugrul Ercan, Istemihan Tengiz, Mustafa Akin, Abdi Sagcan, Cevad Sekuri, Erhan Eser, and Ege Üniversitesi
Subjects: Adult, Male, Medicine (General), medicine.medical_specialty, Time Factors, Genotype, Turkey, Angiotensinogen, Coronary Disease, Peptidyl-Dipeptidase A, 030204 cardiovascular system & hematology, Polymerase Chain Reaction, Receptor, Angiotensin, Type 1, 03 medical and health sciences, R5-920, 0302 clinical medicine, Endocrinology, Gene Frequency, Polymorphism (computer science), Internal medicine, Odds Ratio, Internal Medicine, Humans, Medicine, Genetic Predisposition to Disease, 030212 general & internal medicine, Allele frequency, Cardiovascular risk factors, Polymorphism, Genetic, Angiotensin II receptor type 1, business.industry, ComputerSystemsOrganization_COMPUTER-COMMUNICATIONNETWORKS, Gene polymorphism, Case-control study, Middle Aged, Angiotensin II, ComputingMilieux_MANAGEMENTOFCOMPUTINGANDINFORMATIONSYSTEMS, ComputingMethodologies_PATTERNRECOGNITION, Premature coronary heart disease, Case-Control Studies, Female, InformationSystems_MISCELLANEOUS, Renin-angiotensin system, Restriction fragment length polymorphism, business, Polymorphism, Restriction Fragment Length
Abstract: PubMed ID: 16088850, Introduction: Experimental and clinical studies demonstrated that the renin-angiotensin system (RAS) affects the pathogenesis of atherosclerosis and prognosis of coronary heart disease (CHD). The aim of this study was to investigate the genotype distribution and the allele frequencies of three RAS genes polymorphisms and their effects on premature CHD in a Turkish population. Materials and methods: One-hundred and fifteen Turkish patients with premature CHD and 128 controls were included into the study. Angiotensin-converting enzyme (ACE), angiotensin II type 1 (AT1) receptor and angiotensinogen (AGT) gene polymorphisms were analysed by polymerase chain reaction (PCR) and restriction fragment length polymorphism (RFLP). Results: The patients group showed an increased frequency of the ACE D allele compared with controls (65% vs. 35%, p-0.0001). There was a significant association between the DD genotype and premature CHD (ACE DD vs. ID and II; odds ratio [OR]=2.82 [CI 95% 1.33-2.91, P=0.002]). Also, we observed increased premature CHD risk associated with higher frequencies of the AGT MM genotype in patients when compared with controls (AGT MM vs. TT and MT, OR=1.92 [CI 95% 1.11-3-33, p=0.018]). We found a significant association between AT1-receptor AA genotype and decreased risk of premature CHD (AT1R AA vs. AC and CC, OR=0.57[CI 95% 0.34-0.95, p=0.03]). Conclusions: We demonstrated that increased premature CHD risk is associated with higher frequencies of the ACE DD and AGT MM genotypes. These findings indicate a synergistic contribution of ACE DD and AGT MM polymorphisms to the development of premature CHD. Also, our results suggest that family history, smoking, diabetes, hypertension, obesity and ACE DD genotype were independent risk factors for premature CHD.
Published: 2005
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32. Relation of RAS Gene Polymorphisms with Impaired Glucose Tolerance in Patient with End Stage Renal Failure

Author: F. Sirri Cam, Seyhun Kürşat, Hulya Colak Bahadir, Aysun Toraman Avcu, and Derya Oz
Subjects: medicine.medical_specialty, Angiotensin II receptor type 1, Biology, medicine.disease, Angiotensin II, Impaired glucose tolerance, Insulin receptor, Blood pressure, Endocrinology, Insulin resistance, Internal medicine, Diabetes mellitus, Renin–angiotensin system, Genetics, medicine, biology.protein, Genetics (clinical)
Abstract: Renin angiotensin system (RAS) is a system that is the role of renal hemodynamics, blood pressure and fluid electrolyte balance regulation with progression to end-stage renal disease (ESRD). Angiotensin II (AII),the main mediator of this system, is thought to cause a functional impairment in insulin receptor and post receptor signaling pathways. Our aim; was to show the relation of angiotensin-converting enzyme (ACE), angiotensin II receptor type 1 (AT1 R) and angiotensinogen gene (AGT) polymorphisms, that cause genetic susceptibility to over activation of RAS, to glucose intolerance in patient with end stage renal failure. The study included one hundredth patients. Based on fasting plasma glucose values and oral glucose tolerance test second hours glucose values, patients were grouped as normal patients and patients with glucose intolerance (impaired fasting glycemia (IFG), impaired glucose tolerance (IGT) and diabetes mellitus (DM)). Insulin resistance was calculated by HOMA-IR method and insulin sensitivity index was calculated by ISI-S method. ACE, AT1 R, AGT polymorphisms were detected by polymerase chain reaction (PCR) method. There was no statistically significant difference between distribution of ACE, AT1R and AGT genotypes and glucose intolerance groups, and insulin resistance. There was a statistically significant difference between MM and TT genotypes and average insulin resistance values (p < 0,04). We have obtained results suggesting a relation between ACE gene D allele and glucose intolerance in patients with end stage renal failure (p=0.06). Moreover, a relation has been shown between AGT gene T allele and insulin resistance (p=0.04).
Published: 2013
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33. Contributor contact details

Author: F. Van Immerseel, Y. Nys, M. Bain, M.T. Musgrove, M. Chemaly, G. Salvat, R. Raspoet, I. Gantois, R. Devloo, F. Pasmans, F. Haesebrouck, R. Ducatelle, C. Jondreville, C. Feidt, A. Fournier, A. Travel, B. Roudaut, R. Davies, J.J. Carrique-Mas, J. Dewulf, S. Van Hoorebeke, K. Richard, null Gast, W. Messens, J. Gittins, S. Leleu, N. Sparks, A. Berardinelli, C. Cevoli, A. Fabbri, L. Ragni, G. Manfreda, F. Pasquali, M.E. Guerzoni, L. Vannini, I. Seuss-Baum, F. Nau, C. Guérin-Dubiard, B.A. Griffin, Y. Mine, M. Yang, F. Sirri, A. Meluzzi, A. Schiavone, A.C. Barroeta, M. Anton, J. Kovacs-Nolan, and V. Guyonnet
Published: 2011
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34. Alpha Adducin Gene Polymorphism İn The Turkish Hypertensives

Author: Alioglu, Emin, Turk, Ugur, Tuzun, Nurullah, Cam, F Sirri, Berdeli, Afig, and Ege Üniversitesi
Abstract: …
Published: 2007

35. Association of platelet-activating factor acetylhydrolase gene polymorphism with premature coronary artery disease in Turkish patients

Author: Cevad, Sekuri, F Sirri, Cam, Istemihan, Tengiz, Ertuğrul, Ercan, Ozgür, Bayturan, and Afig, Berdeli
Subjects: Male, Polymorphism, Genetic, Turkey, Coronary Artery Disease, Middle Aged, Polymerase Chain Reaction, White People, Case-Control Studies, 1-Alkyl-2-acetylglycerophosphocholine Esterase, Mutation, Prevalence, Humans, Female, Polymorphism, Restriction Fragment Length
Abstract: Platelet-activating factor (PAF) is a phospholipid with multiple actions that is involved in inflammatory diseases as well as in atherogenesis. It is inactivated by a plasma enzyme, PAF-acetylhydrolase (PAF-AH). Deficiency of this enzyme in plasma is caused by a missense mutation in the gene (G994T). The aim of this study was to investigate association of this mutation with premature coronary artery disease (CAD).One hundred and fifteen unrelated Turkish patients with a diagnosis of premature CAD and 128 unrelated healthy subjects were enrolled in this study. Genotyping was performed by polymerase chain reaction (PCR) and restriction fragment length polymorphism (RFLP).The prevalence of the G994T mutation in the patients was 2.60 % (heterozygote), and 0 % in the controls. There was no significant difference in allele frequency and genotype distribution among the study groups.The G9943T mutation in the plasma PAF acetylhydrolase gene is not associated with premature CAD in Turkish subjects.
Published: 2006

36. Association between the ACE I/D gene polymorphism and physical performance in a homogeneous non-elite cohort

Author: Çagatay Sahan, F. Sirri Cam, Afig Berdeli, Sule Colakoglu, Cevad Sekuri, and Muzaffer Colakoglu
Subjects: Oncology, Male, medicine.medical_specialty, Anaerobic Threshold, Genotype, Weight Lifting, Physiology, Peptidyl-Dipeptidase A, Genetic determinism, Running, Cohort Studies, Oxygen Consumption, Gene Frequency, Polymorphism (computer science), Internal medicine, medicine, Humans, Orthopedics and Sports Medicine, Allele, Allele frequency, Alleles, Genetics, Polymorphism, Genetic, business.industry, Cohort, DNA Transposable Elements, Physical Endurance, Gene polymorphism, business, Gene Deletion, Cohort study
Abstract: Background: I/D polymorphism of the ACE gene may be associated with better endurance performance and a stronger response to exercise training. The aim of this study was to investigate the association between ACE gene polymorphism and athletic performance in a homogeneous cohort. Methods: Eighty-eight male non-elite Caucasian Turkish athletes with similar training backgrounds for at least for 6 months were studied for ACE gene polymorphisms by PCR analysis. Performance on the 60-meter sprint and middle-distance running tests were evaluated. Results: The distributions of the ACE I/D genotypes were 20.5%, 40.9%, and 38.6% for II, ID, and DD polymorphisms in the whole group (N = 88), respectively. The ACE DD genotype frequency was significantly higher in the superior group (56.7%) than in the poor (37.9%) and mediocre (20.7%) group in middle-distance running performance (χ2 = 11.778; p = 0.019). Conclusion: The ACE DD genotype may be related to better short-duration aerobic endurance performance. Larger homogeneous cohorts may help clarify the association between ACE I/D polymorphism and physical performance. Key words: genetics, endurance performance, DNA, sprint
Published: 2005

37. Hypertrophic cardiomyopathy: pathological features and molecular pathogenesis

Author: F Sirri, Cam and Merih, Güray
Subjects: Phenotype, Genotype, Humans, Cardiomyopathy, Hypertrophic
Abstract: Hypertrophic cardiomyopathy (HCM) is a heterogeneous genetic cardiac disorder with various genotypic and phenotypic manifestations, and is often a diagnostic challenge. Although more than forty years have passed since the first description of HCM, a variety of mutations in genes encoding sarcomeric proteins, that cause the disease have been defined by laboratory and clinical studies over the past few years. The fact that HCM is the most common cause of sudden death in young competitive athletes and that, it is actually an important cause of morbidity and mortality in people of all ages, has made the researchers to concentrate more on the molecular basis and treatment strategies of the disease. This study aims to summarize both pathological features and rapidly evolving molecular genetics of HCM, and so to understand this not infrequently seen, complex disorder better.
Published: 2004

38. Hypertrophic cardiomyopathy: pathological features and molecular pathogenesis

Author: Güray, MERİH and Cam, F Sirri
Published: 2004

39. Association between the eNOS (Glu298Asp) and the RAS genes polymorphisms and premature coronary artery disease in a Turkish population

Author: Cevad Sekuri, Abdi Sagcan, Istemihan Tengiz, Ertugrul Ercan, Erhan Eser, Afig Berdeli, F. Sirri Cam, and Mustafa Akin
Subjects: Adult, Male, medicine.medical_specialty, Turkish population, Genotype, Nitric Oxide Synthase Type III, Turkey, Arteriosclerosis, Clinical Biochemistry, Coronary Artery Disease, Biochemistry, Pathogenesis, Coronary artery disease, Renin-Angiotensin System, Gene Frequency, Enos, Risk Factors, Internal medicine, Renin–angiotensin system, medicine, Odds Ratio, Humans, Age of Onset, Polymorphism, Genetic, biology, Reverse Transcriptase Polymerase Chain Reaction, Biochemistry (medical), General Medicine, DNA, biology.organism_classification, medicine.disease, Endocrinology, Amino Acid Substitution, Female, Gene polymorphism, Restriction fragment length polymorphism, Nitric Oxide Synthase, Polymorphism, Restriction Fragment Length
Abstract: Background The renin–angiotensin system (RAS) and endothelial nitric oxide (NO) affect the pathogenesis of atherosclerosis and prognosis of coronary artery disease (CAD). Previous epidemiologic data suggested that genetic factors are more likely to affect young rather than old people. Our objective was to investigate the association between the polymorphisms of eNOS (Glu298Asp) and the RAS genes and premature CAD in a Turkish population. Methods A total of 115 Turkish patients with premature CAD and 83 controls were included in the study. ACE I/D, AT1R A/C, AGT T/M and eNOS Glu298Asp gene polymorphisms were analysed by polymerase chain reaction (PCR) and restriction fragment length polymorphism (RFLP). Results It was found that increased premature CAD risk is associated with higher frequencies of the ACE DD [OR: 2.600 (CI 95% 1.395–4.847, p =0.002)], AGT MM [OR=2.407 (CI 95% 1.267–4.573, p =0.007)] and eNOS 894TT [OR=17.000 (CI 95% 3.952–73.125, p ACE DD+ eNOS 894TT ( p =0.002), AGT MM+ eNOS 894TT ( p =0.001), AT1R AA+ eNOS 894TT and AT1R non-AA+ eNOS 894TT ( p =0.002) genotypes were significantly associated with the risk of premature CAD. Conclusions This study indicates a synergistic contribution of RAS genes ( ACE I/D, AGT T/M, AT1R T/C) and eNOS Glu298Asp polymorphisms to the development of the premature CAD.
Published: 2004

40. Relation of RAS Gene Polymorphisms with Impaired Glucose Tolerance in Patient with End Stage Renal Failure

Author: Oz, Derya, primary, Avcu, Aysun Toraman, additional, Kursat, Seyhun, additional, Bahadir, Hulya Colak, additional, and Cam, F. Sirri, additional
Published: 2013
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41. Relation of RAS Gene Polymorphisms with Impaired Glucose Tolerance in Patient with End Stage Renal Failure

Author: Cam, F. Sirri, primary
Published: 2013
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42. Deletion of 18(p) in a fetus determinated holoprosencephaly

Author: Boyacioglu, Seda Orenay, primary, Akyar, Halide Gul, additional, Coskunoglu, Aysun, additional, Ulucay, Safiye, additional, Ozkan, Munevver Yegul, additional, and Cam, F Sirri, additional
Published: 2011
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43. Deletion of 18(p) in a fetus determinated holoprosencephaly

Author: Seda Orenay Boyacioglu, F. Sirri Cam, Aysun Coskunoglu, Safiye Uluçay, Munevver Yegul Ozkan, and Halide Gul Akyar
Subjects: medicine.medical_specialty, Fetus, Endocrinology, Holoprosencephaly, Internal medicine, Biomedical Engineering, medicine, Bioengineering, Biology, medicine.disease, Biotechnology
Published: 2011
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44. Renin-angiotensin system gene polymorphisms and premature coronary heart disease

Author: Sekuri, Cevad, primary, Cam, F Sirri, additional, Ercan, Ertugrul, additional, Tengiz, Istemihan, additional, Sagcan, Abdi, additional, Eser, Erhan, additional, Berdeli, Afig, additional, and Akin, Mustafa, additional
Published: 2005
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45. Association Between the ACE I/D) Gene Polymorphism and Physical Performance in a Homogeneous Non-Elite Cohort

Author: Cam, F. Sirri, primary, Colakoglu, Muzaffer, additional, Sekuri, Cevad, additional, Colakoglu, Sule, additional, Sahan, Çagatay, additional, and Berdeli, Afig, additional
Published: 2005
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46. Assoication of angiotensin-converting enzyme I/D and eNOS G894T gene polymorphisms with erectile dysfunction.

Author: Çam, F. Sirri, Gümüs, Bilali H., Var, Ahmet, and Berdeli, Afig
Published: 2011
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47. ACE genotype may have an effect on single versus multiple set preferences in strength training.

Author: Colakoglu, Muzaffer, Cam, F. Sırrı, Kayitken, Bulent, Cetinoz, Fırat, Colakoglu, Sule, Turkmen, Mustafa, Sayin, Metin, Cam, F Sirri, and Cetinoz, Firat
Subjects: MUSCLE strength, PHYSICAL education, ANGIOTENSINS, RENIN-angiotensin system, PHYSICAL fitness, EXERCISE, GENETICS
Abstract: A polymorphic variant of the human angiotensin converting enzyme (ACE) gene was identified. The 'D' (rather than 'I') variant was associated with improvements in strength related to physical training. We set out to determine whether the response to different patterns of strength training might also differ. Ninty-nine Caucasian male non-elite athletes were randomly allocated into one of three groups: 31 non-training/control (CG: 31), single-set (SSG: 35) and multiple-set (MSG: 33). SSG and MSG trained three times a week for 6 weeks. Both training groups were underwent a strength-training program with two mesocycles (12-15 repetition maximum (RM) and 8-12 RM mesocycles). One RM loads in half squat and bench press were assessed before training and after the first and second mesocycles. ACE polymorphisms analysed by polymerase chain reaction (PCR) methods. Subjects with ACE II genotype in the MST group had improved strength development in 12-15 RM, while SST and MST groups had similar gains in 8-12 RM. Subjects with ACE DD genotype in both the SSG and the MSG had similar benefits from both 12-15 RM and 8-12 RM. Strength gains for subjects with ACE ID genotype in the SSG were similar to MSG gains in response to 8-12 RM loads but not with 12-15 RM loads. Additionally, subjects with DD genotype had superior strength gains in both strength training groups. Tailoring strength training programmes (single-set vs. multiple set) according to the athlete's ACE genotype may be advantageous. [ABSTRACT FROM AUTHOR]
Published: 2005
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48. Neutropenic Fever in a Two-and-a-Half Month Old Girl: Severe Congenital Neutropenia

Author: Hüseyin Gülen, Merve Öçalan, Ayşen Türedi Yıldırım, F. Sırrı Çam, and Hamide Betül Gerik Çelebi
Subjects: Congenital neutropenia, ELA-2, G-CSF, Medicine, Pediatrics, RJ1-570
Abstract: Severe congenital neutropenia is a hereditary disease characterized by a low number of neutrophils occurring from the first months of life leading to severe infections. It is rare but threatens life because of severe infections. For this reason, early diagnosis and treatment of this disease is important. Here, we present a case of a two and a half month old girl who was admitted to our clinic with fever and absolute neutropenia was determined in her clinical course.
Published: 2018
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49. G protein beta 3 subunit gene polymorphism in Turkish hypertensives

Author: Alioglu, Emin, Ercan, Ertugrul, Tengiz, Istemihan, Ahmet Yildiz, Tuerk, Ugur Oensel, Saygi, Serkan, Cam, F. Sirri, and Berdeli, Afig

50. Allometric Models to Estimate Leaf Area for Tropical African Broadleaved Forests

Author: N. F. Sirri, M. B. Libalah, S. Momo Takoudjou, P. Ploton, V. Medjibe, N. G. Kamdem, G. Mofack, B. Sonké, and N. Barbier
Subjects: leaf area, allometric models, tropical African broadleaved forests, Geophysics. Cosmic physics, QC801-809
Abstract: Abstract Direct and semidirect estimations of leaf area (LA) and leaf area index (LAI) are scarce in dense tropical forests despite their importance in calibrating remote sensing products, forest dynamics, and biogeochemical models. We destructively sampled 61 trees belonging to 13 most abundant species in a semideciduous forest in southeastern Cameroon. For each tree, all leaves were weighed, and for a subsample of branches, leaves were counted and the LA measured. Allometric models were calibrated to allow semidirect estimation of LAI at tree and stand levels based on forest inventory data (R2 = 0.7, bias = 21.2%, error = 39.5%) and on predictors that could be extracted from very high resolution remote sensing data (R2 = 0.63, bias = 35.1%, error = 58.73). Using twenty‐one 1‐ha forest plots, stand level estimations of LAI ranged from 4.42–13.99. These values are higher than previous estimates generally obtained using indirect methods. These results may have important consequences on ecosystem exchanges and the role of tropical forest in global cycles.
Published: 2019
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