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1. Neither injury induced macrophages within the nerve, nor the environment created by Wallerian degeneration is necessary for enhanced in vivo axon regeneration after peripheral nerve injury

2. The primary macrophage chemokine, CCL2, is not necessary after a peripheral nerve injury for macrophage recruitment and activation or for conditioning lesion enhanced peripheral regeneration

3. Molecular and cellular identification of the immune response in peripheral ganglia following nerve injury

4. Detection of Neutrophils in the Sciatic Nerve Following Peripheral Nerve Injury

5. Macrophage biology in the peripheral nervous system after injury

6. The Conditioning Lesion Response in Dorsal Root Ganglion Neurons Is Inhibited in Oncomodulin Knock-Out Mice

7. Injury-induced gp130 cytokine signaling in peripheral ganglia is reduced in diabetes mellitus

8. Neutrophils Are Critical for Myelin Removal in a Peripheral Nerve Injury Model of Wallerian Degeneration

9. Detection of Neutrophils in the Sciatic Nerve Following Peripheral Nerve Injury

10. Overexpression of the monocyte chemokine CCL2 in dorsal root ganglion neurons causes a conditioning-like increase in neurite outgrowth and does so via a STAT3 dependent mechanism

11. Nerve Growth Factor Regulates Transient Receptor Potential Vanilloid 2 via Extracellular Signal-Regulated Kinase Signaling To Enhance Neurite Outgrowth in Developing Neurons

12. Limited Recovery of Pineal Function after Regeneration of Preganglionic Sympathetic Axons: Evidence for Loss of Ganglionic Synaptic Specificity

13. Heat shock protein that facilitates myelination of regenerating axons

14. Cytokines inhibit norepinephrine transporter expression by decreasing Hand2

15. The dependence on gp130 cytokines of axotomy induced neuropeptide expression in adult sympathetic neurons

16. The Levels of Leukemia Inhibitory Factor mRNA in a Schwann Cell Line Are Regulated by Multiple Second Messenger Pathways

17. Galanin plays a role in the conditioning lesion effect in sensory neurons

18. Activating transcription factor 3 immunoreactivity identifies small populations of axotomized neurons in rat cervical sympathetic ganglia after transection of the preganglionic cervical sympathetic trunk

19. Novel changes in gene expression following axotomy of a sympathetic ganglion: A microarray analysis

20. Polyamines increase in sympathetic neurons and non-neuronal cells after axotomy and enhance neurite outgrowth in nerve growth factor-primed PC12 cells

21. A comparison of the changes in the non-neuronal cell populations of the superior cervical ganglia following decentralization and axotomy

22. The neuroimmunology of degeneration and regeneration in the peripheral nervous system

23. Nicotinic acetylcholine receptor subunit proteins ?7 and ?4 decrease in the superior cervical ganglion after axotomy

24. Differential regulation of levels of nicotinic receptor subunit transcripts in adult sympathetic neurons after axotomy

25. ■ REVIEW : LIF, NGF, and the Cell Body Response to Axotomy

26. Regulation of Neuropeptide Expression in Sympathetic Neurons

27. A Critical Role for Macrophages Near Axotomized Neuronal Cell Bodies in Stimulating Nerve Regeneration

28. Chemical sympathectomy and postganglionic nerve transection produce similar increases in galanin and VIP mRNA but differ in their effects on peptide content

29. Signals Triggering the Induction of Leukemia Inhibitory Factor in Sympathetic Superior Cervical Ganglia and Their Nerve Trunks after Axonal Injury

30. Galanin expression in sympathetic ganglia after partial axotomy is highly localized to those neurons that are axotomized

31. Cytokines that promote nerve regeneration

32. gp130 cytokines are positive signals triggering changes in gene expression and axon outgrowth in peripheral neurons following injury

33. Regulation of vasoactive intestinal peptide expression in sympathetic neurons in culture and after axotomy: The role of cholinergic differentiation factor/leukemia inhibitory factor

34. Galanin and vasoactive intestinal peptide messenger RNAs increase following axotomy of adult sympathetic neurons

35. Regulation of substance P is similar to that of vasoactive intestinal peptide after axotomy or explantation of the rat superior cervical ganglion

36. Vasoactive intestinal peptide and secretin produce long-term increases in tyrosine hydroxylase activity in the rat superior cervical ganglion

37. Depolarization increases vasoactive intestinal peptide- and substance P- like immunoreactivities in cultured neonatal and adult sympathetic neurons

38. The conditioning lesion effect on sympathetic neurite outgrowth is dependent on gp130 cytokines

39. Distribution of vasoactive intestinal peptide- and peptide histidine isoleucine amide-like immunoreactive neurons and fibers in the thoracic spinal cord of the rat

40. Vasoactive Intestinal Peptide Stimulates Catecholamine Biosynthesis in Isolated Adrenal Chromaffin Cells: Evidence for a Cyclic AMP-Dependent Phosphorylation and Activation of Tyrosine Hydroxylase

41. Functional recovery and collateral neuronal sprouting examined in young and aged rats following a partial neural lesion

42. Effects of Peptides of the Secretin-Glucagon Family and Cyclic Nucleotides on Tyrosine Hydroxylase Activity in Sympathetic Nerve Endings

43. Pharmacological and Biochemical Properties of Nicotinic Receptors from Chick Retina

44. Nerve stimulation in vivo acutely increases tyrosine hydroxylase activity in the superior cervical ganglion and its end organs

45. Recovery of function following unilateral denervation, but not unilateral decentralization, of the pineal gland as indicated by measurements of pineal melatonin content and urinary melatonin metabolites

46. Activating transcription factor 3 induction in sympathetic neurons after axotomy: response to decreased neurotrophin availability

47. Reduction in nerve growth factor availability leads to a conditioning lesion-like effect in sympathetic neurons

48. A conditioning lesion enhances sympathetic neurite outgrowth

49. PACAP promotes sensory neuron differentiation: blockade by neurotrophic factors

50. Modulation of NMDA receptor-dependent calcium influx and gene expression through EphB receptors

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