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Your search keyword '"Donnart, Audrey"' showing total 21 results

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1. Genome-wide association analyses identify new Brugada syndrome risk loci and highlight a new mechanism of sodium channel regulation in disease susceptibility

2. Author Correction: Genome-wide association analyses identify new Brugada syndrome risk loci and highlight a new mechanism of sodium channel regulation in disease susceptibility

4. De Novo Truncating Mutations in the Kinetochore-Microtubules Attachment Gene CHAMP1 Cause Syndromic Intellectual Disability

6. Gap-134, a Connexin43 activator, prevents age-related development of ventricular fibrosis in Scn5a− mice

7. Expansion of natural CD8+Tregs for cell therapy

8. Ex Vivo Expanded Human Non-Cytotoxic CD8+CD45RClow/\textminus Tregs Efficiently Delay Skin Graft Rejection and GVHD in Humanized Mice

9. Ex Vivo Expanded Human Non-Cytotoxic CD8+CD45RClow/-Tregs Efficiently Delay Skin Graft Rejection and GVHD in Humanized Mice

11. An intermediate level of CD161 expression defines a novel activated, inflammatory, and pathogenic subset of CD8 + T cells involved in multiple sclerosis

12. Transient Anti-CD45RC mAb Treatment Induces Specific Transplant Tolerance Through Potentiation of Tregs

13. Transient antibody targeting of CD45RC induces transplant tolerance and potent antigen-specific regulatory T cells

14. De Novo Truncating Mutations in the Kinetochore-Microtubules Attachment GeneCHAMP1Cause Syndromic Intellectual Disability

15. Ex Vivo Expanded Human Non-Cytotoxic CD8+CD45RClow/- Tregs Efficiently Delay Skin Graft Rejection and GVHD in Humanized Mice.

16. Parallel derivation of isogenic human primed and naive induced pluripotent stem cells.

19. An intermediate level of CD161 expression defines a novel activated, inflammatory, and pathogenic subset of CD8+ T cells involved in multiple sclerosis.

20. Gap-134, a Connexin43 activator, prevents age-related development of ventricular fibrosis in Scn5a +/ - mice.

21. Ex Vivo Expanded Human Non-Cytotoxic CD8 + CD45RC low/- Tregs Efficiently Delay Skin Graft Rejection and GVHD in Humanized Mice.

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