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2. Apolipoprotein B, Residual Cardiovascular Risk After Acute Coronary Syndrome, and Effects of Alirocumab

3. Lipoprotein(a) and Benefit of PCSK9 Inhibition in Patients With Nominally Controlled LDL Cholesterol

5. Effects of alirocumab on cardiovascular and metabolic outcomes after acute coronary syndrome in patients with or without diabetes: a prespecified analysis of the ODYSSEY OUTCOMES randomised controlled trial

6. Vorsprung

8. Vorsprung

9. Rapid synthesis of RGD mimetics with isoxazoline scaffolds on solid phase: Identification of αvβ3 antagonists lead compounds

10. Polyfunctional pyridines from nitroacetamidine and β-diketones. A useful synthesis of substituted imidazo [4,5-b] pyridines and related compounds

11. Metabolism resistant isothiazolone inhibitors of cartilage breakdown

13. ChemInform Abstract: 2′-Substituted Chalcone Derivatives as Inhibitors of Interleukin-1 Biosynthesis

14. ChemInform Abstract: Polyfunctional Pyridines from Nitroacetamidine and β-Diketones. A Useful Synthesis of Substituted Imidazo(4,5-b)pyridines and Related Compounds

16. Abstracts – AAPS Topical Drug Products Workshop, March 26–28, 1990, USA

17. Rotator cuff injuries and treatment

18. Continuous parenteral and enteral nutrition induces metabolic dysfunction in neonatal pigs

20. Heteroaryl-fused 2-phenylisothiazolone inhibitors of cartilage breakdown

23. 2'-substituted chalcone derivatives as inhibitors of interleukin-1 biosynthesis

24. 1 5-Lipoxygenase Inhibitors and their Anti-inflammatory Activities

25. ChemInform Abstract: 2-Substituted 1-Naphthols as Potent 5-Lipoxygenase Inhibitors with Topical Antiinflammatory Activity

26. 2-substituted-1-naphthols as potent 5-lipoxygenase inhibitors with topical antiinflammatory activity

27. Vorsprung

30. Topical anti-inflammatory activity of DuP 654, a 2-substituted 1-naphthol

32. EFFECT OF PLANE OF NUTRITION ON VISCERAL ORGAN GROWTH AND METABOLISM

34. Action of CDK Inhibitor PHA-848125 in ER-negative Breast Cancer with MicroRNA-221/222 Overexpression

35. LETTERS.

36. Continuous parenteral and enteral nutrition induces metabolic dysfunction in neonatal pigs.

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