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Your search keyword '"Eun, C.K."' showing total 28 results
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28 results on '"Eun, C.K."'

1. Wireless signaling of beta detection using microdischarges

Author

Eun, C.K., Gharpurey, R., and Gianchandani, Y.B.

Subjects
Electric discharges -- Measurement, Electric discharges through gases -- Measurement, Microelectromechanical systems -- Design and construction, Ultra wideband technology -- Usage, Mobile communication systems -- Analysis, Wireless communication systems -- Analysis, Wireless technology, Engineering and manufacturing industries, Science and technology
Published
2010

2. The Study of Cerebral Hemodynamics in the Hyperacute Stage of Fat Embolism Induced by Triolein Emulsion

Author

Kim, Y.W., Kim, H.J., Cho, B.M., Moon, T.Y., and Eun, C.K.

Subjects
Cerebral Cortex, Fat Emulsions, Intravenous, Blood Volume, Hemodynamics, Brain, Contrast Media, Embolism, Fat, Image Enhancement, Disease Models, Animal, Diffusion Magnetic Resonance Imaging, Intracranial Embolism, Regional Blood Flow, Acute Disease, Cats, Animals, Dominance, Cerebral, Blood Flow Velocity, Magnetic Resonance Angiography, Triolein
Abstract

PURPOSE: The purpose of this study was to evaluate the cerebral hemodynamic change in the hyperacute stage of cerebral fat embolism induced by triolein emulsion, by using MR perfusion imaging in cat brains. METHODS: By using the femoral arterial approach, the internal carotid arteries of 14 cats were infused with an emulsion of triolein 0.05 mL. T2-weighted (T2WI), diffusion-weighted (DWI), apparent diffusion coefficient (ADC) map, perfusion-weighted (PWI), and gadolinium-enhanced T1-weighted (Gd-T1WI) images were obtained serially at 30 minutes and 2, 4, and 6 hours after infusion. The MR images were evaluated qualitatively and quantitatively. Qualitative evaluation was performed by assessing the signal intensity of the serial MR images. Quantitative assessment was performed by comparing the signal-intensity ratio (SIR) of the lesions to the contralateral normal side calculated on T2WIs, Gd-T1WIs, DWIs, and ADC maps at each acquisition time and by comparing the relative cerebral blood volume (rCBV), cerebral blood flow (CBF), and mean transit times (MTT) of the lesions to the contralateral normal side calculated on PWI. RESULTS: In the qualitative evaluation of the MR images, the lesions showed hyperintensity on T2WIs, enhancement on the Gd-T1WIs, and isointensity on DWIs and the ADC maps. In the quantitative studies, SIRs on the Gd-T1WIs, DWIs, and ADC maps peaked at 2 hours after infusion. The SIRs on the T2WIs peaked at 4 hours after infusion and decreased thereafter. On PWIs, the rCBV, rCBF, and MTT of the lesion showed no significant difference from the contralateral normal side (P = .09, .30, and .13, respectively) and showed no significant change of time course (P = .17, .31, and .66, respectively). CONCLUSION: The embolized lesions induced by triolein emulsion showed no significant difference in cerebral hemodynamic parameters from those on the contralateral normal side. The result may suggest that consideration of the hemodynamic factor of embolized lesions is not necessary in further studies of the blood-brain barrier with triolein emulsion.

Published
2006

17. Immunochemical identification of the chick HPRTgene transferred from chick erythrocytes to mammalian somatic cells

Author

Eun, C.K., Paik, S.G., Goldwasser, P., Shin, S., and Klinger, H.P.

Abstract

Immunochemical methods were used to identify the genetic origin of hypoxanthine phosphoribosyltransferase (HPRT) expressed in heteroploid, HPRT-deficient mouse (A9) cells and Chinese hamster ovary (K627) cells, after these cells were fused with chick embryo erythrocytes and selected for resistance to hypoxanthine-aminopterin-thymidine (HAT) medium. All of the HAT-selected clones produced HPRT activity which was immunoprecipitable by an antiserum specific for chick HPRT, but not by an antiserum specific for mouse and hamster HPRT. Furthermore, the HPRT activity in these clones was electrophoretically indistinguishable from chick liver HPRT and clearly different from mouse liver HPRT. These data provide evidence that the HPRT activity expressed in cell hybrids produced by the fusion of HPRT-negative mammalian cells and chick erythrocytes containing genetically inactive nuclei is indeed coded by the chick HPRT gene and that an avian gene can be stably incorporated and correctly expressed in a mammalian cell.

Published
1981
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