6 results on '"Elmissiri, AM"'
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2. Club 35 Poster session Friday 13 December: 13/12/2013, 08: 30–18: 00Location: Poster area
- Author
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Elmissiri, AM
- Published
- 2013
3. Club 35 Poster Session Wednesday 11 December: 11/12/2013, 09: 30–16: 00Location: Poster area
- Author
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Elmissiri, AM, El Shahid, GS, and Abdal-Wahhab, S
- Published
- 2013
4. Club 35 Poster session 2: Thursday 4 December 2014, 08:30-18:00 * Location: Poster area
- Author
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Santos, M, Rivero, J, Mccullough, SD, Opotowsky, AR, Waxman, AB, Systrom, D, Shah, AM, Olsen, F J, Jorgensen, PG, Mogelvang, R, Jensen, JS, Fritz-Hansen, T, Bech, J, Sivertsen, J, Biering-Sorensen, T, Santoro, C, Esposito, R, Schiano Lomoriello, V, Raia, R, De Palma, D, Ippolito, R, Ierano, P, Arpino, G, De Simone, G, Galderisi, M, Cameli, M, Lisi, M, Di Tommaso, C, Solari, M, Focardi, M, Maccherini, M, Henein, M, Galderisi, M, Mondillo, S, Simova, I, Katova, T, Galderisi, M, Pauncheva, B, Vrettos, A, Dawson, D, Grigoratos, C, Papapolychroniou, C, Nihoyannopoulos, P, Danylenko, O, Kovalenko, V, Nesukay, E, Polenova, N, Titov, I, Voilliot, D, Huttin, OH, Vaugrenard, TV, Venner, CV, Sadoul, NS, Aliot, EA, Juilliere, YJ, Selton-Suty, CSS, Hamdi, I, Mahfoudhi, H, Ben Mansour, N, Dahmani, R, Lahidheb, D, Fehri, W, Haouala, H, Erken Pamukcu, H, Gerede, DM, Sorgun, M, Akbostanci, C, Turhan, S, Erol, û, Voilliot, D, Magne, JM, Dulgheru, RD, Kou, SK, Henri, CH, Caballero, LC, De Sousa, CDS, Sprynger, MS, Pierard, LP, Lancellotti, PL, Panelo, M L, Rodriguez-Fernandez, A, Escriba-Bori, S, Krol, W, Konopka, M, Burkhard, K, Jedrzejewska, I, Pokrywka, A, Klusiewicz, A, Chwalbinska, J, Dluzniewski, M, Braksator, W, Elmissiri, AM, Eid, M, Sayed, I, Awadalla, H, Schiano-Lomoriello, V, Esposito, R, Santoro, C, Lo Iudice, F, De Simone, G, Galderisi, M, Ibrahimi, P, Jashari, F, Johansson, E, Gronlund, C, Bajraktari, G, Wester, P, Henein, MY, Potluri, R, Aziz, A, Hooper, J, Mummadi, SM, Uppal, H, Asghar, O, Chandran, S, Surkova, E A, Tereshina, O V, Shchukin, U V, Rubanenko, A O, Medvedeva, E A, Hamdi, I, Mahfoudhi, H, Ben Mansour, N, Dahmani, R, Lahidheb, D, Fehri, W, Haouala, H, Krapf, L, Nguyen, V, Cimadevilla, C, Himbert, D, Brochet, E, Iung, B, Vahanian, A, Messika-Zeitoun, D, Danylenko, O, Kovalenko, V, Nesukay, E, Titov, I, Polenova, N, Van De Heyning, C M, Magne, J, Pierard, LA, Bruyere, PJ, Davin, L, De Maeyer, C, Paelinck, BP, Vrints, CJ, Lancellotti, P, Bertrand, PB, Groenendaels, Y, Vertessen, VJ, Mullens, W, Pettinari, M, Gutermann, H, Dion, RA, Verhaert, D, Vandervoort, PM, Guven, S, Sen, T, Tufekcioglu, O, Gucuk, E, Uygur, B, Kahraman, E, Valuckiene, Z, Jurkevicius, R, Pranevicius, R, Marcinkeviciene, J, Zaliaduonyte-Peksiene, D, Stoskute, N, and Zaliunas, R
- Abstract
Introduction: Among patients with unexplained dyspnea, left ventricular (LV) filling pressures (LVFP) is commonly estimated non-invasively by the E/e' ratio using Doppler echocardiography. However the accuracy of E/e' is controversial. We evaluated the correlation of E/e' ratio with invasively measured LVFP and of change in E/e' (ΔE/e') with change in LVFP. Methods: Supine and upright transthoracic echocardiography was performed in patients with unexplained dyspnea undergoing right heart catheterization. Patients with significant valvular disease and reduced LV ejection fraction (LVEF < 50%) were excluded. Pulmonary artery wedge pressure (PAWP) was used as the invasive indicator of LVFP. The mean of septal and lateral e' velocities was used for the calculation of E/e' ratio. Results: We studied 98 subjects with a mean age of 52 ± 20 years (69% of female gender). The supine E/e' and PAWP were 9.2 ± 3.2 and 12.1 ± 4.9 mmHg (range: 4-27 mmHg) respectively and were modestly correlated (r=0.38; p<0.001). With position change (supine to upright), ΔPAWP was -5.1 ± 4.3 mmHg and ΔE/e' was 0.17 ± 2.6, with no significant association between these two measures (r=0.003; p=0.98). Both E-wave (80 ± 22 to 65 ± 22 cm/s) and mean average e' (10.2 ± 3.6 to 7.3 ± 2.0 cm/s) decreased with the upright position. The ΔPAWP was correlated with ΔE-wave velocity (r=0.33; p=0.01), but not with Δe' (r=0.14; p=0.26). Conclusions: In patients with unexplained dyspnea and a preserved LVEF, E/e' is modestly, though significantly, correlated with PAWP. ΔE/e' is not correlated with ΔPAWP, partially related to the preload sensitivity of e'.
Figure Figure 1 - Supine and delta E/e' plotted - Published
- 2014
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5. Club 35 Poster Session Wednesday 11 December: 11/12/2013, 09:30-16:00 * Location: Poster area
- Author
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Montoro Lopez, M, Pons De Antonio, I, Itziar Soto, C, Florez Gomez, R, Alonso Ladreda, A, Rios Blanco, JJ, Refoyo Salicio, E, Moreno Yanguela, M, Lopez Sendon, JL, Guzman Martinez, G, Van De Heyning, C M, Magne, J, Pierard, LA, Bruyere, PJ, Davin, L, De Maeyer, C, Paelinck, BP, Vrints, CJ, Lancellotti, P, Michalski, BW, Krzeminska-Pakula, M, Lipiec, P, Szymczyk, E, Chrzanowski, L, Kasprzak, JD, Leao, R N, Florencio, A F, Oliveira, A R, Bento, B, Lopes, S, Calaca, J, Palma Reis, R, Krestjyaninov, MV, Gimaev, RH, Razin, VA, Arangalage, D, Chiampan, A, Cimadevilla, C, Touati, A, Himbert, D, Brochet, E, Iung, B, Nataf, P, Vahanian, A, Messika-Zeitoun, D, Guvenc, TS, Karacimen, D, Erer, HB, Ilhan, E, Sayar, N, Karakus, G, Eren, M, Iriart, X, Tafer, N, Roubertie, F, Mauriat, P, Thambo, JB, Wang, J, Fang, F, Yip, G WK, Sanderson, J, Feng, W, Yu, CM, Lam, YY, Assabiny, A, Apor, A, Nagy, A, Vago, H, Toth, A, Merkely, B, Kovacs, A, Castaldi, B, Vida, VL, Guariento, A, Padalino, M, Cerutti, A, Maschietto, N, Biffanti, R, Reffo, E, Stellin, G, Milanesi, O, Baronaite-Dudoniene, K, Urbaite, L, Smalinskas, V, Veisaite, R, Vasylius, T, Vaskelyte, J, Puodziukynas, A, Wieczorek, J, Rybicka-Musialik, A, Berger-Kucza, A, Hoffmann, A, Wnuk-Wojnar, A, Mizia-Stec, K, Melao, F, Ribeiro, V, Amorim, S, Araujo, C, Torres, JP, Cardoso, JS, Pinho, P, Maciel, MJ, Storsten, P, Eriksen, M, Boe, E, Estensen, ME, Erikssen, G, Smiseth, OA, Skulstad, H, Miglioranza, MH, Gargani, L, Sant`Anna, RT, Rover, M, Martins, VM, Mantovanni, A, Kalil, RK, Leiria, TL, Luo, XX, Fang, F, Lee, PW, Zhang, ZH, Lam, YY, Sanderson, JE, Kwong, J SW, Yu, CM, Borowiec, A, Dabrowski, R, Wozniak, J, Jasek, S, Chwyczko, T, Kowalik, I, Janas, J, Musiej-Nowakowska, E, Szwed, H, Palinsky, M, Petrovicova, J, Pirscova, M, Baricevic, Z, Lovric, D, Cikes, M, Skoric, B, Ljubas Macek, J, Reskovic Luksic, V, Separovic Hanzevacki, J, Milicic, D, Elmissiri, AM, El Shahid, GS, Abdal-Wahhab, S, Vural, M G, Yilmaz, M, Cetin, S, Akdemir, R, Yoldas, T K, Yeter, E, Karamanou, AG, Hamodraka, ES, Lekakis, IA, Paraskevaidis, IA, Kremastinos, DT, Appiah-Dwomoh, E K, Wang, VC, Otto, C, Mayar, F, Bonaventura, K, Sunman, H, Canpolat, U, Kuyumcu, M, Yorgun, H, Sahiner, L, and Ozer, N
- Abstract
Purpose: It is known the higher prevalence of structural heart disease in HIV patients, mostly diastolic dysfunction and pulmonary hypertension. In spite of that, there are few data about predisposing factors. Our objective was to evaluate whether HIV stage or detectable blood viral load correlate with the degree of heart disease. Methods: We conducted a prospective cohort study with HIV patients monitored by the internal medicine unit of our institution. We selected symptomatic patients with functional class ≥ II of NYHA scale. Viral blood load and CD4 count were systematically determined in order to obtain the HIV stage. Patients underwent a transthoracic echocardiogram to assess ventricular hypertrophy, systolic and diastolic dysfunction and pulmonary hypertension, according to the limits set by ESC guidelines. Results: Data were obtained from 65 HIV patients with dyspnea (63% male) with a mean age of 48 years. 50% were in NYHA grade II, 32.3% III and 17.7% IV. 46.7% of patients had some data of structural heart disease (figure). Belong to AIDS group (65.3%) did not correlate with the degree of heart disease. However, patients with positive blood viral load had a significantly higher incidence of structural heart disease than those with undetectable load (75% vs. 43% p <0.04), independent of their cardiovascular risk profile or type of antiretroviral therapy (Table). Conclusion: In our experience, half of HIV patients with dyspnea show echocardiographic data of structural heart disease. Detectable viral load in blood doubles the prevalence of heart disease, so that HIV itself may be an independent causal agent. These data should be taken into account in the screening of structural heart disease in these patients.
Figure Prevalence of structural heart disease - Published
- 2013
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6. Club 35 Poster session Friday 13 December: 13/12/2013, 08:30-18:00 * Location: Poster area
- Author
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Ben Abda, A, Hachulla, E, Polge, AS, Richardson, M, Duva Penthia, A, De Groote, P, Montaigne, D, Lamblin, N, Lamer, M, Cinotti, R, Delater, A, Asehnoune, K, Blanloeil, Y, Le Tourneau, T, Rozec, B, Piriou, N, Moon, J, Kim, TH, Ahn, T, Chung, WJ, Chimura, M, Oonishi, T, Tukishiro, Y, Yamada, S, Taniguchi, Y, Yasaka, Y, Kawai, T, Elmissiri, AM, Andres Lahuerta, A, Alonso Fernandez, P, Igual Munoz, B, Osca Asensi, J, Cano Perez, O, Jimenez Carreno, R, Sancho-Tello De Carranza, MJ, Olague De Ros, J, Salvador Sanz, A, Atas, H, Samadov, F, Kepez, A, Sunbul, M, Cincin, A, Direskeneli, H, Tigen, K, Yildiz, A, Karakas, MF, Cimen, T, Tuncez, A, Korkmaz, A, Uygur, B, Isleyen, A, Tufekcioglu, O, Melao, F, Paiva, M, Goncalves, A, Pinho, T, Madureira, A, Martins, E, Macedo, F, Maciel, MJ, Guvenc, TS, Erer, HB, Kul, S, Oz, D, Koroglu, B, Kaya, Y, Koc, S, Sayar, N, Degirmencioglu, A, Eren, M, Stapor, M, Condemi, F, Bapat, V, Gianstefani, S, Catibog, N, Monaghan, M J, Carro, A, Pijuan, A, Dos, L, Huguet, F, Abad, C, Gonzalez, N, Miranda, B, Galian, L, Casaldaliga, J, Evangelista, A, Gurzun, M M, Ionescu, A, Kahraman, E, Sen, T, Guven, S, Keskin, G, Topaloglu, S, Korkmaz, S, Moatemri, F, Mahdhaoui, A, Bouraoui, H, Jeridi, G, Ernez, S, Basaran, O, Gozubuyuk, G, Dundar, C, Tasar, O, Bulut, M, Karaahmet, T, Pala, S, Tigen, K, Izgi, A, Kirma, C, Baronaite-Dudoniene, K, Urbaite, L, Smalinskas, V, Veisaite, R, Vasylius, T, Vaskelyte, J, Puodziukynas, A, Carro, A, Teixido-Tura, G, Rodriguez-Palomares, JF, Cuellar, H, Pineda, V, Gruosso, D, Gutierrez, L, Moral, S, Gonzalez-Alujas, MT, Evangelista, A, Oprescu, N, Micheu, M, Calmac, L, Pitic, D, Dorobantu, M, Brugger, N, Huerzeler, M, Wustmann, K, Wahl, A, Steck, H, Seiler, C, Ismail, H, Linde, JJ, Kofoed, KF, Dixen, U, Soergaard, M, Hove, JD, Willis, J, Oxborough, D, Augustine, DX, Knight, D, Coghlan, G, Shah, R, Easaw, J, Verseckaite, R, Pilkauskaite, G, Lapinskas, T, Miliauskas, S, Sakalauskas, R, Jurkevicius, R, Ozeke, O, Turak, O, Ozcan, F, Cay, S, Topaloglu, S, Aras, D, Tufekcioglu, O, Golbasi, Z, and Aydogdu, S
- Abstract
Background: Systemic sclerosis (SSc) is a rare connective tissue disease that can lead to severe heart complications, i.e. congestive heart failure, arrhythmia and sudden cardiac death. The latter are associated with poor prognosis in SSc. The aim of this study was to test whether infra-clinical myocardial dysfunction can be detected in patients with SSc free from cardiovascular symptoms. Methods: Global LV, and segmental right ventricular longitudinal strain (2D-speckle tracking strain) were quantified by transthoracic echocardiography to assess ventricular deformations in 48 patients suffering from SSc and 40 matched control subjects. Results: Despite normal LVEF, patients presenting SSc had significantly impaired LV GLS in compared to controls (17.9 +/- 3.04 % vs -19.4+/- 2.3 %; p= 0.01). By contrast, there was no significant difference on lateral and inferior RV strain between patients and controls. Decreased LV deformation was detected in patients regardless of pulmonary injury, i.e. restrictive syndrome and pulmonary arterial hypertension (PAH). Patients with diffuse cutaneous SSc had impaired (GLV?) LV and RV strains in 4 and 2 chamber views compared to those presenting a limited form of cutaneous SSc, with respectively, -18.9 +/- 3.47% vs -16.6+/- 3.70%, p=0.039 and -19.7+/- 2.93% versus -17.6+/- 3.40% p=0.047. Conclusion: Speckle-tracking strain analysis can detect infra-clinical impairment of LV myocardial function in patients with SSc who are free from any cardiovascular symptoms regardless of SSc impact on pulmonary function.
Right and Left ventricular strain in Ssc - Published
- 2013
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