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2. Dominant missense variants in SREBF2 are associated with complex dermatological, neurological, and skeletal abnormalities

3. Identification of potential non-invasive biomarkers in diastrophic dysplasia

8. Defining the clinical phenotype of Saul–Wilson syndrome

12. GGC Repeat Expansion and Exon 1 Methylation of XYLT1 Is a Common Pathogenic Variant in Baratela-Scott Syndrome

13. A Recurrent De Novo Heterozygous COG4 Substitution Leads to Saul-Wilson Syndrome, Disrupted Vesicular Trafficking, and Altered Proteoglycan Glycosylation

14. RAB1A haploinsufficiency phenocopies the 2p14–p15 microdeletion and is associated with impaired neuronal differentiation

24. Extreme Growth Failure is a Common Presentation of Ligase IV Deficiency

31. Biallelic variants in DNA2 cause microcephalic primordial dwarfism

32. Dominant missense variants in SREBF2are associated with complex dermatological, neurological, and skeletal abnormalities

37. The expanding phenotype of <italic>RNU4ATAC</italic> pathogenic variants to Lowry Wood syndrome.

39. Extreme Growth Failure is a Common Presentation of Ligase IV Deficiency

40. Paternally inherited microdeletion at 15q11.2 confirms a significant role for the SNORD116 C/D box snoRNA cluster in Prader–Willi syndrome

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