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1. Pharmacologic ATF6 activation confers global protection in widespread disease models by reprograming cellular proteostasis

2. Noncanonical Form of ERAD Regulates Cardiac Hypertrophy

3. Optimization of Large‐Scale Adeno‐Associated Virus (AAV) Production

4. Design and Production of Heart Chamber-Specific AAV9 Vectors

5. The peroxisomal enzyme, FAR1, is induced during ER stress in an ATF6-dependent manner in cardiac myocytes

6. PRAS40 prevents development of diabetic cardiomyopathy and improves hepatic insulin sensitivity in obesity

7. Proteomic analysis of the cardiac myocyte secretome reveals extracellular protective functions for the ER stress response

9. Pharmacologic ATF6 activation confers global protection in widespread disease models by reprograming cellular proteostasis

10. Optimizing Adeno-Associated Virus Serotype 9 for Studies of Cardiac Chamber-Specific Gene Regulation

11. Mesencephalic astrocyte–derived neurotrophic factor is an ER‐resident chaperone that protects against reductive stress in the heart

12. Mesencephalic astrocyte-derived neurotrophic factor is an ER-resident chaperone that protects against reductive stress in the heart

13. ATF6 Decreases Myocardial Ischemia/Reperfusion Damage and Links ER Stress and Oxidative Stress Signaling Pathways in the Heart

14. Junctophilin-2 gene therapy rescues heart failure by normalizing RyR2-mediated Ca2+ release

17. ATF6 Regulates Cardiac Hypertrophy by Transcriptional Induction of the mTORC1 Activator, Rheb

18. Abstract 352: Manf, a Structurally Unique Redox-sensitive Chaperone, Restores Er-protein Folding in the Ischemic Heart

19. Abstract 547: Pharmacologic ATF6 Activation Confers Global Protection in Widespread Disease Models by Reprogramming Cellular Proteostasis

21. Hrd1 and ER-Associated Protein Degradation, ERAD, Are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes

22. Abstract 21206: Manf, a Structurally Unique Redox-Sensitive Chaperone, Restores ER-Protein Folding in the Ischemic Heart

23. Mechanistic Target of Rapamycin Complex 2 Protects the Heart From Ischemic Damage

24. PRAS40 prevents development of diabetic cardiomyopathy and improves hepatic insulin sensitivity in obesity

25. Pathological hypertrophy amelioration by PRAS40-mediated inhibition of mTORC1

26. Junctophilin-2 gene therapy rescues heart failure by normalizing RyR2-mediated Ca

27. S100A4 protects the myocardium against ischemic stress

28. Regulation of microRNA expression in the heart by the ATF6 branch of the ER stress response

29. Roles for Endoplasmic Reticulum–Associated Degradation and the Novel Endoplasmic Reticulum Stress Response Gene Derlin-3 in the Ischemic Heart

30. Ischemia Activates the ATF6 Branch of the Endoplasmic Reticulum Stress Response

31. Coordination of Growth and Endoplasmic Reticulum Stress Signaling by Regulator of Calcineurin 1 (RCAN1), a Novel ATF6-inducible Gene

32. Abstract 402: Selective Protein Secretion Upon Endoplasmic Reticulum Stress Protects Cardiac Myocytes

33. Activation of the Unfolded Protein Response in Infarcted Mouse Heart and Hypoxic Cultured Cardiac Myocytes

34. Opposing Roles for ATF6α and ATF6β in Endoplasmic Reticulum Stress Response Gene Induction

35. Mimicking Phosphorylation of αB-Crystallin on Serine-59 Is Necessary and Sufficient to Provide Maximal Protection of Cardiac Myocytes From Apoptosis

36. MANF, a structurally unique redox-sensitive chaperone, restores ER-protein folding in the ischemic heart

37. Sarco/endoplasmic Reticulum Calcium ATPase-2 Expression Is Regulated by ATF6 during the Endoplasmic Reticulum Stress Response

38. αB-crystallin Gene Induction and Phosphorylation by MKK6-activated p38

39. p38 Mitogen-activated Protein Kinase Mediates the Transcriptional Induction of the Atrial Natriuretic Factor Gene through a Serum Response Element

40. A Role for the p38 Mitogen-activated Protein Kinase Pathway in Myocardial Cell Growth, Sarcomeric Organization, and Cardiac-specific Gene Expression

41. Differential Effects of Protein Kinase C, Ras, and Raf-1 Kinase on the Induction of the Cardiac B-type Natriuretic Peptide Gene through a Critical Promoter-proximal M-CAT Element

42. Regulation of cardiac hypertrophic signaling by prolyl isomerase Pin1

43. Abstract 18: Synoviolin, an E3 Ubiquitin Ligase, Modulates Cardiac Myocyte Size and Restores Heart Function in Hypertrophic Cardiomyopathy

44. Protein disulfide isomerase-associated 6 is an ATF6-inducible ER stress response protein that protects cardiac myocytes from ischemia/reperfusion-mediated cell death

45. Brain natriuretic peptide is induced by alpha 1-adrenergic agonists as a primary response gene in cultured rat cardiac myocytes

46. Regulation of rat brain natriuretic peptide transcription. A potential role for GATA-related transcription factors in myocardial cell gene expression

47. Abstract P134: Synoviolin Is a Stress-Inducible Endoplasmic/Sarcoplasmic Reticulum E3 Ubiquitin Ligase that Preserves Cardiac Function

48. Effects of the isoform-specific characteristics of ATF6 alpha and ATF6 beta on endoplasmic reticulum stress response gene expression and cell viability

49. Endoplasmic reticulum stress gene induction and protection from ischemia/reperfusion injury in the hearts of transgenic mice with a tamoxifen-regulated form of ATF6

50. MAP kinase kinase 6-p38 MAP kinase signaling cascade regulates cyclooxygenase-2 expression in cardiac myocytes in vitro and in vivo

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