42 results on '"Eastman, Alison J."'
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2. Streptococcus agalactiae cadD alleviates metal stress and promotes intracellular survival in macrophages and ascending infection during pregnancy
3. Reproductive organ on-a-chip technologies and assessments of the fetal-maternal interface.
4. The Influence of Obesity and Associated Fatty Acids on Placental Inflammation
5. Environmental Toxicant Exposure Paralyzes Human Placental Macrophage Responses to Microbial Threat
6. Impact of the Levonorgestrel-Releasing Intrauterine System on the Progression of Chlamydia trachomatis Infection to Pelvic Inflammatory Disease in a Baboon Model
7. Matrix metalloproteinases in preterm prelabor rupture of membranes in the setting of chorioamnionitis: A scoping review
8. Matrix metalloproteinases in preterm prelabor rupture of membranes in the setting of chorioamnionitis: A scoping review.
9. Leveraging bioengineering to assess cellular functions and communication within human fetal membranes.
10. Front Cover: Antibacterial and Anti‐biofilm Activity of the Human Breast Milk Glycoprotein Lactoferrin against Group B Streptococcus (ChemBioChem 12/2021)
11. Group B Streptococcus cpsE Is Required for Serotype V Capsule Production and Aids in Biofilm Formation and Ascending Infection of the Reproductive Tract during Pregnancy
12. Antibacterial and Anti‐biofilm Activity of the Human Breast Milk Glycoprotein Lactoferrin against Group B Streptococcus
13. Presenilin 2 influences miR146 level and activity in microglia
14. Vitamin D and Streptococci: The Interface of Nutrition, Host Immune Response, and Antimicrobial Activity in Response to Infection
15. Cytotrophoblasts suppress macrophage‐mediated inflammation through a contact‐dependent mechanism
16. Fas Activation in Alveolar Epithelial Cells Induces KC (CXCL1) Release by a MyD88-Dependent Mechanism
17. Leveraging bioengineering to assess cellular functions and communication within human fetal membranes
18. Palmitate induces apoptotic cell death and inflammasome activation in human placental macrophages
19. Levonorgestrel modulates innate immune activation in human endocervical epithelium and monocytes during Chlamydia trachomatis infection
20. Epigenetic stabilization of DC and DC precursor classical activation by TNFα contributes to protective T cell polarization
21. Palmitate induces apoptotic cell death and inflammasome activation in human placental macrophages
22. Cytotrophoblasts suppress macrophage‐mediated inflammation through a contact‐dependent mechanism.
23. Erratum for Xu et al., “Disruption of Early Tumor Necrosis Factor Alpha Signaling Prevents Classical Activation of Dendritic Cells in Lung-Associated Lymph Nodes and Development of Protective Immunity against Cryptococcal Infection”
24. Impact of the Levonorgestrel-Releasing Intrauterine System on the Progression of Chlamydia trachomatis Infection to Pelvic Inflammatory Disease in a Baboon Model
25. Modulation of IL-1β axis by levonorgestrel in the female reproductive tract during chlamydia trachomatis infection in a baboon model and in vitro
26. Immune regulation in the female reproductive tract by the levonorgestrel-releasing intrauterine device during a baboon model of Chlamydia trachomatis infection
27. Scavenger Receptor MARCO Orchestrates Early Defenses and Contributes to Fungal Containment during Cryptococcal Infection
28. Cryptococcus neoformans-induced macrophage lysosome damage crucially contributes to fungal virulence1
29. Disruption of Early Tumor Necrosis Factor Alpha Signaling Prevents Classical Activation of Dendritic Cells in Lung-Associated Lymph Nodes and Development of Protective Immunity against Cryptococcal Infection
30. Early TNFα signaling promotes classical activation of dendritic cells in lung associated lymph nodes to prime development of protective immunity against cryptococcal infection
31. Dual roles of scavenger receptor MARCO in innate versus adaptive immune responses during Cryptococcus neoformans infection
32. Early TNFα signaling results in pulmonary DC1 polarization and programing of murine myeloid precursor cells in the bone marrow towards DC1 polarization throughout Cryptococcus neoformans infection
33. CD4 T cell expression of PTIP is necessary for epigenetic maintenance of Th1/Th17 responses during Cryptococcus neoformans infection
34. Role of dendritic cell–pathogen interactions in the immune response to pulmonary cryptococcal infection
35. Cryptococcal Heat Shock Protein 70 Homolog Ssa1 Contributes to Pulmonary Expansion of Cryptococcus neoformans during the Afferent Phase of the Immune Response by Promoting Macrophage M2 Polarization
36. Cryptococcus neoformans–Induced Macrophage Lysosome Damage Crucially Contributes to Fungal Virulence
37. PKR-dependent CHOP induction limits hyperoxia-induced lung injury
38. Presenilin 2 Is the Predominant γ-Secretase in Microglia and Modulates Cytokine Release
39. P4-058: Presenilin 2 is the Predominant Gamma-secretase in Microglia and Regulates Cytokine Release
40. Presenilin 2 is the Predominant Gamma-secretase in Microglia and Regulates Cytokine Release
41. Scavenger Receptor MARCO Orchestrates Early Defenses and Contributes to Fungal Containment during Cryptococcal Infection.
42. PKR-dependent CHOP induction limits hyperoxia-induced lung injury.
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