849 results on '"Eilers, Martin"'
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2. PAF1c links S-phase progression to immune evasion and MYC function in pancreatic carcinoma
3. The MYCN oncoprotein is an RNA-binding accessory factor of the nuclear exosome targeting complex
4. USP28 controls SREBP2 and the mevalonate pathway to drive tumour growth in squamous cancer
5. Drugging the “Undruggable” MYCN Oncogenic Transcription Factor: Overcoming Previous Obstacles to Impact Childhood Cancers
6. Beyond gene expression: how MYC relieves transcription stress
7. MYC multimers shield stalled replication forks from RNA polymerase
8. MYC determines lineage commitment in KRAS-driven primary liver cancer development
9. LRP8‐mediated selenocysteine uptake is a targetable vulnerability in MYCN‐amplified neuroblastoma
10. MYC promotes immune-suppression in triple-negative breast cancer via inhibition of interferon signaling
11. Immune evasion: An imperative and consequence of MYC deregulation.
12. Targeting MYC effector functions in pancreatic cancer by inhibiting the ATPase RUVBL1/2.
13. eLife assessment: Ribosome subunit attrition and activation of the p53–MDM4 axis dominate the response of MLL-rearranged cancer cells to WDR5 WIN site inhibition
14. Combined inhibition of Aurora-A and ATR kinases results in regression of MYCN-amplified neuroblastoma
15. Association with TFIIIC limits MYCN localisation in hubs of active promoters and chromatin accumulation of non-phosphorylated RNA polymerase II.
16. Association with TFIIIC limits MYCN localization in hubs of active promoters and chromatin accumulation of non-phosphorylated RNA Polymerase II
17. NEAT1 promotes genome stability via m6A methylation-dependent regulation of CHD4
18. USP10 drives cancer stemness and enables super-competitor signalling in Colorectal Cancer
19. Reprogramming of host glutamine metabolism during Chlamydia trachomatis infection and its key role in peptidoglycan synthesis
20. Target gene-independent functions of MYC oncoproteins
21. Drugging MYCN through an Allosteric Transition in Aurora Kinase A
22. Regulation of BCR-mediated Ca2+ mobilization by MIZ1-TMBIM4 safeguards IgG1+ GC B cell–positive selection.
23. eLife assessment: Ribosome subunit attrition and activation of the p53–MDM4 axis dominate the response of MLL-rearranged cancer cells to WDR5 WIN site inhibition
24. A MYC–GCN2–eIF2α negative feedback loop limits protein synthesis to prevent MYC-dependent apoptosis in colorectal cancer
25. Recruitment of BRCA1 limits MYCN-driven accumulation of stalled RNA polymerase
26. Myc Regulates Keratinocyte Adhesion and Differentiation via Complex Formation with Miz1
27. Direct RNA-binding by MYCN mediates feedback from RNA processing to transcription control
28. Regulation of BCR-mediated Ca2+ mobilization by MIZ1-TIMBIM4 safeguards IgG1+ GC B cell positive selection
29. Maintaining protein stability of ∆Np63 via USP28 is required by squamous cancer cells
30. Stabilisation of β-Catenin-WNT signalling by USP10 in APC-truncated colorectal cancer drives cancer stemness and enables super-competitor signalling
31. Supplementary Figure 3 from Inflammation-Induced NFATc1–STAT3 Transcription Complex Promotes Pancreatic Cancer Initiation by KrasG12D
32. Data from Targeting Translation Initiation Bypasses Signaling Crosstalk Mechanisms That Maintain High MYC Levels in Colorectal Cancer
33. Supplementary Figure 1 from Inflammation-Induced NFATc1–STAT3 Transcription Complex Promotes Pancreatic Cancer Initiation by KrasG12D
34. Supplementary Figure 5 from Inflammation-Induced NFATc1–STAT3 Transcription Complex Promotes Pancreatic Cancer Initiation by KrasG12D
35. Data from Inflammation-Induced NFATc1–STAT3 Transcription Complex Promotes Pancreatic Cancer Initiation by KrasG12D
36. Supplementary Figure 4 from Inflammation-Induced NFATc1–STAT3 Transcription Complex Promotes Pancreatic Cancer Initiation by KrasG12D
37. Supplementary Information from Inflammation-Induced NFATc1–STAT3 Transcription Complex Promotes Pancreatic Cancer Initiation by KrasG12D
38. Supplementary Figure 2 from Inflammation-Induced NFATc1–STAT3 Transcription Complex Promotes Pancreatic Cancer Initiation by KrasG12D
39. Supplementary Figure Legends, Table Legends, Table 1 from Targeting Translation Initiation Bypasses Signaling Crosstalk Mechanisms That Maintain High MYC Levels in Colorectal Cancer
40. Supplementary Figure 4 from Targeting Translation Initiation Bypasses Signaling Crosstalk Mechanisms That Maintain High MYC Levels in Colorectal Cancer
41. Supplementary Figure 7 from Targeting Translation Initiation Bypasses Signaling Crosstalk Mechanisms That Maintain High MYC Levels in Colorectal Cancer
42. Supplementary Figure 3 from Targeting Translation Initiation Bypasses Signaling Crosstalk Mechanisms That Maintain High MYC Levels in Colorectal Cancer
43. Supplementary Figure 6 from Targeting Translation Initiation Bypasses Signaling Crosstalk Mechanisms That Maintain High MYC Levels in Colorectal Cancer
44. Supplementary Figure 2 from Targeting Translation Initiation Bypasses Signaling Crosstalk Mechanisms That Maintain High MYC Levels in Colorectal Cancer
45. Supplementary Figure 6 from Inflammation-Induced NFATc1–STAT3 Transcription Complex Promotes Pancreatic Cancer Initiation by KrasG12D
46. Supplementary Figure 5 from Targeting Translation Initiation Bypasses Signaling Crosstalk Mechanisms That Maintain High MYC Levels in Colorectal Cancer
47. Supplementary Figure 1 from Targeting Translation Initiation Bypasses Signaling Crosstalk Mechanisms That Maintain High MYC Levels in Colorectal Cancer
48. Supplementary table S3 from UBR5 Is Coamplified with MYC in Breast Tumors and Encodes an Ubiquitin Ligase That Limits MYC-Dependent Apoptosis
49. Supplementary materials and methods from UBR5 Is Coamplified with MYC in Breast Tumors and Encodes an Ubiquitin Ligase That Limits MYC-Dependent Apoptosis
50. Figure S6 from MYC- and MIZ1-Dependent Vesicular Transport of Double-Strand RNA Controls Immune Evasion in Pancreatic Ductal Adenocarcinoma
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